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1 trol site (i.e., natural colonization of the tooth surface).
2 the presence of decayed, missing, or filled tooth surfaces).
3 supragingival plaque (PI > or =2 on >40% of tooth surfaces).
4 sting and newly formed alveolar bone and the tooth surface.
5 mutans attachment to and accumulation on the tooth surface.
6 liva, is a bacterial receptor that coats the tooth surface.
7 ortant role in microbial colonization of the tooth surface.
8 luid-derived protein layer that forms on the tooth surface.
9 sease induced by a biofilm that forms on the tooth surface.
10 uw ) distribution caused by the drops on the tooth surface.
11 dhesion of this gram-positive species to the tooth surface.
12 droxyapatite (sHA), an in vitro model of the tooth surface.
13 hanisms to colonize and form biofilms on the tooth surface.
14 oped that correlate with mineral loss of the tooth surface.
15 olonizers that initiate biofilm formation on tooth surfaces.
16 ing to be 27% for decayed and 14% for filled tooth surfaces.
17 protection against S. mutans colonization of tooth surfaces.
18 the salivary pellicle coating supragingival tooth surfaces.
19 the ability of some streptococci to colonize tooth surfaces.
20 may modulate oral bacterial colonization of tooth surfaces.
21 ty of S. sputigena to colonize supragingival tooth surfaces.
22 were collected from caries-free and carious tooth surfaces.
23 r that has a seamless interface with natural tooth surfaces.
24 antimicrobial protection to the mucosal and tooth surfaces.
25 ily exert their effects uniformly across all tooth surfaces.
26 odulate the dynamic formation of biofilms on tooth surfaces.
27 on cumulative caries increments on different tooth surfaces.
28 ry (d(2,3)fs) and total (d(2,3)fs+D(2,3)MFS) tooth surfaces.
29 the multispecies environment in biofilms on tooth surfaces.
30 and mediate sucrose-independent adherence to tooth surfaces.
31 rganisms, such as Streptococcus gordonii, to tooth surfaces.
32 anaerobes that initiate biofilm formation on tooth surfaces.
33 ed separate criteria for smooth and occlusal tooth surfaces.
34 ion of the acquired salivary pellicle on the tooth surface, a conditioned film that provides the crit
35 protein layer formed from natural saliva on tooth surfaces, acquired enamel pellicle (AEP), protects
36 outcomes within individual patients, such as tooth surfaces affected by a caries lesion, tended to be
37 hypothesize that such polishing may protect tooth surfaces against the damage caused by cariogenic b
38 Forty-two consented young adults, blinded to tooth surface allocation, were treated with resin infilt
39 the children had at least 1 unfilled decayed tooth surface and 33% had at least 1 filled surface.
40 bacteria that accumulate in a biofilm on the tooth surface and affect the adjacent periodontal tissue
41 occus sanguinis is an early colonizer of the tooth surface and competes with oral pathogens such as S
42 sucrose to initiate biofilm formation on the tooth surface and consequently produces lactic acid to d
43 romote accumulation of microorganisms on the tooth surface and further establishment of pathogenic bi
44 are initiated by bacteria that colonize the tooth surface and gingival sulcus, the host response is
46 us parasanguis is a primary colonizer of the tooth surface and plays a pivotal role in the formation
47 cocci and actinomyces and their adherence to tooth surface and the associated host cells are key earl
48 s streptococci are primary colonizers of the tooth surface and thus form the foundation for the compl
50 ciation between caries of the mid- dentition tooth surfaces and AJAP1 (p value = 2e-8), a gene possib
51 val dental plaque differ substantially among tooth surfaces and children of different caries activiti
53 f filled (p < 0.001) and decayed (p = 0.036) tooth surfaces and negatively associated with number of
54 les that selectively adsorb from saliva onto tooth surfaces and provides a protective interface betwe
55 tial adherence of S. mutans to saliva-coated tooth surfaces and subsequent development of dental cari
56 elies on the bacterium's ability to colonize tooth surfaces and survive a strongly acidic environment
57 er of decayed, missing, and filled permanent tooth surfaces and teeth, and the number of decayed, and
59 ocesses are important in different groups of tooth surfaces and that innate liability to some pattern
60 utcomes (e.g., number of carious and missing tooth surfaces), and oral health-related quality of life
61 yces are the major initial colonizers of the tooth surface, and the interactions between them and the
62 sms are not so effective against biofilms on tooth surfaces, and oral hygiene measures such as brushi
63 tached gingiva, the calculus deposition over tooth surfaces, and the subgingival calculus that enable
64 al pH fluctuations within the biofilm on the tooth surface are a ubiquitous and natural phenomenon, t
67 ilm formation of Streptococcus mutans on the tooth surface as it directly regulates the extracellular
68 ying the 3D surface textures that develop on tooth surfaces as a consequence of feeding, we show that
69 silica nanoparticles were used to polish the tooth surfaces, as compared with conventional polishing
71 g the permanent dentition into categories of tooth surfaces based on co-occurrence of caries) to nomi
72 rgeting care to groups, individuals, or even tooth surfaces based on their caries risk has become a r
73 al remained over the dentin and at the outer tooth surface, but between these regions were invading f
75 ted sequential nature of colonization of the tooth surface by members of different genera can be inve
76 parable to the shear stress generated at the tooth surface by salivary flow ( approximately 0.8 dyn/c
77 gen membrane that readily adapts to bone and tooth surfaces by a gel formation of collagen fibers and
78 al, sub-micrometre scale textures created on tooth surfaces by interactions with food, reveals that t
82 g a split-mouth design, with half the buccal tooth surfaces coated with serum and the other half with
83 S. sanguinis, which yields understanding of tooth surfaces colonization and contributions to dental
84 her, we find that a rigorous cleaning of the tooth surface combining diluted bleach and UV light irra
86 ies scores in groups of biologically similar tooth surfaces derived from hierarchical clustering of t
88 tion time points, the microbiomes of healthy tooth surfaces differed substantially from those found d
89 e with baseline decayed, missing, and filled tooth surfaces (dmfs) (intercept) and rate of change in
91 to compute the decayed, missing, and filled tooth surfaces (DMFS) index and the number of remaining
92 umber of decayed, missing and filled primary tooth surfaces (dmfs) were used as a repeated outcome me
95 tococcus mutans uses adhesin P1 to adhere to tooth surfaces, extracellular matrix components, and oth
97 ed significantly more cellular attachment to tooth surfaces formerly covered by subgingival calculus
98 ary colonizer of the multispecies biofilm on tooth surfaces forming dental plaque and a potential age
99 ctinomyces that initiate colonization of the tooth surface frequently coaggregate with each other as
101 arasanguis FW213, a primary colonizer of the tooth surface, has been purified from the culture medium
103 , clusters 1-5, groups of similarly behaving tooth surfaces identified through hierarchical clusterin
104 , clusters 1-5, groups of similarly behaving tooth surfaces identified through hierarchical clusterin
106 growth and creates horizontal grooves on the tooth surface in humans and other mammals, yet there is
108 5-2.9) and filled (OR, 1.4; 95% CI, 1.1-1.8) tooth surfaces in deciduous but not in permanent teeth.
110 cterized by a dysbiotic shift at the biofilm-tooth surface interface, yet comprehensive biochemical c
111 cus gordonii to the acquired pellicle of the tooth surface involves specific interactions between bac
112 rasanguis FW213, a primary colonizer, to the tooth surface is mediated mainly by peritrichous long fi
114 n is initiated by bacteria that colonize the tooth surface, leading to inflammation and bone resorpti
115 y different (P < 0.001) when comparing sound tooth surfaces, lesion areas identified as arrested, and
116 herefore, we used hierarchical clustering on tooth surface-level caries data for 1,068 Appalachian ad
118 o stabilize disease or even remineralize the tooth surface, maximizing retention of tooth tissue and
120 ar trial period, children had a mean of 18.7 tooth surfaces (median, 16) restored in the amalgam grou
122 ion in its ability to adhere in the in vitro tooth surface model, saliva-coated hydroxylapatite (SHA)
123 restorations placed in previously unrestored tooth surfaces of 4,672 patients by 222 clinicians in th
125 teeth (OR = 0.86; 95% CI, 0.60 to 1.22) and tooth surfaces (OR = 0.85; 95% CI, 0.59 to 1.21) were ex
128 C-terminal AgI/II fragments to the putative tooth surface receptor salivary agglutinin (SAG), as mon
133 three coaggregating early colonizers of the tooth surface (Streptococcus gordonii DL1, Streptococcus
134 harides to enhance bacterial adhesion on the tooth surface; subsequent lactic acid production reduces
136 t dentition can be subdivided into groups of tooth surfaces that are useful for understanding the fac
137 sis yielded evidence of 5 distinct groups of tooth surfaces that differ with respect to caries: (C1)
138 pragingival dental plaque was collected from tooth surfaces that were caries-lesion-free (PF) and fro
139 can be applied to at least three levels: the tooth surface, the individual, or the group/population.
141 proteins can promote cell attachment to the tooth surface; therefore, attempts have been made to uti
143 nctional epithelium directly attaches to the tooth surface via hemidesmosomes, a soft diet requires m
145 essary first step toward colonization of the tooth surface, we sought to determine what effect deleti
146 The number of untreated carious and missing tooth surfaces were associated with oral health-related
149 sed on different ECC definitions and sets of tooth surfaces) were quantified using Brownian distance
150 ccus mutans proliferates as a biofilm on the tooth surface, where it obtains nutrients and metabolize
151 , initiate the formation of oral biofilms on tooth surfaces, which requires differential expression o
153 e that 23.8 million persons have one or more tooth surfaces with > or = 3 mm gingival recession; 53.2
154 during oral surgery should saturate exposed tooth surfaces with amounts of fibronectin adequate for
155 ales had a lower number of untreated coronal tooth surfaces with caries (1.5), but a higher mean numb
156 the primary trait was the number of primary tooth surfaces with caries experience (i.e., dmfs index)
157 on using a dataset with 3 levels of nesting: tooth surfaces within an interproximal (IP) region, IP r