ライフサイエンスコーパス: tonsil

1 common site (60.4%), followed by tongue and tonsil.

2 been identified in human blood, spleen, and tonsil.

3 f T cell development occurs within the human tonsil.

4 rge intestine, bursa of Fabricius, and cecal tonsil.

5 ent in the variant Creutzfeldt-Jakob disease tonsil.

6 r germinal centre (GC) B cells compared with tonsil.

7 largest reservoir in the spleen, followed by tonsils.

8 an colon tissue as well as hyperplasia human tonsils.

9 ies can persist in pediatric adenoids and/or tonsils.

10 vivo in T-cell areas of lingual and palatine tonsils.

11 induction to food and aeroallergens in human tonsils.

12 e identified both in lingual and in palatine tonsils.

13 ic cell-compartments in palatine and lingual tonsils.

14 CL6-expressing CD4(+) T-cell subset in human tonsils.

15 al-associated lymphoid tissue, adenoids, and tonsils.

16 ally those arising in the oropharynx and the tonsils.

17 keratinocytes from the foreskin, cervix, and tonsils.

18 o potent chemokines made abundantly in human tonsils.

19 ted B cell-helping effector T cells in human tonsils.

20 e salivary glands, oral mucosa, and palatine tonsils.

21 atal cord blood, adult peripheral blood, and tonsils.

22 source of TGF-beta1 in chronically infected tonsils.

23 rus 1 (HBoV1) can persist in nasopharynx and tonsils.

24 close connection with each other in palatine tonsils.

25 teraction that probably takes place in human tonsils.

26 increased germinal center reactivity in the tonsils.

27 isolated, stimulated T cells of 27 palatine tonsils (10 RAT, 7 PTA, 10 tonsils without inflammation)

28 igher than that of contralateral cancer-free tonsils (2.54 + or - 0.88; P < .0001) and tonsils in con

29 Furthermore, we identify within the tonsil a cNK precursor population that is characterized

30 ased innate antiviral factors may render the tonsil a potential site for oral transmission.

31 CD34) isolated from a human lymphoid organ, (tonsils), adding to our understanding of how L-selectin

32 Tonsil allergen-specific FOXP3(+) regulatory T (Treg) ce

33 The spleen and tonsil, although harboring the largest number of overall

34 l and umbilical cord blood (UCB), thymus and tonsil, although mRNA levels were reduced compared with

35 In this study, human tonsil and adenoid tissues were analyzed to determine th

36 d the concentration of released cytokines in tonsil and blood as well as in different forms of inflam

37 Both tonsil and blood PDCs expressed several genes necessary

38 ally cytopathic to other immune cells), both tonsil and blood PDCs supported IAV infection.

39 stant to IAV infection, was detected in both tonsil and blood PDCs.

40 nti-inflammatory cytokines IL-4 and IL-10 in tonsil and blood samples in RAT, PTA, and samples withou

41 analysis indicates that, in comparison with tonsil and bone marrow (BM) PCs, these PCs distinctively

42 illar memory B cells (MB) and PCs, from both tonsil and bone marrow tissues, express BCMA.

43 of interleukin-22 (IL-22) production in the tonsil and colon; an increase in the levels of CD107a, g

44 Tonsil and hypermutating Ramos B-cells convert C-->U in

45 r and spleen, spleen and cecal tonsil, cecal tonsil and ileum, liver and cecal tonsil, liver and ileu

46 oid cell populations expressing CD155 in the tonsil and intestine, as well as in spinal cord neurons.

47 Droppings, cecal tonsils and 5 internal organs were sampled and cultured

48 ens were searched for in tissues of palatine tonsils and adenoids removed from patients without ARI s

49 consists of diffuse lymphoid cells and lacks tonsils and adenoids.

50 hes a latent infection in lymphocytes of the tonsils and adenoids.

51 ceptor phenotype of the Th17 cell subsets in tonsils and adult blood.

52 ry B-cell (MBC) responses were enumerated in tonsils and blood.

53 influenza virus-specific B-cell responses in tonsils and blood.

54 in oligodendrocyte glycoprotein) in palatine tonsils and cervical lymph nodes of 28 acute stroke pati

55 strated that MHC class II may be detected in tonsils and EBV-negative Hodgkin disease but not in EBV-

56 niquely express lysozyme and can be found in tonsils and in tumors.

57 e mature than other CD56(bright) NK cells in tonsils and less mature than other NK cells in blood, sh

58 ner in single-cell suspensions of both human tonsils and NALT.

59 Virus was, however, isolated from the tonsils and nasal swabs of the asymptomatic T15 pigs at

60 and an increase in the levels of NK cells in tonsils and oral lymph nodes.

61 was absent on human ILCs expressing NKp44 in tonsils and PB of Crohn disease patients, and relatively

62 Tonsils and peripheral blood (pre- and postvaccination)

63 allergen-specific T-cell tolerance in human tonsils and peripheral blood through a mechanism depende

64 responses of allergen-specific T cells from tonsils and peripheral blood were measured by using trit

65 include organized MALT (O-MALT) such as the tonsils and Peyer patches.

66 tinal tract) and lymphoid/monocytic cells in tonsils and Peyer's patches (explaining viremia), extend

67 tinal tract) and lymphoid/monocytic cells in tonsils and Peyer's patches (explaining viremia), thereb

68 mucosa-associated lymphoid tissues, such as tonsils and Peyer's patches, which is hard-wired to secr

69 in children and adolescents and colonize the tonsils and pharynx of up to 20% of healthy children.

70 rimary Tfh and non-Tfh T effector cells from tonsils and prepared genome-wide maps of active, interme

71 uely report the mucosal humoral responses in tonsils and saliva after intranasal live attenuated infl

72 Tonsillectomy removes only the palatine tonsils and sometimes adenoids.

73 CNS invasion, prions spread centrifugally to tonsils and spleen at an advanced stage of the incubatio

74 aive patients with FL compared with reactive tonsils and the peripheral blood of healthy donors.

75 sh stage III immature NK cells isolated from tonsils and these cells expressed CXCL8 protein on PMA s

76 red with those of healthy TILs from reactive tonsils and this can be induced on healthy T cells by FL

77 stages in a human secondary lymphoid organ (tonsils) and adult, but not in neonatal, blood.

78 ific) mBCs in peripheral blood (PB), spleen, tonsil, and bone marrow.

79 l LNs analyzed, as well as in the spleen and tonsil, and correspond to the two known blood DC subtype

80 ometry and cell stimulation assays in colon, tonsil, and oral lymph node samples.

81 By day 4, vDNA was detected in the throat, tonsil, and spleen, and monkeypox antigen was detected i

82 of Fabricius, gastrointestinal tract, cecal tonsil, and trachea.

83 e contact to NK cells in T-cell areas of the tonsils, and a subpopulation of the pDCs expressed GITRL

84 airways usually occur in the upper airways, tonsils, and adenoid structures that make up the Waldeye

85 id tissues (O-MALT) such as Peyer's patches, tonsils, and adenoids.

86 other regions, such as the salivary glands, tonsils, and bone marrow.

87 sity, and ontogeny in paired donor blood and tonsils, and in blood after vaccination.

88 Germinal centers and plasma cells in tonsils appeared normal, as were serum immunoglobulin le

89 (-)) CD127(+)RORC(+) LTi-like cells in human tonsil are precursors to CD56(+)CD127(+)RORC(+)NKp46(+)

90 s with isolated infarction of the cerebellar tonsil are unknown.

91 T cells in tonsils are fully responsive and competent for antigen-i

92 going a germinal center (GC) reaction in the tonsils are limited to the follicles and proliferate ext

93 Tonsils are strategically located in the gateway of both

94 Tonsils are the lymph nodes serving the upper respirator

95 an tissue-resident macrophages isolated from tonsil as a tractable cell model.

96 n tonsillar follicles and support the use of tonsils as lymphoid sites for the study of germinal cent

97 ephalosynapsis correlates with fusion of the tonsils, as well as midbrain abnormalities including aqu

98 ed with the percentage of eGC B cells in the tonsil at the outset of the culture.

99 noted between radiotracer uptake in spleen, tonsil, axillary lymph nodes, and peripheral blood CD4 T

100 lation between the phenotypic composition of tonsil B cells and the CSR to IgE ex vivo.

101 When purified tonsil B cells are incubated with IL-4 and anti-CD40 to

102 his study shows that the maturation state of tonsil B cells determines their capacity to undergo clas

103 Total tonsil B cells give rise to IgE(+) PCs by direct and seq

104 Human tonsil B cells were analyzed by flow cytometry (FACS) an

105 inal center (GC), early GC (eGC), and memory tonsil B cells were isolated by FACS, and their capaciti

106 To generate human IgE(+) cells, we cultured tonsil B cells with IL-4 and anti-CD40.

107 SphK1 inhibition in human tonsil B cells, as well as inhibition or deletion of Sph

108 e of generated IgE(+) cells, the capacity of tonsil B-cell subsets to generate IgE(+) PCs and the cla

109 LC differentiation was not observed from tonsil BDCA-1(+) and BDCA-3(+) subsets.

110 sues close to the viral entry site, with the tonsil being the primary site of virus replication and i

111 racterized by displacement of the cerebellar tonsils below the base of the skull, resulting in signif

112 Tonsil biopsies were analyzed by confocal microscopy.

113 ected antemortem as an adjunct to testing of tonsil biopsy specimens and surveillance by necropsy for

114 functionally immature transitional B cells, tonsil biopsy tissues revealed active germinal center (G

115 linical diagnosis of probable vCJD was made; tonsil biopsy was not done.

116 at the spleen harbors most mBCs, followed by tonsils, BM, and PB, and we detected no major difference

117 We found that tonsil but not blood leukocytes were responsive to S1P g

118 as not only detected and isolated from human tonsils but displayed unique genetic features in compari

119 e expression of EphA4 was detected in EBV(-) tonsils but not in EBV(+) PTLD.

120 herein we investigated slan(+) -cell fate in tonsils by using a molecular-based approach.

121 lated from four major chicken tissues: cecal tonsil (C), ileum (I), liver (L), and spleen (S) were us

122 e driven predominantly by base-of-tongue and tonsil cancers in men.

123 ng molecular features distinct from those of tonsil CD11b(+) CD14(+) -macrophages and cDC2.

124 In tonsils, CD138(+) plasma cells (PCs) are surrounded by C

125 , using imaging mass cytometry, we find that tonsil CD14(+) macrophages localize in situ in the B cel

126 Here we find that tonsil cDC2 and CD14(+) macrophages are the best inducer

127 d between liver and spleen, spleen and cecal tonsil, cecal tonsil and ileum, liver and cecal tonsil,

128 However, in tonsil cell cultures, HIV-2, HIV-2 DeltaVpx, and HIV-1 i

129 Using tonsil cell lysates to repair a G.U mismatch, A/T and G/

130 HIV-1 ex vivo by spinoculating and culturing tonsil cells with HIV-1 GFP reporter viruses.

131 munodeficiency virus type 1 (HIV-1), such as tonsil, cervical, or rectal tissue.

132 ified squamous epithelia of human esophagus, tonsil, cervix, larynx, and cornea.

133 de, nasopharyngeal lymph node and pharyngeal tonsil collected at the peak of clinical disease from be

134 en processing were increasingly expressed in tonsil compared with the epithelium of another oropharyn

135 FOXP3(+) Treg cells, are identified in human tonsils compared with peripheral blood.

136 These data reveal that human tonsil contains long-lived plasma cells, the majority of

137 Little is known about how tonsils contribute to the local immune response after in

138 Functionally, tonsil DC also only stimulated low levels of antigen-spe

139 We show that tonsil DC are able to sample their antigenic environment

140 n and brain activity in the right cerebellar tonsil, declive, culmen, lingual gyrus and cuneus.

141 immunity, by studying the responses of human tonsil-derived DC to Neisseria meningitidis as a model o

142 fore, examined the susceptibility of a human tonsil-derived follicular dendritic cell-like cell line

143 We have compared CD4 T cell death in tonsil-derived human lymphoid aggregate cultures (HLACs)

144 Phylogenetic analysis placed tonsil-derived IAV in clusters clearly segregated from c

145 als, we found BSE in 50% and 12% of gut- and tonsil-derived samples, respectively.

146 e demonstrate that Notch activation in human tonsil-derived stage 3 (CD34(-)CD117(+)CD94(-)NKp80(-))

147 ulating TFR are phenotypically distinct from tonsil-derived TFR in humans.

148 ation status of blood TFR is comparable with tonsil-derived TFR.

149 irmed the increased presence of CXCR4 in the tonsil epithelium compared with multiple oral epithelial

150 Tonsil epithelium has been implicated in human immunodef

151 Importantly, tonsil epithelium highly expressed genes associated with

152 e with anti-HIV activity, was minimal in the tonsil epithelium, in contrast to oral mucosa.

153 In the cervical and tonsil epithelium, we observe significant downregulation

154 HIV, we performed microarray analysis of the tonsil epithelium.

155 KG2A(+)CD94(+)CD54(+)CD62L(-), is present in tonsils ex vivo and is more mature than other CD56(brigh

156 B cells from different tonsils exhibited varying capacities for CSR to IgE ex v

157 isolated from peripheral blood and palatine tonsils, expanded, and cocultured with naive B cells.

158 oinflammatory signaling receptors in a human tonsil explant model.

159 ne (AZT) were tested in HIV-1-infected human tonsil explants to compare levels of inhibition of HIV-1

160 that B lymphocytes from peripheral blood and tonsils express DC-SIGN and that this expression increas

161 (five male, 12 female; aged 4-43 years) with tonsils extending more than 5 mm below the foramen magnu

162 asion by GAS was reproduced in primary human tonsil fibroblasts, which could be a source of TGF-beta1

163 was stimulated in GAS-infected primary human tonsil fibroblasts.

164 nal centers and plasma cells were studied in tonsils from 4 additional children with Down syndrome.

165 Tfh cells in tonsils from control individuals displayed the active fo

166 gene BZLF1 were more frequently detected in tonsils from EBV carriers colonized with GAS than from E

167 FOXP3(+) Treg cells and pDCs is observed in tonsils from nonatopic individuals.

168 Tonsils from patients (n = 617) undergoing tonsillectomy

169 the first time to our knowledge in children, tonsils from seasonal influenza-vaccinated children.

170 Generation of IgE(+) PCs from tonsil GC B cells occurs mainly via sequential switching

171 rted by the presence of CCR10(+)IgA(+)PBs in tonsil GCs.

172 ct on chronic psoriasis because the palatine tonsils generate effector T cells that recognize keratin

173 Human tonsils grafted into immunodeficient mice were therefore

174 asional viremia, and virus was isolated from tonsils, gut mucosa, and draining lymph nodes.

175 ree cell types, and viral replication in the tonsil/gut was associated with histopathologic destructi

176 ybridization (ISH) assays confirmed that the tonsil harbors SVA RNA during the persistent phase of in

177 The developing Th17 cells in tonsils highly expressed both Th1- (CCR2, CXCR3, CCR5, a

178 An ex vivo human tonsil histoculture infection model was developed to sup

179 l subsets include a novel in vivo-stimulated tonsil IL17+ T cell subset detected without any artifici

180 a number of tissue-selective genes for cecal tonsil, ileum, liver, and spleen identified (95, 71, 535

181 we provide evidence of a role for the human tonsil in a comprehensive program of extrathymic T cell

182 ee tonsils (2.54 + or - 0.88; P < .0001) and tonsils in control subjects (2.98 + or - 1.08; P < .0001

183 The mean SUV(max) ratio between tonsils in patients with carcinoma was 3.79 + or - 1.69,

184 e found in close proximity to ILC3s in human tonsils in situ.

185 tivated phenotypes, expressed TSLPR in human tonsils in vivo.

186 fferentiation, which takes place in palatine tonsils in vivo.

187 er--specifically of the lingual and palatine tonsils--in white men younger than age 50 years who have

188 orted with unilateral flocculus and anterior tonsil infarction.

189 ys to address the mucosal immune dynamics in tonsils investigating the spatial positioning, frequency

190 c evidence, suggest that EBV infection in IM tonsils involves extrafollicular B cells expressing CD38

191 Lingual tonsil is anatomically big and remains lifelong intact.

192 regs in human peripheral blood, spleens, and tonsils is similar, but they display heterogenous surfac

193 sil, cecal tonsil and ileum, liver and cecal tonsil, liver and ileum, spleen and ileum (P < 10-7), re

194 mately 3-7%, in spleen was 10%, and in cecal tonsil, lung, and bone marrow was approximately 15%.

195 tal details for setting up cultures of human tonsils, lymph nodes and cervicovaginal and rectosigmoid

196 sistently observed, with occasional viremia; tonsil, mesentery lymph nodes, and intestinal mucosa ser

197 Tonsil naive T cells were readily chemoattracted by S1P

198 Moreover, baseline tonsil obstruction detected by either DISE or mean snori

199 Tonsil obstruction was significantly, inversely correlat

200 monstrated the persistence of SVA RNA in the tonsil of experimentally or naturally infected animals l

201 tantly, infectious SVA was isolated from the tonsil of two animals on day 60 p.i., confirming the occ

202 olonged cytokine stimulation, accumulates in tonsils of EBV carriers, and is able to potently restric

203 se cells are present in peripheral blood and tonsils of healthy subjects and display a degree of hype

204 re, we identified GAS-specific Th17 cells in tonsils of humans naturally exposed to GAS, prompting us

205 this study was to examine T cell function in tonsils of patients with recurrent acute tonsillitis (RA

206 level and duration of IL-6 production in the tonsils of pigs intranasally inoculated with NS4B.VGIv w

207 d (ii) asymptomatic carriage on the palatine tonsils of pigs on UK farms.

208 tion of high-dose Zika virus directly to the tonsils of three rhesus macaques results in detectable p

209 hat LAIV elicits humoral B-cell responses in tonsils of young children.

210 (RLNs), and sections of tonsil (sections of tonsil only from captive animals were tested).

211 initially thought to represent SCCUP is the tonsil or base of the tongue, and an increasing percenta

212 We show here that human Ms (isolated from tonsils or generated from monocytes in vitro) drive acti

213 We analysed B cells from NP or tonsil, or after ILC2 coculture, by flow cytometry.

214 but not uninfected, CD4 T cells from blood, tonsil, or spleen and only when armed with anti-HIV anti

215 xposed to donor saliva via the conjunctivae, tonsils, or nostrils did not become infected.

216 e HPV16 infection of cervical, foreskin, and tonsil organotypic rafts.

217 34) and caudate body, and in the cerebellar tonsils (P < 0.001).

218 34) and caudate body, and in the cerebellar tonsils (p<0.001).

219 Tonsil PDCs also had a dampened cytokine response to pur

220 Tonsil pDCs have the ability to generate functional CD4(

221 Our findings suggest that tonsil PDCs may be less responsive to IAV than blood PDC

222 In this study, we investigated how human tonsil PDCs, likely exposed to virus because of their lo

223 e IFN-alpha in response to IAV compared with tonsil PDCs.

224 E pathology samples with known CN, including tonsil, placentae, and FFPE melanoma cell lines.

225 We collected tonsils, plasma, and saliva samples from children and ad

226 Tonsils play a key role in eliciting immune responses ag

227 CD4(+)CCR6(+)IL-7R(+)T cells from human tonsils produced IL-10 following stimulation by naive B

228 at CXCR5(lo)ICOS(lo) CD4(+) T cells in human tonsils represent Tfh lineage-committed cells that provi

229 study we found that ILC3s and ILC1s in human tonsils represented the ends of a spectrum that included

230 allergic patients the immune profile of the tonsils represents the atopic status of patients, with l

231 Here, we show that freshly isolated tonsil-resident BDCA1(+) DCs, BDCA3(+) DCs, and pDCs all

232 memory population, a class-switched CD39(+) tonsil-resident population, and a CD19(hi)CD11c(+) memor

233 ow that in vivo, EBV-infected B cells in the tonsils retain expression of functional and phenotypic m

234 Palatine tonsil samples were obtained from 143 elective tonsillec

235 Blood, saliva, and tonsils samples were collected from 39 children before a

236 Immunohistochemistry of human tonsil sections demonstrates that tonsillar interdigitat

237 aryngeal lymph nodes (RLNs), and sections of tonsil (sections of tonsil only from captive animals wer

238 infection in SVA-infected animals, with the tonsil serving as one of the sites of virus persistence.

239 Human tonsils show very low levels of allergen-induced T-cell

240 ing for age, sex, body mass index (BMI), and tonsil size (TS), the grade IV individuals had a 4.4-fol

241 Hence, RNA from tonsil slan(+) -cells, conventional CD1c(+) DCs (cDC2) a

242 , we and other groups have reported that, in tonsils, slan marks dendritic cell (DC)-like cells, as d

243 f the incubation period, thus explaining why tonsil specimens were not reliable for detection of simi

244 iased analysis of human DC subsets in blood, tonsil, spleen, and skin.

245 ve immunostaining, were present in the lung, tonsil, spleen, lymph nodes, and colon.

246 Tonsil standardized uptake values (SUVs) were measured b

247 with altered memory B cell subpopulations in tonsils, suggests that peripheral blood memory cell reco

248 Deep sequencing of virus from probang or tonsil swab samples harvested prior to postmortem showed

249 obang samples (oropharyngeal scrapings), and tonsil swabs to determine if sufficient virus variation

250 SAT serotypes from field buffaloes, palatine tonsil swabs were the sample of choice for recovering in

251 Subsequently, serum samples, tonsil swabs, and feces were collected from sows (n = 22

252 Human Tregs in blood, tonsil, synovial fluid, colon, and lung tissues did not

253 Tonsil T cells did not proliferate to common food and ae

254 ombinants in melanoma cells or primary human tonsil T cells in vitro.

255 ate exhibited by the majority of resting CD4 tonsil T cells leads to accumulation of incomplete rever

256 s phenomenon was not tumor specific, because tonsil T cells were similar to FL TILs.

257 nables the allergen-induced proliferation of tonsil T cells, indicating an active role of Treg cells

258 POKA in infectivity levels for primary human tonsil T cells.

259 In lymph nodes and tonsils, T-follicular helper cells have been identified

260 In fact, purified circulating and tonsil Tfh cells increased IgG secretion by blood Ag-ind

261 ture human B-cell subpopulation in the human tonsil that has characteristics of both naive B cells an

262 erize a distinct human NK cell population in tonsils that produces high amounts of the immunomodulato

263 m plays a gatekeeper role, as lesions of the tonsil, the lobus semilunaris inferior, and parts of the

264 R was clearly expressed on PC from the human tonsil, the lymph node, and the spleen (secondary lympho

265 ar structure in the oral mucosa and palatine tonsils, the high rate of oral blood flow, and innate fa

266 cell carcinomas (HPV-HNSCC) originate in the tonsils, the major lymphoid organ that orchestrates immu

267 teeth; mucosal swab samples from the tongue, tonsils, throat, and buccal mucosa; and stimulated and u

268 inized gingiva, hard palate; saliva, tongue, tonsils, throat; sub- and supra-gingival plaques; and st

269 Human NK cells develop in tonsils through discrete NK cell developmental intermedi

270 lymphoid hyperplasia, extensive hyperplastic tonsils, thymus hyperplasia, autoimmune lymphocytic thyr

271 ngue base, and pharyngeal wall brushes, then tonsil tissue (tonsillectomy).

272 gh pneumococcal-specific T-cell responses in tonsil tissue and peripheral blood.

273 r was also robust, approaching that of human tonsil tissue and the human germinal center B cell line,

274 of human slan(+) -monocytes infiltrating the tonsil tissue is toward a macrophage-like population, di

275 ILC3 frequencies were measured in tonsil tissue of allergic and nonallergic patients and i

276 ILC3 frequency was reduced in tonsil tissue of allergic patients and in peripheral blo

277 In tonsil tissue, although EBV reactivities outnumbered the

278 In ex vivo cultures of human tonsil tissue, CD4 T cells undergo a pronounced cytopath

279 77-positive centroblasts isolated from human tonsil tissue.

280 , cecal, jejunal, ileal, duodenal, and cecal tonsil tissues.

281 e HPV16 infection of cervical, foreskin, and tonsil tissues.

282 eristic of habitat groupings such as throat, tonsils, tongue dorsum, hard palate, and saliva.

283 The mean maximum SUV (SUV(max)) of tonsil tumors was 9.36 + or - 4.54, which was significan

284 In tonsils, unlike in blood, PDCs are the most frequent DC

285 cer, the mean difference in SUV(max) between tonsils was 10.43 + or - 7.07, which was significantly g

286 Uptake in bone marrow, parotid glands, and tonsils was slightly but statistically significantly hig

287 es (a B cell line, a monocyte line and human tonsils) was reactive with HECA-452, a mAb that recogniz

288 a patient with acute infarction of the right tonsil, we found (1) nearly completely abolished ipsilat

289 B cells from peripheral blood and tonsils were assessed using multicolor flow cytometry, a

290 h head and neck carcinomas not involving the tonsils were included as control subjects.

291 sting IgM(+)IgD(+)CD27(-) B cells from human tonsils were labeled with CFSE and stimulated in vitro w

292 We found that 51% of tonsils were positive for Hi, and in 95% of cases analyz

293 ture in secondary lymphoid organs, including tonsils, where common pathogens, such as EBV, enter the

294 low levels in various tissues, including the tonsils, whereas the wild-type virus was not.

295 , tongue, cheeks, hard and soft palates, and tonsils, which are colonized by bacteria.

296 raises the possibility of IAV shedding from tonsils, which may have an impact on host-to-host transm

297 could initiate infection either through the tonsil, with spread to respiratory tissues, or through i

298 o evaluate association of FDG uptake between tonsils within control subjects.

299 ose that arise from the lingual and palatine tonsils within the oropharynx.

300 ls of 27 palatine tonsils (10 RAT, 7 PTA, 10 tonsils without inflammation) was measured via a bead-ba