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1 del of bone marrow transplantation (BMT) for tolerance induction.
2 riming capacity, which supports its role for tolerance induction.
3 on DC subsets can be exploited for improved tolerance induction.
4 tins) and are probably of prime relevance to tolerance induction.
5 repertoire, weakens the platform for central tolerance induction.
6 e host thymus implying an impairment in self-tolerance induction.
7 g a critical role of Tef cell elimination in tolerance induction.
8 ll engraftment and then evaluated transplant tolerance induction.
9 as not all alloantigens are revealed during tolerance induction.
10 e to deliver Ags to DCs for presentation and tolerance induction.
11 have been shown to be a critical barrier to tolerance induction.
12 y affects immune system development and oral tolerance induction.
13 ity could be a potential strategy for walnut tolerance induction.
14 apeutic manipulation of MDSCs for transplant tolerance induction.
15 organ, the liver also has a central role in tolerance induction.
16 epithelial cells, a key process for central tolerance induction.
17 innate and adaptive immune systems in Ag-SP tolerance induction.
18 c anatomical sites may preferentially favour tolerance induction.
19 ay important roles in antiviral immunity and tolerance induction.
20 II, the primed cells remained susceptible to tolerance induction.
21 ch contributes to intestinal homeostasis and tolerance induction.
22 auses of graft failure or remove barriers to tolerance induction.
23 mpact of immunosenescence on chimerism-based tolerance induction.
24 o assess whether PA can be prevented by oral tolerance induction.
25 ome and so was crucial for effective central tolerance induction.
26 rsity do not constitute a uniform barrier to tolerance induction.
27 lf-antigens in the medulla in the context of tolerance induction.
28 ) thymocytes, a process important for T cell tolerance induction.
29 iscuous gene expression required for central tolerance induction.
30 the effect of corticosteroids on respiratory tolerance induction.
31 d gene expression signatures associated with tolerance induction.
32 matory quality of IgG molecules formed on TD tolerance induction.
33 nvestigate whether donor brain death affects tolerance induction.
34 matory sialylated IgGs that are formed on TD tolerance induction.
35 thogenic T(H)2 cells is an essential step in tolerance induction.
36 etween an effector inflammatory response and tolerance induction.
37 y molecule PD-L1 that is essential for Ag-SP tolerance induction.
38 essary step in some but not all protocols of tolerance induction.
39 CTLA4/B7.1/B7.2 and for PD-1 for CD8 T-cell tolerance induction.
40 n-treated mice disrupts alloantigen-specific tolerance induction.
41 cell death provides an important signal for tolerance induction.
42 e profound effects on the insulin autoimmune tolerance induction.
43 findings and has important implications for tolerance induction.
44 a productive response and avoid death and/or tolerance induction.
45 intravenous injection as a model for immune tolerance induction.
46 ave been implicated in T cell activation and tolerance induction.
47 ion with major implications for immunity and tolerance induction.
48 able barrier to long-term graft survival and tolerance induction.
49 perprolactinemia on the mechanisms of B cell tolerance induction.
50 tate the likelihood of success or failure of tolerance induction.
51 may exhibit differential susceptibilities to tolerance induction.
52 trategy for the development of protocols for tolerance induction.
53 controls the fate of developing B cells and tolerance induction.
54 ons are critical for long-term chimerism and tolerance induction.
55 ne marrow cell (BMC) engraftment and promote tolerance induction.
56 elerating allograft rejection and preventing tolerance induction.
57 ased DCs suggests their nonredundant role in tolerance induction.
58 f immunomodulating agents in transplantation tolerance induction.
59 dioxygenase (IDO), an enzyme associated with tolerance induction.
60 n hematopoietic cells was necessary for oral tolerance induction.
61 ed HSC mobilization and completely abrogated tolerance induction.
62 totic body ingestion by CD8(+) DCs initiates tolerance induction.
63 ing the crucial role of B cells in perinatal tolerance induction.
64 microenvironments for T-cell development and tolerance induction.
65 f carp parvalbumin to digestion affects oral tolerance induction.
66 -specific B cells before and after bee venom tolerance induction.
67 In vivo, HDAC11KO T cells were refractory to tolerance induction.
68 s for the use of antigen-specific therapy in tolerance induction.
69 an contribute to graft rejection and also to tolerance induction.
70 erimental approach to immune suppression and tolerance induction.
71 a non-inflammatory context is beneficial for tolerance induction.
72 within the lymph node (LN) are integral for tolerance induction.
73 apoptotic cell antigens and failure of long-tolerance induction.
74 eractions provides a new research target for tolerance induction.
75 g B cell development, selection, and central tolerance induction.
76 inst mitochondrial content, and amenable for tolerance induction.
77 suggests that they are possible targets for tolerance induction.
78 ecific CD4(+) T cells transferred at time of tolerance induction (7 days before transplantation) beca
79 epletion of CD4+CD25+ cells did not abrogate tolerance induction, adoptive transfer of CD4+ cells fro
80 tween iNKT cells and Tregs in the context of tolerance induction after allogeneic HCT and set the sta
82 ings extend the notion that requirements for tolerance induction after lung transplantation differ fr
83 iated acceptance of most tissues and organs, tolerance induction after lung transplantation is critic
84 However, the precise cellular mechanisms of tolerance induction against particle-bound antigens, the
86 ts, novel treatment options may include oral tolerance induction, although most authors do not curren
88 B in DCs further results in an impaired oral tolerance induction and a marked type 2 immune bias amon
89 itional deletion of Pten, resulted in failed tolerance induction and abundant autoantibody production
91 hymus medulla formation for alphabeta T cell tolerance induction and demonstrated a Rank-mediated rec
92 , implicating TCR revision as a mechanism of tolerance induction and demonstrating that TCR-dependent
93 gh self-antigen doses required for effective tolerance induction and elicits anergic, interleukin (IL
94 mice and found they were resistant to immune tolerance induction and exhibited greater susceptibility
95 rect clinical implications for understanding tolerance induction and for rationally developing novel
96 rvations have implications for understanding tolerance induction and how B cell depletion may prevent
97 ermore, reducing alpha4 laminin circumvented tolerance induction and induced cardiac allograft inflam
98 chronic liver injury and fibrosis abrogated tolerance induction and led to an immunogenic reprogramm
99 and strengthen the cadre of Tregs to promote tolerance induction and long-term allograft survival.
100 ction of BALT in pulmonary allografts during tolerance induction and may provide a platform for the d
101 nowledge, this is the first demonstration of tolerance induction and persistence of chimerism in a la
103 ade of B7-H1/CD80 interaction prevented oral tolerance induction and restored T-cell responsiveness t
105 perties and hepatic gene transfer results in tolerance induction and suppression of autoimmune diseas
106 ) versus donor-specific transfusion (DST) in tolerance induction and sustenance in living donor renal
107 roinflammatory cytokines interfere with oral tolerance induction and that blocking the IL-6 pathway i
108 ends great appeal as a strategy for targeted tolerance induction and treatment of autoimmune diseases
110 lt in enhanced alloreactivity, resistance to tolerance induction, and destabilization of the establis
111 Treg population, were essential for in vivo tolerance induction, and expressed a biased, restricted
113 ammatory phenotype, were less susceptible to tolerance induction, and released more tumor necrosis fa
114 lergen-specific T cell epitopes required for tolerance induction, and upon immunization of animals in
118 alter host susceptibility to transplantation tolerance induction as well as impair the quality and st
119 g-loaded dendritic cells (DCs) delays T cell tolerance induction as well as refunctionalizes already
120 ular, we discuss phenotypic diversity during tolerance induction as well as signals that drive effect
121 re of interest for diagnosis, prognosis, and tolerance induction, as well as improving our understand
123 that aGVHD weakens the platform for central tolerance induction because individual TRAs are purged f
124 T regulatory cells that are dispensable for tolerance induction but required for long-term tolerance
125 f protein antigens is a preferred method for tolerance induction, but degradation during gastrointest
126 itional developmental stage that facilitates tolerance induction, but inflammation converts antigen-e
127 nted repressive nucleosome repositioning and tolerance induction, but the "open" promoter required en
128 enhance anti-transplant responses and impair tolerance induction, but the effect of Qa-1 deficiency o
129 ed regulation of gene expression and central tolerance induction, but this influence is unlikely to r
130 LA(-/-) OT-II cells were less susceptible to tolerance induction by a high-dose OVA peptide administe
133 owed by rapid tumor regrowth, reminiscent of tolerance induction by CD3 mAb-mediated T-cell depletion
137 serum prolactin levels interfere with B cell tolerance induction by impairing BCR-mediated clonal del
140 o not develop allergic contact dermatitis is tolerance induction by repeated exposure to low doses of
142 n of CD4(+)CD25(+)FOXP3(+) Treg cells during tolerance induction completely abolishes the development
143 ing pathways that are being investigated for tolerance induction, detailing preclinical studies and t
146 ptophan degradation pathway is important for tolerance induction during systemic allergen immunothera
147 ne conditions for stable mixed chimerism and tolerance induction following combined hematopoietic cel
148 e in the control of T-cell alloresponses and tolerance induction following hepatocyte transplantation
150 rial therapy may be a promising strategy for tolerance induction for clinical xenogeneic islet transp
151 ent review discusses current developments in tolerance induction for solid organ transplantation with
154 ffectiveness and additional requirements for tolerance induction for xenogeneic islet transplantation
155 progress to be made in moving strategies for tolerance induction from the bench to the bedside and di
159 intrathymically and was essential for robust tolerance induction in a mouse model of spontaneous ence
161 icted TCR responsible for Ag recognition and tolerance induction in CD8(+) T cells can, in the absenc
163 e system that allows interrogation of T-cell tolerance induction in endogenous naive and memory CD8 T
165 mmunotherapy may be a promising approach for tolerance induction in experimental arthritis and perhap
166 In order to elucidate cellular mechanisms of tolerance induction in healthy and injured liver, we dev
167 gs and is a promising reagent for studies of tolerance induction in hematopoietic cell transplantatio
169 rolled trials exploring the efficacy of oral tolerance induction in infancy for the prevention of FA.
170 e ability of intestinal grafts to facilitate tolerance induction in major histocompatibility complex
171 ndings highlight the critical role of LCs in tolerance induction in mice to the prototype innocuous h
175 Our data show that NK cells are required for tolerance induction in recipients given donor splenocyte
180 of xenogeneic thymus tissue allows xenograft tolerance induction in the highly disparate pig-to-mouse
185 that Aire plays an important role in T cell tolerance induction in the thymus, mainly by promoting e
189 DCs and invariant NKT cells are required for tolerance induction in this system that was translated i
192 DC subset may result in a higher efficacy of tolerance induction in vivo and may support the developm
193 ll activation, its role in peripheral T cell tolerance induction in vivo has not been addressed.
195 chanisms of CD4 cell tolerance in a model of tolerance induction involving establishment of mixed hem
198 ar whether the efficiency of engraftment and tolerance induction is influenced by targeting antigen t
199 lectively, these findings indicate that full tolerance induction is largely dependent on substantial
203 idity in patients before they undergo immune tolerance induction (ITI) and in those with persistent h
204 to attempt to eliminate inhibitors is immune tolerance induction (ITI) via a protocol requiring inten
207 tive randomized, multicenter study of immune tolerance induction(ITI) in patients with hemophilia A r
211 lating therapies (including antigen-specific tolerance induction) need to be further delineated.
212 trate that NFAT1 is neither required for CD4 tolerance induction nor for their regulatory function on
213 initial induction of MRP expression and the tolerance induction of monocytes associated with nuclear
215 mediated inflammation, maintenance of immune tolerance, induction of the two suppressive cytokines in
216 (acute, chronic, or latent) and the stage of tolerance (induction or maintenance) in this infection-t
217 Treg) cells, as their depletion early during tolerance induction or late after established tolerance
221 However, IFN-gamma can also participate in tolerance-induction pathways, indicating it is not solel
222 series of experiments that implicate LCs in tolerance induction, positioning these cells as key regu
223 o siglecs, CD22 and Siglec-G, contributed to tolerance induction, preventing plasma cell differentiat
224 autoreactive B-lymphocytes bypassing normal tolerance induction processes to be the subset of antige
225 erwent LDRT between 2007 and 2011: 606 under tolerance induction protocol (TIP) and 310 with our usua
226 ach in demographically well-balanced groups, tolerance induction protocol (TIP) was used with SCT in
228 y led to the first clinical application of a tolerance induction protocol for kidney transplantation.
229 the remaining obstacles to introduction of a tolerance induction protocol to clinical practice in han
231 n, "What do we need to have in place to make tolerance induction protocols a 'standard of care' for o
232 e a useful tool in the development of future tolerance induction protocols in non-sensitized patients
233 cute rejections and are an essential part of tolerance induction protocols in various animal models;
234 sed to accomplish this goal, both as part of tolerance induction protocols or to reduce the requireme
235 process, test new antirejection treatments, tolerance induction protocols or to understand basic imm
237 s been demonstrated in mixed chimerism-based tolerance induction protocols; however, the development
238 nfections at the time of transplantation and tolerance induction provide a pro-inflammatory milieu th
239 rgy show promise for oral immunotherapy, but tolerance induction remains elusive, and additional ther
241 tients with such enzyme deficiencies, immune tolerance induction should be advocated in the patients
242 tions, there is an immediate need to develop tolerance induction strategies and to elucidate the mech
243 iple myeloma and ESRD and, more recently, to tolerance induction strategies involving combined bone m
248 umors result in either complete ignorance or tolerance induction, suggesting inadequate DC function.
249 y DC10 are requisite, but not sufficient for tolerance induction, suggesting that DC10 and Th2 effect
251 ing from the TCR to control thymic iNKT cell tolerance induction, terminal differentiation, and effec
253 we focused on identifying regimens of immune tolerance induction that may be readily available for cl
254 pment of more robust and safer protocols for tolerance induction that will be guided by innovative im
255 > 1:80) in patients after successful immune tolerance induction therapy (n = 23), and 100% (n = 20,
256 on, host CD8(+) DCs play a requisite role in tolerance induction through interactions with NKT cells.
258 Moreover, these Tregs were capable of serial tolerance induction through modulation of macrophages th
262 upt gut immune homeostasis and prevents oral tolerance induction to bystander food antigen through th
263 that clearance of apoptotic cells results in tolerance induction to cleared Ags by dendritic cells (D
266 ein tested the effect of B-cell depletion on tolerance induction to factor VIII (FVIII) in a mouse mo
267 te cellular and molecular mechanisms of oral tolerance induction to food and aeroallergens in human t
268 control the ability of TLR agonists to block tolerance induction to hematopoietic and skin allografts
271 er studies that explore the efficacy of oral tolerance induction to other common food allergens and t
274 hat the approach can also be used for immune tolerance induction to prevent or eliminate inhibitory a
275 argets for depletion, immunosuppression, and tolerance induction to promote long-term graft survival.
276 ine a period of heightened susceptibility to tolerance induction to tissue-restricted antigens (TRAs)
277 tokines, and local inflammation demonstrated tolerance induction toward the secondary antigen in the
280 medicine, offering approaches for developing tolerance-induction treatments relevant to cell therapie
283 tic understanding, and clinical potential of tolerance induction using apoptotic Ag-coupled apoptotic
293 2,3 dioxygenase (IDO)-tryptophan pathway in tolerance induction were assessed, and the frequency and
295 bility of DCs to delay tumor-mediated T cell tolerance induction, whereas interfering with 4-1BBL, CD
296 d alloimmune response and are susceptible to tolerance induction, which is associated with a clonal d
297 ining lymph nodes (sdLNs) and defective skin tolerance induction, while talin1-deficient dermal DCs u
299 ted patients, and patients undergoing immune tolerance induction with large doses of factor VIII to e
300 f autoimmune conditions can be reinforced by tolerance induction with peptide epitopes, but the mecha