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1 n while the other two received PhMT-s before tooth movement.
2 herapies for patients undergoing orthodontic tooth movement.
3 stigate the effect of obesity on orthodontic tooth movement.
4 OT with hard tissue augmentation accelerated tooth movement.
5 tients and associated with observed rates of tooth movement.
6 d and analyzed: hypoloading and experimental tooth movement.
7 acclimatization can be exploited to optimize tooth movement.
8 neration, and root resorption in orthodontic tooth movement.
9 benefits for patients undergoing orthodontic tooth movement.
10 and metalloprotease domain; OTM, orthodontic tooth movement.
11 n of certain cytokines decreases the rate of tooth movement.
12 eralized osteoporosity and increased rate of tooth movement.
13 e, increases the rate of bone remodeling and tooth movement.
14 r bone reactions, which can be exploited for tooth movement.
15 es of alveolar bone cells during orthodontic tooth movement.
16  bone remodeling associated with orthodontic tooth movement.
17 Osteoclasts play a vital role in orthodontic tooth movement.
18             Two studies reported an expanded tooth movement.
19 ession in a rat model system of experimental tooth movement.
20 ion, periodontal remodeling, and orthodontic tooth movement.
21 steoclast differentiation during orthodontic tooth movement.
22 movement for patients undergoing orthodontic tooth movement.
23 eful for experimentally limiting orthodontic tooth movement, a process involving perturbations of nor
24                              The orthodontic tooth movement-ABR model was used to move a molar into t
25         BE reduced the amount of orthodontic tooth movement achieved in rats after 28 days.
26 ption of the palatal alveolar bone caused by tooth movement after the maxillary incisors were retract
27 larity index when compared with conventional tooth movement alone.
28 alone, osteotomy-assisted tooth movement, or tooth movement alone.
29 ned the TRPV2 expression during experimental tooth movement and assessed the effect of TRPV2 on osteo
30  force as applied to bone during orthodontic tooth movement and bone remodeling.
31 r bone alterations influenced by orthodontic tooth movement and can help determine risk assessment pr
32 t, while secondary outcomes included rate of tooth movement and change in clinical parameters (plaque
33 mineral metabolism contributing to disrupted tooth movement and exfoliation.
34 modeled throughout life, particularly during tooth movement and following tooth loss.
35  tongue, cheeks, and lips are known to cause tooth movement and in some situations can cause PTM.
36 f the alveolus has been an approach to speed tooth movement and is referred to as periodontally accel
37      Despite their hypothesized functions in tooth movement and maintenance, these 2 regions have not
38 tudying late stages of tooth development and tooth movement and the lack of good model systems.
39 ar spline regression to construct models for tooth movement and to identify factors associated with d
40 nical stress during mastication, orthodontic tooth movement, and wound healing following periodontal
41 ction osteogenesis in the osteotomy-assisted tooth movement animals.
42 nders them inadequate for proper orthodontic tooth movement at a later stage.
43            This study shows that orthodontic tooth movement can be inhibited with the use of matrix m
44                                  Orthodontic tooth movement can result into iatrogenic sequelae to th
45  patients had a significantly higher rate of tooth movement compared with normal-weight patients (+0.
46 e smoke while the control group was not, and tooth movement distance and osteoclast numbers were asse
47            Osseous morphometric analysis and tooth movement distance were evaluated with micro-CT.
48  with mastication, eruption, and orthodontic tooth movement-does the tissue increase its rate of cell
49  palatal implants that are placed to control tooth movement during orthodontic treatment and removed
50 nhancing facial bone thickness, accelerating tooth movement, expanding the scope of safe tooth moveme
51 reatment approaches in patients where buccal tooth movement (expansion) is planned in the anterior ma
52 e likely biologic mechanism underlying rapid tooth movement following selective alveolar decorticatio
53  tooth movement, expanding the scope of safe tooth movement for patients undergoing orthodontic tooth
54           "Fast movers" (herein cases with a tooth movement >=1 mm per month) developed a GC in >90%
55  inhibitors adjacent to teeth to limit local tooth movement in response to orthodontic forces.
56  the chief complaint and patient awareness o tooth movement in the last 5 years.
57 ry dentofacial therapy involving orthodontic tooth movement in the management of malocclusion with as
58          The probability of recession during tooth movement in thin biotype is high to justify gingiv
59 7 was increased in osteocytes in response to tooth movement in vivo.
60 nd seems to occur more frequently with early tooth movement initiation and in "fast movers."
61 olar bone regeneration (ABR) and orthodontic tooth movement into bovine bone (BB) regenerated sites.
62              The distance of the orthodontic tooth movement into the bovine bone was significantly re
63  Corticotomy-assisted and osteotomy-assisted tooth movement involves surgical incisions through the a
64                           During orthodontic tooth movement, mechanical forces acting on periodontal
65 PV2 was detected on day 1 after experimental tooth movement on the compression side, and the number o
66 ounding natural teeth undergoing orthodontic tooth movement or influenced by orthopedic forces throug
67 r physiologic compressive stress that causes tooth movement or pathologic stress that causes OIIRR.
68 n significantly increase the rate of initial tooth movement or reduce the amount of time required to
69 ovement, osteotomy alone, osteotomy-assisted tooth movement, or tooth movement alone.
70 ups: corticotomy alone, corticotomy-assisted tooth movement, osteotomy alone, osteotomy-assisted toot
71                                  Orthodontic tooth movement (OTM) causes transient pain and changes i
72                We tested whether orthodontic tooth movement (OTM) could be blocked by local administr
73                   Periodontal tissues during tooth movement (OTM) in T1D and under diabetic treatment
74 hosphate (beta-TCP) and evaluate orthodontic tooth movement (OTM) into the augmented sites.
75                                  Orthodontic tooth movement (OTM) is a prime example of mechanically
76                                  Orthodontic tooth movement (OTM) occurs through proteolytic remodell
77 in signaling plays a key role in orthodontic tooth movement (OTM), a common clinical practice for mal
78 g mechanisms in a mouse model of orthodontic tooth movement (OTM).
79 ition, cases in which there will be a facial tooth movement outside of the alveolar process need to b
80 o the maxillary molars inhibited orthodontic tooth movement (p < 0.01).
81 RANKL expression was seen at the orthodontic tooth movement pressure zone, without any change in OPG
82 ry teeth, ankylosis, and/or slow orthodontic tooth movement, suggesting altered mineral metabolism co
83           We used a rat model of orthodontic tooth movement to test the hypothesis that periodontal l
84                                  Orthodontic tooth movement was inhibited by local delivery of Ilomas
85 increased number of osteoclasts, orthodontic tooth movement was not altered in these mice.