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1 c induction and mechanical ventilation via a tracheal tube.
2 ration values necessitating uncapping of the tracheal tube.
3 cent of tracheal intubation were with cuffed tracheal tubes.
4 of the lumen matrix during maturation of the tracheal tubes.
5 ide full barrier function and produce normal tracheal tubes.
6 o asphyxiation brought on by fluid-congested tracheal tubes.
7 nd causes expansion forces that elongate the tracheal tubes.
8 sponding to the membrane-driven expansion of tracheal tubes.
9 e pressure ventilation by an endotracheal or tracheal tube, a PaO2:FiO2 less than 200 mm Hg with at l
10 ed fifty-two adult patients intubated with a tracheal tube allowing subglottic secretion suctioning w
11 ow and pressure measured at the inlet of the tracheal tube and expressed as resistance (Rrs) and reac
14 of mechanical ventilation, the prevalence of tracheal tubes, and behavioral "learned nonuse" may all
18 n produce some ventilation with or without a tracheal tube, because the straight upper airways of ani
21 Second, Yorkie controls water tightness of tracheal tubes by transcriptional regulation of the delt
22 xchange respiratory gases in their system of tracheal tubes by using either diffusion or changes in i
25 ression appeared in late-stage embryos after tracheal tube formation, with individual PPK genes showi
28 osis included transplant stricture (n = 13), tracheal tube injury (n = 10), inflammation (n = 6), tra
31 the remaining 13 patients, the magnitude of tracheal tube leak increased by > or = 10% after delayed
34 thway in promoting cell intercalation during tracheal tube morphogenesis in Drosophila embryogenesis,
35 Here we show that three genes required for tracheal tube morphogenesis in Drosophila melanogaster e
36 e underlying mechanisms using the developing tracheal tube network of Drosophila indirect flight musc
40 r, those patients who failed to tolerate the tracheal tube occlusion protocol had clinically importan
41 was undertaken to test the hypothesis that a tracheal tube occlusion protocol predicts clinically imp
43 or without cardiac arrest, with or without a tracheal tube, showed essentially no ventilation by ster
44 o not disrupt several processes required for tracheal tube size control, including septate junction f
46 al extracellular matrix proteins involved in tracheal tube size control: Crumbs, Uninflatable, Kune-K
49 known septate junctions genes cause the same tracheal tube-size defects as ATPalpha and nrv2 mutation
50 sophila Dorsal Air Sac Primordium (ASP) is a tracheal tube that grows toward Branchless FGF-expressin
51 pgant35A) are recessive lethal and result in tracheal tubes that are irregular in diameter and morpho
52 larval development, which generate the adult tracheal tubes, the spiracle and the epidermis surroundi
53 sinuous have previously been shown to cause tracheal tubes to be elongated and have diameter increas
54 bunits of the Na+/K+ ATPase cause Drosophila tracheal tubes to have increased lengths and expanded di
55 who developed respiratory distress when the tracheal tube was occluded were deemed to have failed th