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1 hrenia is associated with very high rates of tobacco smoking.
2 , for example, the "pack-years" variable for tobacco smoking.
3 tational signatures that are associated with tobacco smoking.
4 diseases, respectively, were attributable to tobacco smoking.
5 models adjusted for age, sex, and cumulative tobacco smoking.
6 increased with each additional pack-year of tobacco smoking.
7 Risk factors for periodontal disease include tobacco smoking.
8 r levels by 6 to 12 weeks of abstinence from tobacco smoking.
9 xplore the mutational burden associated with tobacco smoking.
10 is a major cause for starting or increasing tobacco smoking.
11 ntribute to the perpetuation of addiction to tobacco smoking.
12 on between the level of fasting glycemia and tobacco smoking.
13 ich despite some success have not eliminated tobacco smoking.
14 use of death, and almost all of it is due to tobacco smoking.
15 the youngest infants and in those exposed to tobacco smoking.
16 exposure of the upper aerodigestive tract to tobacco smoking.
17 on with the effect of nonsmoking and regular tobacco smoking.
18 sted waist-to-hip ratio, alcohol intake, and tobacco smoking.
19 of the genetic effect of body mass index and tobacco smoking.
20 inconclusive due to confounding by previous tobacco smoking.
21 a indicate that vaping may not be safer than tobacco smoking.
22 ically involved in the reinforcing effect of tobacco smoking.
23 x disease, Barrett's esophagus, obesity, and tobacco smoking.
24 ion treatments and reduce the harm caused by tobacco smoking.
25 association with risk for IBD is modified by tobacco smoking.
26 t disease, diseases strongly associated with tobacco smoking.
27 , but this does not seem to be made worse by tobacco smoking.
28 ical tumors and that LOY could be induced by tobacco smoking.
29 e, the principal addictive component driving tobacco smoking.
30 geting prevention and cessation of waterpipe tobacco smoking.
31 ntified strong epigenetic signals related to tobacco smoking.
32 (beta +/- SE 0.07 +/- 0.01; P < 0.0001) and tobacco smoking (0.64 +/- 0.26; P = 0.016), as well as w
33 risk ratio [RR] 1.41 [95% CI 1.26-1.57]) and tobacco smoking (1.46 [1.30-1.65]) than in HIV-negative
34 use (RR 1.36 [95% CI 1.10-1.69]; p=0.0050), tobacco smoking (1.90 [1.38-2.62]; p<0.0001), and smokel
35 positive women was 1.3% (95% CI 0.8-1.9) for tobacco smoking, 2.1% (1.1-3.4) for smokeless tobacco us
37 ositive men was 24.4% (95% CI 21.1-27.8) for tobacco smoking, 3.4% (1.8-5.6) for smokeless tobacco us
38 SBS4, which has previously been linked with tobacco smoking(9), and a 76% increase in the clock-like
39 The primary outcome was 7 months continuous tobacco smoking abstinence as measured at 9 months after
42 e no significant differences in gender, age, tobacco smoking, Acute Physiology and Chronic Health Eva
43 st associations, with the exception of daily tobacco smoking (adjusted OR 1.74, 95% CI 1.08-2.81), an
44 There is growing evidence that water pipe tobacco smoking affects heart rate, blood pressure regul
46 y (aHR = 2.757 [1.616-4.704], p < 0.001) and tobacco smoking (aHR = 2.150 [1.319-3.503], p < 0.01) we
47 lected on education, household overcrowding, tobacco smoking, alcohol and drugs use, and history of h
48 adult exposures to carcinogens, such as from tobacco smoking, alcohol consumption and various industr
49 rders in general), including breast-feeding, tobacco smoking, alcohol consumption, and exposure to do
50 ,115) and tested the association of RBA with tobacco smoking, alcohol consumption, and genetic varian
51 extensive questionnaire containing items on tobacco smoking, alcohol consumption, and occupational e
52 gnancy was identified based on self-reported tobacco smoking, alcohol consumption, and use of cannabi
53 sion models that were adjusted for age, sex, tobacco smoking, alcohol drinking, and hypertension.
54 terms for age, sex, study center, education, tobacco smoking, alcohol drinking, hepatitis B surface a
56 ze existing data on the prevalence of active tobacco smoking among patients with hypertension or diab
58 from the general population, in relation to tobacco smoking, an exposure with well-characterised hea
59 o specific risk factors, such as alcohol and tobacco (smoking and smokeless), but dietary factors and
62 ted with the widely accepted risk factors of tobacco smoking and alcohol use and is the end result of
63 ants of health (SDHs, including drug misuse, tobacco smoking and alcohol), and TB, taking into accoun
64 mokers to assess whether baseline water pipe tobacco smoking and baseline snus use were associated wi
65 models to assess whether baseline water pipe tobacco smoking and baseline snus use were associated wi
68 population density areas, and pack-years of tobacco smoking and decreases in subjects with history o
72 rience co-occurring substance use, including tobacco smoking and heavy cannabis use, and substance us
73 h decreased risk of ADA development, whereas tobacco smoking and infections during the study were ass
75 anged by at least 2 percentage points (e.g., tobacco smoking and mental health conditions) were assoc
76 s to investigate the differential effects of tobacco smoking and nicotine on changes in GABAA recepto
77 fidence intervals, controlling for age, sex, tobacco smoking and opium usage history, alcohol consump
80 childhood behavior might reduce early onset tobacco smoking and risk of tobacco dependence among smo
81 This study examined the associations between tobacco smoking and serum cotinine levels, an objective
84 emographic risk factors, baseline water pipe tobacco smoking and snus use were independently associat
89 , physical activity, nutrition, alcohol, and tobacco smoking) and were ranked based on how frequently
91 pollutants, built environment, green spaces, tobacco smoking, and biomarkers of chemical pollutants (
99 Associations of cognitive function, obesity, tobacco smoking, and medical conditions with natural cau
100 scular risk factors: hypertension, diabetes, tobacco smoking, and obesity across 230 cities in six co
102 residualization, adjusting for air nicotine, tobacco smoking, and other air particle generating or ve
103 le), past-month binge alcohol use, past-year tobacco smoking, and past-year suicidal ideation or suic
105 es regarded as a less harmful alternative to tobacco smoking, and there is some evidence of their pot
107 enitor-like cells likely due to the combined tobacco smoking- and APOBEC3A-associated DNA damage and
109 decrement: 1.02 (95% CI 1.01-1.03)], current tobacco smoking [aOR versus never: 1.81 (95% CI 1.26-2.5
112 al cell carcinoma, obesity, hypertension and tobacco smoking are risk factors, but they do not explai
113 tablished risk factors, that is, obesity and tobacco smoking, are overrepresented among patients who
114 in Nha Trang, Vietnam, to evaluate paternal tobacco smoking as a risk factor for infectious and non-
117 tios (HRs) for lung cancer incidence by sex, tobacco smoking, asbestos exposure, presence of asbestos
118 Aberrant AID/APOBEC activity-associated and tobacco smoking-associated mutational signatures were re
119 s conducted at the Clinical Research Lab for Tobacco Smoking at Florida International University in M
121 nd non-communicable diseases); health risks (tobacco smoking, binge drinking, overweight, and anaemia
122 are hypothesized to contribute to relapse to tobacco smoking but mechanisms underlying and linking su
123 5 million deaths a year are attributable to tobacco smoking, but attempts to help people either quit
124 Modeled age-standardized prevalence of daily tobacco smoking by age, sex, country, and year; cigarett
125 nicotine consumption and prevent relapse to tobacco smoking by modulating glutamate transmission.
132 the evidence on the use of e-cigarettes for tobacco smoking cessation in adults, including pregnant
133 vidence on pharmacotherapy interventions for tobacco smoking cessation in pregnant persons is insuffi
134 net benefit of behavioral interventions for tobacco smoking cessation on perinatal outcomes and smok
135 F reviewed the evidence on interventions for tobacco smoking cessation that are relevant to primary c
136 ssociated (FDA)-approved pharmacotherapy for tobacco smoking cessation, alone or combined, in nonpreg
138 obstructive pulmonary disease (COPD) due to tobacco smoking commonly presents when extensive lung da
143 ibutable and unattributable to hypertension, tobacco smoking, diabetes mellitus, and obesity using an
148 losis risk was independently associated with tobacco smoking, drugs use (especially injectable drugs
149 has been reported that pre-conditioning with tobacco smoking during adolescence predisposes those you
153 ChRs) may underlie differential nicotine and tobacco smoking effects and related behaviors in women v
154 e and other risk factor trends continue, the Tobacco Smoking Elimination as of 2023 scenario quantifi
155 ng prevalence from 2023 onwards, whereas the Tobacco Smoking Elimination by 2050 scenario provides es
157 nd chemotherapy-responsive tumor, related to tobacco smoking, environmental arsenic exposure, industr
160 ding age, sex, genetic ancestry, second-hand tobacco smoking exposure, LCINS histology, stage, and ov
161 ss the world, reflecting varying patterns of tobacco smoking, exposure to environmental risk factors
162 t risk reduction, followed by a reduction in tobacco smoking for men and obesity for women, but these
163 ine is clearly recognized by the tenacity of tobacco smoking for most users, and has prompted extensi
164 variety of psychiatric disorders, including tobacco smoking, for which there is strong evidence supp
165 % confidence interval [CI], 1.7-49.3), heavy tobacco smoking (>20 pack-years vs none; OR, 9.2; 95% CI
166 ught to be the key substance responsible for tobacco-smoking habits and appears to trigger reinforcem
174 (reflux), higher body mass index (BMI), and tobacco smoking have been individually associated with e
175 factors related to DALYs were dietary risks, tobacco smoking, high body mass index, high blood pressu
177 N3), Zubrod performance status (0 vs 1), and tobacco smoking history (<=10 pack-years vs >10 pack-yea
178 A 62-year-old man with a 45 pack per year tobacco-smoking history presented with painless gross he
179 CI, 1.83-4.47; z = 4.62; adjusted P < .001), tobacco smoking (HR, 2.26; 95% CI, 1.55-3.30; z = 4.23;
181 ars (wave 10), mental health problems, daily tobacco smoking, illicit drug use, and dependence were a
183 ne, a noradrenergic alpha2a agonist, reduced tobacco smoking in a 4-week trial and in animal models h
185 elated neuroadaptations of cAMP signaling to tobacco smoking in human subjects and suggest that smoki
188 modeled age-standardized prevalence of daily tobacco smoking in the population older than 15 years de
189 lysis of covariance with age (in months) and tobacco smoking (in pack-years) as covariates showed tha
190 from solid fuels (HAP; 7.0% [5.6-8.3]), and tobacco smoking including second-hand smoke (6.1% [5.4-6
191 ertainty interval 6.2-7.7] of global DALYs), tobacco smoking including second-hand smoke (6.3% [5.5-7
195 These results provide further support that tobacco smoking increases the risk of developing aortic
197 tors, such as the microbiome, nutrition, and tobacco smoking, induce alterations in the epigenome of
198 ]PHNO PET may be more sensitive to measuring tobacco smoking-induced DA release in human tobacco smok
199 latory effects of nicotine in the brain (ie, tobacco smoking-induced upregulation of beta(2)*-nAChRs)
200 ttributable risk (PAR) of bladder cancer for tobacco smoking is 50% to 65% in men and 20% to 30% in w
225 ove to be one of the key mechanisms by which tobacco smoking leads to increased periodontitis suscept
227 to help them quit, the benefits of quitting tobacco smoking may outweigh any negative oral health im
229 s study was to investigate whether different tobacco smoking metrics and smoking cessation was associ
230 ing that there are various pathways by which tobacco smoking might contribute to development of Barre
234 lcohol withdrawal with or without concurrent tobacco smoking/nicotine consumption resulted in signifi
235 e, calendar period, and diagnoses related to tobacco smoking, obesity, and alcohol overconsumption.
241 risk, we sought to investigate the impact of tobacco smoking on the risk of premature death and its c
242 ere we report on the sex-specific effects of tobacco smoking on transcriptomic and epigenetic feature
248 This blunting may contribute to relapse to tobacco smoking, particularly in depression-vulnerable i
250 moking status, demographics, alcohol intake, tobacco smoking, physical activity, and included a food-
252 and a 96.6% response rate, found an overall tobacco smoking prevalence of 19.4%, with higher rates a
254 problems later in life, we generated ES for tobacco smoking, psychosis, autism, diabetes, and obesit
255 ver, this excess risk may be attributable to tobacco smoking rather than the three-fold higher preval
261 0.9-4.0) for 29 to 39 years and >39 years of tobacco smoking, respectively, as compared with those th
263 significantly increase risk for SP included tobacco smoking (RR, 2.47; 95% CI, 2.12-2.87), alcohol i
264 her odds of lung cancer after adjustment for tobacco smoking, serum cotinine levels, educational atta
266 , and overall relative prevalence ratios for tobacco smoking, smokeless tobacco use, and any tobacco
268 gender, age at time of initial examination, tobacco smoking status at time of initial examination, p
269 l confounders, particularly body mass index, tobacco smoking status, and socioeconomic status, part o
270 e likely to smoke, and, after adjustment for tobacco-smoking status and other potential confounders,
271 y factors, including childhood maltreatment, tobacco smoking, substance dependence, psychiatric medic
272 a substantial decrease in the prevalence of tobacco smoking, the adverse health effects of tobacco s
275 and cause-specific mortality associated with tobacco smoking using adjusted hazard ratios and their 9
276 CI, 0.82-1.14, P = .69) after adjusting for tobacco smoking, vaccination, comorbidity, diagnosis dat
277 between ages 40-60 after adjusting for age, tobacco smoking, viral load, and traditional risk factor
278 ts with validated continuous abstinence from tobacco smoking was 28.9% in the intervention group and
279 studies established for the first time that tobacco smoking was a risk factor for the development of
280 r, prevalence of psychiatric comorbidity and tobacco smoking was alarmingly high in severe patients w
281 healthy infants whether exposure to parental tobacco smoking was associated with airway hyperreactivi
285 istories, the mutational signature caused by tobacco smoking was essentially absent in NSRO-driven NS
292 eristics, especially alcohol consumption and tobacco smoking, we examined promoter methylation of the
296 DA) system drives the reinforcing effects of tobacco smoking, whereas the mesocortical DA system-incl
297 gh concentrations of nicotine resulting from tobacco smoking will cause an abnormal activation, a des