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1 d risk factors for NCDs (such as obesity and tobacco use).
2 amily history, obesity, type 2 diabetes, and tobacco use.
3 male sex, white race, abdominal obesity, and tobacco use.
4 s tobacco use, and 27.1% (22.8-31.7) for any tobacco use.
5 t class analysis indicated 5 classes of poly-tobacco use.
6 etes, and 19.4% reported current or previous tobacco use.
7 re, both forms of disease are exacerbated by tobacco use.
8 ntation of evidence-based measures to reduce tobacco use.
9 bidities, physical activity, and alcohol and tobacco use.
10 n phenotypes, including hypercoagulation and tobacco use.
11 arkinson's disease and a person's history of tobacco use.
12 106 never exposed, matched for age, sex, and tobacco use.
13 ables after adjusting for sex, diabetes, and tobacco use.
14 yzing the impact of pre transplant recipient tobacco use.
15 of negative events increases with increasing tobacco use.
16 guard future generations from the ravages of tobacco use.
17 nt effective public health policies limiting tobacco use.
18 ovascular risk factors, including especially tobacco use.
19 re paradigm that may better model adolescent tobacco use.
20  some regions for years of cocaine and daily tobacco use.
21  systemic inflammation, pericardial fat, and tobacco use.
22  than currently agreed should be adopted for tobacco use.
23 early age of initiation for both alcohol and tobacco use.
24 gets for all three of these measures and for tobacco use.
25 , in many cases cannabis use develops before tobacco use.
26 h-burden countries for both tuberculosis and tobacco use.
27 l, high-density lipoprotein cholesterol, and tobacco use.
28  sex, body mass index, recent infection, and tobacco use.
29 forts to reduce morbidity and mortality from tobacco use.
30 drawal may have a critical role in promoting tobacco use.
31 ving diet, increasing activity, and reducing tobacco use.
32 bits, increase physical activity, and reduce tobacco use.
33 n other physiological effects of smoking and tobacco use.
34 a diagnosis of bladder cancer on patterns of tobacco use.
35 etes, and 19.4% reported current or previous tobacco use.
36 symptoms representing a major burden to quit tobacco use.
37 omote novel therapeutic strategies to reduce tobacco use.
38 e considered a safe alternative to combusted tobacco use.
39  population health consequences of continued tobacco use.
40 ntial adverse health effects of this form of tobacco use.
41 Rs) of OPC, genital HPV in healthy women, or tobacco use.
42  the acceleration of biological aging due to tobacco use.
43 r-specific mortality risks of dual- and poly-tobacco use.
44 bacco use, and 3.6% (95% CI 2.3-5.2) for any tobacco use.
45  increased age, prior radiation therapy, and tobacco use.
46 oth toxicants and carcinogens resulting from tobacco use.
47 us cerebrovascular disease) + 0.352 x (prior tobacco use) + 0.376 x (number of devices >2) + 0.016 x
48  an open fire (1.32; 1.22-1.43) and maternal tobacco use (1.23; 1.18-1.27).
49  (1.90 [1.38-2.62]; p<0.0001), and smokeless tobacco use (1.32 [1.03-1.69]; p=0.030) than in HIV-nega
50  than 50 years [5.19, (3.05-8.29)]; previous tobacco use [1.40 (1.18-1.66)]; and complicated initial
51 ascular disease (2.4% versus 3.3%; P=0.002), tobacco use (36.5% versus 52.3%; P<0.001), and prior car
52 ; lipid disorders, 12%-21%; diabetes, 4%-7%; tobacco use, 5%-16%; and obesity, 4%-9%).
53  with older age, cytotoxic chemotherapy, and tobacco use; 75% of survivors had symptomatic improvemen
54                   A more ambitious target on tobacco use (a 50% reduction) will almost reach the targ
55                                              Tobacco use, a primary HNSCC risk factor, was evaluated
56  persistent opioid use included preoperative tobacco use (adjusted odds ratio [aOR], 1.35; 95% CI, 1.
57 that clinicians ask all pregnant women about tobacco use, advise them to stop using tobacco, and prov
58 ommends that clinicians ask all adults about tobacco use, advise them to stop using tobacco, and prov
59 essation before LTx were at greatest risk of tobacco use after LTx.
60 spective study, we describe the frequency of tobacco use after lung transplantation (LTx), pretranspl
61                                              Tobacco use after solid organ transplantation is associa
62 gestational weight gain, high underlying BP, tobacco use, alcohol consumption, and sedentary lifestyl
63 hnicity, body mass index, physical activity, tobacco use, alcohol consumption, formal education, clin
64 lmonary disease and coronary artery disease, tobacco use, alcohol use, and body mass index were also
65 ultiple measures of socioeconomic status and tobacco use, alcohol use, diet, and physical activity.
66       Pooled prevalence estimates of current tobacco use, alcohol use, low fruit and vegetable intake
67 d with a cluster of behavioural factors (ie, tobacco use, alcohol, diet, physical activity, and sodiu
68 ing was associated with an increased risk of tobacco use among adolescent offspring.
69                We assessed the prevalence of tobacco use among people living with HIV in LMICs.
70 er, very little is known about the extent of tobacco use among people living with HIV in low-income a
71                                              Tobacco use among people living with HIV results in exce
72 r brief counseling, to prevent initiation of tobacco use among school-aged children and adolescents.
73 -feasible interventions for the cessation of tobacco use among school-aged children and adolescents.
74 y, and the potential to create a new form of tobacco use among those no longer interested in taking u
75 rent tobacco use (smoked, smokeless, and any tobacco use) among 6729 HIV-positive men from 27 LMICs (
76 the potential ENDS have to alter patterns of tobacco use and affect the health of the public; however
77 Although the most important risk factors are tobacco use and alcohol consumption, the disease is also
78 e to use or problem use, as were alcohol and tobacco use and disorders, major depression, and schizot
79 tand the cancer risks of exclusive smokeless tobacco use and dual use of smokeless tobacco with other
80                 Prevention and management of tobacco use and exposure to second-hand smoke in pregnan
81                Previous studies on smokeless tobacco use and head and neck cancer (HNC) have found in
82 onally representative, longitudinal study of tobacco use and health in the United States.
83 tween 2003 and 2014 from 28 LMICs where both tobacco use and HIV test data were made publicly availab
84 er of lifetime oral sexual partners, current tobacco use and immunosuppression.
85 l strategies that have significantly reduced tobacco use and initiation in the United States.
86    Excessive proteolysis occurs with chronic tobacco use and is causative for bronchiectasis and emph
87  by the reversal of socioeconomic gradients; tobacco use and low fruit and vegetable intake have beco
88 l groups; and was partially accounted for by tobacco use and mean arterial pressure.
89                      They also had increased tobacco use and more comorbidities.
90 phic characteristics, lifestyle (alcohol and tobacco use and physical activity), and medication, were
91 ncluded studies presented clear patterns for tobacco use and physical activity, heterogeneity between
92  insomnia, asthma, rhinitis, weight, height, tobacco use and physical activity.
93  Goal 3, setting a global target of reducing tobacco use and premature mortality from non-communicabl
94     The possibility of a causal link between tobacco use and psychosis merits further examination.
95 mprove health behaviors (for example, reduce tobacco use and sedentary behavior and improve diet) of
96 20 years of age were queried regarding their tobacco use and serum cotinine was measured.
97 a diagnosis of bladder cancer on patterns of tobacco use and smoking cessation among patients with in
98 iovascular risk factors, as well as reducing tobacco use and substance abuse, should be leading prior
99 se correlation between a person's history of tobacco use and susceptibility to developing Parkinson's
100 king current perceptions of service members' tobacco use and unmasking the forces perpetuating those
101 obacco smoking, 3.4% (1.8-5.6) for smokeless tobacco use, and 27.1% (22.8-31.7) for any tobacco use.
102 obacco smoking, 2.1% (1.1-3.4) for smokeless tobacco use, and 3.6% (95% CI 2.3-5.2) for any tobacco u
103 e pulmonary disease, valvular heart disease, tobacco use, and alcohol abuse.
104 alence ratios for tobacco smoking, smokeless tobacco use, and any tobacco use separately for males an
105  other vegetables, age, sex, race/ethnicity, tobacco use, and caffeine.
106 d the health risks associated with nicotine, tobacco use, and electronic nicotine delivery systems in
107 f traditional risk factors (such as obesity, tobacco use, and genetic predisposition) and HIV-specifi
108 ms, hiatal hernia, elevated body mass index, tobacco use, and intra-abdominal distribution of fat) to
109 leep disturbance, heavy alcohol use, current tobacco use, and larger initial opioid prescription size
110  microcatheter type, smoking history, active tobacco use, and other risk factors for arterial disease
111 s, obesity, dyslipidemia, drug dependence or tobacco use, and renal disease.
112 ociated with early initiation of alcohol and tobacco use, and rs6495309 has been associated with nico
113 ace/ethnicity, education, employment status, tobacco use, and scanner technology.
114 ng for sex, race, education, income, region, tobacco use, and select chronic medical conditions (0-1
115 lant patient characteristics associated with tobacco use, and the safety, efficacy, and outcomes of p
116 perative group clinical trials do not assess tobacco use, and there is no observable trend in improve
117  residence, applicator license type, chewing tobacco use, and total lifetime days of all pesticide us
118                                    Smokeless tobacco use appears to be associated with HNC, especiall
119 se health effects of intermittent water pipe tobacco use are critical to strengthen the evidence base
120                       Rates of dual and poly-tobacco use are high among trainees, and while these gro
121  prevent initiation and promote cessation of tobacco use are needed to reduce associated morbidity an
122 ng suboptimal diet, physical inactivity, and tobacco use, are leading causes of preventable diseases
123 history, systemic co-morbidities, alcohol or tobacco use as well as serum levels of calcium and vitam
124 similar in magnitude to that associated with tobacco use, as compared with no use (hazard ratio, 1.54
125 tors, including excess sugar consumption and tobacco use, as well as underlying infection and inflamm
126 Cooperative Group Program were evaluated for tobacco use assessment at enrollment and follow-up by us
127    The purpose of this study was to evaluate tobacco use assessment in patients enrolled onto activel
128 al laboratories as well as for point-of-care tobacco use assessment.
129 ace and ethnicity or those with a history of tobacco use, asthma, or diabetes mellitus type II.
130 ials reviewed, 45 (29%) assessed any form of tobacco use at enrollment, but only 34 (21.9%) assessed
131                           Weight loss >= 5%, tobacco use at LEMS onset and age at onset >= 50 years w
132                 Patients who reported active tobacco use at the time of PAVM embolization had a 5-yea
133 -demographic factors, CD4 count, alcohol and tobacco use, baseline WHO stage 4 disease, social suppor
134                              As the risks of tobacco use become recognized and smoking becomes stigma
135 Respondents completed a survey on history of tobacco use, beliefs regarding bladder cancer risk facto
136                                     Stopping tobacco use benefits virtually every smoker.
137 ied according to centre, performance status, tobacco use, best response to previous EGFR tyrosine-kin
138 nts also reveal hidden relationships between tobacco use, body mass index, and blood pressure.
139 are effective measures to reduce alcohol and tobacco use, but are not available to target illicit dru
140                 This study hypothesized that tobacco use by PAD patients would be associated with hig
141                       Methamphetamine and/or tobacco use by pregnant women remains prevalent.
142 ints were biomarker-calibrated self-reported tobacco use by the mother at late pregnancy, birthweight
143                      Tobacco use initiation; tobacco use cessation; health outcomes; harms.
144 health at age 38, even after controlling for tobacco use, childhood health, and childhood socioeconom
145                    Age, sex, race/ethnicity, tobacco use, chronic conditions, influenza vaccination,
146                         Risk factors include tobacco use, coronary artery disease, and respiratory fa
147 ssued a separate recommendation statement on tobacco use counseling in adults and pregnant women.
148 archers should use caution when interpreting tobacco use data obtained from check-all-that-apply form
149 rable changes in lifestyle such as increased tobacco use, decreased physical activity, and consumptio
150       Linear regression adjusted for income, tobacco use, dental insurance, and previous radiation an
151 , if risk factor targets for blood pressure, tobacco use, diabetes mellitus, and obesity are achieved
152 ce/ethnicity, and income level), alcohol and tobacco use, diabetes mellitus, and past periodontal tre
153                                              Tobacco use did not change significantly, but the 10-yea
154 , P < 10(-30)), followed by individuals with tobacco use disorder (TUD) (AOR = 8.222 ([7.925-8.530],
155  was significantly inversely associated with tobacco use disorder comorbidities (adjusted odds ratio
156  and caffeine withdrawal syndromes, aligning tobacco use disorder criteria with other substance use d
157                                              Tobacco use disorder is associated with dysregulated neu
158 ission and enhancing IC during the course of tobacco use disorder treatment.
159 ne the co-occurrence of past-year alcohol or tobacco use disorder with past-year anxiety disorders, m
160 rders, largely driven by an association with tobacco use disorder.
161 among those who met criteria for a past-year tobacco use disorder.
162 ase; 6) hyperlipidemia; 7) alcohol abuse; 8) tobacco use disorder; and 9) random effect of geographic
163 tudy of Nicotine Dependence (ascertained for tobacco use disorder; n = 918 cases; 988 control subject
164 es associated with alcohol use disorders and tobacco use disorders among heterosexual, bisexual, and
165 nly seven trials (4.5%) assessed any form of tobacco use during follow-up.
166  nicotine replacement therapy, and smokeless tobacco use during pregnancy are associated with cogniti
167 fluid balance, obesity, hypoxemia, acidosis, tobacco use, emergent hematoma evacuation, and vasopress
168 which might include behaviors such as lesser tobacco use, greater physical activity, and/or enhanced
169 art failure, peripheral arterial disease, or tobacco use had the largest predicted differences in sur
170           Substantial evidence suggests that tobacco use has adverse effects on cancer treatment outc
171                                              Tobacco use has fallen in countries that are members of
172 ent outcomes; however, routine assessment of tobacco use has not been fully incorporated into standar
173 To date, most genetic association studies of tobacco use have been conducted in European American sub
174 ets for reducing systolic blood pressure and tobacco use have more substantial effects on future scen
175 t were relevant to primary care and reported tobacco use, health outcomes, or harms were included.
176 l concentrated among patients with diabetes, tobacco use, heart failure, or peripheral arterial disea
177 s for SCD: older age, African-American race, tobacco use, higher pulse, higher waist-to-hip ratio, el
178 icantly associated with STAT3 activation and tobacco use history in non-small cell lung cancer (NSCLC
179 ients with diabetes (HR, 0.88), a history of tobacco use (HR, 0.82), heart failure (HR, 0.84), and pe
180 RNA (HR, 1.19 [95% CI, 1.02-1.38], P = .02), tobacco use (HR, 1.37 [95% CI, 1.02-1.85], P = .04), CD4
181       Among patients with ERAF, only current tobacco use (HR, 3.84; 95% CI, 1.82-8.11; P<0.001) was a
182 has in the past been largely associated with tobacco use, human papillomavirus (HPV+) oropharynx canc
183                                    Age, sex, tobacco use, hypercholesterolemia, hypertension, and typ
184 odels included adjustment for race, obesity, tobacco use, hypertension (HTN), atrial fibrillation (AF
185 antly to the global burden of CVD, including tobacco use, hypertension, and secondary prevention for
186 ed annual 480 000 deaths are attributable to tobacco use in adults, including from secondhand smoke.
187 s on the rates of initiation or cessation of tobacco use in children and adolescents and on health ou
188 sociated with an increased risk of offspring tobacco use in later life.
189 otential for creating an alternative form of tobacco use in light of declining cigarette sales and so
190 acco industry's future depends on increasing tobacco use in low-income and middle-income countries (L
191 nificantly higher smoking and male smokeless tobacco use in most Appalachian disparity states.
192 ically associated with the long-term risk of tobacco use in offspring.
193                                              Tobacco use in PAD is associated with substantial increa
194       INTERPRETATION: The high prevalence of tobacco use in people living with HIV in LMICs mandates
195 weden 2005-2012 with information on maternal tobacco use in pregnancy, followed until December 2015.
196                     Pooled prevalence of any tobacco use in pregnant women in LMICs was 2.6% (95% CI
197                                     Overall, tobacco use in pregnant women in LMICs was low; however
198                 We assessed the magnitude of tobacco use in pregnant women in LMICs.
199                       However, the extent of tobacco use in pregnant women in low-income and middle-i
200   The pooled prevalence of current smokeless tobacco use in pregnant women was lowest in the European
201 nction as a potential mechanism for elevated tobacco use in schizophrenia and also identify activatio
202 ng education or brief counseling, to prevent tobacco use in school-aged children and adolescents have
203 r brief counseling, to prevent initiation of tobacco use in school-aged children and adolescents.
204 never having smoked a full cigarette, and no tobacco use in the prior 3 years.
205 etween maternal smoking during pregnancy and tobacco use in young adulthood, an association that was
206 ed model regarding the effects of emotion on tobacco use, in particular, as well as on addictive beha
207 ctors associated with poly-tobacco (vs. mono-tobacco) use included lower confidence to remain tobacco
208 ities for significantly reducing the toll of tobacco use, including: (1) the need for novel, nonlinea
209 ny cancer cases in India are associated with tobacco use, infections, and other avoidable causes.
210  fair-quality) that were designed to prevent tobacco use initiation or promote cessation (or both) an
211                                              Tobacco use initiation; tobacco use cessation; health ou
212  406 loci associated with multiple stages of tobacco use (initiation, cessation, and heaviness) as we
213 cological interventions with no or a minimal tobacco use intervention control group (eg, usual care,
214                                              Tobacco use is a leading cause of preventable death worl
215                                              Tobacco use is a major driver of carcinogenesis in HNSCC
216 ctive, one beneficial consequence of chronic tobacco use is a reduced risk for Parkinson's disease.
217                                              Tobacco use is an established risk factor for the develo
218                                              Tobacco use is an important preventable cause of periphe
219                                              Tobacco use is an underappreciated risk for Barrett's es
220                            First, that daily tobacco use is associated with an increased risk of psyc
221                                        Daily tobacco use is associated with increased risk of psychos
222  but whether or not they help people to quit tobacco use is controversial.
223 vernments can create an environment in which tobacco use is reduced and citizens maintain good levels
224                                              Tobacco use is responsible for a large proportion of the
225                                              Tobacco use is the leading cause of preventable death in
226                                              Tobacco use is the leading cause of preventable death in
227                                              Tobacco use is the leading preventable cause of death wo
228                                              Tobacco use is the leading preventable cause of mortalit
229                                              Tobacco use is the main risk factor for HNSCC, and tobac
230 tional individual level risk factors (mainly tobacco use, lipids, and elevated blood pressure) and so
231 e that health professionals have in reducing tobacco use, many have a smoking habit themselves.
232              Furthermore, antipsychotics and tobacco use may increase CB1R availability in this popul
233  that disparities in hypertension related to tobacco use might be masked by differences in body mass
234 ionally representative sources that measured tobacco use (n = 2102 country-years of data) were system
235 rovides evidence-based policies for reducing tobacco use, no global policy exists for the control of
236 x (BMI), hypertension, diabetes, alcohol and tobacco use, ocular conditions (including blindness, cat
237 ctors for developing colon pathology include tobacco use (odds ratio (OR), 2.0; 95% confidence interv
238 n integral role in affecting the patterns of tobacco use of those newly diagnosed.
239  accurately understand the precise effect of tobacco use on cancer treatment outcomes.
240 f coronary artery disease, but the impact of tobacco use on cardiac structure and function in the gen
241 total and direct causal roles of alcohol and tobacco use on CVD risk factors and events.
242 t the effect of comorbid methamphetamine and tobacco use on human fetal brain development.
243 alth (nutrition, environment/occupation, and tobacco use) on day 2.
244 r point, 1.92; 95% CI, 1.55-2.37; P < .001), tobacco use (OR, 3.64; 95% CI, 1.36-9.73; P = .01), ence
245 nt with offspring's smoking, age at onset of tobacco use, or changes in use between 2006 and 2010.
246 dence intervals for the associations between tobacco use, oral health indicators, and PD risk.
247 e likely to report initiation of combustible tobacco use over the next year.
248 ependent of age, diabetes, hypertension, and tobacco use (P < 0.001).
249 , more years on dialysis before waitlisting, tobacco use, panel-reactive antibody greater than 0, pub
250                                           As tobacco use patterns continue to change and diversify, f
251 nmental factors, such as risk perceptions of tobacco use, peer use, and tobacco-company influences.
252 from a short list of risk factors--including tobacco use, poor diet and physical inactivity (both str
253 re found to be significantly associated with tobacco use posttransplant.
254       Four percent of our LTx cohort resumed tobacco use posttransplant.
255   In conclusion, the genetics of alcohol and tobacco use potentially has broader implications on phys
256  pressure, heart rate, change in heart rate, tobacco use, presence of diabetes mellitus, alcohol use,
257                  The difference in smokeless tobacco use prevalence between HIV-positive and HIV-nega
258 e to preventive practices from 1999 to 2010, tobacco use remained high, and almost half of U.S. adult
259                                  Combustible tobacco use remains the number-one preventable cause of
260                                              Tobacco use remains the single most common modifiable ca
261 had 100% sensitivity and 45% specificity for tobacco use resumption.
262         The PCI may be a useful predictor of tobacco use resumption.
263 higher prevalence in HIV-positive men of any tobacco use (risk ratio [RR] 1.41 [95% CI 1.26-1.57]) an
264  hypertension, diabetes, hyperlipidemia, and tobacco use, risk differences comparing participants wit
265 gher prevalence in HIV-positive women of any tobacco use (RR 1.36 [95% CI 1.10-1.69]; p=0.0050), toba
266 formation on several risk factors, including tobacco use, secondhand exposure to cigarette smoke, obe
267 acco smoking, smokeless tobacco use, and any tobacco use separately for males and females to study di
268      Using serum cotinine values to classify tobacco use showed that smokers have higher serum concen
269                        Prevalence of current tobacco use (smoked and smokeless) was estimated for eve
270  regional, and overall prevalence of current tobacco use (smoked, smokeless, and any tobacco use) amo
271 ression analyses, adjusted for education and tobacco use (snuff use in the smoking analysis and smoki
272 ic blood pressure, diastolic blood pressure, tobacco use, statin use, body mass index, urine microalb
273 participants without a history of alcohol or tobacco use, suggesting that observed associations with
274 ed minority adults surveyed in the 1992-2015 Tobacco Use Supplement to the Current Population Survey
275 rictions, augmenting access to treatment for tobacco use through insurance coverage and telephone hel
276 ourth priority is a UN high-level meeting on tobacco use to galvanise global action towards the 2040
277 amples is of great importance to testing for tobacco use, tobacco cessation treatment, and studies on
278 However, global efforts to control smokeless tobacco use trail behind the progress made in curbing ci
279 Parties collect or present data on smokeless tobacco use under global or national surveillance mechan
280                                   Older age, tobacco use, underlying cardiovascular disease, diabetes
281          Behavioural risk factors, including tobacco use, unhealthy diets, and physical inactivity ar
282 oeconomic status and harmful use of alcohol, tobacco use, unhealthy diets, and physical inactivity wi
283                                Reductions in tobacco use, uptake of preventive measures, adoption of
284 ong children in the US since the late 1990s, tobacco use via electronic cigarettes (e-cigarettes) is
285                                We found that tobacco use was associated with a lower risk of PD in ma
286                         Unlike cannabis use, tobacco use was associated with worse lung function, sys
287                                              Tobacco use was observed to be associated with a lower r
288 ation and treatment of individuals who smoke tobacco using water pipes.
289 complications, high triglyceride levels, and tobacco use were additional independent predictors for g
290 ger lesion size, nonupper lobe location, and tobacco use were associated with increased diagnostic yi
291                                Marijuana and tobacco use were each independently associated with high
292 en, hospital stays in the past 6 months, and tobacco use were included, the adjusted HR was 0.73 (95%
293           A high body mass index and current tobacco use were the only independent risk factors for d
294 cotine and cotinine in hair as biomarkers of tobacco use where in the past the detection of cotinine
295 nary function and other covariates including tobacco use, which was analyzed in parallel as a positiv
296 been described, including chronic heartburn, tobacco use, white race, and obesity.
297  older, peripheral artery disease, diabetes, tobacco use, white race, stroke, atrial fibrillation, co
298 er, education, tooth brushing, flossing, and tobacco use with a questionnaire.
299 ppressants and other medications, and 4% for tobacco use, with 31% clinic appointment nonadherence in
300                                  Prohibiting tobacco use would be entirely consistent with other mili

 
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