ライフサイエンスコーパス: trabecular

1 l-endocardial distributions were as follows: trabecular 26.1% and subendocardial 20.2%, midwall 33.4%

2 of dysregulated RhoA GTPase activity in the trabecular AH outflow pathway increases intraocular pres

3 ark the Nppa(+) or Hey2(+) cardiomyocytes as trabecular and compact components of the ventricular wal

4 that is composed of cells derived from both trabecular and compact layers.

5 en support previous reports that deficits in trabecular and cortical bone density and structure indep

6 ted tomography (HR-pQCT), we demonstrate low trabecular and cortical bone density contributing to low

7 tical osteoid mineralization rate and higher trabecular and cortical bone formation rate was displaye

8 el, vertical sleeve gastrectomy (VSG) caused trabecular and cortical bone loss that was independent o

9 layed reduced peak bone mass and age-related trabecular and cortical bone loss.

10 Treatment with estradiol increased the trabecular and cortical bone mass to a similar extent in

11 salpha(OsxKO) mice) yielded markedly reduced trabecular and cortical bone mass.

12 Trabecular and cortical bone mineral density (BMD) and c

13 ized BMPR1a-FC receptor ligand trap prevents trabecular and cortical bone volume loss caused by myelo

14 We analyse metacarpal trabecular and cortical bone, which provide insight into

15 le mice that results in exceptionally strong trabecular and cortical bones, whose density surpasses o

16 elated reductions in bone volume in both the trabecular and cortical compartments.

17 OVX produced the expected changes in trabecular and cortical parameters consistent with post-

18 nd bone volume fraction, negatively impacted trabecular and cortical parameters, and resulted in shor

19 Trabecular and total BMD at the tibia trended toward hig

20 -term SPI diet diminished the loss of total, trabecular, and cortical bone mineral density, whereas S

21 Although trabecular anisotropy was improved in all the materials

22 hepatocellular carcinoma, which exhibited a trabecular architecture, closely resembling human hepato

23 (cortical thickness, P = 0.008 and < 0.001; trabecular area, P = 0.001 and < 0.001, respectively).

24 mative ranges for first lumbar vertebra (L1) trabecular attenuation values across all adult ages to m

25 Conclusion Normative ranges of L1 vertebra trabecular attenuation were established across all adult

26 l [CI]: 0.78, 0.93), as well as trochanteric trabecular BMD combined with neck cortical thickness (mo

27 Tibial trabecular bone among participants completing 70% or mor

28 These disorders result in excess growth of trabecular bone and collagen fibers that replace hematop

29 s important in bone remodeling, particularly trabecular bone and endogenous Epor expression in osteob

30 pe characterized by osteopenia of epiphyseal trabecular bone and subchondral cortical plate.

31 th Pyle's disease, have increased amounts of trabecular bone and unusually thin cortical bone, as a r

32 SBP (subchondral bone plate) and B.Ar/T.Ar (trabecular bone area to total tissue area).

33 In this case-control study, trabecular bone biopsies from iliac crest were collected

34 The present study was aimed to evaluate the trabecular bone changes between healthy individuals and

35 Assessment of microdamage within the trabecular bone core was performed using synchrotron X-r

36 ne in humans may significantly underestimate trabecular bone damage sustained by ART.

37 gher trabecular connectivity density, higher trabecular bone density and thicker structures, somethin

38 ic histomorphometry showed no differences in trabecular bone formation between WT and Col6alpha2-KO m

39 indicating that Bmpr1a signaling suppresses trabecular bone formation through effectors beyond Smad4

40 ministration (1200 U/kg) for 10 days reduced trabecular bone in control mice but not in Tg mice.

41 of muscle mass and strength, and the loss of trabecular bone in femurs and vertebrae following Folfir

42 strogen response in NOER mice), cortical and trabecular bone in long bones, as well as uterus and thy

43 results demonstrated an increased amount of trabecular bone in MULTI calluses at 21 days post-injury

44 the age-related changes in the cortical and trabecular bone in old age, and assessed whether we can

45 n response was highly tissue-dependent, with trabecular bone in the axial skeleton being strongly dep

46 icroarchitecture deteriorated, mainly in the trabecular bone in the first 6 months and cortical bone

47 in vitro, suggesting that the restoration of trabecular bone in vivo was due to decreased bone resorp

48 e protected against PTH-induced cortical and trabecular bone loss as well as from increases in serum

49 benefit from zoledronate); HR-pQCT detected trabecular bone loss at the peripheral skeleton, which z

50 our data imply that Col6a2 deficiency causes trabecular bone loss by enhancing osteoclast differentia

51 m supplementation on peripheral cortical and trabecular bone loss during pregnancy and bone gain post

52 CD8+ T cells may further contribute to trabecular bone loss in some patients with advanced AIDS

53 Osteoporosis is characterised by trabecular bone loss resulting from increased osteoclast

54 not only muscle aging-like deficit but also trabecular bone loss, a feature of osteoporosis.

55 ntribute to post-transplant central skeleton trabecular bone loss, and zoledronate does not induce AB

56 uction, stimulate bone resorption, and cause trabecular bone loss, demonstrating that the gut microbi

57 L-6 expression, hyperosteoclastogenesis, and trabecular bone loss, uncovering a pathological mechanis

58 formation, bone resorption, and cortical and trabecular bone loss.

59 tion of ART, causes significant cortical and trabecular bone loss.

60 is central in sex steroid deficiency-induced trabecular bone loss.

61 caused by sex steroid deficiency, leading to trabecular bone loss.

62 lar joints (TMJs), displaying as subchondral trabecular bone loss.

63 ncreased osteoclast numbers, and significant trabecular bone loss.

64 c BMD and bone microstructure indicated that trabecular bone mainly contributed to the positive assoc

65 lamin A/C-KO mice diminishes the deficits in trabecular bone mass but not muscle.

66 ssion were increased in bones, and increased trabecular bone mass from pre-osteoblast specific Ezh2 d

67 Dmp1-Cre, we observed a dramatic increase in trabecular bone mass in postnatal mice, which was due to

68 cted Hdac3 deficiency decreased cortical and trabecular bone mass parameters, suggesting that Hdac3 r

69 hes the osteoclastogenic deficit and reduces trabecular bone mass.

70 n wild type animals causes a 50% decrease in trabecular bone mass.

71 lls specifically in postnatal mice increased trabecular bone mass.

72 ssion of WNT16 surprisingly increases mainly trabecular bone mass.

73 nt LT showed severe deficits in cortical and trabecular bone microarchitecture.

74 odifications of the aBMD and of cortical and trabecular bone microstructures.

75 A significant decrease in global trabecular bone mineral density (38.1%) and cortical thi

76 ne mineral content and density, cortical and trabecular bone mineral density (BMD), BMC, and bone are

77 The local trabecular bone mineral density decreased in both high s

78 Total and trabecular bone mineral density were significantly lower

79 odronate was less than the large increase in trabecular bone observed in a nonwounded long bone.

80 re decreased in the proximal tibia and spine trabecular bone of dKO-Hom mice compared to wild-type (W

81 .Dn), and by increased separation (Tb.Sp) in trabecular bone of the femur and vertebra.

82 mid-L5 level, the mean CT attenuation of the trabecular bone of the L5 vertebral body (L5HU) was meas

83 habitual tool manufacture, have a human-like trabecular bone pattern in the metacarpals consistent wi

84 SRK-015P treatment improves the cortical and trabecular bone phenotypes in these mice.

85 Pb and HFD each reduced trabecular bone quality and together had a further detri

86 The trabecular bone region was divided into 37 volumes of in

87 Further, muCT analysis of trabecular bone revealed that, compared with the vehicle

88 neck BMD, lumbar spine BMD, and lumbar spine trabecular bone score (TBS) as secondary outcomes, all a

89 nd periodontitis remains unresolved; and the trabecular bone score (TBS) is a new index for assessing

90 Trabecular bone score and in vivo microindentation are n

91 Trabecular bone score was borderline lower (1.21 +/- 0.1

92 Trabecular bone score was measured by specific software

93 udy the association between KTR and BMD/BMSi/trabecular bone score.

94 In a rodent sepsis model, trabecular bone strength is functionally reduced within

95 ion of bortezomib (Bzb) reversed the loss of trabecular bone structure and strength in mice at 4 wk a

96 at more highly mobile human populations have trabecular bone structure similar to what would be expec

97 alyses were performed to assess cortical and trabecular bone structure, density, and rigidity.

98 ut it failed in normalizing growth plate and trabecular bone structures.

99 companied by increased osteoclast number per trabecular bone surface.

100 utant PTH1R exhibited a dramatic increase in trabecular bone that was dependent upon expression of Gs

101 ronate treatment increased extraction socket trabecular bone thickness at 14 d, which correlated with

102 Femoral head trabecular bone tissue digests were sorted into CD45-CD2

103 Increased subchondral trabecular bone turnover due to imbalanced bone-resorbin

104 Analyses of mass-corrected trabecular bone variables reveal that the forager popula

105 d 22.2% and female Tg mice had 29.6% reduced trabecular bone volume (BV) compared to controls.

106 matically recognized compact bone volume and trabecular bone volume (IBV) in CT slices.

107 <.05) and resulted in a 2.5-fold increase in trabecular bone volume (p<.001).

108 th CLP for 2 weeks had significantly reduced trabecular bone volume and cortical bone thickness, asso

109 in human milk to this diet increased femoral trabecular bone volume and cortical thickness, reduced o

110 S3 has been ablated in osteocytes, have high trabecular bone volume and poorly defined metaphyseal co

111 g ACVR2A had significantly increased femoral trabecular bone volume at 6 weeks of age.

112 tomography revealed robust deterioration of trabecular bone volume by both subsets, while CD4+ T cel

113 in signaling in vivo and completely restored trabecular bone volume by increasing bone formation and

114 egative effects on bone, as shown by reduced trabecular bone volume fraction (BV/TV), thickness (Tb.T

115 the marrow, which correlated with increased trabecular bone volume fraction after 7 and 14 d.

116 rly Rhbdf2, whose close association with the trabecular bone volume fraction and number was strongly

117 treated only with loading showed the highest trabecular bone volume fraction at week 22.

118 lted in virtually identical losses in tibial trabecular bone volume fraction, BV/TV (24-28% reduction

119 olic effect of intermittent PTH treatment on trabecular bone volume is blunted by deletion of Gsalpha

120 0 mug/kg/day) for 4 weeks failed to increase trabecular bone volume or cortical thickness in male and

121 ed tomography analysis showed a reduction of trabecular bone volume, bone mineral density, and number

122 to increase osteoclast numbers and decrease trabecular bone volume, cortical thickness and mechanica

123 d ACVR2B demonstrated sustained increases in trabecular bone volume, similar to those in ACVR2A singl

124 d thoracic spine, with an associated loss of trabecular bone volume.

125 In this study, PEDF delivery increased trabecular bone volume/total volume by 52% in 6-mo-old P

126 ortical bone was -0.250, and between BMI and trabecular bone was -0.143 (all P < 0.001).

127 Trabecular bone was likewise reduced and was accompanied

128 e osteogenic benefits of co-treatment on the trabecular bone were lower than loading alone.

129 ic-specific deletion of Epor exhibit reduced trabecular bone with age without change in marrow adipoc

130 lly migrated and colonized tenascin-C-coated trabecular bone xenografts in a novel system that employ

131 Besides increasing osteoblast number in the trabecular bone, deletion of Bmpr1a by Dmp1-Cre also not

132 med quantitative differences in cortical and trabecular bone, including decreased cortical thickness

133 ese included fracture sites with predominant trabecular bone, not previously reported as being associ

134 pha antagonist all protect cortical, but not trabecular bone, revealing complex effects of T-cell rec

135 logical analysis showed reduced cortical and trabecular bone, suggesting cell-autonomous functions of

136 that accompany prostate cancer metastasis to trabecular bone, with potential implications to therapeu

137 ogenically-committed BMSC subset in human OA trabecular bone.

138 es and hindered accrual of peak cortical and trabecular bone.

139 astic bone consisting of mature cortical and trabecular bone.

140 expansion and were present in control non-OA trabecular bone.

141 l size and perfection in remnant metaphyseal trabecular bone.

142 gions are present in the disordered phase of trabecular bone.

143 and no statistically significant changes in trabecular bone.

144 OA in the superior subchondral cortical and trabecular bone.

145 deficiency of sFRP4, that cortical-bone and trabecular-bone homeostasis were governed by different m

146 resolution scans and histopathology revealed trabecular bones in two cases, hyaline cartilage in anot

147 ulation occurred in the epiphyseal plates of trabecular bones.

148 inant of intraocular pressure and success of trabecular bypass glaucoma surgeries.

149 yet how tissue symmetry is broken to specify trabecular cardiomyocytes is unknown.

150 Parietal bone trabecular compartment was mildly altered.

151 to bone quality in the femoral cortical and trabecular compartments of male and female mice.

152 racellular matrix contacts and modulated the trabecular component of aqueous outflow whereas another

153 iopsies showed post-transplant impairment of trabecular connectivity (and no benefit from zoledronate

154 towards thicker cortical bone plate, higher trabecular connectivity density, higher trabecular bone

155 py was improved in all the materials groups, trabecular connectivity was diminished when the biomater

156 ession with those in epithelial, stromal and trabecular corneal cells, we selected 9 structural or fu

157 Thus, the low trabecular density of the recent modern human skeleton e

158 n humans today appear to have relatively low trabecular density, but little is known about how that d

159 Moreover, deletion of PKD1 in vivo reduced trabecular development and activity of osteoblast develo

160 sms underlying appropriate downregulation of trabecular ERBB2 signaling are little understood.

161 is lineage in vivo, the capacity to induce a trabecular fate in immature cardiomyocytes in vitro, and

162 The trabecular iris angles of MPS types I, II, and VI were s

163 scleral spur: angle opening distance (AOD), trabecular iris space area (TISA), and scleral spur angl

164 rameters, including angle recess area (ARA), trabecular iris space area (TISA), iris area, iris curva

165 angle opening distance (AOD500, AOD750), and trabecular iris space area (TISA500, TISA750) were measu

166 increases in AOD750, angle recess area, and trabecular iris surface area (P < 0.05 for all).

167 Trabecular-iris angle (TIA) and angle opening distance 5

168 The iridocorneal angle measurements: trabecular-iris angle (TIA), angle opening distance (AOD

169 Trabecular-iris angle (TIA), angle opening distance 500

170 ce (AOD), trabecular-iris space area (TISA), trabecular-iris circumference volume (TICV), length of i

171 Trabecular-iris contact (TIC) was observed in 8 eyes of

172 ACA parameters angle opening distance (AOD), trabecular-iris space area (TISA), trabecular-iris circu

173 e (TIA), angle opening distance (AOD500) and trabecular-iris space area (TISA500) 500 mum from the sc

174 and 750 mum (AOD750) from the scleral spur; trabecular-iris space area at 500 mum (TISA500) and 750

175 and 750 mum anterior from scleral spur), the trabecular-iris-space area (TISA, measured 500 and 750 m

176 ish, we find that cardiomyocytes seeding the trabecular layer actively change their shape while compa

177 oncentrically encircles the ridge-like inner trabecular layer.

178 her contractility to delaminate and seed the trabecular layer.

179 distinct layers: the outer compact and inner trabecular layers.

180 ered in the ciliary muscle (46 +/- 5.6%) and trabecular meshwork (37 +/- 8.3%) of treated eyes relati

181 nd severity of glaucoma, pigmentation of the trabecular meshwork (PTM), total energy delivered, and b

182 conventional outflow pathway comprising the trabecular meshwork (TM) and Schlemm's canal (SC).

183 The IOP is maintained by the trabecular meshwork (TM) and the elevation of IOP in ope

184 ls (VRAC) in the modulation of the volume of trabecular meshwork (TM) cells and, in turn, the aqueous

185 se blindness by compromising the function of trabecular meshwork (TM) cells in the anterior eye, but

186 l-to-mesenchyme-like transition (Endo-MT) of trabecular meshwork (TM) cells is known to be associated

187 We have previously shown that trabecular meshwork (TM) cells might detect aqueous humo

188 ying pathological mechanisms of glaucomatous trabecular meshwork (TM) damage and elevation of intraoc

189 of primary open angle glaucoma and is due to trabecular meshwork (TM) damage, which leads to impaired

190 Aberrant remodeling of trabecular meshwork (TM) extracellular matrix (ECM) may

191 GC-induced myocilin expression in the trabecular meshwork (TM) has been suggested to play an i

192 To determine if trabecular meshwork (TM) height differs between primary

193 Trabecular meshwork (TM) is a highly mechanosensitive ti

194 The trabecular meshwork (TM) is a specialized ocular tissue,

195 The trabecular meshwork (TM) is an ocular tissue that mainta

196 f extracellular matrix (ECM) proteins in the trabecular meshwork (TM) is associated with TM dysfuncti

197 ctional changes in the unlasered portions of trabecular meshwork (TM) of laser-treated primate eyes a

198 Given that the trabecular meshwork (TM) provides most of aqueous humor

199 The trabecular meshwork (TM) tissue controls drainage of aqu

200 Trabecular meshwork (TM) tissue in the eye is under cons

201 rrogated the biomechanical properties of the trabecular meshwork (TM), including the inner wall of Sc

202 d fibroblast-like) from cells located in the trabecular meshwork (TM), the primary structural compone

203 to endoplasmic reticulum (ER) stress in the trabecular meshwork (TM), the tissue that regulates IOP.

204 liary body (CB) and its drainage through the trabecular meshwork (TM).

205 50]) and angle opening (all 4 quadrants with trabecular meshwork [TM] visible on gonioscopy after LPI

206 ulated to adversely affect the health of the trabecular meshwork and outflow resistance.

207 sufficient aqueous humor outflow through the trabecular meshwork and Schlemm's canal (SC) is the most

208 comatous subjects were analyzed to determine trabecular meshwork anteroposterior length and 3 anterio

209 d a significant positive association between trabecular meshwork anteroposterior length and all anter

210 d a significant positive association between trabecular meshwork anteroposterior length and all anter

211 ere used to evaluate the association between trabecular meshwork anteroposterior length and anterior

212 Mean trabecular meshwork anteroposterior length was 824.86 +/

213 ior chamber angle is associated with greater trabecular meshwork anteroposterior length.

214 In this clinic-based population, trabecular meshwork height is shorter in PACG patients c

215 defined as inability to visualize pigmented trabecular meshwork in 3 or more quadrants.

216 Oxidative stress/damage to the trabecular meshwork in such post-vitrectomy cases may co

217 ression is enriched in the smooth muscle and trabecular meshwork in the eye.

218 ance in the aqueous drainage tract distal to trabecular meshwork is potentially an important determin

219 h more advanced glaucoma suggesting that the trabecular meshwork is the primary impediment to outflow

220 mily members (8/10) showed moderate to heavy trabecular meshwork pigmentation and either Krukenberg s

221 iris transillumination defects, and/or heavy trabecular meshwork pigmentation.

222 he juxtacanalicular connective tissue of the trabecular meshwork together with inner wall endothelium

223 eyes were classified as narrow if posterior trabecular meshwork was not visible and open if the angl

224 The trabecular meshwork's (TM) physiological role is to main

225 ced damage to the anterior chamber angle and trabecular meshwork, and reduced postoperative use of st

226 of severely hypomorphic Schlemm's canal and trabecular meshwork, as well as elevated IOP, demonstrat

227 s also one of the most abundant genes in the trabecular meshwork, the eye tissue responsible for main

228 ounger patients may be more localized to the trabecular meshwork, therefore making GATT a particularl

229 low structures, Schlemm's canal (SC) and the trabecular meshwork.

230 of caveolae in the Schlemm's canal (SC) and trabecular meshwork.

231 n of SLT was performed to 360 degrees of the trabecular meshwork.

232 d-mediated outflow obstruction distal to the trabecular meshwork.

233 Trabecular metaphyseal and cortical midshaft morphometri

234 stent (Ivantis, Inc, Irvine, CA) or 2 iStent Trabecular Micro Bypass devices (Glaukos Inc, San Clemen

235 IL-27 treatment prevented the loss of trabecular micro-architecture and preserved cortical bon

236 We assessed outflow enhancement by trabecular micro-bypass (TMB) implantation or by ab inte

237 ts were carried out comparing 1 and 2 iStent Trabecular Micro-Bypass Stents and 2 iStent Inject impla

238 s of an ab interno implanted (iStent inject) Trabecular Micro-Bypass System (Glaukos Corporation, San

239 n are novel techniques that directly measure trabecular microarchitecture and mechanical properties o

240 Despite persistent decrease in BMD, trabecular microarchitecture and tissue quality remain n

241 e, which is mainly related to changes in the trabecular microstructure.

242 ation, we find a causal relationship between trabecular morphology and risk of cardiovascular disease

243 of trabeculae using the fractal analysis of trabecular morphology in 18,096 participants of the UK B

244 from human participants, we demonstrate that trabecular morphology is an important determinant of car

245 of the palm in Australopithecus sediba have trabecular morphology most like orangutans and consisten

246 mal formation of the ventricular walls.Fetal trabecular muscles in the heart undergo a poorly describ

247 heart development as it functions to induce trabecular myocardium, the first heart tissue to form, a

248 Failure of trabecular myocytes to undergo appropriate cell cycle wi

249 LT recipients had lower trabecular number (-9.7%; P = .004) and lower trabecular

250 ue volume (BV/TV), trabecular thickness, and trabecular number (all P < 0.01) as well as an increase

251 Indeed, women with an Acid-D had higher trabecular number (P = 0.010 vs. Alk-D; P = 0.001 vs. Ne

252 histomorphometry showed consistently reduced trabecular number and connectivity.

253 ch showed lower bone volume/total volume and trabecular number in Col6alpha2-KO mice compared with WT

254 This effect is due to a higher trabecular number in these mice.

255 becular thickness, trabecular separation and trabecular number of femur and lumbar, serum osteocalcin

256 including cortical volumetric bone density, trabecular number, and trabecular thickness at the dista

257 over, the newly formed bone contained higher trabecular number, connective density, and bone mineral

258 WT mice had reduced trabecular thickness and trabecular number.

259 amax) was proportionate to a 38% increase in trabecular number.

260 ower bone resorption with either maintained (trabecular) or higher (cortical) bone formation as compa

261 categorized into surgeries that increase the trabecular outflow [Trabectome, iStent (first and second

262 We measured cortical and trabecular parameters from micro-computed tomography sca

263 d across species or how and when the present trabecular patterns emerged over the course of human evo

264 , which induces a thinning of trabeculae and trabecular perforations.

265 rimordium and subsequent persistence of deep trabecular recesses in the myocardial wall.

266 ing from the subchondral region into the mid trabecular region.

267 ITS revealed predominant change in trabecular rods, and EDX confirmed less mineralization.

268 tissue-level mechanics were compared across trabecular sections from the proximal femora of three gr

269 ber (all P < 0.01) as well as an increase in trabecular separation (P < 0.001).

270 and Headaches, Inverse Difference Moment and Trabecular Separation accurately diagnose early stages o

271 mineral density (BMD), trabecular thickness, trabecular separation and trabecular number of femur and

272 Group EP presented greater bone porosity, trabecular separation, and connective tissue attachment

273 icant change in bone volume/total volume and trabecular spacing, but it simultaneously damaged the bo

274 and trabecular thickness, with a decrease in trabecular spacing.

275 l-mediated RV stiffness was determined in RV trabecular strips.

276 The trabecular structure of a proximal femur (StW 522) attri

277 analysis to discriminate the changes in the trabecular structure of interdental bone between individ

278 ith no differences in femur length, cortical/trabecular structure or mineral density, or mechanical p

279 expressed in adult tibiae, including at the trabecular surfaces and in cortical osteocytes, epiphyse

280 I, UL and TBI-UL groups showed reduced tibia trabecular (Tb) bone mass by 15%, 70%, and 75%, respecti

281 These findings suggest that trabecular texture analysis might contribute information

282 integrity by using computed tomographic (CT) trabecular texture analysis of the lumbar spine in patie

283 Trabecular texture analysis was performed by using softw

284 performed to determine associations between trabecular texture and body composition.

285 to determine body composition predictors of trabecular texture.

286 rabecular number (-9.7%; P = .004) and lower trabecular thickness (-8.1%; P = .025).

287 covered the bone mineral density, -volume, -trabecular thickness and -separation.

288 d group, the PTH-treated WT mice had reduced trabecular thickness and trabecular number.

289 umetric bone density, trabecular number, and trabecular thickness at the distal radius and a model in

290 Trabecular thickness was significantly associated with o

291 At 24 months, bone formation rate, trabecular thickness, and bone volume were higher in the

292 crease in bone volume/tissue volume (BV/TV), trabecular thickness, and trabecular number (all P < 0.0

293 ight/obese hip OA patients exhibited reduced trabecular thickness, increased bone surface/bone volume

294 ing weight loss, bone mineral density (BMD), trabecular thickness, trabecular separation and trabecul

295 sed bone volume fraction, tissue density and trabecular thickness, with a decrease in trabecular spac

296 o previous reports, the present study showed trabecular thinning, higher numbers of apoptotic osteocy

297 d between GLIS3 and CLPTM1L with hyalinizing trabecular tumors (HTTs) and fibrolamellar hepatocellula

298 ted participants had lower mean z scores for trabecular volumetric BMD (-0.85), cortical volumetric B

299 Trabecular volumetric BMD was lower in coinfected than i

300 1.58 (1.45-1.72) per 1 SD decrease in radius trabecular volumetric bone density.