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1 (TCF)-lymphoid enhancer factor (LEF) family transcription factor complex.
2 dependent activation of the GABP(alpha/beta) transcription factor complex.
3 he subcellular localization of the Rtg1/Rtg3 transcription factor complex.
4 tinct biological activities of the NF-kappaB transcription factor complex.
5 locator (ARNT/HIF-1beta) to form an AhR/ARNT transcription factor complex.
6 ity by inducing a unique conformation in the transcription factor complex.
7 hway signaling which converges upon the AP-1 transcription factor complex.
8 that were reconstituted by rapamycin into a transcription factor complex.
9 able to independently bind its corresponding transcription factor complex.
10 meric, basic helix-loop-helix/leucine zipper transcription factor complex.
11 , in assembly of a myeloid-restricted, basal transcription factor complex.
12 p65 (RelA) forms the prototypical NF-kappaB transcription factor complex.
13 ceptor (AhR)/AhR nuclear translocator (ARNT) transcription factor complex.
14 A1 represses DNMT3a expression via HDAC1/YY1 transcription factor complex.
15 strongly inhibited binding of the respective transcription factor complex.
16 he MuvB:B-MYB complex functions as a pioneer transcription factor complex.
17 forms part of the activator protein 1 (AP-1) transcription factor complex.
18 its stabilization and the formation of HIF-1 transcription factor complex.
19 lipogenic genes through the Mondo/ChREBP-Mlx transcription factor complex.
20 NFATc2 is critical for assembly of this transcription factor complex.
21 erning is dependent on the WER-GL3/EGL3-TTG1 transcription factor complex.
22 through activation of the beta-catenin/TCF4 transcription factor complex.
23 ression of HIF1 (hypoxia-inducible factor 1) transcription factor complex.
24 (PRC1), binds directly to the Runx1/CBFbeta transcription factor complex.
25 ative affinities to any DNA sequence for any transcription factor complex.
26 being directly responsive to a core Pol III transcription factor complex.
27 d Rbpj, no neural targets are known for this transcription factor complex.
28 ignal transduction machinery, ribosomes, and transcription factor complexes.
29 ted assembly of Smad-containing multiprotein transcription factor complexes.
30 ter activity and altered binding of specific transcription factor complexes.
31 re also recruited, possibly as components of transcription factor complexes.
32 embers of the Jun family to form active AP-1 transcription factor complexes.
33 xpression also alters the composition of E2F transcription factor complexes.
34 dinated assembly and disassembly of distinct transcription factor complexes.
35 ukaryotic genes are regulated by multivalent transcription factor complexes.
36 toplasmic inhibitor of select NF-kappa B/Rel transcription factor complexes.
37 lterations in the expression of E2F and AP-1 transcription factor complexes.
38 rk of genes under the direct control of SOC1 transcription factor complexes.
39 art by the coordinated action of a series of transcription factor complexes.
40 d, and Hth binding sites mediate cooperative transcription factor complexes.
42 r, our findings illustrate how SHR-dependent transcription factor complexes act in different domains
43 osis factor-alpha, acting via its downstream transcription factor complex activator protein-1, functi
44 1 association with c-Jun, a component of the transcription factor complex, activator protein 1 (AP-1)
45 nctions through a stable AML1-ETO-containing transcription factor complex (AETFC) that contains sever
46 BTF122 is a subunit of the Xenopus CCAAT box transcription factor complex and a member of a family of
47 with inhibition of the canonical NF-kappaB1 transcription factor complex and activation of the alter
48 form part of the activating protein-1 (AP-1) transcription factor complex and are implicated in regul
49 cell fates are determined by a MYB-bHLH-WD40 transcription factor complex and are regulated by many i
50 Fos, a key component of the ubiquitous AP-1 transcription factor complex and as such could influence
51 al component of the activator protein (AP)-1 transcription factor complex and can promote contact-ind
52 e due to decreased activation of the E2F1/Dp transcription factor complex and delayed progression thr
54 member of the multimeric activator protein 1 transcription factor complex and plays an important role
55 l cycle progression, including the E2F-1/DP1 transcription factor complex and the retinoblastoma tumo
58 e the DNA binding subunits of MBF cell-cycle transcription factor complexes and contain an N terminal
59 )17 responses through the activation of AP-1 transcription factor complexes and the histone deacetyla
60 ells in culture, to dissociate the p130-E2F4 transcription factor complex, and to stimulate ATP hydro
61 n of size control, localization of inhibitor/transcription factor complexes, and the nutritional effe
65 ological action of the eukaryotic NF-kappa B transcription factor complex are tightly regulated throu
66 tions suggests that NLI-dependent LIM domain transcription factor complexes are involved in communica
67 protein, and the heterodimeric RUNX1/CBFbeta transcription factor complex, are critical for definitiv
69 egulation involving chromatin accessibility, transcription factor complex assembly, and protein phosp
71 cells to enhancer occupancy by the BATF-IRF4 transcription factor complex at varying strengths of TCR
72 tress regulated in part by a BMPR2 dependent transcription factor complex between PPARgamma and p53.
74 e demonstrate that this protein is part of a transcription factor complex binding to extended sequenc
79 ons could affect not only the composition of transcription factor complexes but also their degradatio
80 same signal transduction components (R-Smad transcription factor complexes), but whether and how the
81 n is therefore accompanied by replacement of transcription factor complexes by a repressive chromatin
82 ets of Cln3/CDK, we analyzed the SBF and MBF transcription factor complexes by multidimensional prote
83 physically interacted with Aiolos to form a transcription factor complex capable of inducing the exp
84 athways (the Rb-like protein RBF and the E2F transcription factor complex components dE2F and dDP) co
85 rs (CBFs) are a small group of heterodimeric transcription factor complexes composed of DNA binding p
86 ulated by a conserved anthocyanin-regulating transcription factor complex consisting of a MYB, a bHLH
90 This is the first example of a eukaryotic transcription factor complex containing both a MADS-box
91 nism whereby mutations in BRCA1, via a novel transcription factor complex containing BRCA1, c-Myc, an
92 st that Hmo1 is required for the assembly of transcription factor complexes containing Fhl1 and Ifh1
94 -like antigen 1 (FOSL1), a component of AP-1 transcription factor complexes, contributes to the regul
95 wen (2019) finds that a PBX/MEIS homeodomain transcription factor complex controls a transcriptional
96 acute leukemia that affect the AML-1/CBFbeta transcription factor complex create dominant inhibitory
99 tionary conservation of the function of this transcription factor complex during metazoan neural deve
101 Area III contains a binding site for PTF1, a transcription factor complex essential for pancreas deve
103 d extends the argument that CO utilizes NF-Y transcription factor complexes for the activation of FLO
104 d intrinsic signals to control heterodimeric transcription factor complex formation provides a robust
106 the 2.1-A crystal structure of the archaeal transcription factor complex formed by the TATA-box-bind
107 appaB elements that are activated by a novel transcription factor complex formed when U-STAT3 binds t
108 ted Gabpa, the DNA-binding component of this transcription factor complex, from mouse embryonic fibro
110 2 lineages are specified and in general how transcription factor complexes govern hematopoiesis.
115 T cells and implicates the integrity of this transcription factor complex in developmental events ess
117 data highlight the key role of the Yap/Tead transcription factor complex in maintaining inner ear pr
118 amined the expression of members of the AP-1 transcription factor complex in response to stimulation
120 ve potential of a newly identified PPARy-p53 transcription factor complex in the pulmonary endotheliu
121 otential of a newly identified PPARgamma-p53 transcription factor complex in the pulmonary endotheliu
122 The Core Binding Factor is a heterodimeric transcription factor complex in vertebrates that is comp
123 r the nucleus and combine with TCF to form a transcription factor complex in which TCF binds DNA and
125 nce for the functional role of highly mobile transcription factor complexes in transcription regulati
126 tivation of RELA, a subunit of the NF-kappaB transcription factor complex, in gingival macrophages of
127 otein complexes, in particular chromatin and transcription factor complexes, in a rapid and robust ma
129 ) is a prerequisite for the formation of the transcription factor complex interferon-stimulated gene
140 tion factor c-Jun, which is part of the AP-1 transcription factor complex, is also important for mono
141 s-like antigen 1 (FOSL1), a component of AP1 transcription factor complexes, is a key player in regul
144 which led to activation of a heterotrimeric transcription factor complex known as IFN-stimulated gen
146 riation in activity of the Myb, bHLH and WDR transcription factor complex (MBW) that regulates anthoc
147 ic helix-loop-helix-leucine zipper (bHLHZip) transcription factor complex MondoA-Mlx plays a central
148 mplex formed between the prominent oncogenic transcription factor complex Myc/MAX and the tumor suppr
149 tains an inverted CCAAT box motif, binds the transcription factor complex NF-Y (also referred to as C
150 of these cytokines is often regulated by the transcription factor complex, nuclear factor-kappa B (NF
151 PTF1 is an atypical basic helix-loop-helix transcription factor complex of pancreatic acinar cells
155 characterize the DNA binding activity of Hox transcription factor complexes on eight experimentally v
156 this stage of myelopoiesis, the formation of transcription factor complexes on the promoter was compl
157 s that CIITA is required for the assembly of transcription factor complexes on the promoters of this
158 TA1 indicating the recruitment of CTCF/GATA1 transcription factor complex onto the HPIP promoter.
159 cific cellular processes by associating with transcription factor complexes or binding to mRNAs.
160 otein c-Jun, a major constituent of the AP-1 transcription factor complex, or expression of a c-Jun-s
161 These data provide evidence that Fos/Jun transcription factor complexes play a role in modulating
162 cts genetically with: (1) genes for multiple transcription factor complexes predominantly involving M
163 udy of the structural details of an archaeal transcription factor complex presents the opportunity to
164 h is compromised for assembly into the Notch transcription factor complex, primarily modifies burst s
165 along with an E protein and Rbpj, forms the transcription factor complex PTF1-J that is essential fo
167 or Tal1) protein forms part of a multimeric transcription factor complex required for normal megakar
169 onse gene c-fos, these pathways activate the transcription factor complex serum response factor (SRF)
170 hrough the hypoxia-inducible factor (HIF), a transcription factor complex stabilized under low oxygen
171 oid receptor (GR) can tether to inflammatory transcription factor complexes, such as NFkappaB and AP-
172 ltorphin also increased accumulation of AP-1 transcription factor complexes, suggesting that DOR1 aug
173 compared with other Stat protein-containing transcription factor complexes suggests distinct roles f
174 eractions between proteins that comprise the transcription factor complex TFIIH raises the possibilit
175 rom the genomic DNA and recruits the general transcription factor complex, TFIIH, for subsequent lesi
177 orm of STAT3 interacted with DNMT1 to form a transcription factor complex that bound to and methylate
178 heterodimerize, thereby generating an active transcription factor complex that commits mated cells to
180 or GA binding protein (GABP) is a tetrameric transcription factor complex that contains GABPalpha and
181 anding of the complexity and dynamics of the transcription factor complex that forms at the MT-I prom
182 the primary blue-light photoreceptor and the transcription factor complex that initiates light-regula
183 ore binding factor [CBF]) is a heterodimeric transcription factor complex that is frequently involved
184 appaB corresponds to an inducible eukaryotic transcription factor complex that is negatively regulate
185 embly and activation of the RUNX2-TAZ master transcription factor complex that is required for osteob
186 rticipate as components of a multi-component transcription factor complex that is required for regula
187 hese results suggest the presence of a novel transcription factor complex that mediates the glucose-r
188 are targets of the DREAM complex, which is a transcription factor complex that regulates expression o
189 ucleoprotein complex configuration, and that transcription factor complexes that bind the same enhanc
190 ed a series of multisubunit, tissue-general, transcription factor complexes that bound to the GHF3 ac
191 sult of decreased expression of unidentified transcription factor complexes that interact with PRE1 a
192 itions with overexpressed E2F and Cabut, two transcription factor complexes that promote ectopic cell
193 2 is a component of multisubunit DNA-binding transcription factor complexes that regulate gene expres
194 is a component of activator protein-1 (AP-1) transcription factor complexes that regulates processes
195 nserved chromatin-remodeling and homeodomain transcription factor complexes that work with somi-1 to
196 ster, where it targets the Mcm1p-Fkh2p-Ndd1p transcription factor complex, through direct phosphoryla
197 d with MAPK signaling through the c-Jun/AP-1 transcription factor complex to activate CD73 transcript
198 king example of the repurposing of a general transcription factor complex to aid in genome defense ag
199 reprograms the composition of the TEA domain transcription factor complex to suppress their downstrea
200 lated the binding of the activator protein-1 transcription factor complex to the cyclic AMP response
201 evealed the binding of such a p204-pRb-Cbfa1 transcription factor complex to the promoter of the oste
203 al systems to be mediated through binding of transcription factor complexes to TRE and EpRE elements.
204 ATA binding protein (TBP) interacts with two transcription factor complexes, upstream activating fact
206 and the transcriptional activity of the AP-1 transcription factor complex were markedly reduced in th
207 binding sites for the Nuclear Factor Y (NFY) transcription factor complex were significantly bound du
209 binds a multi-protein hematopoietic-specific transcription factor complex which includes GATA-1, SCL/
210 ding factor alpha subunit of a heterodimeric transcription factor complex which plays critical roles
211 mmalian cells induces formation of the ISGF3 transcription factor complex, which binds to interferon
212 before forming the heterodimeric NFATc3-FosB transcription factor complex, which bound the proximal A
213 formation of an oncogenic NFATc1/SMAD3/cJUN transcription factor complex, which drives the expressio
214 un is a component of the activator protein-1 transcription factor complex, which is involved in cellu
215 ead box protein H1-Sma and Mad homolog 2/3/4 transcription factor complex, which is promoted by the T
216 ead box protein H1-Sma and Mad homolog 2/3/4 transcription factor complex, which is promoted by the T
217 ry trajectory for the heterodimeric TMO5/LHW transcription factor complex, which is rate-limiting for
218 n and interaction with the TEA domain (TEAD) transcription factor complex, which led to upregulated e
219 forms part of the activator protein 1 (AP-1) transcription factor complex, which plays a pivotal role
220 th Jun family proteins to create active AP-1 transcription-factor complexes, which bind to DNA specif
221 in is a target gene of beta-catenin-Tcf, the transcription factor complex whose activity is thought t
222 binding site for a GA binding protein (GABP) transcription factor complex, whose assembly at the prom
223 he nucleus where it functions in a bipartite transcription factor complex, whose targets include inva
224 nd adaptation to iron starvation by the same transcription factor complex with activating and repress
225 sidues allow FIT to interact and form active transcription factor complexes with subgroup Ib bHLH fac