ライフサイエンスコーパス: tobacco exposure

1 d to prenatal cannabis (mostly combined with tobacco exposure).

2 the TIME, which is potentially modulated by tobacco exposure.

3 or concurrent BMI, pubertal development, and tobacco exposure.

4 n imperfect and biased measure of cumulative tobacco exposure.

5 e sequence context 5'-ATN-3' correlated with tobacco exposure.

6 ts in lung function associated with in utero tobacco exposure.

7 Exposures: Prenatal methamphetamine and/or tobacco exposure.

8 ly, smokers were stratified by pack-years of tobacco exposure.

9 results were largely mirrored with increased tobacco exposure.

10 PCE; a subset of each group additionally had tobacco exposure.

11 data regarding the cardiovascular effects of tobacco exposure.

12 isk of lung cancer increased with cumulative tobacco exposure.

13 s, nor considered other risk factors such as tobacco exposure.

14 suggesting a common airway-wide response to tobacco exposure.

15 e types of DNA damage, including damage from tobacco exposure.

16 havioral anomalies associated with perinatal tobacco exposure.

17 hs but not thereafter, and may reflect heavy tobacco exposure.

18 h weights for women with different levels of tobacco exposure.

19 r smokers was strongly related to cumulative tobacco exposure.

20 icipant characteristics, asthma history, and tobacco exposure.

21 olic syndrome, and prevalence increased with tobacco exposure: 1.2% for nonexposed, 5.4% for those ex

22 genes conditionally essential for surviving tobacco exposure, abscess formation and epithelial invas

23 ressure was 1.31 (95% CI, 1.06-1.61) for any tobacco exposure after adjustment; odds were similar acr

24 a causal relationship between environmental tobacco exposure and adverse behavioral and cognitive ou

25 There were interactions between early-life tobacco exposure and age, sex, deprivation, and diet on

26 0.001, p < 0.001 respectively) regardless of tobacco exposure and associated strongly with differenti

27 Reported associations between gestational tobacco exposure and autism spectrum disorders (ASDs) ha

28 the relations between these two measures of tobacco exposure and birth weight.

29 We analyzed the relationship between TIME, tobacco exposure and clinical outcomes in OPSCC patients

30 that there is a positive association between tobacco exposure and elevated blood pressure in the stud

31 ng and further assessed the joint effects of tobacco exposure and genetic susceptibility.

32 ion of factors that increase PAI-1 including tobacco exposure and obesity.

33 Associations between tobacco exposure and overall survival (OS) and progressi

34 med a modest (inverse) relation between true tobacco exposure and serum beta-carotene.

35 Understanding the relationship between tobacco exposures and specific mutations may yield etiol

36 errors in reported smoking (relative to true tobacco exposure) and assumed a modest (inverse) relatio

37 its of mm Hg-years (similar to pack-years of tobacco exposure) and related to the presence of coronar

38 ake of retinol and provitamin A carotenoids, tobacco exposure, and asbestos exposure.

39 uding those linked to its formation, such as tobacco exposure, and progression, such as homologous re

40 rds models adjusted for age, black ancestry, tobacco exposure, and socioeconomic position.

41 uvenile idiopathic arthritis, visual acuity, tobacco exposure, and socioeconomic status.

42 some individuals with a history of prolonged tobacco exposure, and that expression of the GRP recepto

43 stment for age, race, tumor and nodal stage, tobacco exposure, and treatment assignment, had a 58% re

44 hildren with prenatal methamphetamine and/or tobacco exposure are present at birth before childhood e

45 scular disease the cardiovascular effects of tobacco exposure are substantial.

46 l update recent findings supporting diet and tobacco exposure as etiologic factors in the development

47 ized to develop a non-invasive biomarker for tobacco exposure as well as a non-invasive screening or

48 nd death increases directly as a function of tobacco exposure at diagnosis and during therapy and is

49 we used two assays with mutagens relevant to tobacco exposure (benzo[a]pyrene diol epoxide (BPDE) and

50 data demonstrates that the current burden of tobacco exposure both in the United States and worldwide

51 Tobacco exposure, both current and lifetime, was linearl

52 hylation in certain genes is associated with tobacco exposure but it is unknown whether these methyla

53 Marijuana exposure was nearly as common as tobacco exposure but was mostly light (median, 2-3 episo

54 Environmental tobacco exposure, but not allergic sensitization, also h

55 ed birth weights for women who reduced their tobacco exposure by 50 percent or more and for those who

56 Prenatal cocaine or tobacco exposure can differentially affect structural br

57 Prenatal tobacco exposure compared with nonexposed control subjec

58 Despite reportedly having less tobacco exposure compared with whites, African Americans

59 We explore how tobacco exposure contributes to cancer development by mu

60 Tobacco exposure correlated significantly (p < 0.001) wi

61 Tobacco exposure, defined as serum cotinine levels great

62 ates and abroad, the cardiovascular risks of tobacco exposure despite improvements in medical therapy

63 s, only a small fraction of individuals with tobacco exposure develop cancer.

64 ce of pleiotropic effects and accounting for tobacco exposure did not alter the association (OR of sc

65 undertaken to determine 1) whether reducing tobacco exposure during pregnancy increases the birth we

66 or multiple risks (IPV, smoking, depression, tobacco exposure) found significantly fewer recurrent ep

67 on elucidating the complex interaction among tobacco exposure, genetics and environmental factors.

68 n utero exposure, participants with in utero tobacco exposure had an increase in Klemera-Doubal biolo

69 In utero tobacco exposure has been associated with fetal growth r

70 ificant subset of HPV tumors associated with tobacco exposure have diminished treatment response and

71 While all forms of tobacco exposure have negative health effects, the signi

72 hibited a mutational profile consistent with tobacco exposure; human papillomavirus was detectable by

73 iency but weaker exogenous processes such as tobacco exposure in AYAs.

74 Tobacco exposure in cardiac transplant recipients, befor

75 r experiments reveal that pretransplantation tobacco exposure in donors and/or recipients results in

76 HPV infection may interact with alcohol and tobacco exposure in tumor promotion.

77 activation of signaling pathways relevant to tobacco exposure, including ATM, BCL2, GPX1, NOS2, IKBKB

78 The cardiovascular effects of tobacco exposure, including even low levels from SHS, re

79 Birth weight declined as tobacco exposure increased; however, the relation was no

80 ogether, these new findings demonstrate that tobacco exposure induces the abnormal expression of SNCG

81 This study suggests that tobacco exposure is associated with elevated blood press

82 Rationale: In utero tobacco exposure is associated with reduced lung functio

83 Tobacco exposure is the leading cause of lung cancer, bu

84 year 1 but not with race/ethnicity, income, tobacco exposure, maternal stress, or early introduction

85 Prenatal tobacco exposure may be one suspected cause of bipolar d

86 ollectively, prenatal methamphetamine and/or tobacco exposure may lead to delayed motor development a

87 ions and Relevance: Prenatal methamphetamine/tobacco exposure may lead to delays in motor development

88 ly thought to result almost exclusively from tobacco exposure, may have an inherited predisposition a

89 and FVC compared with those with no in utero tobacco exposure (mean difference, -6.2% predicted, P =

90 As in the case of alcohol use, intensity of tobacco exposure (measured as packs per day) was not ass

91 s in OPSCC patients (n = 143) with extensive tobacco exposure (median pack-years = 40).

92 l, 3,042 (71.7%) were ever-smokers with mean tobacco exposure of 33 pack-years.

93 t (LT) recipients to determine the impact of tobacco exposure on 10-year survival and de novo cancer

94 The impact of tobacco exposure on health varies by race and ethnicity

95 However, the impact of tobacco exposure on the TIME in OPSCC patients remains u

96 ideration the potential modifying effects of tobacco exposure on treatment effectiveness and clinical

97 s) with or without COPD and controls without tobacco exposure or airflow obstruction.

98 cioeconomic variables such as race, poverty, tobacco exposure, or general nutritional status.

99 as smaller in adolescents following prenatal tobacco exposure (P = .03).

100 e following either PCE (P = .05) or prenatal tobacco exposure (P = .04).

101 sure and is one potential mechanism by which tobacco exposure predisposes to adverse health outcomes,

102 -ethnicity, education, poverty-income ratio, tobacco exposure, previous diagnosis of diabetes, and bo

103 Prenatal tobacco exposure (PTE) affected speech processing, level

104 Prenatal alcohol exposure (PAE) and prenatal tobacco exposure (PTE) are risk factors associated with

105 prenatal alcohol exposure (PAE) and prenatal tobacco exposure (PTE) with adolescent neuroanatomical d

106 risk of PAIS in term neonates: maternal age, tobacco exposure, recreational drug exposure, preeclamps

107 This association was related to past tobacco exposure, regardless of whether the subjects wer

108 Tobacco exposure remains the main but not exclusive caus

109 Tobacco exposure results in chronic inflammation, tissue

110 Early-life tobacco exposure serves as a non-negligible risk factor

111 cancer mutational signatures associated with tobacco exposure, supporting its contribution to the car

112 n the same way they are trained to ask about tobacco exposure to assess cancer and heart disease risk

113 hlight the importance of reducing early-life tobacco exposure to improve healthy aging.

114 edings from 2007 and 2008 that link in-utero tobacco exposure to neurodevelopmental outcomes in expos

115 Parent and child's passive tobacco exposure was assessed using interview-assisted q

116 th alterations in two texture features while tobacco exposure was associated with alterations in five

117 In utero tobacco exposure was associated with deficits in lung fu

118 zygous for the GSTM1-null genotype, in utero tobacco exposure was associated with lower FEV(1) and FV

119 ity of studies reviewed were prospective and tobacco exposure was quantified biologically.

120 Additionally, tobacco exposure was significantly associated with great

121 e potential confounding variables, including tobacco exposure, was 9.4 (95% CI, 2.6 to 33.8).

122 , we explored the associations of early-life tobacco exposure with accelerated biological aging and f

123 ubstantially larger and to quantify lifetime tobacco exposure with more precision than have past stud