ライフサイエンスコーパス: total peripheral resistance

1 vous system, and increased vascular tone and total peripheral resistance.

2 ers, additive declines in blood pressure and total peripheral resistance.

3 ortic pulsatile load and concurrently lowers total peripheral resistance.

4 umans despite reduced diastolic pressure and total peripheral resistance.

5 uced an increase in blood pressure (43%) and total peripheral resistance (65%) without any change in

6 ics changed significantly with a decrease in total peripheral resistance and an increase in common fe

7 terized by elevated heart rate and decreased total peripheral resistance and arterial blood pressure.

8 etention were hypotensive, with decreases in total peripheral resistance and filtration fraction on d

9 stimulate vasoconstriction, which increases total peripheral resistance and mean arterial pressure.

10 Total peripheral resistance and perivascular fibrosis in

11 women during EFP than MLP (P = 0.030), while total peripheral resistance and plasma noradrenaline wer

12 Cardiac index, total peripheral resistance, and blood volume were not d

13 ut, systemic oxygen delivery, stroke volume, total peripheral resistance, and organ blood flow in the

14 onses (P < 0.001), diastolic blood pressure, total peripheral resistance, and stroke volume compared

15 emic blood pressure is acutely controlled by total peripheral resistance as determined by the diamete

16 l pressure, portal pressure, cardiac output, total peripheral resistance, central blood volume, and e

17 ripheral hemodynamics were determined by the total peripheral resistance, common femoral artery flow,

18 ed lower (P = 0.09), heart rate was similar, total peripheral resistance decreased (2172 +/- 364 vs.

19 enol infusion before any symptoms developed, total peripheral resistance decreased 24% +/- 20% in gro

20 ponse to tilt before any symptoms developed, total peripheral resistance decreased 9% +/- 14% in grou

21 Cardiac index and total peripheral resistance decreased in MSA patients by

22 Total peripheral resistance decreased with rosuvastatin

23 n the EP group and associated with increased total peripheral resistance (difference in means, 96.4 [

24 Total peripheral resistance during tilt post flight was

25 iac output, and calculated stroke volume and total peripheral resistance, during supine rest and 10 m

26 as defined by LV end-diastolic dimension and total peripheral resistance estimated by thoracic impeda

27 on on autonomic nerves may impair control of total peripheral resistance giving rise to symptomatic o

28 copal subjects, accompanied by a decrease in total peripheral resistance in 16 of them (64%, group A)

29 In vivo, human U-II markedly increases total peripheral resistance in anaesthetized non-human p

30 and cardiac output (CO) but minor changes in total peripheral resistance in saline-treated rats.

31 d VVS (P<0.05) compared with baseline, while total peripheral resistance increased (P<0.05).

32 1.24 and 1.15 versus 1.02 mm Hg/mL x m2) and total peripheral resistance index (3027 and 2805 versus

33 .01) in cardiac index, a 28% increase in the total peripheral resistance index (p < .01), and a 33% d

34 The total peripheral resistance index and stroke volume inde

35 Although total peripheral resistance markedly increased, mean art

36 es in mean arterial pressure of 18 mm Hg and total peripheral resistance of 665 AU and increases in h

37 of approximately 20 mm Hg and an increase in total peripheral resistance of approximately 30%.

38 olic pressure (7 +/- 4 mmHg, P <= 0.001) and total peripheral resistance (P = 0.013) concomitantly wi

39 (P<0.001) while accentuating the increase in total peripheral resistance (P=0.012).

40 junction with a 94.5 dynes x sex/cm(5) lower total peripheral resistance (P=0.0368).

41 Although total peripheral resistance remains decreased, Na+ reten

42 e of sepsis), cardiac output, stroke volume, total peripheral resistance, systemic oxygen delivery, a

43 of stroke volume (SV), cardiac output (CO), total peripheral resistance (TPR) and arterial complianc

44 rve activity (MSNA) is positively related to total peripheral resistance (TPR) and inversely related

45 hic measures of preejection period (PEP) and total peripheral resistance (TPR) in healthy black (n=76

46 ecreased, whereas heart rate (HR), MSNA, and total peripheral resistance (TPR) increased during HUT (

47 Total peripheral resistance (TPR) increased during LBNP

48 Baseline total peripheral resistance (TPR) was not different betw

49 ciated decrease in cardiac contractility and total peripheral resistance (TPR) were similar in TRPV(1

50 is, stroke volume (SV), heart rate (HR), and total peripheral resistance (TPR), in 163 patients with

51 Patients with VAH show a greater rise in total peripheral resistance (TPR), suggesting a compensa

52 , and a sympathetically-mediated increase in total peripheral resistance (TPR).

53 es (OF; n = 12) underwent assessments of BP, total peripheral resistance (TPR; Modelflow) and MSNA ac

54 Total peripheral resistance was calculated from mean art

55 s. 5.02 +/- 0.40 l min(-1), P = 0.01), while total peripheral resistance was greater (1327 +/- 117 vs

56 stroke volume (r=0.88+/-0.13, P<0.05), while total peripheral resistance was related to MSNA during 4

57 pared with control rats, but the increase in total peripheral resistance was significantly attenuated

58 In the other 9 subjects, total peripheral resistance was well maintained even at

59 Heart rate, mean arterial pressure, and total peripheral resistance were greater, whereas cardia

60 Upright heart rate and total peripheral resistance were greater, whereas stroke

61 Blood pressure and total peripheral resistance were higher during rest and