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1           RCM images were taken to visualize transepidermal application of topically-applied zinc.
2 ectance confocal microscopy (RCM) to compare transepidermal application of zinc in sensitive and non-
3  vivo studies, E-cadherin expression and the transepidermal delivery (TED) of human epidermal growth
4     However, the ability of LICAP to enhance transepidermal delivery in sub-cytotoxic conditions has
5 nvestigate the effects of LICAP treatment on transepidermal drug delivery (TED) and mechanisms using
6                                              Transepidermal inoculation of vaccinia virus (VV), or sc
7 be a sampling tool with a depth component, a transepidermal microprojection array (MPA), which captur
8 y demonstrated that LICAP treatment enhances transepidermal permeation of hEGF, apparently via both p
9 meters (time and concentration) for PARPi-FL transepidermal permeation through intact skin were succe
10  fetal rats displayed no measurable barrier (transepidermal water loss > 10 mg per cm2 per h), a meas
11 children had higher lesional and nonlesional transepidermal water loss (both P < .001) as well as dec
12 cantly improved skin hydration and decreased transepidermal water loss (NCT04253704).
13 asol, respectively, with parallel changes in transepidermal water loss (P < .05).
14  .03) and increased lesional and nonlesional transepidermal water loss (P = .01, P = .03).
15 r associations with skin filaggrin (FLG) and transepidermal water loss (TEWL) (assesses skin barrier
16                                 The lip skin transepidermal water loss (TEWL) and capacitance of CC p
17 ] and urocanic acid [UCA]) using UPLC-MS/MS, transepidermal water loss (TEWL) and epidermal pH.
18 ipid barrier as measured by skin resistance, transepidermal water loss (TEWL) and Fourier transform i
19                                              Transepidermal water loss (TEWL) and levels of epidermal
20            The effects were quantified using transepidermal water loss (TEWL) and were correlated wit
21 determine whether eczema, dry skin, and high transepidermal water loss (TEWL) at 3 months were associ
22 ering techniques of laser Doppler imaging, a transepidermal water loss (TEWL) device and a skin therm
23  We have evaluated the content of lipids and transepidermal water loss (TEWL) in lesional and non-les
24 his methylation is associated with increased transepidermal water loss (TEWL) in risk allele carriers
25                                              Transepidermal water loss (TEWL) is a measure of skin ba
26 ough parameters like skin hydration (SH) and transepidermal water loss (TEWL) is vital for diagnosing
27 tional Endpoints (BASELINE) birth cohort had transepidermal water loss (TEWL) measured in the early n
28                                              Transepidermal water loss (TEWL) measurement provides a
29 by electric impedance spectroscopy (EIS) and transepidermal water loss (TEWL) measurements after the
30 ing test (LAST):'stinger' and 'non-stinger'; transepidermal water loss (TEWL) measurements; and sensi
31                                              Transepidermal water loss (TEWL) measures were collected
32 healing endpoint and recurrence by measuring transepidermal water loss (TEWL) post-closure at the sit
33 rthermore, to determine whether increases in transepidermal water loss (TEWL) predate the development
34                      Significantly increased transepidermal water loss (TEWL) was found in MRSA-colon
35 were applied to the upper arm; impedance and transepidermal water loss (TEWL) were measured at baseli
36                                       EI and transepidermal water loss (TEWL) were measured before an
37                                      EIS and transepidermal water loss (TEWL) were measured in lesion
38  of the skin and measurements of the rate of transepidermal water loss (TEWL) were recorded sequentia
39                                         RCM, transepidermal water loss (TEWL), and fluorescence excit
40  function, impaired skin barrier function by transepidermal water loss (TEWL), eczema, and filaggrin
41        We measured water flux across the SC, transepidermal water loss (TEWL), in six women, in vivo.
42 rneum was assessed by measuring capacitance, transepidermal water loss (TEWL), rates of absorption-de
43 sure in wild-type and Serpinb3a-null mice on transepidermal water loss (TEWL), sensitization, and inf
44 e associated with atopic dermatitis (AD) and transepidermal water loss (TEWL).
45 n barrier function was assessed by measuring transepidermal water loss (TEWL).
46 versely correlated (r=-0.654, P=0.0004) with transepidermal water loss (TEWL).
47 ion examining the permeability barrier using transepidermal water loss (TEWL).
48  recovery of barrier function as measured by transepidermal water loss after tape stripping.
49  the delayed barrier recovery as measured by transepidermal water loss after tape-stripping.
50 unction, as measured by corneometry, pH, and transepidermal water loss also normalized with treatment
51                                              Transepidermal water loss and a(*) (skin redness) decrea
52                                              transepidermal water loss and allergic sensitization wer
53  STS proteins was highly correlative to skin transepidermal water loss and allergic sensitization.
54 rier development, evidenced by a decrease in transepidermal water loss and an enhanced outside-in bar
55            We assessed here, first, baseline transepidermal water loss and barrier recovery kinetics
56                     These deposits increased transepidermal water loss and caused irritation, particu
57                    Here, we report increased transepidermal water loss and compromised expression of
58           This was associated with increased transepidermal water loss and development of eczematous
59  skin assessments, including improvements in transepidermal water loss and disease severity.
60 interactions with the TGM1 genotype included transepidermal water loss and emollient and retinoid use
61 of the epidermis associated with a decreased transepidermal water loss and increased proinflammatory
62 oms, accompanied by significant decreases in transepidermal water loss and increases in water content
63  skin barrier dysfunction leads to increased transepidermal water loss and inflammation.
64          Bacterial diversity correlated with transepidermal water loss and pH level but not with corn
65 to barrier disruption, indicated by elevated transepidermal water loss and reduced lipid synthesis en
66 S1pr2(-/-) mouse showed significantly higher transepidermal water loss and required another 24 hours
67  was to investigate the relationship between transepidermal water loss and skin permeability to triti
68 certained using confocal Raman spectroscopy; transepidermal water loss and skin surface pH were measu
69 more prematurely have elevated rates of both transepidermal water loss and transcutaneous heat loss,
70 on of the barrier function of skin increases transepidermal water loss and up-regulates inflammatory
71 44, P = .0006) and skin barrier dysfunction (transepidermal water loss area under the curve r = 0.31,
72 d to interpret the significance of measuring transepidermal water loss by evaporimetry.
73                     These data indicate that transepidermal water loss cannot be unconditionally ascr
74 ximately 0.5 U); (ii) enhanced SC integrity (transepidermal water loss change with sequential tape st
75              Measured in ambient conditions, transepidermal water loss did not signal disease risk at
76                 We then assessed skin pH and transepidermal water loss every 12 hours on the burn wou
77 lents in SCID/NOD mice demonstrated enhanced transepidermal water loss following s.c. administration
78                     Previous measurements of transepidermal water loss have suggested that, regardles
79 arrier physiology, as indicated by increased transepidermal water loss in obese animals.
80  active plaque phenotypes displayed elevated transepidermal water loss levels, increased numbers of e
81 required another 24 hours to normalize their transepidermal water loss levels.
82 quency impedance spectroscopy, to complement transepidermal water loss measurements.
83 lg(-/-) neonates showed little alteration in transepidermal water loss or lipid- or corneocyte-relate
84 tic administration of all activators tested (transepidermal water loss range 4.0-8.5 mg per cm2 per h
85             There was no correlation between transepidermal water loss rate and 3H2O permeability fol
86  Similarly, there was no correlation between transepidermal water loss rates and the 3H2O permeabilit
87       No correlation was found between basal transepidermal water loss rates and the permeability of
88                                  While basal transepidermal water loss rates are near normal, barrier
89                                              Transepidermal water loss rates declined during explant
90                             Whereas baseline transepidermal water loss rates were elevated by approxi
91 orneocytes and its contribution to increased transepidermal water loss was confirmed by tape strippin
92                                              Transepidermal water loss was measured on unaffected for
93 od samples were assessed for hormone levels, transepidermal water loss was measured to assess skin ba
94                                              Transepidermal water loss was normal in all conditions.
95                   In an acute AD model, skin transepidermal water loss was significantly attenuated i
96 y normal, and exhibited an increase in basal transepidermal water loss without alteration in basal mi
97 se curve, with maximal sweating (measured as transepidermal water loss) (mean 70 g m(-2) hour(-1)) af
98 ssessed, and skin physiology (pH, hydration, transepidermal water loss) was measured.
99 rations, skin barrier dysfunction (increased transepidermal water loss), and AD in infancy.
100                                              Transepidermal water loss, a functional barrier measure,
101                        Our study showed that transepidermal water loss, a parameter of skin barrier i
102  KLK5 activity in TgKLK5 mouse skin, reduced transepidermal water loss, and decreased proinflammatory
103 l epidermis, corneocyte fragility, increased transepidermal water loss, and local inflammation in the
104  measurements of percentage of skin redness, transepidermal water loss, and participant-assessed pain
105 permeability indicated by higher pH, greater transepidermal water loss, and reduced lipid synthesis e
106         Changes in total sign scores (TSSs), transepidermal water loss, and tissue biomarkers (determ
107 phils, cutaneous expression of Il4 and Il13, transepidermal water loss, antigen-specific IgE antibody
108 ing IL-13Ralpha2 had significantly increased transepidermal water loss, cutaneous inflammation, perip
109 mal permeability barrier defects with severe transepidermal water loss, decreased intercellular lipid
110 d in Ric(EKO) mice, as revealed by increased transepidermal water loss, enhanced corneocyte fragility
111 on, and cytokine production were measured by transepidermal water loss, histopathology, molecular bio
112 m, with SerpinB2(-/-) mice showing increased transepidermal water loss, increased overt loss of strat
113 hat cutaneous barrier formation, measured as transepidermal water loss, is delayed in male fetal rats
114 sebopsoriasis displayed a lesser increase in transepidermal water loss, normal numbers of lamellar bo
115  present in these dogs, as well as increased transepidermal water loss, particularly in sites charact
116  exercise, on several dermatologic measures: transepidermal water loss, recovery of skin barrier func
117 re), Scoring for Atopic Dermatitis (SCORAD), transepidermal water loss, skin filaggrin (FLG) expressi
118 e markers also significantly correlated with transepidermal water loss, suggesting a link between the
119  by reduced transglutaminase (TGM) activity, transepidermal water loss, up-regulation of the proinfla
120 nce analysis to measure hydration status and transepidermal water loss, we show that the epidermal ba
121 ith severe dehydration, suggesting excessive transepidermal water loss, which was confirmed by in vit
122 ion mutations in skin barrier genes increase transepidermal water loss.
123 lated with the SC water content but not with transepidermal water loss.
124        Skin barrier function was measured by transepidermal water loss.
125 n barrier function as evidenced by increased transepidermal water loss.
126 robial activity, paracellular diffusion, and transepidermal water loss.
127 ells in blood, correlating with severity and transepidermal water loss.
128 primary cell population responsible for high transepidermal water loss.
129 oglobulin levels, skin symptom scores, or on transepidermal water loss.
130 id envelope and die shortly after birth from transepidermal water loss.
131 icle-treated animals), assessed as increased transepidermal water loss.
132 tures improve barrier repair, as assessed by transepidermal water loss.
133 es (body surface area/BSA, pruritus ADQ, and transepidermal water loss/TEWL) with immune and barrier
134 ates erythema (IASI-E) and scaling (IASI-S); transepidermal water loss; and pruritus.
135 ut allergy (PA) is associated with increased transepidermal water loss; low urocanic acid (UCA) and p
136 ut allergy (PA) is associated with increased transepidermal water loss; low urocanic acid (UCA) and p

 
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