ライフサイエンスコーパス: trans-retinoic acid

1 metabolism by HSCs and was dependent on all-trans retinoic acid.

2 ncubated with rapamycin or rapamycin and all-trans retinoic acid.

3 is rapidly down-regulated in response to all-trans retinoic acid.

4 ent of acute promyelocytic leukemia with all-trans retinoic acid.

5 was rescued by treatment with exogenous all-trans retinoic acid.

6 this is counteracted in the presence of all-trans retinoic acid.

7 The top hit identified in the screen was all-trans-retinoic acid.

8 e, the immediate precursor for bioactive all-trans-retinoic acid.

9 The addition of all-trans retinoic acid, a commonly used agent for the treat

10 Accordingly, all-trans-retinoic acid, alone or together with gemfibrozil,

11 Treatment of APL blasts with all-trans retinoic acid also did not result in immediate DNA

12 portant therapeutic agents; for example, all-trans retinoic acid, an activating ligand for retinoic a

13 tients had significantly lower levels of all-trans retinoic acid and 13-cis retinoic acid than contro

14 ans retinol), and its geometric isomers, all-trans retinoic acid and 9-cis retinoic acid, in a focal

15 Upon treating APL with all-trans retinoic acid and achieving complete remission, th

16 ents with t(15;17) treated with extended all-trans retinoic acid and anthracycline-based chemotherapy

17 ents with APL homogeneously treated with all-trans retinoic acid and anthracycline-based chemotherapy

18 ns high despite the wide availability of all-trans retinoic acid and appears significantly higher tha

19 Thanks to modern treatment with all-trans retinoic acid and chemotherapy, acute promyelocyti

20 cells from these mice were responsive to all-trans retinoic acid and had virtually no differences in

21 All patients received All-trans retinoic acid and idarubicin according to the GIME

22 We hypothesize that topical all-trans retinoic acid and interferon alfa-2b may act syner

23 ates of apoptosis in FBAE cells than did all-trans retinoic acid and the control (P = 0.004).

24 cute promyelocytic leukemia treated with all-trans retinoic acid and/or arsenic trioxide.

25 ocytic leukemia (APL) who were receiving all-trans-retinoic acid and anthracycline-based chemotherapy

26 ulated during differentiation induced by all-trans-retinoic acid and brain-derived neurotrophic facto

27 so increased Bcl2a1 expression, although all-trans-retinoic acid and ligands for other RXR partner re

28 rapid stimulation of dendritic growth by all-trans-retinoic acid and reveal that the ligand-dependent

29 h other targeted therapies such as ATRA (all trans retinoic acid) and arsenic trioxide.

30 Finally, vitamin A (all-trans retinoic acid) and vitamin D (1,25-dihydroxyvitami

31 Novel mutual prodrugs (MPs) of ATRA (all- trans-retinoic acid) and HDIs (histone deacetylase inhib

32 Retinoids (i.e., vitamin A, all-trans retinoic acid, and related signaling molecules) in

33 oncentration-dependent, 2) selective for all-trans-retinoic acid, and 3) requires the presence of apo

34 Treatment with all-trans retinoic acid antagonizes stress-induced activatio

35 In nude mice models, combination of all-trans retinoic acid (ATRA) and AEG-1 knockdown synergist

36 a model for oncogene-targeted therapies: all-trans retinoic acid (ATRA) and arsenic both target and d

37 We examined whether combining all-trans retinoic acid (ATRA) and arsenic trioxide (ATO) mi

38 down of NPM1 also sensitized OCI-AML3 to all-trans retinoic acid (ATRA) and cytarabine.

39 We used all-trans retinoic acid (ATRA) and histone deacetylase (HDAC

40 g data indicates that retinoids, such as all trans retinoic acid (ATRA) and its precursor all trans r

41 ave identified a novel pathway involving all-trans retinoic acid (ATRA) and its receptor (RARgamma) s

42 Here we show that all-trans retinoic acid (ATRA) and other agonists of the ret

43 tment until the recent identification of all-trans retinoic acid (ATRA) as a Pin1 inhibitor.

44 The identification of all-trans retinoic acid (ATRA) as a potent Pin1 inhibitor pr

45 om 5 large clinical trials that included all-trans retinoic acid (ATRA) as part of induction, we asse

46 We show that treatment with all-trans retinoic acid (ATRA) at clinically achievable dose

47 ematologist because early institution of all-trans retinoic acid (ATRA) at the first suspicion of the

48 ct was reversed for K1 and K10 by adding all-trans retinoic acid (ATRA) but increased for K2 in the a

49 ment of PML-RARalpha leukemic cells with all-trans retinoic acid (ATRA) causes them to differentiate

50 oblastoma cells following treatment with all-trans retinoic acid (ATRA) compared to controls.

51 All patients received all-trans retinoic acid (ATRA) during induction, each consol

52 The treatment of patients with all-trans retinoic acid (ATRA) effectively ameliorates the d

53 demonstrated that treatment of AML with all-trans retinoic acid (ATRA) enhanced FRbeta expression, r

54 t differentiate along this lineage after all-trans retinoic acid (ATRA) exposure.

55 0(9)/L (or for a maximum of 28 days) and all-trans retinoic acid (ATRA) for 90 days.

56 Recent studies have shown that all-trans retinoic acid (ATRA) had a potent activity in elim

57 All-trans retinoic acid (ATRA) has been used in several clin

58 ent of acute promyelocytic patients with all-trans retinoic acid (ATRA) has improved the survival of

59 cancer stem cells through treatment with all-trans retinoic acid (ATRA) have yielded limited success,

60 nds to enhance the biological effects of all-trans retinoic acid (ATRA) in a retinoid-responsive neur

61 inical outcomes of patients treated with all-trans retinoic acid (ATRA) in combination with anthracyc

62 ion relieved by pharmacological doses of all-trans retinoic acid (atRA) in culture and in vivo.

63 tone deacetylase inhibitor valproate and all-trans retinoic acid (ATRA) in treatment-naive elderly pa

64 Treatment with all-trans retinoic acid (ATRA) increased both the expression

65 All-trans retinoic acid (ATRA) increased IRF4 expression, re

66 The vitamin A metabolite all-trans retinoic acid (ATRA) induces a gut-homing phenotyp

67 All-trans retinoic acid (ATRA) induces clinical remission in

68 All-trans retinoic acid (ATRA) induces differentiation in va

69 provides the first evidence showing that all-trans retinoic acid (ATRA) induces the interaction and c

70 Additionally, we reveal that all trans retinoic acid (ATRA) induces VCP expression, creat

71 be dissociated by pharmacologic doses of all trans retinoic acid (ATRA) inducing differentiation and

72 We determined the mechanism by which all-trans retinoic acid (ATRA) inhibits experimental autoimm

73 In mammals, all-trans retinoic acid (ATRA) is instrumental to spermatoge

74 All trans retinoic acid (atRA) is one of the most potent the

75 All-trans retinoic acid (ATRA) neutralizes the differentiati

76 RA, 9-cis retinoic acid (9-cis RA), and all-trans retinoic acid (ATRA) on cell proliferation, apopto

77 Pdx-1-Cre (KPC) mice and the effects of all-trans retinoic acid (ATRA) on these processes.

78 , we evaluated the impact of exposure to all-trans retinoic acid (ATRA) on wild-type NK and CD38KO NK

79 Comparison of monocytes stimulated with all-trans retinoic acid (ATRA) or 1,25-dihydroxyvitamin D3 (

80 ZF/RAR alpha leukemia, was responsive to all-trans retinoic acid (ATRA) or As2O3 treatments.

81 promyelocytic leukemia by treatment with all-trans retinoic acid (ATRA) plus arsenic trioxide (ATO),

82 We recently reported that all-trans retinoic acid (atRA) rapidly (within minutes) acti

83 This study found that all-trans retinoic acid (atRA) reverses the effects of HIV-1

84 All-trans retinoic acid (atRA) reverses the HIV-induced podo

85 Treatment of monocytes with all-trans retinoic acid (ATRA) significantly decreased basel

86 the eyeballs are a source of diffusible all-trans retinoic acid (ATRA) that allow their targeting by

87 We investigated the benefit of adding all-trans retinoic acid (ATRA) to chemotherapy for younger p

88 We used all-trans retinoic acid (ATRA) to differentiate MDSCs in mic

89 Dendritic cells (DCs) use all-trans retinoic acid (ATRA) to promote characteristic int

90 nitiated from the nongenomic activity of all-trans retinoic acid (atRA) to stimulate complex formatio

91 While all-trans retinoic acid (ATRA) treatment in acute promyelocy

92 We have also found that all-trans retinoic acid (ATRA) treatment increased AGAP2 pro

93 Interestingly, all-trans retinoic acid (ATRA) treatment induced potent cell

94 In acute promyelocytic leukemia (APL), all-trans retinoic acid (ATRA) treatment induces granulocyti

95 All-trans retinoic acid (ATRA) upregulated TRAIL-R1 expressi

96 All-trans retinoic acid (ATRA) was negatively connected with

97 The effects of all-trans retinoic acid (ATRA) were studied in LSL-KrasG12D/

98 vulnerable to a novel strategy combining all-trans retinoic acid (ATRA) with arsenic trioxide (ATO).

99 All-trans retinoic acid (ATRA) with chemotherapy is the stan

100 All-trans retinoic acid (ATRA), a derivative of vitamin A, i

101 All-trans retinoic acid (ATRA), a natural retinoid, arrests

102 All-trans retinoic acid (ATRA), a potent derivative of vitam

103 as to evaluate the therapeutic effect of all-trans retinoic acid (ATRA), an active metabolite of vita

104 th daunorubicin, cytarabine (Ara-C), and all-trans retinoic acid (ATRA), and complete remission was d

105 response to its natural agonist ligand, all-trans retinoic acid (atRA), and is repressed by SHP.

106 cted to examine the interactive roles of all-trans retinoic acid (ATRA), Ets-1, Sp1, and histone acet

107 Its biologically active metabolite, all-trans retinoic acid (ATRA), exhibits a potent antiviral

108 he short half-life vitamin A metabolite, all-trans retinoic acid (atRA), in an amount sufficient to s

109 h phosphate-buffered saline (PBS), UDCA, all-trans retinoic acid (atRA), or UDCA and atRA by gavage.

110 esent standard of care, chemotherapy and all-trans retinoic acid (ATRA), results in a high proportion

111 All-trans retinoic acid (atRA), the active derivative of vit

112 In response to challenge with all-trans retinoic acid (atRA), the balance of daughter cell

113 oter and is stimulated by treatment with all-trans retinoic acid (ATRA), the biologically active form

114 or metabolism into an active metabolite, all-trans retinoic acid (atRA), where atRA is essential to c

115 ource of the immunoregulatory metabolite all-trans retinoic acid (ATRA), which may contribute to the

116 anism-based screening, here we find that all-trans retinoic acid (ATRA)--a therapy for acute promyelo

117 Despite the great success of all-trans retinoic acid (ATRA)-based therapy, which results

118 his study, we present an effective model All-Trans Retinoic Acid (ATRA)-induced differentiation of HL

119 s of NTAL were associated with increased all-trans retinoic acid (ATRA)-induced differentiation, gene

120 pleted of Ajuba are highly sensitized to all-trans retinoic acid (atRA)-induced transcription and dif

121 Mutations in the all-trans retinoic acid (ATRA)-targeted ligand binding domai

122 bstantially in the marrow and spleens of all-trans retinoic acid (ATRA)-treated C57BL6 mice, while ly

123 estigated the gene expression profile of all-trans retinoic acid (ATRA)-treated human CD4(+) T cells.

124 nditions, TNIP1 expression is induced by all trans retinoic acid (ATRA).

125 is directly and potently upregulated by all-trans retinoic acid (atRA).

126 a (SK-N-SH) cells to neuronal cells with all-trans retinoic acid (ATRA).

127 d insensitive response to this effect of all-trans retinoic acid (ATRA).

128 activity and may restore sensitivity to all-trans retinoic acid (ATRA).

129 e up-regulation in the leukemic cells by all-trans retinoic acid (ATRA).

130 , a phenotype reversed by treatment with all trans retinoic acid (ATRA).

131 itivity to another known limb teratogen, all-trans retinoic acid (atRA).

132 g (low-dose) or 1,000 microg (high-dose) all-trans retinoic acid (ATRA)/kg body weight in corn oil or

133 PURPOSE Event-free survival following all-trans-retinoic acid (ATRA) -based therapy for acute prom

134 06 trial showed that the combination of all- trans-retinoic acid (ATRA) and arsenic trioxide (ATO) is

135 romyelocytic leukemia (APL) treated with all-trans-retinoic acid (ATRA) and arsenic trioxide (ATO) wi

136 RARa-associated APL is sensitive to both all-trans-retinoic acid (ATRA) and arsenic trioxide (ATO), a

137 The use of all-trans-retinoic acid (ATRA) and arsenic trioxide, both of

138 e P450 CYP26 enzymes are responsible for all-trans-retinoic acid (atRA) clearance.

139 All-trans-retinoic acid (ATRA) combined with chemotherapy, t

140 All-trans-retinoic acid (ATRA) did not stimulate osteoclasto

141 All-trans-retinoic acid (atRA) has been implicated in the lo

142 All-trans-retinoic acid (ATRA) has been shown to act physiol

143 All-trans-retinoic acid (ATRA) has been shown to arrest the

144 L), the use of the differentiation agent all-trans-retinoic acid (ATRA) has revolutionized the therap

145 The all-trans-retinoic acid (atRA) hydroxylase Cyp26a1 is essent

146 Treatment of APL with all-trans-retinoic acid (ATRA) induces disease remission by

147 All-trans-retinoic acid (ATRA) induces growth arrest of many

148 Although all-trans-retinoic acid (atRA) is a key regulator of intesti

149 All-trans-retinoic acid (ATRA) is a natural compound propose

150 All-trans-retinoic acid (ATRA) is an active vitamin A deriva

151 All-trans-retinoic acid (atRA) is an important morphogen inv

152 All-trans-retinoic acid (atRA) is the active metabolite of v

153 All-trans-retinoic acid (atRA) is the active metabolite of v

154 The all-trans-retinoic acid (atRA) isomer, 9-cis-retinoic acid (

155 family is believed to be responsible for all-trans-retinoic acid (atRA) metabolism and elimination in

156 n2 KO mice exhibited a blunted effect of all-trans-retinoic acid (ATRA) on body weight and fat mass,

157 The combination of all-trans-retinoic acid (ATRA) plus arsenic trioxide (ATO) h

158 mpared efficacy and toxicity of standard all-trans-retinoic acid (ATRA) plus chemotherapy versus ATRA

159 We investigated the actions of all-trans-retinoic acid (atRA) signaling in pancreatic beta-

160 All-trans-retinoic acid (atRA) stimulates neurogenesis, dend

161 All-trans-retinoic acid (atRA) supports embryonic developmen

162 ned only about a 0.6 nm concentration of all-trans-retinoic acid (atRA) that is the most active natur

163 cessfully treated with therapy utilizing all-trans-retinoic acid (ATRA) to differentiate leukemic bla

164 leukemia patients and cell lines during all-trans-retinoic acid (ATRA) treatment by using a miRNA mi

165 All-trans-retinoic acid (atRA), an autacoid derived from ret

166 fter recognition of the effectiveness of all-trans-retinoic acid (ATRA), anthracycline-based chemothe

167 Vitamin A derivatives, including all-trans-retinoic acid (ATRA), have a well-established role

168 defect can be overcome by treatment with all-trans-retinoic acid (ATRA), leading to complete clinical

169 All-trans-retinoic acid (ATRA), retinol, retinalaldehyde, an

170 All-trans-retinoic acid (atRA), the active metabolite of vit

171 All-trans-retinoic acid (atRA), the active metabolite of vit

172 All-trans-retinoic acid (atRA), the active metabolite of vit

173 All-trans-retinoic acid (atRA), the major active metabolite

174 Subsequent oxidation produces all-trans-retinoic acid (ATRA), which functions as a ligand

175 with fibrillar fibronectin and show that all trans-retinoic acid (ATRA), which induces PSC quiescence

176 different statins were found to enhance all-trans-retinoic acid (ATRA)-dependent differentiation of

177 rmation and leukemogenesis, and inhibits all-trans-retinoic acid (ATRA)-induced AML cell differentiat

178 RAR) target gene expression and inhibits all-trans-retinoic acid (ATRA)-induced myeloid differentiati

179 site in the OLFM4 promoter and mediates all-trans-retinoic acid (ATRA)-induced transactivation of th

180 veral developmental genes, including the all-trans-retinoic acid (ATRA)-responsive ones, through its

181 the formation of the essential morphogen all-trans-retinoic acid (ATRA).

182 system require the vitamin A metabolite all-trans-retinoic acid (atRA).

183 on of myeloid cell differentiation using all-trans-retinoic acid (ATRA).

184 targeting pancreatic stellate cells with all-trans-retinoic-acid (ATRA) reprograms pancreatic stroma

185 Vitamin A (all trans retinoic acid, ATRA) treated leukemic cells had in

186 the outcome of 155 patients treated with all-trans retinoic acid-based therapy on 3 clinical trials,

187 -apo-8'-carotenal, 13-cis-retinoic acid, all-trans-retinoic acid, beta-carotene-5,6-epoxide, all-tran

188 Rdh10 catalyzes the first step of all-trans-retinoic acid biogenesis physiologically, conversi

189 nal dehydrogenases (critical enzymes for all-trans retinoic acid biosynthesis) and were significantly

190 efficiently (V(m)/K(m)) in a pathway of all-trans-retinoic acid biosynthesis in cells and recognizes

191 When differentiation was induced by all-trans retinoic acid, CEACAM6 expression strongly correla

192 romyelocytic leukemia model treated with all-trans retinoic acid combined with the histone-deacetylas

193 o neutrophils and in vivo treatment with all-trans retinoic acid decreased plasminogen binding to acu

194 blasts in response to 5 toxic compounds (all-trans retinoic acid, dexamethasone, doxorubicin, 5'-fluo

195 ly demonstrated using both ibuprofen and all-trans retinoic acid; drugs with anti-inflammatory and an

196 tes, and knocking down RARalpha prevents all-trans-retinoic acid effects on dendritic growth.

197 cycles with idarubicin, cytarabine, and all-trans retinoic acid either with VPA or without (STANDARD

198 cute promyelocytic leukemia (APL) in the all-trans retinoic acid era.

199 or vision as well as the biosynthesis of all-trans-retinoic acid for differentiation and development.

200 The major active retinoid, all-trans retinoic acid, has long been recognized as critica

201 granulocyte colony-stimulating factor or all-trans-retinoic-acid have shown improvement in decreasing

202 of all-trans-retinol for biosynthesis of all-trans-retinoic acid, however, initial assays suggested t

203 Consistent with the role of all-trans retinoic acid in inducing gut-homing T cells, OT c

204 d that response to targeted therapy with all-trans retinoic acid in vivo was dependent on NB integrit

205 ce that vitamin A uptake is regulated by all-trans-retinoic acid in non-ocular tissues of mice.

206 oduce visual chromophore in the eyes and all-trans-retinoic acid in other tissues.

207 All-trans retinoic acid increased CD38 levels and decreased

208 macrophage differentiation, but blocked all-trans-retinoic acid induced granulocytic differentiation

209 auses acute myeloid leukemia and impairs all-trans retinoic acid-induced granulocytic differentiation

210 n-induced mortality (GRIM)-19, as an IFN/all-trans retinoic acid-induced growth suppressor.

211 ound that the major vitamin A metabolite all-trans-retinoic acid induces histone acetylation at the F

212 AR) alpha, alone and in conjunction with all-trans-retinoic acid is capable of enhancing TFEB in brai

213 Administration of all-trans-retinoic acid led to a significant decrease in the

214 se retinoid (retinol, retinyl ester, and all-trans-retinoic acid) levels were observed.

215 All-trans-retinoic acid may be an important molecular signal

216 yeloid leukemias respond dramatically to all-trans retinoic acid mediated differentiation therapy, wh

217 Chiral pentamers of all-trans-retinoic acid molecules have been prepared on Au(1

218 Temporary discontinuation of all-trans retinoic acid or arsenic trioxide is indicated onl

219 rigger PML-RARalpha degradation, such as all-trans retinoic acid or arsenic trioxide, restore nuclear

220 All-trans retinoic acid or related compounds may also play a

221 randomization were randomly assigned to all-trans-retinoic acid or not.

222 of SH-SY5Y human neuroblastoma cells by all-trans retinoic acid, or oxidative stress induced by mito

223 differentiation following treatment with all-trans retinoic acid (P = 3.1 x 10(-13) to 2.4 x 10(-30))

224 Treg)-polarizing molecules TGF-beta1 and all-trans retinoic acid, particularly during states of infla

225 able only in the context of conventional all-trans retinoic acid plus chemotherapy regimens.

226 ll as IL10 and the vitamin A metabolite; all-trans retinoic acid (RA [at-RA]) has been found to enhan

227 genes are transcriptionally activated by all-trans retinoic acid (RA) and display increased levels of

228 al cord phenotype using a combination of all-trans retinoic acid (RA) and epidermal growth factor (EG

229 All-trans Retinoic acid (RA) and its derivatives are potent

230 (CYP26B1) regulates the concentration of all-trans retinoic acid (RA) and plays a key role in germ ce

231 Reduced generation of all-trans retinoic acid (RA) by CD103(+) intestinal dendriti

232 We demonstrate that all-trans retinoic acid (RA) induces FoxP3(+) adaptive T reg

233 odel human myeloid leukemia cells, where all-trans retinoic acid (RA) induces granulocytic differenti

234 We have previously shown that all-trans retinoic acid (RA) mediates activity blockade-indu

235 The major vitamin A metabolite all-trans retinoic acid (RA) not only enforces the generatio

236 However, the effects of all-trans retinoic acid (RA) on cellular expression of VEGF

237 cation of active vitamin D3 (VD3) and/or all-trans retinoic acid (RA) on wild-type mouse skin induces

238 All-trans retinoic acid (RA) plays crucial roles in embryoge

239 hanisms whereby the vitamin A metabolite all-trans retinoic acid (RA) promotes the formation of plasm

240 All-trans retinoic acid (RA) stimulates cellular proliferati

241 the ability of the vitamin A metabolite all-trans retinoic acid (RA) to restore the defective immune

242 Here, we report that all-trans retinoic acid (RA), a well-known developmental mor

243 Vitamin A and its active metabolite, all-trans retinoic acid (RA), regulate the antibody response

244 oma (EC) is relieved upon treatment with all-trans retinoic acid (RA), resulting in enhanced p53 tran

245 Hox gene expression is activated by all-trans retinoic acid (RA), through binding to retinoic ac

246 ity, in part by stimulating synthesis of all-trans retinoic acid (RA), which in turn increases AMPA r

247 tic cells (DC) metabolize vitamin A into all-trans retinoic acid (RA), which is required to induce ly

248 HtrA2 and augments cell death in an IFN/all-trans retinoic acid (RA)-dependent manner.

249 In all-trans retinoic acid (RA)-induced differentiation of acut

250 e or presence of the RAR-specific ligand all trans retinoic acid (RA).

251 lioblastoma stem-like cells (GBM-SCs) to all-trans retinoic acid (RA).

252 oblastoma cells following treatment with all-trans retinoic acid (RA).

253 ion of retinoic acid receptor (RAR) with all-trans-retinoic acid (RA) ameliorates glucose intolerance

254 e P450 enzyme Cyp26a1, which metabolizes all-trans-retinoic acid (RA) and thereby reduces RA levels,

255 Many biological activities of all-trans-retinoic acid (RA) are mediated by the ligand-acti

256 APL cell treatment with all-trans-retinoic acid (RA) degrades the chimeric, dominant

257 tudy was to examine the possibility that all-trans-retinoic acid (RA) in the eye is a signal related

258 We have previously demonstrated that all-trans-retinoic acid (RA) induction of RIP140 constitutes

259 1, a cytochrome P450 enzyme, metabolizes all-trans-retinoic acid (RA) into polar metabolites, e.g. 4-

260 All-trans-retinoic acid (RA) is a vitamin A metabolite that

261 HBE) cells to evaluate stabilities after all-trans-retinoic acid (RA) or cycloheximide treatments.

262 The vitamin A metabolite all-trans-retinoic acid (RA) regulates multiple biological p

263 t not Tbx18 or NFATC1, is activated with all-trans-retinoic acid (RA) treatment of isolated chick EPD

264 We found that all-trans-retinoic acid (RA) treatment of mouse epiphyseal c

265 All-trans-retinoic acid (RA), a bioactive derivative of vita

266 We report herein that all-trans-retinoic acid (RA), an active metabolite of vitami

267 Vitamin A metabolite, all-trans-retinoic acid (RA), induces cell growth, different

268 tamin A (retinol/ROL) can be oxidized to all-trans-retinoic acid (RA), which plays a critical role in

269 DGL-alpha and DGL-beta may contribute to all-trans-retinoic acid (RA)-induced neurite outgrowth in ne

270 Whereas both are all-trans-retinoic acid (RA)-responsive in ES cells, the dow

271 itory and pro-differentiating effects of all-trans-retinoic acid (RA).

272 l epithelial (HCjE) cell line grown with all-trans-retinoic acid (RA).

273 A3 complexed with NAD(+) and the product all-trans retinoic acid (REA).

274 contrast, treatment of apc mutants with all-trans retinoic acid rescued retinal differentiation defe

275 gs should be considered for treatment of all-trans retinoic acid-resistant APL patients as well as th

276 uce cell death and induces expression of all-trans-retinoic acid-responsive genes that can be blocked

277 Short-term treatment of mice with all-trans retinoic acid resulted in increased PreB lymphopoi

278 All-trans-retinoic acid stimulates dendritic growth in hippo

279 n of the retinoic acid receptor agonist, all-trans-retinoic acid, suggesting that the ability of ALDH

280 All -trans-Retinoic acid ( t-RA) regulates leukocyte differen

281 erate both all-trans-retinal (t-RAL) and all-trans-retinoic acid (t-RA) from the precursor all-trans-

282 vascular endothelial growth factor, cis- and trans-retinoic acids, thalidomide, and tyrosine kinase i

283 All-trans retinoic acid therapy of acute promyelocytic leuke

284 d cell proliferation and synergized with all-trans retinoic acid to promote differentiation.

285 Topical treatment of human skin with all-trans retinoic acid (tRA) induces EGFR ligands heparin-b

286 All-trans retinoic acid (tRA) induces NIS gene expression an

287 All-trans-retinoic acid (tRA) markedly induces NIS activity

288 on of c-SRC as early as 15 min following all-trans-retinoic acid treatment in LA-N-5 cells.

289 All trans-retinoic acid treatment of A404 cells induced a st

290 arly forced maturation of MDSC by either all-trans-retinoic acid treatment or active immunoreceptor t

291 tilizing in-vitro experimental models of all-trans retinoic acid triggered myeloid leukemia different

292 oth promoters are partially regulated by all-trans retinoic acid via RARA and other RARs.

293 r CCR9 by cell sorting or culturing with all-trans retinoic acid, we measured chemotaxis, intracellul

294 Isotretinoin is a pro-drug for all-trans retinoic acid, which can induce long-term remissio

295 sociated dendritic cells (DC) synthesize all-trans retinoic acid, which is required for inducing gut-

296 tinaldehyde, the visual chromophore, and all-trans-retinoic acid, which is involved in the regulation

297 Furthermore, treatment of NB4 cells with all-trans-retinoic acid, which promotes PML-RARalpha degrada

298 istic effects of valproic acid (VPA) and all-trans retinoic acid with chemotherapy.

299 h idarubicin, cytarabine, etoposide, and all-trans-retinoic acid with or without GO.

300 retinoic acid receptor (RAR) compared to all-trans-retinoic acid, with select analogues also reducing