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1 hrenia is associated with very high rates of tobacco smoking.
2 , for example, the "pack-years" variable for tobacco smoking.
3 tational signatures that are associated with tobacco smoking.
4 diseases, respectively, were attributable to tobacco smoking.
5 models adjusted for age, sex, and cumulative tobacco smoking.
6  increased with each additional pack-year of tobacco smoking.
7 Risk factors for periodontal disease include tobacco smoking.
8 r levels by 6 to 12 weeks of abstinence from tobacco smoking.
9 xplore the mutational burden associated with tobacco smoking.
10  is a major cause for starting or increasing tobacco smoking.
11 ntribute to the perpetuation of addiction to tobacco smoking.
12 on between the level of fasting glycemia and tobacco smoking.
13 ich despite some success have not eliminated tobacco smoking.
14 use of death, and almost all of it is due to tobacco smoking.
15 the youngest infants and in those exposed to tobacco smoking.
16 exposure of the upper aerodigestive tract to tobacco smoking.
17 on with the effect of nonsmoking and regular tobacco smoking.
18 sted waist-to-hip ratio, alcohol intake, and tobacco smoking.
19 of the genetic effect of body mass index and tobacco smoking.
20  inconclusive due to confounding by previous tobacco smoking.
21 a indicate that vaping may not be safer than tobacco smoking.
22 ically involved in the reinforcing effect of tobacco smoking.
23 x disease, Barrett's esophagus, obesity, and tobacco smoking.
24 ion treatments and reduce the harm caused by tobacco smoking.
25 association with risk for IBD is modified by tobacco smoking.
26 t disease, diseases strongly associated with tobacco smoking.
27 , but this does not seem to be made worse by tobacco smoking.
28 ical tumors and that LOY could be induced by tobacco smoking.
29 e, the principal addictive component driving tobacco smoking.
30 geting prevention and cessation of waterpipe tobacco smoking.
31 ntified strong epigenetic signals related to tobacco smoking.
32  (beta +/- SE 0.07 +/- 0.01; P < 0.0001) and tobacco smoking (0.64 +/- 0.26; P = 0.016), as well as w
33 risk ratio [RR] 1.41 [95% CI 1.26-1.57]) and tobacco smoking (1.46 [1.30-1.65]) than in HIV-negative
34  use (RR 1.36 [95% CI 1.10-1.69]; p=0.0050), tobacco smoking (1.90 [1.38-2.62]; p<0.0001), and smokel
35 positive women was 1.3% (95% CI 0.8-1.9) for tobacco smoking, 2.1% (1.1-3.4) for smokeless tobacco us
36                                              Tobacco smoking (3.806, one degree of freedom; P < .015,
37 ositive men was 24.4% (95% CI 21.1-27.8) for tobacco smoking, 3.4% (1.8-5.6) for smokeless tobacco us
38  SBS4, which has previously been linked with tobacco smoking(9), and a 76% increase in the clock-like
39  The primary outcome was 7 months continuous tobacco smoking abstinence as measured at 9 months after
40                              In 2004, active tobacco smoking accounted for approximately 15.8% (95% C
41                                     Although tobacco smoking accounts for the majority of lung cancer
42 e no significant differences in gender, age, tobacco smoking, Acute Physiology and Chronic Health Eva
43 st associations, with the exception of daily tobacco smoking (adjusted OR 1.74, 95% CI 1.08-2.81), an
44    There is growing evidence that water pipe tobacco smoking affects heart rate, blood pressure regul
45 2 therapy was influenced by PPD at baseline, tobacco smoking, age, and type of tooth.
46 y (aHR = 2.757 [1.616-4.704], p < 0.001) and tobacco smoking (aHR = 2.150 [1.319-3.503], p < 0.01) we
47 lected on education, household overcrowding, tobacco smoking, alcohol and drugs use, and history of h
48 adult exposures to carcinogens, such as from tobacco smoking, alcohol consumption and various industr
49 rders in general), including breast-feeding, tobacco smoking, alcohol consumption, and exposure to do
50 ,115) and tested the association of RBA with tobacco smoking, alcohol consumption, and genetic varian
51  extensive questionnaire containing items on tobacco smoking, alcohol consumption, and occupational e
52 gnancy was identified based on self-reported tobacco smoking, alcohol consumption, and use of cannabi
53 sion models that were adjusted for age, sex, tobacco smoking, alcohol drinking, and hypertension.
54 terms for age, sex, study center, education, tobacco smoking, alcohol drinking, hepatitis B surface a
55                  Given the coincident use of tobacco smoking among HIV-infected intravenous drug user
56 ze existing data on the prevalence of active tobacco smoking among patients with hypertension or diab
57                                      Heavier tobacco smoking among people with schizophrenia (SCZ) ha
58  from the general population, in relation to tobacco smoking, an exposure with well-characterised hea
59 o specific risk factors, such as alcohol and tobacco (smoking and smokeless), but dietary factors and
60 nicotine accumulates in the intestine during tobacco smoking and activates intestinal AMPKalpha.
61                                   Rationale: Tobacco smoking and air pollution are primary causes of
62 ted with the widely accepted risk factors of tobacco smoking and alcohol use and is the end result of
63 ants of health (SDHs, including drug misuse, tobacco smoking and alcohol), and TB, taking into accoun
64 mokers to assess whether baseline water pipe tobacco smoking and baseline snus use were associated wi
65 models to assess whether baseline water pipe tobacco smoking and baseline snus use were associated wi
66 or numerous potential confounders, including tobacco smoking and C-reactive protein levels.
67                                         Both tobacco smoking and circulating cardiac troponin I (cTnI
68  population density areas, and pack-years of tobacco smoking and decreases in subjects with history o
69                        Outcomes were current tobacco smoking and e-cigarette use.
70           We studied the association between tobacco smoking and epigenome-wide methylation in non-tu
71                                              Tobacco smoking and female sex were associated with high
72 rience co-occurring substance use, including tobacco smoking and heavy cannabis use, and substance us
73 h decreased risk of ADA development, whereas tobacco smoking and infections during the study were ass
74  the documentation of an association between tobacco smoking and lung cancer.
75 anged by at least 2 percentage points (e.g., tobacco smoking and mental health conditions) were assoc
76 s to investigate the differential effects of tobacco smoking and nicotine on changes in GABAA recepto
77 fidence intervals, controlling for age, sex, tobacco smoking and opium usage history, alcohol consump
78                      We investigated whether tobacco smoking and other factors increase risk for Barr
79  and 95% confidence intervals, adjusting for tobacco smoking and other potential confounders.
80  childhood behavior might reduce early onset tobacco smoking and risk of tobacco dependence among smo
81 This study examined the associations between tobacco smoking and serum cotinine levels, an objective
82                                              Tobacco smoking and smoke exposure during pregnancy and
83 t in the neurochemical mechanisms underlying tobacco smoking and smoking-related behaviors.
84 emographic risk factors, baseline water pipe tobacco smoking and snus use were independently associat
85        Aware of the negative consequences of tobacco smoking and the challenges of smoking cessation,
86                                   Water pipe tobacco smoking and the use of snus at baseline.
87                                   Water pipe tobacco smoking and the use of snus independently predic
88  HIV-infected men ages 40-60, independent of tobacco smoking and traditional risk factors.
89 , physical activity, nutrition, alcohol, and tobacco smoking) and were ranked based on how frequently
90                              Clinician type, tobacco smoking, and age may be important clinical facto
91 pollutants, built environment, green spaces, tobacco smoking, and biomarkers of chemical pollutants (
92       Risk factors for CRC in Africa include tobacco smoking, and consumption of red meat, butter, an
93 , age, height, weight, total caloric intake, tobacco smoking, and education.
94                                     Obesity, tobacco smoking, and genetic predisposition increase the
95 G included short maternal stature (<145 cm), tobacco smoking, and HIV infection.
96  in England, including through drugs misuse, tobacco smoking, and homelessness.
97 rhosis, diabetes, obesity, alcohol drinking, tobacco smoking, and host genetic polymorphisms.
98 had higher body mass index, a higher rate of tobacco smoking, and lower lung function.
99 Associations of cognitive function, obesity, tobacco smoking, and medical conditions with natural cau
100 scular risk factors: hypertension, diabetes, tobacco smoking, and obesity across 230 cities in six co
101 obiome were observed with poor oral hygiene, tobacco smoking, and oral cancer.
102 residualization, adjusting for air nicotine, tobacco smoking, and other air particle generating or ve
103 le), past-month binge alcohol use, past-year tobacco smoking, and past-year suicidal ideation or suic
104 rence, body mass index, alcohol consumption, tobacco smoking, and physical activity.
105 es regarded as a less harmful alternative to tobacco smoking, and there is some evidence of their pot
106 for recurrent reflux, body mass index (BMI), tobacco smoking, and type of antireflux surgery.
107 enitor-like cells likely due to the combined tobacco smoking- and APOBEC3A-associated DNA damage and
108 sociated with >2 times the odds of taking up tobacco smoking [aOR 2.33, 1.28-4.24]).
109 decrement: 1.02 (95% CI 1.01-1.03)], current tobacco smoking [aOR versus never: 1.81 (95% CI 1.26-2.5
110 al interventions to reduce alcohol abuse and tobacco smoking are also needed.
111            Life-course persistent asthma and tobacco smoking are risk factors for irreversible airflo
112 al cell carcinoma, obesity, hypertension and tobacco smoking are risk factors, but they do not explai
113 tablished risk factors, that is, obesity and tobacco smoking, are overrepresented among patients who
114  in Nha Trang, Vietnam, to evaluate paternal tobacco smoking as a risk factor for infectious and non-
115                                 Results show tobacco smoking as a risk factor for oral HPV among PLWH
116                     Current cannabis use and tobacco smoking, as documented in the medical encounter.
117 tios (HRs) for lung cancer incidence by sex, tobacco smoking, asbestos exposure, presence of asbestos
118  Aberrant AID/APOBEC activity-associated and tobacco smoking-associated mutational signatures were re
119 s conducted at the Clinical Research Lab for Tobacco Smoking at Florida International University in M
120 bladder cancers are chemically induced, with tobacco smoking being the leading risk factor.
121 nd non-communicable diseases); health risks (tobacco smoking, binge drinking, overweight, and anaemia
122 are hypothesized to contribute to relapse to tobacco smoking but mechanisms underlying and linking su
123  5 million deaths a year are attributable to tobacco smoking, but attempts to help people either quit
124 Modeled age-standardized prevalence of daily tobacco smoking by age, sex, country, and year; cigarett
125  nicotine consumption and prevent relapse to tobacco smoking by modulating glutamate transmission.
126                                 The rates of tobacco smoking by people living with HIV vastly exceed
127                                              Tobacco smoking can account for some of the excess morta
128        Epidemiological studies indicate that tobacco smoking can be protective against neurodegenerat
129             Although residual confounding by tobacco smoking cannot be ruled out, this finding may re
130                                              Tobacco smoking causes lung cancer(1-3), a process that
131  important aim in public health worldwide as tobacco smoking causes many preventable deaths.
132  the evidence on the use of e-cigarettes for tobacco smoking cessation in adults, including pregnant
133 vidence on pharmacotherapy interventions for tobacco smoking cessation in pregnant persons is insuffi
134  net benefit of behavioral interventions for tobacco smoking cessation on perinatal outcomes and smok
135 F reviewed the evidence on interventions for tobacco smoking cessation that are relevant to primary c
136 ssociated (FDA)-approved pharmacotherapy for tobacco smoking cessation, alone or combined, in nonpreg
137 hanism contributing to its ability to aid in tobacco smoking cessation.
138  obstructive pulmonary disease (COPD) due to tobacco smoking commonly presents when extensive lung da
139                                              Tobacco smoking compromises the prognosis of dental impl
140 thylation in 5243 cancers of types for which tobacco smoking confers an elevated risk.
141                          Known signatures of tobacco smoking correlated with tobacco consumption, but
142                  DNA adducts associated with tobacco smoking could provide a marker of biologically e
143 ibutable and unattributable to hypertension, tobacco smoking, diabetes mellitus, and obesity using an
144                             Data on lifetime tobacco smoking, diet, education, and anthropometric mea
145         Hazardous lifestyle choices (notably tobacco smoking) differ in prevalence between genders, a
146              These findings demonstrate that tobacco smoking differentially affects the mesocortical
147                                              Tobacco smoking, driven by the addictive properties of n
148 losis risk was independently associated with tobacco smoking, drugs use (especially injectable drugs
149 has been reported that pre-conditioning with tobacco smoking during adolescence predisposes those you
150 y through year-4 and by frequent exposure to tobacco smoking during chemotherapy.
151                         Associations between tobacco smoking during pregnancy and offspring asthma ha
152                                              Tobacco smoking during this age can compromise the norma
153 ChRs) may underlie differential nicotine and tobacco smoking effects and related behaviors in women v
154 e and other risk factor trends continue, the Tobacco Smoking Elimination as of 2023 scenario quantifi
155 ng prevalence from 2023 onwards, whereas the Tobacco Smoking Elimination by 2050 scenario provides es
156                                              Tobacco smoking enhances peri-implant soft tissue inflam
157 nd chemotherapy-responsive tumor, related to tobacco smoking, environmental arsenic exposure, industr
158  findings suggest a possible route to reduce tobacco smoking-exacerbated NAFLD progression.
159 divergent evolutionary trajectories based on tobacco smoking exposure, ancestry and sex.
160 ding age, sex, genetic ancestry, second-hand tobacco smoking exposure, LCINS histology, stage, and ov
161 ss the world, reflecting varying patterns of tobacco smoking, exposure to environmental risk factors
162 t risk reduction, followed by a reduction in tobacco smoking for men and obesity for women, but these
163 ine is clearly recognized by the tenacity of tobacco smoking for most users, and has prompted extensi
164  variety of psychiatric disorders, including tobacco smoking, for which there is strong evidence supp
165 % confidence interval [CI], 1.7-49.3), heavy tobacco smoking (&gt;20 pack-years vs none; OR, 9.2; 95% CI
166 ught to be the key substance responsible for tobacco-smoking habits and appears to trigger reinforcem
167                    Cigarette and alternative tobacco smoking had a significant impact on the supragin
168                     The spread of water pipe tobacco smoking has been abetted by the marketing of fla
169                                              Tobacco smoking has been associated with impaired pulmon
170                                              Tobacco smoking has been demonstrated to increase the ri
171                                              Tobacco smoking has been linked to an increased risk of
172                                              Tobacco smoking has many adverse health consequences.
173               Exposure to direct and passive tobacco smoking has significant impact on the gingival a
174  (reflux), higher body mass index (BMI), and tobacco smoking have been individually associated with e
175 factors related to DALYs were dietary risks, tobacco smoking, high body mass index, high blood pressu
176                                   Cumulative tobacco smoking histories and spirometry were obtained a
177 N3), Zubrod performance status (0 vs 1), and tobacco smoking history (<=10 pack-years vs >10 pack-yea
178    A 62-year-old man with a 45 pack per year tobacco-smoking history presented with painless gross he
179 CI, 1.83-4.47; z = 4.62; adjusted P < .001), tobacco smoking (HR, 2.26; 95% CI, 1.55-3.30; z = 4.23;
180                Traditional risk factors were tobacco smoking, hypertension, elevated total cholestero
181 ars (wave 10), mental health problems, daily tobacco smoking, illicit drug use, and dependence were a
182 ioural risk factors were unhealthy diets and tobacco smoking in 2015.
183 ne, a noradrenergic alpha2a agonist, reduced tobacco smoking in a 4-week trial and in animal models h
184 2030, which means reducing the prevalence of tobacco smoking in adults to 5% by then.
185 elated neuroadaptations of cAMP signaling to tobacco smoking in human subjects and suggest that smoki
186             The pooled prevalence of current tobacco smoking in pregnant women ranged from 0.6% (0.3-
187  interactions between FLG null mutations and tobacco smoking in relation to asthma.
188 modeled age-standardized prevalence of daily tobacco smoking in the population older than 15 years de
189 lysis of covariance with age (in months) and tobacco smoking (in pack-years) as covariates showed tha
190  from solid fuels (HAP; 7.0% [5.6-8.3]), and tobacco smoking including second-hand smoke (6.1% [5.4-6
191 ertainty interval 6.2-7.7] of global DALYs), tobacco smoking including second-hand smoke (6.3% [5.5-7
192                            Despite declines, tobacco smoking including second-hand smoke remained the
193                                        Thus, tobacco smoking increases mutational burden, cell-to-cel
194                                              Tobacco smoking increases the risk of at least 17 classe
195   These results provide further support that tobacco smoking increases the risk of developing aortic
196                                     Paternal tobacco smoking independently increased the risk of LRTI
197 tors, such as the microbiome, nutrition, and tobacco smoking, induce alterations in the epigenome of
198 ]PHNO PET may be more sensitive to measuring tobacco smoking-induced DA release in human tobacco smok
199 latory effects of nicotine in the brain (ie, tobacco smoking-induced upregulation of beta(2)*-nAChRs)
200 ttributable risk (PAR) of bladder cancer for tobacco smoking is 50% to 65% in men and 20% to 30% in w
201                                              Tobacco smoking is a chronic, relapsing disorder that co
202                                              Tobacco smoking is a major risk factor for many diseases
203                                              Tobacco smoking is a risk factor for numerous disorders,
204                                              Tobacco smoking is a well-known risk factor for subseque
205                                              Tobacco smoking is a worldwide public health problem.
206                                              Tobacco smoking is associated with a substantially eleva
207                                              Tobacco smoking is associated with chronic obstructive p
208                                              Tobacco smoking is associated with differential methylat
209                                     Parental tobacco smoking is associated with lower airway function
210                                              Tobacco smoking is common in HIV-infected patients, and
211                                              Tobacco smoking is considered a major modifiable risk fa
212                           Once dependence on tobacco smoking is established, evidence suggests that t
213                                              Tobacco smoking is frequently abused by schizophrenia pa
214                                    Waterpipe tobacco smoking is growing in popularity despite adverse
215                                              Tobacco smoking is highly addictive and causes respirato
216 ented in countries where ETS due to maternal tobacco smoking is negligible.
217                                              Tobacco smoking is the dominant risk factor for chronic
218                                              Tobacco smoking is the leading cause but not the only on
219                                              Tobacco smoking is the leading cause of preventable deat
220                                              Tobacco smoking is the leading risk factor for bladder c
221                                              Tobacco smoking is the major cause of lung cancer, and p
222                                              Tobacco smoking is the most important and well-establish
223                                   Worldwide, tobacco smoking is the principal lifestyle exposure that
224                                     Although tobacco smoking is well-known to increase esophageal aci
225 ove to be one of the key mechanisms by which tobacco smoking leads to increased periodontitis suscept
226                     Our results suggest that tobacco smoking may alter the genetic susceptibility to
227  to help them quit, the benefits of quitting tobacco smoking may outweigh any negative oral health im
228                                              Tobacco smoking may play an important role in both the o
229 s study was to investigate whether different tobacco smoking metrics and smoking cessation was associ
230 ing that there are various pathways by which tobacco smoking might contribute to development of Barre
231 tients with lung cancer report no history of tobacco smoking (never-smokers).
232                                              Tobacco smoking, nicotine in particular, is one of the r
233              At blood levels comparable with tobacco smoking, nicotine infusion does not appear to al
234 lcohol withdrawal with or without concurrent tobacco smoking/nicotine consumption resulted in signifi
235 e, calendar period, and diagnoses related to tobacco smoking, obesity, and alcohol overconsumption.
236                                      Besides tobacco smoking, occupation, and other factors, diet may
237 e disorders, the most robust risk factor was tobacco smoking (odds ratio, OR=3.07).
238                 Understanding the effects of tobacco smoking on neuroadaptations in GABAA receptor le
239  needed to fully characterize the effects of tobacco smoking on the brain immune system.
240                               The effects of tobacco smoking on the immune system of the brain are no
241 risk, we sought to investigate the impact of tobacco smoking on the risk of premature death and its c
242 ere we report on the sex-specific effects of tobacco smoking on transcriptomic and epigenetic feature
243  These associations were not attributable to tobacco smoking or clozapine medication.
244 -base substitutions were generated by either tobacco smoking or exposure to ultraviolet light.
245 cancers, which could indicate confounding by tobacco smoking or reverse causality.
246  secondary causes of erythrocytosis, such as tobacco smoking or sleep apnea.
247                                 In contrast, tobacco smoking (OR = 0.96, 95% CI: 0.36-2.55), alcohol
248   This blunting may contribute to relapse to tobacco smoking, particularly in depression-vulnerable i
249 er among males and females reflect differing tobacco smoking patterns.
250 moking status, demographics, alcohol intake, tobacco smoking, physical activity, and included a food-
251 ome variable (the percentage point change in tobacco smoking prevalence between 2005 and 2015).
252  and a 96.6% response rate, found an overall tobacco smoking prevalence of 19.4%, with higher rates a
253                                       Higher tobacco smoking, psychosis, and diabetes ES were found i
254  problems later in life, we generated ES for tobacco smoking, psychosis, autism, diabetes, and obesit
255 ver, this excess risk may be attributable to tobacco smoking rather than the three-fold higher preval
256 D (chronic obstructive pulmonary disease), a tobacco smoking-related disease.
257 that acrolein is a major etiologic agent for tobacco smoking-related lung cancer.
258          We sought to quantify the burden of tobacco-smoking-related deaths in Asia, in parts of whic
259                                              Tobacco smoking remains an important public health issue
260                                              Tobacco smoking represents the leading preventable cause
261 0.9-4.0) for 29 to 39 years and >39 years of tobacco smoking, respectively, as compared with those th
262                                 Addiction to tobacco smoking results from the binding of nicotine to
263  significantly increase risk for SP included tobacco smoking (RR, 2.47; 95% CI, 2.12-2.87), alcohol i
264 her odds of lung cancer after adjustment for tobacco smoking, serum cotinine levels, educational atta
265                                    No strong tobacco smoking signatures were detected, even in cases
266 , and overall relative prevalence ratios for tobacco smoking, smokeless tobacco use, and any tobacco
267                                To assess how tobacco smoking status affects baseline dopamine D2/D3 (
268  gender, age at time of initial examination, tobacco smoking status at time of initial examination, p
269 l confounders, particularly body mass index, tobacco smoking status, and socioeconomic status, part o
270 e likely to smoke, and, after adjustment for tobacco-smoking status and other potential confounders,
271 y factors, including childhood maltreatment, tobacco smoking, substance dependence, psychiatric medic
272  a substantial decrease in the prevalence of tobacco smoking, the adverse health effects of tobacco s
273 s of four factors: HPV status, pack-years of tobacco smoking, tumor stage, and nodal stage.
274          Chronic exposure to nicotine, as in tobacco smoking, up-regulates nicotinic acetylcholine re
275 and cause-specific mortality associated with tobacco smoking using adjusted hazard ratios and their 9
276  CI, 0.82-1.14, P = .69) after adjusting for tobacco smoking, vaccination, comorbidity, diagnosis dat
277  between ages 40-60 after adjusting for age, tobacco smoking, viral load, and traditional risk factor
278 ts with validated continuous abstinence from tobacco smoking was 28.9% in the intervention group and
279  studies established for the first time that tobacco smoking was a risk factor for the development of
280 r, prevalence of psychiatric comorbidity and tobacco smoking was alarmingly high in severe patients w
281 healthy infants whether exposure to parental tobacco smoking was associated with airway hyperreactivi
282                                     Parental tobacco smoking was associated with lower baseline airwa
283                                              Tobacco smoking was associated with treatment escalation
284               In the PATH Study, combustible tobacco smoking was consistently associated with lifetim
285 istories, the mutational signature caused by tobacco smoking was essentially absent in NSRO-driven NS
286               The strongest association with tobacco smoking was found for lung cancer: a 3- to 4-fol
287                                      Current tobacco smoking was reported at one or more timepoints b
288                                      Current tobacco smoking was significantly associated with an inc
289                                      Current tobacco smoking was significantly associated with increa
290                                        Thus, tobacco smoking was the main explanation for poor progno
291                                              Tobacco smoking was the major influence on mutational bu
292 eristics, especially alcohol consumption and tobacco smoking, we examined promoter methylation of the
293        Survey data that did not report daily tobacco smoking were adjusted using the average relation
294                           Infants exposed to tobacco smoking were approximately half as likely to dev
295                         Median pack-years of tobacco smoking were lower among p16-positive than p16-n
296 DA) system drives the reinforcing effects of tobacco smoking, whereas the mesocortical DA system-incl
297 gh concentrations of nicotine resulting from tobacco smoking will cause an abnormal activation, a des
298                                              Tobacco smoking with a water pipe or hookah is increasin
299 ng with gastresophageal reflux, obesity, and tobacco smoking with genome-wide significance.
300            Clinical observations have linked tobacco smoking with increased type 2 diabetes risk.

 
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