ライフサイエンスコーパス: transcription by

1 -copies of the MSR1 element can repress gene transcription by 50 to 115-fold; (3) the higher-expressi

2 owed that CRY inhibits CLOCK-BMAL1-activated transcription by a "blocking"-type mechanism and that CR

3 The G4 stabilized R-loop leads to increased transcription by a mechanism involving successive rounds

4 is positively autoregulated at the level of transcription by a mechanism that requires cAMP receptor

5 studies demonstrate that MUC1-C drives BMI1 transcription by a MYC-dependent mechanism in breast and

6 induced GTPase GBP1 occurred at the level of transcription by a partially MINCLE-dependent mechanism

7 Both proteins regulate transcription by a variety of local effects on transcrip

8 mouse O4(+) cells inhibited TH-induced mMog transcription by a yet unknown mechanism.

9 MLL-ENL and MLL-AF10 constitutively activate transcription by aberrantly inducing both AEP-dependent

10 ed chromatin, reconstituted in vitro, blocks transcription by abrogating core histone modifications b

11 expression, H3K4me3 facilitates histone gene transcription by acting as a boundary to restrict the sp

12 showed that CBFA2T3 inhibits RAR target gene transcription by acting at an early step to regulate his

13 active forms of Notch can directly stimulate transcription by activating enhancers, the effect of the

14 manganese superoxide dismutase (MnSOD) gene transcription by activating MKK4 via redox control of Cy

15 ltaneously, fumarate increases ferritin gene transcription by activating the NRF2 (nuclear factor [er

16 Enhanced AR gene transcription by ADT provides a prerequisite condition t

17 o acid determinant for maximal activation of transcription by AgrA and provides novel insights into s

18 tion, DNA packing and repair, as well as for transcription by allowing changes in DNA topology.

19 e transcript, working in cis, regulated HAS2 transcription by altering the chromatin structure around

20 in vitro, suggesting that IdeR induces bfrB transcription by antagonizing the repressor activity of

21 tion of the 25-kb Lockd locus reduced Cdkn1b transcription by approximately 70% in an erythroid cell

22 elayed by the apparent lack of resumption of transcription by arrested polymerases.

23 nal regulators RbpA and CarD act to modulate transcription by associating to the initiation complex a

24 se (RNAP) accomplishes multiple tasks during transcription by assuming different structural forms.

25 At the same time, chromatin impacts transcription by at least five distinct mechanisms: (i)

26 talk logic imparting differential control of transcription by AT1R, integrins, and calcium channels.

27 te that epigenetic upregulation of Npr1 gene transcription by ATRA and NaBu leads to attenuation of r

28 histone phosphorylation, in turn, regulates transcription by attenuating the effect of local arginin

29 tein produced in the very late phase of gene transcription by Autographa californica multiple nucleop

30 NA, which are purified after ribozyme-fusion transcription by automated size exclusion chromatography

31 interplay between these two elements during transcription by bacterial RNAP.

32 Tat, a small protein that facilitates viral transcription by binding an RNA structure (trans-activat

33 Bacterial 6S RNAs globally regulate transcription by binding RNA polymerase (RNAP) holoenzym

34 Under normal conditions, AmpR represses ampC transcription by binding the PG precursor UDP-N-acetylmu

35 BBR inhibits gene transcription by binding the TATA boxes in the transcrip

36 racts with its target mRNAs initially during transcription by binding through its N-terminal prion-li

37 Most activated response regulators regulate transcription by binding tightly to promoter DNA via a p

38 tailed in this review, estrogen can regulate transcription by binding to 2 nuclear receptors, ERalpha

39 series antimicrobials allosterically inhibit transcription by binding to a conserved alpha helix (bet

40 Last, we uncovered that FXR suppresses MMP7 transcription by binding to a negative FXR-responsive el

41 Intranuclear uPA modulates gene transcription by binding to a subset of transcription fa

42 ranscription factor complex to activate CD73 transcription by binding to an intronic enhancer.

43 assical biotype V. cholerae repressed vieSAB transcription by binding to its promoter.

44 RA activates transcription by binding to nuclear RA receptors (RARs)

45 DksA alters transcription by binding to RNA polymerase and allosteri

46 tors (TFs) regulate complex programs of gene transcription by binding to short DNA sequence motifs.

47 D), a master regulator for SMC-specific gene transcription by binding to SRF to form the MYOCD/SRF/CA

48 Furthermore, ISL2 regulated ANGPT2 transcription by binding to the ANGPT2 promoter.

49 we demonstrate that FOXO3a regulates its own transcription by binding to the conserved response eleme

50 SsrB also increases ugtL transcription by binding to the ugtL promoter region, wh

51 lity, in erythroid cells, Sp1 activates PIGM transcription by binding upstream of (but not to) the -2

52 SsrB promotes phoP transcription by binding upstream of the phoP promoter.

53 ption of BCL11A, a repressor of gamma-globin transcription, by binding to its enhancer and fostering

54 hromosomes while likely also repressing late transcription by blocking access to necessary regulatory

55 enhancer would be expected to repress early transcription by blocking access to part of the SP1 bind

56 We found that p38gamma increases EGFR transcription by c-Jun-mediated promoter binding and sti

57 e context, however, the reduction of COOLAIR transcription by cdkc;2 disrupts a COOLAIR-mediated repr

58 Ongoing transcription by cellular RNA polymerase II (Pol II) is

59 Repression of RNA pol III-dependent transcription by chemical inhibition or knockdown of BRF

60 protein involved in the initiation of viral transcription by cleaving capped mRNA bound to PB2.

61 transcription factors play key roles in HAEC transcription by co-binding enhancers associated with EC

62 pha was shown to directly regulate Mfn1 gene transcription by coactivating the estrogen-related recep

63 ase pair regulates the efficiency of initial transcription by controlling multiple steps including do

64 tion functions to indirectly maintain active transcription by counteracting H3K9 dimethylation and ge

65 CBP; an essential cofactor for activation of transcription by CREB) impair long-term synaptic plastic

66 cate a mechanism for selective regulation of transcription by CtBP and LSD1 involving their associati

67 nistically, Sirt6 inhibits Notch1 and Notch4 transcription by deacetylating histone H3K9.

68 nhibitor K7174 inhibits enzalutamide-induced transcription by decreasing binding of the GATA2/AR/Medi

69 organs depends on modular regulation of dsx transcription by dedicated cell type-specific enhancers.

70 inding factor ETS-1, and it represses miR-31 transcription by delivering the H3K4me3 demethylase JARI

71 sis further shows that FACT facilitates gene transcription by destabilizing the tetranucleosomal unit

72 NAP is thought to initiate the next round of transcription by detaching from DNA and rebinding a new

73 nsistent with a role for USP42 in regulating transcription by deubiquitylating histones.

74 Lastly, activation of Zp transcription by DFO and MLN-4924 mapped to its HRE.

75 y Klf5 as an activator of Dmp1 and Dspp gene transcriptions by different mechanisms and demonstrate t

76 ry protein CueR both represses and activates transcription by differentially modulating local DNA str

77 ining DNA binding domains generally regulate transcription by direct interaction with DNA.

78 Our results suggest that Xist silences transcription by directly interacting with SHARP, recrui

79 Mechanistically, PBX1 plays a dual role in transcription by directly repressing or activating genes

80 esence of CRY, nuclear entry of PER inhibits transcription by displacing CLOCK-BMAL1 from the promote

81 c transcription factors (GSTFs) control gene transcription by DNA binding and specific protein comple

82 s long been thought to influence the rate of transcription by DNA-dependent RNA polymerases, but the

83 sis, reveals a new mechanism regulating Edn1 transcription by Dot1a and HDAC2, and reinforces endothe

84 We find that repression of reaper transcription by Dpp is mediated by Schnurri.

85 lar mechanism of nutrient regulation of gene transcription by dynamic O-GlcNAcylation of TBP.

86 rmined that reduced Notch signaling mediated transcription by ectopic expression of dominant-negative

87 that Olfm2 mediates TGF-beta-induced SM gene transcription by empowering SRF binding to CArG box in S

88 ts greatest antiviral potency during reverse transcription by enhancement of G-to-C transversion muta

89 hat testosterone robustly represses hepcidin transcription by enhancing Egfr signaling in the liver a

90 histone methyltransferase to repress NEDD4L transcription by enhancing histone H3 lysine 27 trimethy

91 suggested that A20 knockdown increased STAT1 transcription by enhancing TBK1 activation and subsequen

92 we developed a chemical approach for halting transcription by Escherichia coli RNAP.

93 hal initiation, suggesting that Hir1 affects transcription by establishing transcriptional thresholds

94 chromatin environment is thought to inhibit transcription by excluding transcription factors and RNA

95 These factors switch on and off transcription by exerting dynamic control of DNA stereoc

96 GATA2 globally directs enzalutamide-induced transcription by facilitating AR, Mediator and Pol II lo

97 sults suggest that CTCF promotes HSV-1 lytic transcription by facilitating the elongation of RNA Pol

98 s usually achieved by exogenous induction of transcription by factors acting in the nucleus.

99 ve uncovered a mechanism for effective viral transcription by focal assembly of RNA polymerase II aro

100 hat AT-rich segments may signal the start of transcription by forming less stable nucleosomes.

101 d, glucocorticoid response element-dependent transcription by formoterol was displaced to the left by

102 Direct repression of anoctamin 1 (ANO1) gene transcription by Gli proteins.

103 We investigated regulation of ANO1 transcription by Gli.

104 wn regulation of Cln3 translation and Cln1,2 transcription by glucose is sufficient to explain the ex

105 a previously unrecognized regulation of gene transcription by GPCR-cAMP signaling through augmentatio

106 element mediated cooperative enhancement of transcription by GR and NF-kappaB that required the pres

107 RNAs modify chromatin structure and silence transcription by guiding Argonaute-containing complexes

108 ily of methyltransferases that controls gene transcription by H3K4 methylation (H3K4me).

109 Major features of transcription by human RNA polymerase II (Pol II) remain

110 reveal new insights into cell type-specific transcription by identifying novel transcription units,

111 es cerevisiae ATPase Mot1 globally regulates transcription by impacting the genomic distribution and

112 i and CDK7 inhibitor treatment abolished MYC transcription by impeding RNAPII loading without affecti

113 demonstrated that entinostat enhanced NKG2D transcription by increasing acetylation of Histones H3 a

114 the BH conformation, whereas CBRs deregulate transcription by increasing coupling between the BH and

115 First, increased NICD levels alter transcription by increasing duration rather than frequen

116 A6 has an essential role in ERalpha-mediated transcription by increasing enhancer-promoter interactio

117 gX regulation revealed that it can stimulate transcription by increasing the binding of RNA polymeras

118 SAB1 inactivates ABI5 transcription by increasing the level of histone H3K27me

119 s release of paused polymerase and activates transcription by increasing the number of transcribing p

120 AT can prevent TRAP-mediated termination of transcription by inducing dissociation of TRAP from the

121 and suggest a possibility that CAP activates transcription by inducing intermediate state, in additio

122 e and also to the nascent RNA and influences transcription by inducing pausing and facilitating the p

123 Many RAPs inhibit transcription by inducing Rho-dependent termination in E

124 , antisense transcription can regulate sense transcription by induction of epigenetic modifications.

125 H4K16 acetylation (H4K16Ac) activates gene transcription by influencing both chromatin structure an

126 his binding promotes the initiation of viral transcription by influenza RdRP.

127 that p21 can indirectly block HIV-1 reverse transcription by inhibiting host cofactors supporting HI

128 r results demonstrate that PKA regulates frq transcription by inhibiting RCM-1 activity through RCM-1

129 d, we found that filgotinib suppresses HIV-1 transcription by inhibiting T cell activation and by mod

130 rions, it also significantly restricts HIV-1 transcription by inhibiting the NF-kappaB pathway.IMPORT

131 Production of IL-1beta requires transcription by innate immune receptor signaling and ma

132 on increased, whereas inhibition of INS gene transcription by INS promoter targeting siRNA decreased

133 unliganded PRA paradoxically activates Cx43 transcription by interacting with FRA2/JUND heterodimers

134 ription factors or as negative regulators of transcription by interacting with the nucleosome remodel

135 rthermore, we show that holo-WhiB1 represses transcription by interaction with sigmaA4in vitro and th

136 MAF1 represses Pol III-mediated transcription by interfering with TFIIIB and Pol III.

137 rsional stress from DNA and facilitates gene transcription by introducing transient DNA double-strand

138 The enhancing effect on reverse transcription by IP6 and the consequences of interaction

139 LIM substrates, we found that LANA modulates transcription by LHX3-LDB1 complex and suggest additiona

140 brain is masculinized by direct induction of transcription by ligand-activated nuclear steroid recept

141 ivity, suggesting that MYC regulates its own transcription by limiting BRD4-mediated chromatin remode

142 x plays a critical role in the regulation of transcription by linking transcription factors to RNA po

143 biological response toward DNA damage during transcription by manipulating their gene expression.

144 These regulate transcription by mechanisms that have not been fully elu

145 1) has been reported to repress and activate transcription by mediating histone H3K4me1/2 and H3K9me1

146 Induction of Duox2 transcription by microbial colonization did not require

147 roth are required for activation of vps gene transcription by Mlc.

148 whether intergenic lncRNAs commonly regulate transcription by modulating chromatin at genomically dis

149 a counterbalancing mechanism governing Edn1 transcription by modulating H3 K79 dimethylation and H3

150 that Sp100 isoforms differentially regulate transcription by modulating lysine acetylation.

151 trand reduces the yield of T7 RNA polymerase transcription by more than an order of magnitude when po

152 mensional chromosomal conformations regulate transcription by moving enhancers and regulatory element

153 Although the regulation of BDNF transcription by neuronal activity has been widely studi

154 ults suggest that the downregulation of Ezh2 transcription by NFIB is an important component of the p

155 This suggests transcription by novel promoters for 72% of Arabidopsis

156 ostasis is regulated largely at the level of transcription by nuclear receptors, particularly the pri

157 The process begins with template DNA transcription by NUCLEAR RNA POLYMERASE IV (Pol IV), who

158 tion of RNA polymerase II (Pol II)-dependent transcription by nucleating pre-initiation complex (PIC)

159 nique strategy for selectively targeting MYC transcription by pharmacological means as a potential tr

160 n/Wnt activities and p38gamma stimulates Wnt transcription by phosphorylating beta-catenin at Ser605.

161 h the 10-subunit TFIIH complex and regulates transcription by phosphorylating the C-terminal domain (

162 clin-dependent kinase, CDK7, which regulates transcription by phosphorylating the carboxy-terminal do

163 Mediator kinase module regulates eukaryotic transcription by phosphorylating transcription-related t

164 been partly attributed to the increased rDNA transcription by Pol I in cancer.

165 elicits promoter-dependent and -independent transcription by Pol III.

166 Here we show that non-coding transcription by Pol V is controlled by preexisting chro

167 oactivator complexes SAGA and NuA4 stimulate transcription by post-translationally modifying chromati

168 those described previously for regulation of transcription by ppGpp.

169 P(XD)-N(TAIL) interaction results in reduced transcription by preformed transcriptases, suggesting re

170 sults in modulation of estrogen-induced gene transcription by preventing Estrogen Receptor chromatin

171 c DNA lesions that block DNA replication and transcription by preventing strand separation.

172 ome remodeler SWI/SNF, facilitated divergent transcription by promoting rapid nucleosome turnover.

173 cluster potentiated bursts in CREB-mediated transcription by promoting recruitment of the CREB coact

174 discuss the potential benefits of activating transcription by promoting RNA polymerase isomerization

175 into chromosome segregation genes and alter transcription by providing a premature termination site,

176 ed the stability of the viral genome and TAg transcription by quantitative reverse transcriptase PCR

177 k to understand the control of mycobacterial transcription by RbpA and CarD.

178 entary to exon 1 of GNG12-AS1 suppresses its transcription by recruiting Argonaute 2 and inhibiting R

179 Steroid receptors activate gene transcription by recruiting coactivators to initiate tra

180 activate ADD1 and CCND1 but repress p53 gene transcription by recruiting differential chromatin modif

181 nscription factor SLUG/SNAI2, repressing its transcription by recruiting HDAC1 and licensing the remo

182 phosphorylation states coordinate eukaryotic transcription by recruiting protein regulators.

183 ns showed that MUC1-C acted to elevate PD-L1 transcription by recruitment of MYC and NF-kappaB p65 to

184 r localization, DNA binding, and target gene transcription by reducing AKT-dependent FOXO1 phosphoryl

185 We show that SP rapidly inhibits HIV-1 transcription by reducing RNAPII recruitment to the HIV-

186 s aggregation, and promote AR-dependent gene transcription by regulating AR-cofactor interactions.

187 hromatin remodeling complex facilitates gene transcription by remodeling chromatin using the energy o

188 w that enCRISPRa and enCRISPRi modulate gene transcription by remodeling local epigenetic landscapes

189 sition from elongation to termination during transcription by removing RNAPII CTD serine 5 phosphoryl

190 nto the elongation and termination phases of transcription by removing the phosphate marker on serine

191 t Myc, to mediate inhibition of MYC-mediated transcription by replacing MYC/MAX heterodimers with Omo

192 ntributes to temporal regulation of myogenic transcription by restricting late gene expression during

193 how different forms of DNA alkylation impair transcription by RNA Pol II in cells and with the isolat

194 the highest levels of template uracilation, transcription by RNA pol II was completely blocked.

195 Initiation of gene transcription by RNA polymerase (Pol) III requires the a

196 a tiny number are estimated to be active in transcription by RNA polymerase (Pol) III.

197 c DNA associated with proteins and RNAs) and transcription by RNA polymerase (RNAP) in all domains of

198 Transcription by RNA polymerase (RNAP) in bacteria requi

199 Transcription by RNA polymerase (RNAP) is interrupted by

200 regulation in organisms minimally depends on transcription by RNA polymerase and on the stability of

201 ion factor A (CITFA), which is essential for transcription by RNA polymerase I (Pol I) in the parasit

202 Ribosomal RNA (rRNA) transcription by RNA polymerase I (Pol I) is the first k

203 show that the THO complex positively affects transcription by RNA polymerase I (Pol I).

204 5S and 5S rRNA, indicating their independent transcription by RNA polymerase I and III, respectively.

205 te that DNA binding by Rrn3 is essential for transcription by RNA polymerase I.

206 Transcription by RNA polymerase II (Pol II) in eukaryote

207 Transcription by RNA polymerase II (Pol II) in metazoans

208 Transcription by RNA polymerase II (Pol II) is a complex

209 Transcription by RNA polymerase II (Pol II) is carried o

210 Transcription by RNA polymerase II (Pol II) is dictated

211 Transcription by RNA polymerase II (Pol II) is required

212 kely required for nucleosome survival during transcription by RNA polymerase II (Pol II) through chro

213 t of this process is repression of host cell transcription by RNA polymerase II (Pol II), which also

214 SHM requires IgV transcription by RNA polymerase II (Pol II).

215 scopy, we probed the spatial organization of transcription by RNA polymerase II (RNAP II) molecules a

216 Pre-mRNA splicing is coupled to transcription by RNA polymerase II (RNAP II).

217 Initiation and regulation of transcription by RNA polymerase II (RNAPII) in eukaryote

218 Transcription by RNA polymerase II (RNAPII) is a dynamic

219 Transcription by RNA polymerase II (RNAPII) is accompani

220 yclin-dependent kinase 9 (Cdk9) occur during transcription by RNA polymerase II (RNAPII), and are mut

221 p50 in both locations is dependent on active transcription by RNA polymerase II and requires the N-te

222 l repeat (LTR) retrotransposons, begins with transcription by RNA polymerase II followed by reverse t

223 The molecular process of transcription by RNA Polymerase II is highly conserved a

224 Gene transcription by RNA polymerase II is regulated by activ

225 We propose that transcription by RNA polymerase II is tuned to optimize

226 mplex is known to be a master coordinator of transcription by RNA polymerase II, and this complex is

227 quently occurs at the same time and place as transcription by RNA polymerase II.

228 DNA and identification of the start site for transcription by RNA polymerase II.

229 tRNA-gene mediated (tgm) silencing of local transcription by RNA polymerase II.

230 to chromatin, in the biochemical analysis of transcription by RNA polymerase II.

231 Transcription by RNA polymerase is a highly dynamic proc

232 Transcription by RNA polymerase may be interrupted by pa

233 Transcription by RNA polymerase V (Pol V) in plants is r

234 ein complex, is crucial to the regulation of transcription by RNA-polymerase II, via its interaction

235 Analysis of host cell transcription by RNA-seq during latency in AGS cells ide

236 factors DksA and GreA modulated translesion transcription by RNAP, depending on changes in the trigg

237 During transcription by RNAP, we directly observed rotational s

238 s involved in DNA repair(9), as well as from transcription by RNAP.

239 e evidence for an involvement of the TEFs in transcription by RNAPI (or RNAPIII).

240 licase DDX21, thereby restricting productive transcription by RNAPII at master transcription factor (

241 ssing of mRNA and the coupled termination of transcription by RNAPII requires the CF IA complex.

242 y of dimeric transcription factors regulates transcription by selectively binding to DNA response ele

243 BET proteins regulate transcription by selectively recognizing acetylated lysi

244 nt mediator of the regulation of PPAR-gamma2 transcription by SHP.

245 ying the circadian regulation of chloroplast transcription by SIG5 are unidentified.

246 and the circadian regulation of chloroplast transcription by SIG5 was predominantly dependent on blu

247 tingly, the selective activation of IL-1beta transcription by SIRT1 deficiency is likely mediated thr

248 ical model includes a description of genetic transcription by SREBP-2 which is subsequently translate

249 n regulator in MTB, binds RNAP and activates transcription by stabilizing the transcription initiatio

250 1 and likely other factors dampens activated transcription by stabilizing Tup1 binding and stimulatin

251 robe all intermediate RNA transcripts during transcription by stalling elongation complexes at cataly

252 a reduction of KIT expression ablates DNMT1 transcription by STAT3 pathway leading to in-parallel mo

253 Androgens control transcription by stimulating androgen receptor (AR) acti

254 e that glucagon plus insulin increases FGF21 transcription by stimulating ATF4 expression and that ac

255 Suppression of ncRNA transcription by strand-specific CRISPR/dCas9 results in

256 In bacteria, RNA polymerase (RNAP) initiates transcription by synthesizing short transcripts that are

257 Activation of LXR-mediated transcription by synthetic agonists, such as T0901317 an

258 f primer assembly of DNA templates, in vitro transcription by T7 RNA polymerase and kit-based purific

259 and studied the effects of this structure on transcription by T7 RNA polymerase in vitro.

260 Several essential conditions for in vitro transcription by T7 RNAP were confirmed with this assay,

261 recently has the idea of disrupting altered transcription by targeting chromatin-associated proteins

262 ors and CRISPR-Cas9 methods aiming to reduce transcription by targeting DNA.

263 We find that both complexes influence transcription by targeting promoters and TSS-distal enha

264 n, whereas miR-122 indirectly inhibits c-Myc transcription by targeting Tfdp2 and E2f1.

265 Notably, miR-122 also repressed c-Myc transcription by targeting transcriptional activator E2f

266 Uncoupling DNA demethylation from antisense transcription by Tet3 overexpression in mouse olfactory

267 of DNA damage that block DNA replication and transcription by tethering the opposing strands of DNA.

268 promoter as mediating the increase in IGF-1 transcription by TGFbeta in pulmonary fibroblasts; (3) d

269 As are modified at their 5' end early during transcription by the addition of N7-methylguanosine (m(7

270 We found that timing of Shh transcription by the cell cycle clock can be reset, thus

271 teins that mediate the suppression of cccDNA transcription by the cytokine, we found that downregulat

272 of epigenetic events in programming IL-1beta transcription by the distal enhancer priming and eRNA pr

273 NA can mimic B-form DNA to directly regulate transcription by the DNA-dependent RNAP.

274 The repression of E-cadherin transcription by the EMT inducers Snail1 and Zeb2 plays

275 Instead, MarA drives transcription by the housekeeping sigma(70) -associated

276 , many trans-acting factors that orchestrate transcription by the individual Pols have been described

277 r insight into the mechanism of viral genome transcription by the influenza virus polymerase and may

278 ysis and found that Mel-18 increased miR-205 transcription by the inhibition of DNA methyltransferase

279 e insights into the regulation of stop-start transcription by the interplay between the RdRp active s

280 Initiation of transcription by the Mycobacterial RNA polymerase (RNAP)

281 karyotes, the separation of translation from transcription by the nuclear envelope enables mRNA modif

282 In neurons, regulation of activity-dependent transcription by the nuclear factor of activated T-cells

283 of Cavalpha1.2; and promotion of Cavalpha1.2 transcription by the prevention of Kir/Gem-mediated shut

284 in the mgtA mRNA that permits termination of transcription by the Rho protein upstream of mgtA, where

285 y in keratinocytes with 11-fold induction of transcription by the risk allele versus 8-fold by the co

286 s, this is a result of activation of class I transcription by the T cell enhanceosome consisting of R

287 al proteins and activation of endogenous RNA transcription by the TEC inside the virus.

288 le and readily displaced in the direction of transcription by the transcriptional machinery.

289 f MCL-1 mitochondrial localization and BFL-1 transcription by the viral EBNA3A protein.

290 Activation of transcription by the yeast activator Gcn4, for example,

291 lncRNAs that are targeted, at their site of transcription, by the YTH domain of the RNA-binding prot

292 Thus, regulation of transcription by these factors is dependent on divergent

293 r, MITF controls c-MYC implicated in general transcription by transactivation of far upstream binding

294 xpression, in part through inhibition of HIV transcription by TRIM22 and decrease in T-cell activatio

295 ich suggest that HiNmlR enhances target gene transcription by twisting of operator DNA sequences in a

296 CRL4(DCAF1) promotes YAP- and TEAD-dependent transcription by ubiquitylating and, thereby, inhibiting

297 HP initiates reverse transcription by using itself as a protein primer (prote

298 nd RNA G4-ligand interactions during reverse transcription, by using a selective reverse transcriptas

299 global and specific alterations of host cell transcription by viruses.

300 Fenofibrate transactivates MDR3 gene transcription by way of the binding of PPARalpha to thre