戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 a leukemic phenotype through common modes of transcriptional dysregulation.
2 sect TF function in diseases associated with transcriptional dysregulation.
3 ads to increased chromatin accessibility and transcriptional dysregulation.
4 on and induces severe atrophy and widespread transcriptional dysregulation.
5 sion and chromatin binding, correlating with transcriptional dysregulation.
6 ernative therapeutic opportunities to target transcriptional dysregulation.
7 sion are not enriched in MG, indicating post-transcriptional dysregulation.
8  both genome-wide association study loci and transcriptional dysregulation.
9  production, loss of effector functions, and transcriptional dysregulation.
10 lopmental disability mediated by VPA-induced transcriptional dysregulation.
11 types, offering insights into miRNA-mediated transcriptional dysregulation.
12 tes reveals the features of age-related post-transcriptional dysregulation.
13 rinsic membrane excitability and ion channel transcriptional dysregulation.
14 AML and other diseases involving MOZ-induced transcriptional dysregulation.
15 activation augments miRNA induction and post-transcriptional dysregulation.
16 ino acid deficient media and exhibits global transcriptional dysregulation.
17 se chain reaction (PCR) were used to examine transcriptional dysregulation.
18 containing transcripts but do display global transcriptional dysregulation.
19 letion and unique profiles of DNA damage and transcriptional dysregulation.
20 eting, chromatin disruption, and ultimately, transcriptional dysregulation.
21 tional co-activators resulting in widespread transcriptional dysregulation.
22 cal effect of the expanded protein is due to transcriptional dysregulation.
23  to induce cytoplasmic neurodegeneration and transcriptional dysregulation.
24 rt their effect through a complex pattern of transcriptional dysregulation.
25 Although these syndromes are associated with transcriptional dysregulation,(24)(,)(28)(,)(30)(,)(31)(
26 ese structural variants are known to lead to transcriptional dysregulation across cancers, the extent
27      Remarkably, a high degree of sharing of transcriptional dysregulation across sex and modes of tr
28                  To what extent and how post-transcriptional dysregulation affects aging proteome rem
29              Therefore, this pattern of HCMV transcriptional dysregulation allows effective cytotoxic
30 both MSN and nNOS-IN, indicating that global transcriptional dysregulation alone does not account for
31                                              Transcriptional dysregulation and aberrant chromatin rem
32 observe clear progressive, striatal-specific transcriptional dysregulation and accumulation of neuron
33  HSCs lacking miR-29a/b-1 exhibit widespread transcriptional dysregulation and adopt gene expression
34                                      Indeed, transcriptional dysregulation and associated aberrant ep
35 s in vitro, leading to increased cell death, transcriptional dysregulation and cell-type-specific mol
36                                              Transcriptional dysregulation and cellular toxicity may
37 uced histone acetylation in the brain causes transcriptional dysregulation and cognitive impairment t
38                                Mechanisms of transcriptional dysregulation and cortical maldevelopmen
39 ighlight IKZF3 oncogenic function in CLL via transcriptional dysregulation and demonstrate that this
40    Genome-wide analyses identified extensive transcriptional dysregulation and DNA methylation change
41  Huntington's disease results in progressive transcriptional dysregulation and drives the loss of spi
42                                              Transcriptional dysregulation and loss of function of tr
43 egeneration correlating with donor survival, transcriptional dysregulation and pharmacological rescue
44 ization which, upon perturbation, results in transcriptional dysregulation and unleashes ERV2 proviru
45 Huntington's disease (HD) is associated with transcriptional dysregulation, and multiple studies with
46 ted with CHD8 suppression, i.e., genome-wide transcriptional dysregulation, and the reduction of H3K3
47 r, the molecular mechanisms underlying their transcriptional dysregulation are still unclear.
48  demonstrates that histone deacetylation and transcriptional dysregulation are two early, largely ind
49 2, a NRG3 risk polymorphism, suggesting NRG3 transcriptional dysregulation as a molecular mechanism o
50 oids leads to altered neural development and transcriptional dysregulation associated with downregula
51 rious intragenic variants of POU3F2 to cause transcriptional dysregulation associated with hyperphagi
52 e 22q11.2 microdeletion underlies widespread transcriptional dysregulation associated with psychiatri
53 D) in HD mice had a normalizing effect on HD transcriptional dysregulation associated with synaptic f
54 del enabled us to identify novel patterns of transcriptional dysregulation because of HD mutations, i
55                                         Post-transcriptional dysregulation becomes evident especially
56 , proteins with expanded polyglutamine cause transcriptional dysregulation before onset of symptoms,
57                                              Transcriptional dysregulation by mutant huntingtin (Htt)
58 anding the role of promoter-enhancer hubs in transcriptional dysregulation can provide insight into n
59 ated genomic regions, epigenetic mechanisms, transcriptional dysregulation, chemical modifications an
60                     Loss of DOT1L results in transcriptional dysregulation coinciding with the onset
61 irradiation, XP-D/CS cells displayed a gross transcriptional dysregulation compared with "pure" XP-D
62  of genes in which H3K9,14 deacetylation and transcriptional dysregulation concur.
63 he extensive Huntington's disease-associated transcriptional dysregulation, consistent with treatment
64 hould help elucidate the mechanisms by which transcriptional dysregulation contributes to neuronal dy
65 trate a role for Stag2 in transformation and transcriptional dysregulation distinct from its shared r
66 ional response that included modification of transcriptional dysregulation elicited by the Htt(Q111)
67  RNA sequencing analysis shows a reversal of transcriptional dysregulation following AAV5-miHTT in a
68                                              Transcriptional dysregulation has been described as a ce
69                                              Transcriptional dysregulation has been described in spin
70 ration in HD have not been fully elucidated, transcriptional dysregulation has been implicated in dis
71                                              Transcriptional dysregulation has been implicated in HD
72                                              Transcriptional dysregulation has been proposed to play
73                                              Transcriptional dysregulation has emerged as a core path
74                                              Transcriptional dysregulation has emerged as a critical
75                                              Transcriptional dysregulation has emerged as a potential
76                                     Although transcriptional dysregulation has not traditionally been
77 ving abnormalities of protein metabolism and transcriptional dysregulation have emerged as well.
78 rst demonstration of in vivo cell-autonomous transcriptional dysregulation in an HD mouse model.
79 erapies.Significance: Systematic analysis of transcriptional dysregulation in cancer cell lines and p
80 ther, these data shed light on mechanisms of transcriptional dysregulation in cancer, identify specif
81 d recapitulate mutant-specific mechanisms of transcriptional dysregulation in cancer.
82 n (LCM) study to examine the contribution of transcriptional dysregulation in candidate genes involve
83  development and underscore the risk of post-transcriptional dysregulation in co-occurring neurodevel
84 eptibility genes is imperative to understand transcriptional dysregulation in disease etiology.
85 sed disease modelling we identified specific transcriptional dysregulation in first and second heart
86 d inclusions (FTLD-U), suggesting a role for transcriptional dysregulation in FTLD-U pathophysiology.
87              Identifying these regulators of transcriptional dysregulation in HD can be leveraged to
88 le and Hdac2 with implications for targeting transcriptional dysregulation in HD.
89 ractions between Htt and MeCP2 contribute to transcriptional dysregulation in HD.
90            Together, these results show that transcriptional dysregulation in hippocampal interneuron
91 elevant mutation causes synapse deficits and transcriptional dysregulation in human neurons and our f
92 htt to accumulate in the nucleus, leading to transcriptional dysregulation in Huntington disease (HD)
93                                              Transcriptional dysregulation in Huntington's disease (H
94                          There is widespread transcriptional dysregulation in Huntington's disease (H
95                            We show here that transcriptional dysregulation in interneurons, particula
96                                              Transcriptional dysregulation in interneurons, particula
97 d enhancer architecture is a major driver of transcriptional dysregulation in MED12 mutant uterine le
98            Complex diseases are mediated via transcriptional dysregulation in multiple tissues.
99 rus-mediated Elk-1 overexpression alleviated transcriptional dysregulation in R6/1 mice.
100 eal that ERV-derived enhancers contribute to transcriptional dysregulation in response to oncogenic s
101  mice restores PV-IN density and ameliorates transcriptional dysregulation in S1, but not circuit dys
102 d exerts a deleterious effect through remote transcriptional dysregulation in specific progenitor sub
103                     In order to identify the transcriptional dysregulation in Tbx1-expressing lineage
104 enic risk is plausibly manifested by complex transcriptional dysregulation in the brain, involving ne
105 eping genes in PS-deficient MEFs, indicating transcriptional dysregulation in these cells.
106  basis for, and etiological significance of, transcriptional dysregulation in this context is lacking
107  for the first time, identified robust early transcriptional dysregulation in unipolar brush cells an
108  ChIP-seq target genes, explained 42% of the transcriptional dysregulation in WS neurons.
109        In addition, Actn2 mutation triggered transcriptional dysregulation, including abnormal expres
110 ex, results in reduced H3K27me3 and profound transcriptional dysregulation, including that of a set o
111  is associated with increased cell death and transcriptional dysregulation indicative of an inflammat
112                                              Transcriptional dysregulation induced by aberrant transc
113          The pattern of immunomodulation and transcriptional dysregulation induced by the n-alkanol s
114                                              Transcriptional dysregulation is a central pathogenic me
115                                     Although transcriptional dysregulation is a critical pathogenic m
116                                              Transcriptional dysregulation is a hallmark of cancer an
117                                              Transcriptional dysregulation is a key step in oncogenes
118 echanisms of toxicity are poorly delineated, transcriptional dysregulation is a likely contributor.
119 ethylation in Huntington disease (HD), where transcriptional dysregulation is a major factor in patho
120                                              Transcriptional dysregulation is a pivotal feature of HD
121                                              Transcriptional dysregulation is an early feature of Hun
122            Convergent evidence suggests that transcriptional dysregulation is an important component
123                                              Transcriptional dysregulation is an important early feat
124  of polyglutamine toxicity and indicate that transcriptional dysregulation is an important part of th
125                                              Transcriptional dysregulation is cell type-specific, and
126                                         IL-6 transcriptional dysregulation is commonly seen in patien
127                         Furthermore, whereas transcriptional dysregulation is limited to genes within
128            A considerable proportion of this transcriptional dysregulation is specifically caused by
129 n early component of polyglutamine toxicity, transcriptional dysregulation, is conserved in yeast and
130 als to birth and beyond, despite substantial transcriptional dysregulation, is consistent with mammal
131  after 24 months of age and age-related post-transcriptional dysregulation leads to accumulation of c
132 trates a distinct disease mechanism by which transcriptional dysregulation leads to an inborn error o
133 e Lange Syndrome, in which global yet subtle transcriptional dysregulation leads to development of at
134                                              Transcriptional dysregulation may also be involved in po
135 haperones and suggest that context dependent transcriptional dysregulation may contribute to differen
136 utation impairs DNA binding, suggesting that transcriptional dysregulation may contribute to the phen
137                                        Thus, transcriptional dysregulation might be an important path
138 xpression analysis of infected hNPCs reveals transcriptional dysregulation, notably of cell-cycle-rel
139 tions of TSC2 demonstrated hyperactivity and transcriptional dysregulation observed in cortical tuber
140                                 We show that transcriptional dysregulation occurs early during develo
141  To determine whether polyglutamine-mediated transcriptional dysregulation occurs in yeast, we expres
142 e multi-omics data revealed that UVR-induced transcriptional dysregulation of a subset of genes was a
143        This correlated with the sex-specific transcriptional dysregulation of a wide range of metabol
144 nflammatory responses in RAI mice, caused by transcriptional dysregulation of AP-1 and NF-kappaB, and
145            These effects are associated with transcriptional dysregulation of autism-associated genes
146          Offspring from old fathers also had transcriptional dysregulation of developmental genes imp
147  abnormal histone ubiquitination and lead to transcriptional dysregulation of developmental genes.
148 ate inferences to the functional effects and transcriptional dysregulation of driver mutations.
149 ubunits in primary myometrium cells leads to transcriptional dysregulation of extracellular matrix as
150  formation of the majority of lipofuscin and transcriptional dysregulation of genes associated with i
151 tion and increased apoptosis associated with transcriptional dysregulation of genes implicated in car
152 pairment through mechanisms dependent on the transcriptional dysregulation of genes required for memo
153 emporal lobe epilepsy (TLE), suggesting that transcriptional dysregulation of HCNs might contribute t
154   In summary, endothelial TET2 loss leads to transcriptional dysregulation of interferon-regulated ge
155 k analyses demonstrate prominent MIA-induced transcriptional dysregulation of mTOR and EIF4E-dependen
156 ts cause histone acetylation deficiency with transcriptional dysregulation of multiples genes, thereb
157                                              Transcriptional dysregulation of Ninj2 and distal genes
158                          They also implicate transcriptional dysregulation of OVOL2 as a common cause
159 n disrupts lysosomes in neurons and leads to transcriptional dysregulation of ribosomal protein genes
160                                              Transcriptional dysregulation of SCC cells is orchestrat
161 ell line and may represent the major site of transcriptional dysregulation of TGF alpha promoter acti
162   These findings emphasize the importance of transcriptional dysregulation of the autoantigen in auto
163 e hypothalamus, respectively, results in the transcriptional dysregulation of the circadian clock and
164      Lastly, we investigated tissue-specific transcriptional dysregulation of the core genes in two i
165   Since somatic alterations or mutations and transcriptional dysregulation of the FOXO genes are infr
166                                              Transcriptional dysregulation of the IL-10 gene may cont
167 ) mice to investigate effects of interneuron transcriptional dysregulation on the dynamics of the I/E
168                               Epigenetic and transcriptional dysregulation plays a fundamental role i
169                                              Transcriptional dysregulation plays a major role in the
170                                              Transcriptional dysregulation, possibly affected by gene
171 cated in Alzheimer's disease (AD), including transcriptional dysregulation, protein misprocessing and
172 st striatum-selective nuclear inclusions and transcriptional dysregulation resembling those in murine
173 sults, many of which molecularly converge on transcriptional dysregulation, resulting in altered syna
174       This study provides evidence that this transcriptional dysregulation results in a variety of mR
175 ten similar, including protein accumulation, transcriptional dysregulation, somatic CAG repeat instab
176 ion, while also uncovering infection-induced transcriptional dysregulation that contributes to the in
177  is a heterogeneous disease characterized by transcriptional dysregulation that results in a block in
178 ma2 signaling leads to a shared signature of transcriptional dysregulation that underlies these pheno
179                                         This transcriptional dysregulation was associated with an imp
180                                              Transcriptional dysregulation was evaluated using RNA se
181 hymic hematopoietic progenitor cells induces transcriptional dysregulation, which activates a lymphoi
182                                We identified transcriptional dysregulation with 1246 (8%) differentia
183 rbalpha drives only modest physiological and transcriptional dysregulation, with derepressed target g
184                                              Transcriptional dysregulation within PB lesions mirrored
185             Previous studies have implicated transcriptional dysregulation within the hippocampus as
186                                Restoring the transcriptional dysregulation without affecting other AR

 
Page Top