戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (1語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1  or H2Bub1 loss induces intragenic antisense transcription of ~10% of fission yeast genes, with each
2                                          The transcription of 18S, 5.8S, and 18S rRNA genes (45S rDNA
3          In response to RS, DUX triggers the transcription of 2C-like markers such as murine endogeno
4 sion analysis under glyphosate stress showed transcription of 41 of these 59 genes, with high express
5  nuclear CREB phosphorylation, and increased transcription of a BDNF-dependent program of gene expres
6                                              Transcription of a chromatin template involves the conce
7  nucleoplasmic extract (NPE) supports robust transcription of a chromatinized plasmid substrate.
8  5'-UTR led to a significant increase in the transcription of a downstream beta-glucuronidase (GUS) r
9 urthermore, ectopic expression increased the transcription of a Fur-controlled gene in Escherichia co
10 ng that important processing can occur after transcription of a gene, but before translation of the m
11                               AggR activates transcription of a large number of virulence genes, incl
12 tion regulates FUS nuclear translocation and transcription of a major profibrotic collagen gene.
13 orylation of MEK/ERK1/2 and activity-induced transcription of a neuronal immediate early gene; and 3)
14 y sites of target genes and also control the transcription of a receptor-exclusive set of genes.
15                              Thus, increased transcription of a set of host genes contributes to a pr
16 nhardtii) was previously shown to affect the transcription of a subset of genes during nitrogen (N)-r
17 t hippocampal neurons promotes the inducible transcription of a subset of NMDAR-sensitive genes.
18  gene expression programs in AML and repress transcription of a subset of SE-associated leukemic onco
19  enhancer factor 2C (MEF2C) and promotes the transcription of a subset of synaptic activity-induced g
20 tant CRPC cells and associated with enhanced transcription of a subset of tumor promoting genes such
21     The master tumor suppressor p53 controls transcription of a wide-ranging gene network involved in
22                            We report that co-transcription of ACE2 and TMPRSS2 is negligible in the p
23 me of these homeostatic mechanisms depend on transcription of activity-regulated genes, including Arc
24  deficient B12/FA uptake demonstrated higher transcription of AhR target genes and lower transcriptio
25 h response elements to induce or repress the transcription of aldosterone-regulated genes.
26 tion to reprogram the host cell and activate transcription of all other viral genes.
27 raC-type transcription factor ExsA activates transcription of all T3SS-associated genes.
28                 PRMT5 knockdown de-repressed transcription of all three miRNAs, accompanied by loss o
29 erase II complex (pol II) is responsible for transcription of all ~21,000 human protein-encoding gene
30                           HIF-1 promotes the transcription of an ADP ribosyltransferase, TiPARP, whic
31                                              Transcription of an antisense long noncoding RNA (lncRNA
32 iption suppressor function of TDP-43 allowed transcription of an early termination cryptic axon, resu
33 repressed under serine-replete conditions by transcription of an upstream SRG1 lncRNA that traverses
34 -oxidant enzyme genes in addition to reduced transcription of antioxidant genes by HIF-2.
35                        Gill et al. show that transcription of antisense ncRNAs induces 'elongation ma
36 ed-forward interplay between immediate early transcription of AP-1 and Hippo pathway function.
37 c disruption of NELF in macrophages enhanced transcription of AP-1-encoding Fos and Jun and, conseque
38 that cause disease through the disruption of transcription of ARPC1B, GATA1, LRBA and MPL.
39                               LKB1 represses transcription of ATOH1, via PDK4, in ISCs, restricting t
40  phosphorylation, a process known to promote transcription of atrophy genes in skeletal muscle.
41                   The myriad questions about transcription of bacterial chromatin are increasingly an
42   We show that Rho can prematurely terminate transcription of bacterial CRISPR arrays, and we identif
43                     ATF4 directly stimulates transcription of BCL11A, a repressor of gamma-globin tra
44                                              Transcription of BHU72_07145 increased markedly when MLF
45                                              Transcription of both constitutive and periodic genes is
46          As in the mouse, human A-MYB drives transcription of both pachytene piRNA precursor transcri
47 ds at the promoters and positively regulates transcription of CaDefensin and CaWRKY33.
48 lular cAMP and (b) prevent immunosuppressive transcription of cAMP response element- and hypoxia resp
49             FLIP(L) depletion also modulated transcription of canonical p53 target genes, suppressing
50 mechanisms, we found that YY1 suppresses the transcription of CArG box-dependent SMC-specific genes i
51 BV RNA are sensitive biomarkers of continued transcription of cccDNA in HBeAg-negative patients despi
52 BV RNA are sensitive biomarkers of continued transcription of cccDNA in HBeAg-negative patients despi
53  secretion of exosomal PD-L1 by blocking the transcription of CD274, which combined with the anti-PD-
54 Histone H3 at CD274 promoter followed by the transcription of CD274.
55 ly, we show that loss of hnRNP C reduces the transcription of CELF2 mRNA, while loss of CELF2 results
56 structure of the gibbon genome by disrupting transcription of cell cycle genes.
57                   These changes ensure basal transcription of cell-cycle genes and are critical for t
58 acement of super-enhancers (SEs) to activate transcription of cell-identity specifying gene networks,
59 ization of Vibrio cholerae by regulating the transcription of class-III flagellar genes in sigma(54)-
60 BMAL1/CLOCK complex and repress E-box-driven transcription of clock-associated genes.
61                                MRX restricts transcription of coding and noncoding DNA by a mechanism
62 ty acids acetate and butyrate and then alter transcription of colonization factors appropriately for
63       Transcriptome studies reveal pervasive transcription of complex genomes, such as those of mamma
64 one acetylation enables BRD4 recruitment for transcription of constitutively active genes.
65  that antagonizes Rho to facilitate complete transcription of CRISPR arrays.
66 egatively regulating fungal colonisation and transcription of crucial signalling components and conse
67 ed receptor alpha (PPARalpha), which induces transcription of cytochrome P450 4A (CYP4A).
68                                              Transcription of DNA is a fundamental process in all cel
69                     CHK2 depletion increases transcription of DNAPK and RAD54, increases clonogenic s
70 of SREBP proteins (SREBP1 or SREBP2) and the transcription of downstream lipogenesis-related genes, p
71 esults show that NURR1 and ERR1 modulate the transcription of DRD2 coexpression partners and support
72           Moreover, the relationship between transcription of early response genes Nr4a1 and Irf8 and
73 ggered CDNs lead to the CDNs-guided enhanced transcription of either the DFHBI aptamer or the MG apta
74 e induced apoptosis and CRISPR-Cas9-mediated transcription of endogenous genes specifically in ErbB-h
75 uals with a germline polymorphism preventing transcription of ENTPD1, encoding CD39.
76 r paired box 2 protein (PAX2) to repress the transcription of ERBB2/HER2.
77 c conflict on evolving Y chromosomes between transcription of essential genes and silencing of selfis
78                                              Transcription of eukaryotic mRNA-encoding genes by RNA p
79                                              Transcription of F3 was regulated by IL1beta, whose secr
80 echanisms downstream of TGFbeta that mediate transcription of fibrogenic signals.
81 -like nucleoid structuring) protein silences transcription of foreign genes in a variety of Gram-nega
82 educes H-NS binding to foreign DNA, allowing transcription of foreign genes, including those required
83 ZIP transcription factors, which impacts the transcription of Fos/Jun target genes.
84 h ERalpha and FOXM1, it also led to enhanced transcription of FOXM1.
85 ched in the promoter of GDH1 to activate the transcription of GDH1, which then promoted glutamine met
86 n, whereas stimulation with TGFbeta1 induced transcription of genes associated with a myofibroblast p
87 rabidopsis circadian clock not only controls transcription of genes but also affects their posttransc
88 ver transcriptome analysis uncovered altered transcription of genes downstream of lipid-related trans
89 the majority of these regulators repress the transcription of genes encoding class A ARFs.
90    The deletion mutant showed a reduction in transcription of genes encoding highly expressed, secret
91 s butyrate likely by indirect means to alter transcription of genes encoding important colonization d
92 rases H3K27me3 chromatin marks, facilitating transcription of genes encoding the pluripotency factors
93         The loss of H3K27me3 facilitates the transcription of genes essential for spermatogenesis and
94 epigenetics in mediating exposure effects on transcription of genes implicated in mental disorders.
95 epigenetic state at enhancers that amplifies transcription of genes in hepatocytes.
96 how that loss of both Trp53 and Rb1 disables transcription of genes in the autophagic machinery neces
97 n mammals, BMAL1 and CLOCK activate rhythmic transcription of genes including the nuclear receptor RE
98            In nematodes, TRA-1 represses the transcription of genes involved in male differentiation,
99 d demonstrates that nuclear Parkin regulates transcription of genes involved in multiple metabolic pa
100  contributes to both increased and decreased transcription of genes involved in regulating multiple m
101     Consistent with phenotypic observations, transcription of genes involved in SA and JA/ET pathways
102 PSC-CM) demonstrated that ERRgamma activates transcription of genes involved in virtually all aspects
103 strogen-related receptor (ERR) signaling and transcription of genes promoting oxidative metabolism.
104 s but increased phosphorylation of SMAD2 and transcription of genes regulated by SMAD.
105 ion of SHP1, leading to STAT6 activation and transcription of genes that regulate TAM generation and
106 verse effects of hypertonicity by increasing transcription of genes, including those that lead to cel
107  by functional hubness, stem-like cells, and transcription of genetic drivers of gliomagenesis.
108 ccur at any level during the replication and transcription of genetic information.
109                              During mitosis, transcription of genomic DNA is dramatically reduced, be
110                              Replication and transcription of genomic DNA requires partial disassembl
111 but not EPT1, cause significant increases in transcription of glycosylation genes, which may reflect
112 ts provide a framework for understanding the transcription of gonococcal ompA through a regulatory sy
113 s synaptic ribbon architecture, and perturbs transcription of hair cells specific genes during zebraf
114 ociated with initiation of RNA Polymerase II transcription of highly expressed genes, suggesting the
115 strate here that MeCP2 represses nascent RNA transcription of highly methylated long genes in the bra
116 represses the rate at which Pol II initiates transcription of highly methylated long genes.
117  of HLA-DR in infected cells by reducing the transcription of HLA-DR transcripts early during infecti
118 in tumor growth without causing an outage of transcription of housekeeping genes.
119 ins have a high renal expression with active transcription of HPS1, 3, 4 and 5 in human podocyte cell
120 ndogenous timing mechanisms that control the transcription of hundreds of genes.
121 ells against viral pathogens by inducing the transcription of hundreds of IFN-stimulated genes.
122                     Compression elevated the transcription of hypertrophic chondrocyte marker MMP13 a
123                In PBMC, pregnancy stimulated transcription of IFI6, RSAD2, IFI44, IFITM2, CLEC3B, OAS
124  within the bulk population displayed robust transcription of IFN-beta mRNA, and this did not appear
125 ach demonstrated that AW112010 regulated the transcription of IL-10.
126            Neuronal activation induces rapid transcription of immediate early genes (IEGs) and longer
127 essing monocytes and regulatory T-cells; and transcription of immune checkpoint (e.g., PD-1, LAG3) ge
128 lating Krebs' cycle intermediates that alter transcription of immune response genes.
129  transcription; in turn, AcaDC activates the transcription of IncC conjugation genes.
130      We propose that by repressing canonical transcription of individual transposon mRNAs, Mael helps
131 y unappreciated role of NELF in constraining transcription of inflammation inhibitors thereby enablin
132 cts through its receptor RARalpha to repress transcription of inflammatory cytokines, but is also ess
133                                              Transcription of integrated DNA from viruses or transpos
134 and 5'-triphosphorylated RNA to activate the transcription of interferon genes and promote antiviral
135 he PB2-D309N substitution enhanced the early transcription of interferon mRNA, revealing a novel role
136                       Further, LUCAT1 limits transcription of interferon stimulated genes by interact
137 uce PML expression in hBMECs and inhibit the transcription of interferon-stimulated genes (ISG).
138 tional potential of magnesium in controlling transcription of its downstream genes and underscores th
139 sion protein PAX3-FOXO1, allowing downstream transcription of its oncogenic program.
140  tna operon of Escherichia coli controls the transcription of its own operon through an attenuation m
141 rkhead box O1 (FOXO1) at Ser-249, leading to transcription of its proapoptotic target gene, Bcl-2-int
142 nd properly folded and activates basal-level transcription of its target vqmR in the absence of DPO.
143                                          The transcription of key genes prpC and prpD in MCC is activ
144             HNF4alpha functions to stimulate transcription of key gluconeogenic genes.
145 sis in oocytes revealed that Snf2h regulates transcription of key meiotic genes, such as Prkar2b, by
146 ltransferase (HAT) activities that activates transcription of key protooncogenes, including MYC We re
147 d by processes that restrict or activate the transcription of KSHV lytic genes.
148 rthermore, the lldR mutant exhibited reduced transcription of l-lactate utilization genes and impaire
149                             MYC controls the transcription of large numbers of long noncoding RNAs (l
150 appaB kinase activity that drove exaggerated transcription of late-phase nuclear factor-kappaB respon
151 nding protein 1 (SREBP1), which promotes the transcription of lipogenes involved in the biosynthesis
152 plications that include single-cycle reverse transcription of long RNAs, dimethyl sulfate mutational
153 rmonic activation of the 12-hour oscillatory transcription of many rate-limiting metabolic genes know
154                In the absence of Hg(II), the transcription of merA is basal and when Hg(II) is presen
155 onization may be caused, in part, by altered transcription of microbe perception genes and defence ge
156                       We show that errors in transcription of microsatellites (MS) and mis-splicing o
157 is concentrated into foci that depend on the transcription of mitochondrial RNAs that may form double
158  proteins bind to APN and thereby induce the transcription of MMPs.
159 anscriptional mechanisms and showed that the transcription of more than half of the induced genes was
160    However, during early neural development, transcription of most essential neurogenic genes is depe
161                        Our results show that transcription of most P. vivax genes occurs during short
162    Mitochondrial DNA (mtDNA) is damaged, the transcription of mtDNA-encoded genes is impaired, and th
163  stem cells (neuroblasts) by controlling the transcription of multiple cell death genes through a sha
164 es glioma cell sensitivity to TMZ and alters transcription of multiple genes.
165 cation of auxin can dramatically enhance the transcription of MUS, which is largely dependent on AUXI
166 fore identify disruptions in MEF2c-dependent transcription of Myoc as a novel mechanism of cancer-ass
167 ated and stabilized NOTCH1 which upregulated transcription of NANOG essential for TIC expansion.
168  efficiently knock down as well as terminate transcription of nascent lncRNAs and mRNAs, Lee and Mend
169 observed at a majority of these sites, while transcription of nearby genes tracked closely with acces
170 velopmental cues, sigma factors initiate the transcription of necessary genes responsible for maintai
171 matory mediator expression through increased transcription of negative regulators of innate immune ac
172 ted macrophage death associated with reduced transcription of NF erythroid 2-related factor 2 (NRF2)-
173         Mastermind proteins are required for transcription of Notch target genes, yet the molecular b
174 ral elements of innate immunity that control transcription of numerous pro-inflammatory genes.
175 highly in DRG, binds HSV-1 genome, represses transcription of numerous viral genes, and suppresses pr
176 ly binding herpesvirus genome, silencing the transcription of numerous viral genes, and ultimately li
177 rker of low glucose stress and regulates the transcription of nutritional stress-responsive genes.
178 ipt analyses demonstrate that TBPL2 mediates transcription of oocyte-expressed genes, including mRNA
179 e oxidase gene (oox) operon and activate the transcription of ooxB-lacZY, resulting in blue pigmentat
180  (5 vs 12 mo) and/or compression reduced the transcription of osterix and notochordal marker T by 40-
181 anical properties by 45-70%, and reduced the transcription of osterix, notochordal markers and chondr
182 endent late lytic gene transcription but not transcription of other EBV genes or cellular genes.
183 tion of both pathways de-represses antisense transcription of over half the genome.
184  transcription of AhR target genes and lower transcription of pathways implicated in birth defects.
185                EDAL positively regulates the transcription of Pcp4l1 encoding a 10-kDa peptide, which
186 activated T cells (NFAT) and potentiated the transcription of peroxisome proliferator-activated recep
187 on mRNAs, Mael helps Rhi drive non-canonical transcription of piRNA precursors without generating mRN
188 tor is a coregulatory complex that regulates transcription of Pol II-dependent genes.
189 d is functionally and physically tied to the transcription of pre-ribosomal RNA (pre-rRNA) and to lar
190 down-regulated NF-kappaB- and STAT-dependent transcription of prosurvival factors BCL2A1, BCL2L1, and
191 tory complex is essential for Nrf1-dependent transcription of proteasome genes.
192 ) counteracts proteotoxic stress by inducing transcription of proteasome subunit genes, resulting in
193 f a protein with a nuclear function to drive transcription of proteotoxic stress machinery genes.
194                                              Transcription of pth2 exhibited bidirectional dynamics;
195 solation specifically decreased the level of transcription of pth2, the gene that encodes the vertebr
196 pocyte proliferation, in vitro adipogenesis, transcription of Retn, and resistin secretion in 3T3-L1
197 ol and ammonia, DR1/NC2 indirectly regulated transcription of RhBG during ethanol and ammonia treatme
198 nd it was observed that CSA and CSB regulate transcription of ribosomal DNA (rDNA) genes and ribosome
199 nterferon-induced transcription, recovery of transcription of ribosomal proteins, and initiation of w
200 kines CXCL9/10/11, as well as a reduction in transcription of ribosomal proteins.
201 derived stress-induced small RNA (tiRNA) and transcription of ribosomal RNA (rRNA), respectively.
202 first-in-class inhibitor of RNA polymerase I transcription of ribosomal RNA genes (rDNA), induces rep
203 ed programmed synthesis of genes, controlled transcription of RNAs, and dictated transcription/transl
204                           AML1-ETO represses transcription of RUNX1 target genes by competitively dis
205           The group 3 NPR1 proteins regulate transcription of SA-responsive PR genes.
206 trophysiology, reveal that FOS activates the transcription of Scg2, a gene that encodes multiple dist
207  temporal pattern of early-pregnancy-induced transcription of select genes in PBMC and peripheral blo
208  revealed that CDK8 positively regulates the transcription of several ABA-responsive genes, probably
209                            JMJD1A influenced transcription of several genes that were also regulated
210 a transcriptional coactivator to enhance the transcription of several proinflammatory cytokines.
211 urn, P-TEFb relocates to chromatin to induce transcription of short units, including key DDR genes an
212 hat SM utilizes XPB to specifically activate transcription of SM target promoters.
213 r located in intron 1 was necessary to drive transcription of Snorc in the mouse, rat, and human.
214    Of note, MNT-MNT homodimers regulated the transcription of some genes involved in cell proliferati
215 hereby serving to either activate or repress transcription of specific genes involved in nickel homeo
216 tor complex that predominantly represses the transcription of stress-responsive genes in yeast.
217 otein can disrupt EBV latency by driving the transcription of target genes and by interacting with th
218              ABI3 can induce and repress its transcription of target genes directly and some intrigui
219     Wnt/beta-catenin signaling activates the transcription of target genes to regulate stem cells and
220 -catenin and promoting beta-catenin-mediated transcription of target genes, including Myc.
221                 These elements influence the transcription of target genes-many of which demonstrate
222 that binds DNA response elements to regulate transcription of target genes.
223 evelopment via epigenetically repressing the transcription of target genes.
224 phorylation of BumR and its ability to alter transcription of target genes.
225 ent, the response regulator, which activates transcription of target genes.
226 .3 mg.L(-1)) in culture GEO12CF enhanced the transcription of tceA to a statistically significant deg
227 he histone acetyltransferase EP300, enabling transcription of TGF-beta targets.
228                      We find that ERG drives transcription of the anticoagulant thrombomodulin (TM),
229 h Keap1 (FACS), Keap1-Nrf2 interactions, and transcription of the antioxidant response genes (immunof
230 presence of a target contaminant induces the transcription of the aptamer, and a fluorescent signal i
231 teins HPr and Enzyme I (EI) are required for transcription of the atxA gene, rather than phosphorylat
232 onal regulatory element, it will lead to the transcription of the barcode sequence, which is measured
233  not DNA methylation, underlie exon-specific transcription of the Bdnf gene induced by leptin.
234 s, C35-mediated TET inhibition activates the transcription of the BMP-SMAD-ID signaling pathway, whic
235  associated with the parent CDN leads to the transcription of the broccoli aptamer recognizing the DF
236 esponse regulator CusR together regulate the transcription of the cus operon that plays important rol
237 Cell exposure resulted in an increase in the transcription of the cytoprotective Hmox1 and pro-inflam
238 romoter, P (oah) , controls the constitutive transcription of the entire operon and a second promoter
239               The present findings show that transcription of the eutherian CHKB and CPT1B genes is l
240     Acyl-CoA binding of FadR derepresses the transcription of the fad genes and cancels fab gene tran
241 ns and creating a supercomplex that promotes transcription of the floral repressor FLOWERING LOCUS C
242 show in mice that ARID1A binds and regulates transcription of the Foxa2 gene required for endometrial
243 ative and cardiac disorder which occurs when transcription of the FXN gene is silenced due to an exce
244 patially restricted primordium via localized transcription of the G-protein-coupled receptor ligand F
245 tes its nuclear translocation, enhancing the transcription of the gasdermin C (GSDMC) gene.
246             Epigenetic mechanisms govern the transcription of the genome.
247 ncreases glucose import by up-regulating the transcription of the glucose transporter genes GLUT-1 an
248      Transcriptomic analyses reveal enhanced transcription of the HERVs in patients; meanwhile DNA-de
249 evel in CD4(+) T cells and did not drive the transcription of the IL10 promoter or putative local enh
250 h distinct time courses of Ca(2+) influx and transcription of the Il4 gene that were elicited by each
251 -5 were examined for stochastic behaviors in transcription of the lac operon.
252                                 Furthermore, transcription of the licT-blgPB operon was found to be r
253 roduced RNA species important, but also that transcription of the lncRNA locus alone can have regulat
254           PLT proteins and CUC2 regulate the transcription of the local auxin biosynthesis gene YUC4
255 naling cascade also positively regulates the transcription of the MCM6 gene that is involved in DNA r
256 Mechanistically, we demonstrate that zygotic transcription of the micro RNA miR-430 promotes degradat
257                                              Transcription of the MMP9 inhibitor TIMP1 was lower in H
258 B, two repressors of myogenesis that inhibit transcription of the myosin heavy chain (MHC) protein fa
259       These changes require vision-dependent transcription of the receptor Fn14 in thalamic relay neu
260 The synthesis of new ribosomes begins during transcription of the rRNA and is widely assumed to follo
261      Two different cell signals often affect transcription of the same gene.
262 ted that the SRF enhancer CArG box regulates transcription of the SRF gene, and mutation of this cons
263 ar these regulatory sequences did not affect transcription of the target gene.
264               Further, c2-HDA attenuated the transcription of the ToxT-dependent cholera toxin synthe
265                                              Transcription of the TTT-usp (4207) operon was induced i
266 e, requires several host factors for reverse transcription of the viral genomic RNA (gRNA) into DNA s
267    Retroviral infection involves the reverse transcription of the viral RNA genome into DNA, which is
268 prior to the normal MBT activated widespread transcription of the zygotic genome including genes prev
269 omosomes derived from Ae. longissima on gene transcriptions of the wheat landrace Chinese Spring.
270 ption by recruiting coactivators to initiate transcription of their target genes.
271 al cis-regulatory elements that activate the transcription of their target genes.
272                                     However, transcription of these 'coactivator-redundant' genes is
273                                We quantified transcription of these genes during growth on antimonate
274 ant for luminal cell fate, and supported the transcription of these genes in a catalytic-independent
275                                      Zygotic transcription of these genes largely retained features o
276 ing antibody secretion; furthermore, initial transcription of these loci requires the mTORC1 kinase a
277                            Understanding how transcription of these repeats occurs has implications f
278 enes in tumors and FGFR inhibition increased transcription of these same genes in cell culture models
279                                              Transcription of these sequences is tightly controlled d
280             In melanoma patients, restricted transcription of this endolysosomal vesicle pathway is a
281 er (TNBC), associates with SMAD3 to modulate transcription of transforming growth factor beta (TGFbet
282  Polymerase (Pol) III is specialized for the transcription of tRNAs and other short, untranslated RNA
283 tumoral effect by maintaining high levels of transcription of tumor suppressors that promote cell dea
284                                          The transcription of two dozen defence- and health-related g
285 1 and T-bet+ Th17 cells and reinforces their transcription of type 1 signature genes, including Tbx21
286 ukaryotic initiation factor 2alpha halts the transcription of uPA mRNA, leaving unopposed the deleter
287 corresponding TAD boundaries and reduces the transcription of upstream genes, while de novo insertion
288 es are repressed in phosphate-rich medium by transcription of upstream lncRNAs that interferes with a
289 as found to directly bind to and control the transcription of various mTOR and metabolism-related gen
290  accumulation of mutations and regulates the transcription of various oxidative stress-response genes
291 epatocyte and serves as the template for the transcription of viral mRNAs.
292     We conclude that iciHHV-6 results in the transcription of viral RNA in the human placenta and pre
293               The DPO-VqmA complex activates transcription of vqmR, encoding the VqmR small RNA, whic
294 gnaling represents a novel mechanism for the transcription of Wnt target genes and regulation of tumo
295  are beta-catenin cofactors that enhance the transcription of Wnt target genes.
296 stoma cells by epigenetically activating the transcription of Wnt target genes.
297    SUNO1 facilitates the cell-cycle-specific transcription of WTIP, a positive regulator of YAP1, by
298 red both YAP/TEAD-mediated transcription and transcription of YAP target genes in HepG2 and C2C12 cel
299 rm to Zur box operator sites, repressing the transcription of zinc uptake transporters.
300          However, when zinc levels are high, transcription of zrt1 is blocked in a manner that is dep

 
Page Top