ライフサイエンスコーパス: tooth

1 d along the marginal gingiva to encircle the tooth.

2 l-mouth periodontal examination at six sites/tooth.

3 cells, vasculature, and nerves in the inner tooth.

4 njuries; and temporary storage of an avulsed tooth.

5 hanisms that govern inflammatory pain in the tooth.

6 e for the degradation of tissues surrounding tooth.

7 by the loss of mineralised tissues from the tooth.

8 experienced periodontists were asked to push tooth #16 into a buccal position to in a typodont model

9 mouse model of the neuropathy Charcot-Marie-Tooth 4F displays a highly pathological myelin proteome

10 delian syndrome CATIFA (cleft lip, cataract, tooth abnormality, intellectual disability, facial dysmo

11 ve frequently been reported as etiologic for tooth agenesis (TA).

12 entify novel LRP6 mutations in patients with tooth agenesis and investigate the significance of Lrp6

13 tudy broadens the mutation spectrum of human tooth agenesis and is the first to identify a LRP6 mutat

14 significant associations between patterns of tooth agenesis and rugae number, in addition to shape of

15 ttern in the control group and curved in the tooth agenesis group (p = 0.012 and p = 0.004, respectiv

16 Mean number of missing tooth units in the tooth agenesis group was 2.1.

17 = 0.004) was significantly increased in the tooth agenesis group.

18 r direction (p = 0.04 for both rugae) in the tooth agenesis group.

19 55) in control and 13.56 (SD, 1.54) years in tooth agenesis groups (p = 0.576).

20 rol study shows that modern individuals with tooth agenesis have indeed smaller facial configurations

21 ccurrence of missing permanent teeth, namely tooth agenesis, is common.

22 escent subjects with normal tooth number and tooth agenesis.

23 ay play an important role in the etiology of tooth agenesis.

24 associated with autosomal dominant inherited tooth agenesis.

25 Morphological analysis of a tooth and mitochondrial DNA from several hominin bone fr

26 ion pattern suggests a potential role in the tooth and surrounding periodontium.

27 alized tissues that surround and support the tooth, and microbiome dysbiosis.

28 of "frank" periodontitis affecting a single tooth; and assessment of factors that do/do not lead to

29 udy, we aimed to reprogram stem cells from a tooth apical papilla (SCAP) of a patient with OFCD, term

30 of these two signaling centers, resulting in tooth arrest at E14.5.

31 m and alveolar bone (AB), both essential for tooth attachment.

32 prismatic enamel, and a novel enamel-to-bone tooth attachment.

33 in adjacent periodontal tissues 4 days after tooth avulsion injury.

34 ules are (1) Stage is a patient-based, not a tooth-based concept, therefore, a single Stage is assign

35 firm the palatine, vomer and pterygoid to be tooth-bearing palatal bones, but also observed heterodon

36 an expansive edentulous maxilla and a small tooth-bearing premaxilla.

37 We propose the tooth became associated with the coleoid when the pteros

38 While pelagornithid or 'bony-toothed' bird fossils representing multiple species are

39 Tooth bleaching gels containing bromelain, papain, or fi

40 In this study, we conducted the tooth borne rapid palatal expansion model on the mouse,

41 The distal papilla around tooth-bound implant-supported restorations in maxillary

42 Patients receiving single implant in a tooth-bound maxillary non-molar site were recruited.

43 A treatment-driven classification for tooth-bound, facial PMMDs in non-molar sites, consisting

44 hment not only improves the prognosis of the tooth, but it also lessens the severity of the disease c

45 between the enamel and the dentine within a tooth, captures important information about tooth develo

46 therium was a genus of large-bodied scimitar-toothed cats, morphologically distinct from any extant f

47 ron X-ray tomographic imaging of incremental tooth cementum, that they had maximum lifespans consider

48 Professional tooth cleaning before the SRP does not improve the clini

49 t (control) two appointments of professional tooth cleaning but with motivation and instruction were

50 previously unreported cause of Charcot-Marie-Tooth (CMT) disease, a mutation in the mt-tRNA(Val) , in

51 lyses included esthetic outcomes: changes in tooth color and levels of volatile sulfur compounds in b

52 , the average tooth loss for flossers was ~1 tooth compared to ~4 teeth lost for nonflossers (P < 0.0

53 variables including rotation speed, burr-to-tooth contact, and coolant premisting modified and visua

54 n may still occur and is modified by burr-to-tooth contact.

55 eeding on hard foods, demonstrating that the tooth cusps of Port Jackson sharks, hard-shelled prey sp

56 cial for reparative dentinogenesis following tooth damage, and the modulation of this pathway affects

57 l enamel, caused by developmental defects or tooth decay (caries), affect health and quality of life,

58 ment of new technologies for caries control, tooth decay in primary teeth remains a major global heal

59 Measurements from the partial 'toothed' dentary point to a giant body size for the spec

60 stry, anthropology, and archaeology on human tooth development and discuss how teeth preserve a time-

61 tooth, captures important information about tooth development and vertebrate evolution and is inform

62 ial changes of Lrp6 expression during murine tooth development from E11.5 to E16.5.

63 ssion of PAX9, MSX1, AXIN2, and RUNX2 (known tooth development genes) was perturbed in mutant cells a

64 signaling pathways shown to adversely affect tooth development indicates they act downstream of these

65 Pitx2 (the first transcriptional marker for tooth development) controls the embryonic formation and

66 family, member 10A) plays a crucial role in tooth development, and patients with biallelic WNT10A mu

67 al shelf extension, palatal rugae formation, tooth development, and periderm formation.

68 e deficiency causes delayed craniofacial and tooth development, dysplastic facial features and delaye

69 ssion in odontoblasts and dental pulp during tooth development, the BSP-GFPtpz transgene was detected

70 he dynamic expression pattern of Lrp6 during tooth development.

71 ns for the preservation of biomarkers during tooth development.

72 ficance of Lrp6 on the biological process of tooth development.

73 investigate the significance of Lrp6 during tooth development.

74 Charcot-Marie-Tooth disease (CMT) is a neuropathy of the peripheral ne

75 Charcot-Marie-Tooth disease (CMT) reduces health-related quality of li

76 culomotor apraxia-4 (AOA4) and Charcot-Marie-Tooth disease (CMT2B2).

77 eripheral neuropathies such as Charcot-Marie-Tooth disease in humans.

78 ses of the neurologic disorder Charcot-Marie-Tooth disease that are accompanied by nephropathy, mostl

79 causes the most common form of Charcot-Marie-Tooth disease type 1A, whereas the reciprocal deletion o

80 e hereditary axonal neuropathy Charcot-Marie-Tooth disease type 2 (CMT2).

81 only autosomal dominant) cause Charcot-Marie-Tooth disease type 2A (CMT2A), the commonest axonal form

82 iduals with autosomal-dominant Charcot-Marie-Tooth disease type 2Z and spinal muscular atrophy, and t

83 pathic pulmonary fibrosis, and Charcot-Marie-Tooth disease, highlighting their therapeutic potential

84 list of genes associated with Charcot-Marie-Tooth disease, many patients with axonal forms lack a ge

85 In contrast, Charcot-Marie-Tooth disease-causing mutations that disrupt the stabili

86 ause either FSGS alone or with Charcot-Marie-Tooth disease.

87 used a combination of FSGS and Charcot-Marie-Tooth disease.

88 pheral neuropathies, including Charcot-Marie-Tooth disease.

89 nherited peripheral neuropathy Charcot-Marie-Tooth disease; however, the mechanism by which GDAP1 fun

90 but risk of colonization was associated with tooth emergence.

91 erstanding of molecular genetic pathology of tooth enamel formation.

92 ers affecting the quality and/or quantity of tooth enamel.

93 onditions that result in defective or absent tooth enamel.

94 t for the dominant theory that multi-layered tooth enameloid facilitated evolutionary diversification

95 Here we isolated Sox2-GFP+ tooth epithelial SCs which generated highly cellular sph

96 r or regenerate enamel, due to early loss of tooth epithelial SCs.

97 amel is secreted by ameloblasts derived from tooth epithelial stem cells (SCs).

98 , our results provide means of generating 3D tooth epithelium from adult SCs which can be utilized to

99 sess unusual microarchitecture that controls tooth erosion in a way that maintains functional cusp sh

100 al progenitor cell populations that regulate tooth eruption and tooth root formation are beginning to

101 tus, tooth root malformation, and failure of tooth eruption in molars, which essentially recapitulate

102 Tooth eruption is a unique biological process by which h

103 rent-nurse conversation about child's future tooth eruption, with advice given to visit a general den

104 rare genetic disorder exclusively affecting tooth eruption.

105 evalence and severity change over time after tooth eruption.

106 such as malocclusions and delayed or failed tooth eruption.

107 dental caries in children who have a primary tooth extracted.

108 h a nonabsorbable membrane (dPTFE) following tooth extraction (ARP n = 26).

109 ither the control group, which involved only tooth extraction (EXT n = 27), or the experimental group

110 Forty-four patients needing a single rooted tooth extraction and ridge preservation in preparation f

111 outcome between natural healing after molar tooth extraction and two different techniques of RP usin

112 gle local injection of fluvastatin following tooth extraction can potentially reduce the chance of de

113 Tooth extraction results in alveolar bone resorption and

114 study was to examine the effects of MaR1 on tooth extraction socket wound healing in a preclinical r

115 rptive events that occur as a consequence of tooth extraction with the purpose of facilitating tooth

116 s in routine checkup visits but increases in tooth extraction, a procedure that is highly used by pub

117 ic space closure initiation, after permanent tooth extraction, affects the incidence of GC.

118 After minimally traumatic tooth extraction, alveolar ridge dimensions were measure

119 mice that developed osteonecrosis following tooth extraction, there was increased bacterial infiltra

120 can preserve alveolar ridge dimension after tooth extraction.

121 nerally restricted to preventive measures or tooth extraction.

122 alveolar bone resorption as a consequence of tooth extraction.

123 valuated the impact of strontium ranelate on tooth-extraction wound healing in estrogen-deficient and

124 ted BH and the expression of bone markers in tooth-extraction wound in estrogen-deficient rats wherea

125 he following parameters were analyzed inside tooth-extraction wound: proportion of newly formed bone

126 of self-reported oral health-overall rating, tooth extractions, gum bleeding, loose teeth, bone loss

127 resembles the nonmarginal, radially arranged tooth files of arthrodires, an early group of armoured f

128 .1), counted by the decay clock, reveals saw-toothed fluctuations around a Phanerozoic mean of 18.6 m

129 Tooth form is considered to be highly heritable and sele

130 that DDR2 signaling is important for normal tooth formation and maintenance of the surrounding perio

131 Whether tooth formation deep inside the pharynx in extant verteb

132 ttainment estimates for fractional stages of tooth formation differed from those in modern humans.

133 rchestrated deposition of hard tissue during tooth formation in acrodont dentitions probably represen

134 efined the expression pattern of Ddr2 during tooth formation using Ddr2-LacZ knock-in mice.

135 wever, it is not known if Ddr2 has a role in tooth formation.

136 occurs from overingestion of fluoride during tooth formation.

137 lytic digestion that occurred in vivo during tooth formation.

138 en signaling is critical for proper bone and tooth formation.

139 derm-like cells have covered the prospective tooth-forming endodermal epithelium.

140 ard clinical examinations (probing six sites/tooth, full-mouth).

141 processing wears down teeth, thus affecting tooth functionality and evolutionary success.

142 he dilemma between the need for an operative tooth geometry and the unavoidable damage inherent to fe

143 in reptiles can be created from asymmetrical tooth germs.

144 ion rates and Sr marker-lines to reconstruct tooth growth along the enamel/dentine and then cementum/

145 h curves for increasing enamel thickness and tooth height and age-of-attainment estimates for fractio

146 e for the proper attachment of epithelium to tooth/implant surface.

147 the mesio-lingual site of the most posterior tooth in each quadrant, were harvested and pooled.

148 r are both required, but not sufficient, for tooth initiation in the pharynx.

149 Following maxillary tooth injury, Smoc2(-/-) mutants had increased osteoclas

150 tor ibuprofen (30 mg/kg body weight) blocked tooth injury-induced bone loss in Smoc2(-/-) mutants, re

151 e questions regarding the biological role of tooth-innervating fibers.

152 Clinical restoration of tooth integrity, sometimes involving physical and chemic

153 nder ultraviolet light reveals the pterosaur tooth is embedded in the now phosphatised cephalopod sof

154 jaw bones by pleurodont ankylosis, where the tooth is held in place on the labial side only.

155 ndividuals of the elongated strain varied in tooth length and the temporal dynamics of their signalin

156 At 36 months, tooth level analysis (Class III and Class IV groups) fou

157 ures of fluorosis severity at the person and tooth level were calculated: second highest FRI score at

158 Similar patterns were observed at the tooth level.

159 score was used) and highest FRI score at the tooth level.

160 At both the person and tooth levels, a decline in mild to moderate fluorosis se

161 According to its morphology, the tooth likely originates from the anterior to middle regi

162 ck or survived for some time with the broken tooth lodged in its mantle.

163 in the new classification were predictive of tooth loss after a long-term follow-up (>10 years) in pa

164 and 1 case each for numbness, diplopia, and tooth loss during intubation.

165 general anesthesia including 4 deaths and 1 tooth loss during intubation.

166 At the 5-y follow-up visit, the average tooth loss for flossers was ~1 tooth compared to ~4 teet

167 Periodontitis is a major cause of tooth loss globally.

168 as >=2 teeth with >=1 mm ACH loss or >=1 new tooth loss to periodontitis.

169 Five-year incident tooth loss was also evaluated.

170 fully adjusted model, PPC stage VII (Severe Tooth Loss) was moderately significantly related to inci

171 Grade A) at baseline and periodontal related tooth loss, whereas no differences were detected for the

172 periodontal disease characterized by severe tooth loss, while none of the categories of the CDC/AAP

173 ssues, leading to severe bone resorption and tooth loss.

174 teeth/gum health, loose teeth and history of tooth loss.

175 eriodontitis progression and determinant for tooth loss.

176 d a significantly higher periodontal-related tooth loss.

177 ral hygiene, gingival bleeding, and bone and tooth loss.

178 on, alveolar bone resorption, and ultimately tooth loss.

179 lead to the periodontal pocket formation and tooth loss.

180 up data (n = 375) was evaluated for incident tooth loss.

181 e individuals affected by cancer separately, tooth loss/edentulism is associated with SNPs in AXIN2 (

182 PPC stage V (Mild Tooth Loss/High Gingival Inflammation) was significant f

183 Twenty-six patients treated with single-tooth maxillary implants were included in this study.

184 a dental abnormality called maxillary canine-tooth mesioversion (MCM) (P = 1.53 x 10(-7)) as well as

185 nder study; specifically, those related with tooth mobility and gum migration.

186 Tooth mobility assessment is subjective and current tech

187 A novel technique to assess tooth mobility based on intraoral scanner measurements p

188 e reliability of a novel technique to assess tooth mobility.

189 ew recent progress in the field of mammalian tooth morphogenesis and also discuss the mechanisms regu

190 "Fast movers" (herein cases with a tooth movement >=1 mm per month) developed a GC in >90%

191 ption of the palatal alveolar bone caused by tooth movement after the maxillary incisors were retract

192 larity index when compared with conventional tooth movement alone.

193 Despite their hypothesized functions in tooth movement and maintenance, these 2 regions have not

194 Orthodontic tooth movement can result into iatrogenic sequelae to th

195 tooth movement, expanding the scope of safe tooth movement for patients undergoing orthodontic tooth

196 nd seems to occur more frequently with early tooth movement initiation and in "fast movers."

197 nhancing facial bone thickness, accelerating tooth movement, expanding the scope of safe tooth moveme

198 n while the other two received PhMT-s before tooth movement.

199 OT with hard tissue augmentation accelerated tooth movement.

200 benefits for patients undergoing orthodontic tooth movement.

201 Two studies reported an expanded tooth movement.

202 movement for patients undergoing orthodontic tooth movement.

203 neuropathies (HMNs) and axonal Charcot-Marie-Tooth neuropathy (CMT2) are clinically and genetically h

204 P2 gene mutations cause human Charcot-Marie-Tooth neuropathy, but the mature myelin sheath assembly

205 n rugae pattern between subjects with normal tooth number and agenesis suggests potential commonality

206 orphology in adolescent subjects with normal tooth number and tooth agenesis.

207 ermline Smoc2 homozygous mutants are viable, tooth number anomalies, reduced tooth size, altered enam

208 d be attributed to a biological mechanism of tooth number reduction that has evolved during time and

209 echanical stresses like those resulting from tooth occlusion, than teeth with a single root.

210 Plesioteuthis subovata is associated with a tooth of the pterosaur Rhamphorhynchus muensteri from th

211 odification therapy (PhMT-s) at sites with a tooth or an implant supported fixed dental prosthesis.

212 This work demonstrates that tooth pain can be quantified in freely behaving mice usi

213 However, the origin of the multicusped tooth pattern present in haramiyidans has remained enigm

214 activation-inhibition mechanisms underlying tooth patterning.

215 Tooth position was obtained using an intraoral scanner a

216 th the alternate files of teeth, whereas new tooth positions are inserted into the files of sequentia

217 in need of extraction of a single posterior tooth (premolar or molar) and subsequent replacement wit

218 ge, sex, length of follow-up period, initial tooth prognosis, revised tooth prognosis, tooth type, an

219 -up period, initial tooth prognosis, revised tooth prognosis, tooth type, and number of teeth lost at

220 The tooth provides an excellent system for deciphering the m

221 n should assist in uncovering mechanisms for tooth pulp inflammatory pain and other forms of trigemin

222 Injury of the tooth pulp is excruciatingly painful and yet the recepto

223 nt insights toward the development of future tooth replacement therapies.

224 its occurrence and optimize the outcomes of tooth replacement therapy with dental implants in this s

225 extraction with the purpose of facilitating tooth replacement therapy.

226 vertebrates vary in both pattern and type of tooth replacement.

227 nt spacing 13-21 MHz), via interleaving comb-tooth-resolved spectra acquired with a highly-coherent b

228 Tooth resorption (TR) in domestic cats is a common and p

229 l composites are routinely placed as part of tooth restoration procedures.

230 lammation all of which are major problems in tooth restoration.

231 nfrabony defects because it can improve both tooth retention rate and overall clinical outcomes.

232 t support formation of the highly functional tooth root and the periodontal attachment apparatus, whi

233 l regeneration coupled with the provision of tooth root coverage.

234 presenting severe oligodontia, microdontia, tooth root deficiencies, alveolar bone hypoplasia, and a

235 Here, we investigated how Wnt10a affects tooth root development by generating different tissue-sp

236 h size (decreased root/crown ratio), delayed tooth root development, widened PDL space, and interradi

237 ed in the fossil record, the significance of tooth root division in mammaliaforms remains enigmatic.

238 populations that regulate tooth eruption and tooth root formation are beginning to be unraveled.

239 uter world, and it occurs concomitantly with tooth root formation.

240 ion of the periodontal attachment apparatus, tooth root malformation, and failure of tooth eruption i

241 The patterns of dental wear along the tooth rows of nearly one hundred jaws of the small, earl

242 ped and worn molariform dentition with three tooth rows supports the previous inference that the spec

243 pled plasma mass spectrometry (LA-ICP-MS) to tooth samples to generate 500 temporally sequential meas

244 In health, the JE attaches to the tooth, sealing the inside of the body against oral micro

245 poral, spatial and functional differences in tooth signaling center activity, that arise from differe

246 No controlled studies pertaining to tooth sites were identified.

247 r2(slie/slie) mice displayed disproportional tooth size (decreased root/crown ratio), delayed tooth r

248 are viable, tooth number anomalies, reduced tooth size, altered enamel prism patterning, and spontan

249 refore proposed to function as scaffolds for tooth stabilization.

250 liaform precursors were "experimenting" with tooth structure and function, resulting in novel pattern

251 ts of modifying soft tissue phenotype around tooth-supported restorations.

252 inflammation affecting the integrity of the tooth supporting tissues.

253 During periodontitis, tooth-supporting alveolar bone is resorbed when there is

254 uld have possible deleterious effects on the tooth-supporting structures of the periodontium.

255 A total of 115 tooth surface sites of 32 teeth from the 12 patients wer

256 ion of the acquired salivary pellicle on the tooth surface, a conditioned film that provides the crit

257 harides to enhance bacterial adhesion on the tooth surface; subsequent lactic acid production reduces

258 e with baseline decayed, missing, and filled tooth surfaces (dmfs) (intercept) and rate of change in

259 aces derived from hierarchical clustering of tooth surfaces (termed caries clusters).

260 ocesses are important in different groups of tooth surfaces and that innate liability to some pattern

261 rgeting care to groups, individuals, or even tooth surfaces based on their caries risk has become a r

262 ies scores in groups of biologically similar tooth surfaces derived from hierarchical clustering of t

263 sion by monitoring thermal emission from the tooth surfaces during 30 s of air drying.

264 The mean number of tooth surfaces exposed to BisGMA materials (composites/s

265 entation may partially explain why groups of tooth surfaces form clusters within the mouth.

266 the presence of decayed, missing, or filled tooth surfaces).

267 y different (P < 0.001) when comparing sound tooth surfaces, lesion areas identified as arrested, and

268 n prognosis system can predictably determine tooth survivability within a 5-year period.

269 Tooth survival rate at the latest follow-up for those wi

270 iables on clinical attachment gain (CAL) and tooth survival were assessed via Cox proportional-hazard

271 nical attachment level (CAL) changes and the tooth survival were assessed via multi-level regression

272 eriodontal prognosis system by investigating tooth survival within a 64-month period and to compare t

273 in maintenance visits significantly affected tooth survival.

274 eas membrane exposure significantly affected tooth survival.

275 o the three-dimensional sub-micrometre scale tooth textures that formed during food consumption.

276 carious lesion management (complete carious tooth tissue removal and restoration placement) with pre

277 P, biological management (sealing in carious tooth tissue restoratively) with prevention; and PA, pre

278 Forty-four patients with a single-rooted tooth to be extracted and replaced by a dental implant w

279 sal (O) regions of the buccal surface of the tooth to determine the linear deviation in the three axe

280 Third permanent molars (M3s) are the last tooth to form but have not been used to estimate age at

281 e removal protocol, age, sex, dental arch or tooth type (p > 0.05/Cox), but was nearly 5-times higher

282 lar domain of P0), that causes Charcot-Marie-Tooth type 1B (CMT1B) neuropathy in humans and a similar

283 r domain of P0) mouse model of Charcot-Marie-Tooth type 1B (CMT1B), the genetic and pharmacological i

284 al tooth prognosis, revised tooth prognosis, tooth type, and number of teeth lost at the latest exam

285 Mean number of missing tooth units in the tooth agenesis group was 2.1.

286 person level (the maximum FRI score for each tooth was determined and the tooth with the second highe

287 reptile based on repeatable observations of tooth wear in a large sample of intact jaws.

288 mineral dust/grit adhering to plants causes tooth wear in mammalian herbivores.

289 Craniodental morphology, tooth wear, torso vertebral morphology, and body size al

290 linical attachment level (CAL) for six sites/tooth were ascertained by smoking status and plotted usi

291 unrestorable maxillary anterior or premolar tooth were randomized to receive either a provisional cr

292 For echolocating toothed whales, the use of sound to forage exposes them

293 ertical transmission of a foraging tactic in toothed whales.

294 be used in the development of peroxide-free tooth whitening gels.

295 on enamel surface and cytotoxicity of novel tooth-whitening formulations containing papain, ficin, o

296 llected from fifteen subjects presenting one tooth with a root canal infection, and their associated

297 g physical and chemical sterilization of the tooth with nerve fiber ablation (i.e., endodontic therap

298 score for each tooth was determined and the tooth with the second highest maximum FRI score was used

299 e collar acts as a suspense structure of the tooth within the bone during physiological loading.

300 y (OCT) can create cross-sectional images of tooth without X-ray exposure.