ライフサイエンスコーパス: transfectant

1 of HepG2, but no significant change in Hep3B transfectant.

2 eta transfectant and Hep3B-p53/GADD45beta co-transfectant.

3 had serum Abs that brightly labeled the MOG transfectant.

4 system and in CXCR4-expressing human HEK293 transfectants.

5 was reduced by approximately 70% in HAS1-AS transfectants.

6 ely 60% less invasive than vector and HAS1-S transfectants.

7 oli LPS and further decreased in polymorphic transfectants.

8 alveolar macrophages, and DC-SIGN-expressing transfectants.

9 tosis of CXCR3 using T lymphocytes and CXCR3 transfectants.

10 L-8 cDNA and selected IL-8-secreting (IL8-S) transfectants.

11 eping genes (e.g. Cl alpha-tubulin) in these transfectants.

12 PSA and AR levels, when compared with vector transfectants.

13 y protected LPA(2) but not LPA(1) and LPA(3) transfectants.

14 otein and mRNA were also reduced in myocilin transfectants.

15 duced mtDNA damage compared with vector-only transfectants.

16 n enhanced shedding of meprin beta from both transfectants.

17 8 at significantly lower rates than did K1m transfectants.

18 to enhance levels of active p38alpha/beta in transfectants.

19 were > or =2-fold lower than those in vector transfectants.

20 ts but decreased IL-4 production by SD VPAC2 transfectants.

21 prin beta and meprin A in the medium of both transfectants.

22 K1-Tr transfectants and BCCs, but not NK1-FL transfectants.

23 the parental or the control RNA interference transfectants.

24 en cyproterone acetate was abolished in ebp1 transfectants.

25 e kinase activity also decreased in HYAL1-AS transfectants.

26 igand, in four independently derived MCA-205 transfectants.

27 Rbeta but not ERalpha was expressed in A2780 transfectants.

28 by the delayed entry into S phase by the RII transfectants.

29 sitivity of the wild type and drug-resistant transfectants.

30 nding activity compared with wild-type STAT6 transfectants.

31 onal and a binding response to LTE4 in these transfectants.

32 rkedly impaired zinc transport in G87R ZnT-2 transfectants.

33 Geminin, and Cdt1 were increased in v-K-ras transfectants.

34 mulate interleukin-8 (IL-8) release from TLR transfectants.

35 s on monocyte/macrophages and FcgammaRIII(+) transfectants.

36 to a greater extent than control HLA class I transfectants.

37 tudies of enhanced GFP-tagged RelA in stable transfectants.

38 eration, migration and invasion in sprouty-2 transfectants.

39 nhibitor when pfht is overexpressed in these transfectants.

40 mutants rapidly and reproducibly emerged in transfectants.

41 ed epithelial Madin-Darby canine kidney cell transfectants.

42 esponse to leukotriene (LT) D(4) with stable transfectants.

43 uronidase activity when compared with vector transfectants (18-24 milliunits).

44 In mouse corticotroph EGFR transfectants, ACTH secretion was enhanced, and EGF incr

45 Z/ZAP transfectants activated through TCRs underwent a faster

46 partially inhibited in the Lyn unique domain transfectants after Ag stimulation.

47 gene, was among these 11 genes found in the transfectants after CP selection.

48 ith the exception of the full-length Jagged1 transfectant, all other cell lines, including the contro

49 SCC-S2 transfectants also displayed an increase in cell migrati

50 and revealed a decrease in HepG2-GADD45beta transfectant and Hep3B-p53/GADD45beta co-transfectant.

51 ts in calcium flux and Transwell CC receptor transfectant and monocytic THP-1 migration assays.

52 ar patterns of cytokine production by NK1-Tr transfectants and BCCs, but not NK1-FL transfectants.

53 control conditions, Cx36 GJ channels in HeLa transfectants and beta-cells are inhibited by endogenous

54 The invasive capacity of L cell transfectants and cultured fibroblast-like synoviocytes

55 ial depolarization when compared with vector transfectants and expressed activated proapoptotic prote

56 ivates the NF1 promoter in myeloid cell line transfectants and identify an ICSBP-binding NF1 cis elem

57 n of endotoxin tolerance in HEK293/TLR4/MD-2 transfectants and in human monocytes markedly suppressed

58 rmed by binding of soluble BTN2A1 to DC-SIGN-transfectants and its inhibition by a specific Ab.

59 Previous studies with fibroblast transfectants and naturally sensitive Caco-2 cells have

60 blocked T. cruzi infection of the TrkC-NNR5 transfectants and of cells that naturally express TrkC.

61 blocked T. cruzi infection of the TrkC-NNR5 transfectants and of naturally permissive TrkC-bearing S

62 plectin levels in astrocytoma-derived stable transfectants and plectin-positive fibroblasts.

63 induction of transcription by I kappa BNS in transfectants and prevents association of I kappa BNS wi

64 icant augmentation of IL-8 release from both transfectants and primary FLS.

65 mAb-mediated cross-linking of CD161, in both transfectants and primary human NK cells, triggers the a

66 method was confirmed by analysis of KIR3DS1 transfectants and the identification of a novel KIR3DS1

67 hosphorylation, and cell motility of FasL(+) transfectants and tumor cells.

68 erpart, was expressed on the surface of 293T transfectants and was able to bind ligand, but exhibited

69 ew four times slower than vector and HYAL1-S transfectants and were blocked in the G2-M phase of the

70 with small interfering RNA in MCF-7/HRPAP20 transfectants and wild-type MDA-MB-231 cells reduced inv

71 ed after Ag stimulation of Lyn unique domain transfectants, and Ag-induced release of histamine was i

72 ient Abs in Western blots of T cells, S1P(1) transfectants, and S1P(1)-hemagglutinin purified by mono

73 ctants generated more IL-4 than did SD VPAC2 transfectants, and this increment was dependent on endog

74 gration in collagen I as compared to control transfectants ( approximately twofold; n=3; P<0.005).

75 idermal JB6 P+ Cl41 cell line and its stable transfectants as well as on several human tumor cell lin

76 Moreover, only the b(0,+)AT-rBAT transfectants became selectively intoxicated during expo

77 n techniques, including production of stable transfectants; biochemical and other assays of pure RGC-

78 When compared with control vector transfectants, breast cancer cells stably overexpressing

79 rface expression was not only established in transfectants but also confirmed by observations of much

80 urther increased IL-4 production by WT VPAC2 transfectants but decreased IL-4 production by SD VPAC2

81 largely derived from these domains in ABCG1 transfectants but not in cells lacking ABCG1.

82 K1 transfectants but not K1m transfectants exhibited reduce

83 exogenous Ag presentation by wild-type CD1d transfectants, but did not affect NKT autoreactive respo

84 We found that the injection of CD40L transfectants, but not mock cells, resulted in VEGF expr

85 DEC-205-expressing MPhis and transfectants, but not their negative counterparts, phag

86 l lines containing NKTs primed with CD1d+C1R transfectants by flow cytometry and ELISA, respectively.

87 tion of the mutants was assayed in transient transfectants by measurement of [(3)H]substrate influx a

88 rRV strains, as well as MA104-derived stable transfectant cell lines expressing either wild-type NSP5

89 proliferation and invasion of GD3-expressing transfectant cells (GD3+).

90 Exposure of the transfectant cells to TRAIL leads to the recruitment of

91 iated with the integrin alpha6 in Du145-CD82 transfectant cells, suggesting that the formation of the

92 Finally, K1 transfectants cleaved procaspase 8 at significantly lowe

93 d to lower levels of adherence to VECs by AS-transfectants compared to control organisms.

94 ependent kinase (cdk) inhibitor in PKC-delta transfectants compared with empty vector (EV) transfecte

95 tent with accelerated apoptosis in PKC-delta transfectants, compared to EV cells, PKC-delta upregulat

96 Stable U2OS transfectants containing a deletion of the ERalpha activ

97 In contrast, although activated Z/Syk transfectants could increase FasL expression, their Fas

98 In HYAL1-v1 transfectants, cyclin B1, cdc2/p34, and cdc25c levels we

99 with vector-alone control cells, BLU stable transfectants, derived from poorly-differentiated NPC HO

100 In contrast, c-Cbl knockdown stable transfectants differentiated slower than wt cells when t

101 IL8-S transfectants displayed a 3- to 5-fold increased motilit

102 Myocilin transfectants displayed a heightened sensitivity to tryp

103 HS6ST1 transfectants displayed a relative increase in mono-6-O-

104 grew significantly more slowly than control transfectants (DLD-1-V) under normal culture conditions

105 AhR overexpression in stable transfectants downregulated Oct4 and also decreased ALDH

106 The four mutant transfectants each had significantly lower levels of glu

107 Stable transfectants ectopically expressing c-Cbl underwent mye

108 All three transfectants effectively bound Lkt.

109 y 4- to 7-fold, whereas high HYAL1 producing transfectants either did not form tumors (DU145) or grew

110 Human T cells expressing endogenous CCR4 and transfectants engineered to express CCR4 were assessed f

111 AS-CAR transfectants exhibit a profound loss in the ability to

112 K1 transfectants but not K1m transfectants exhibited reduced levels of apoptosis indu

113 tumors, but all animals implanted with Hyal1 transfectants exhibited tumor-positive para-aortic lymph

114 Control SHEP cells and ILK transfectants express high levels of ILK and cav-1.

115 Although HYAL1-v1 transfectants expressed equivalent levels of enzymatical

116 the ability to bind or respond to CPE, while transfectants expressing a Claudin-1 mutant with the cor

117 rsely, CPE bound to, and killed, CPE-treated transfectants expressing a Claudin-2 chimera with a subs

118 Rat fibroblast transfectants expressing a Claudin-4 chimera, where the

119 Transfectants expressing a Claudin-4(N149D) mutant lost

120 ata for laboratory strains, as well as pfcrt transfectants expressing a CQR conferring mutant pfcrt g

121 present study we show that N15alphabeta TCR transfectants expressing a FG loop-deleted chain (betaDe

122 ADAM15-transfected T/C28a4 cells, but not in transfectants expressing an ADAM15 mutant without the cy

123 ignal peptide and abrogation of cytolysis of transfectants expressing bovine CD18 carrying the Q(-5)G

124 Leukotoxin specifically lysed transfectants expressing bovine CD18 fragment encompassi

125 Analysis of transfectants expressing CD18 containing the FLAG epitop

126 D44-deficient COS-7/M6 cell to create stable transfectants expressing CD74, CD44, and a truncated CD4

127 LPA equally protected B82L cells, or transfectants expressing EGFR, the kinase dead EGFR(K721

128 d signaling molecules JAK2 and AKT in HEK293 transfectants expressing EPOR and CD131.

129 Analyses using blocking mAbs and Ba/F3 transfectants expressing gp130 indicate that EBI3 activi

130 dividually, and compared these cells with co-transfectants expressing Hyal1 + HAS2 or Hyal1 + HAS3.

131 Using stable CACO-2 transfectants expressing keratin 8 (K8) antisense RNA un

132 C320A transfectants expressing low molecular weight PSGL-1 had

133 ubcellular fractionation of stable HeLa cell transfectants expressing mEGFP-huPSS-1, we found that HC

134 g to cellular activation, was seen only with transfectants expressing monomeric bovine CD18 or LFA-1.

135 Lkt-induced cytolysis was observed only with transfectants expressing monomeric bovine CD18 or LFA-1.

136 Transfectants expressing mutant Arg(27)Ala STAT6 display

137 transfectants is diminished in comparison to transfectants expressing only AQP4.

138 Higher levels are seen for 221-cell transfectants expressing Patr-AL, but in these cells a l

139 L cell transfectants expressing SE-positive DR molecules on the

140 In two stable transfectants expressing survivin-specific short hairpin

141 In contrast, transfectants expressing the DeltaEx10 transcript displa

142 Transfectants expressing the full-length transcript had

143 Incubation with transfectants expressing the HLA-G homodimer (but not wi

144 expressing the HLA-G homodimer (but not with transfectants expressing the HLA-G monomer) resulted in

145 We generated T. cruzi transfectants expressing the N-terminal 24 or 12 amino a

146 iated internalization of E. coli by CHO cell transfectants expressing these receptors.

147 Conversely, in transfectants expressing tyrosinase mutants that are com

148 lysis of 10,000 human genes, comparing BT549 transfectants expressing wild-type and those expressing

149 HEK293T transfectants expressing wild-type or polymorphic TLR4 w

150 We generated stable transfectants-expressing wild-type, K303R ERalpha or a d

151 7 dimer protein and tetramers stained LILRB2 transfectants five times more strongly than B27 heterotr

152 hese compounds were screened in HEK293T-TLR2 transfectants for the ability to inhibit TLR2-mediated I

153 Sprouty-2 transfectants formed significantly larger tumor xenograf

154 WT VPAC2 transfectants generated more IL-4 than did SD VPAC2 tran

155 HYAL1-v1 transfectants grew 3- to 4-fold slower due to cell cycle

156 HAS1-AS transfectants grew 5-fold slower and were approximately

157 Compared with the vector control clone, DcR3-transfectants grew faster and resulted in TAM infiltrati

158 HYAL1-AS transfectants grew four times slower than vector and HYA

159 d approximately 1.7-fold more HA than vector transfectants, HA production was reduced by approximatel

160 HYAL1-AS transfectants had a G2-M block due to decreased cyclin B

161 In contrast to RII transfectants, HCT116 cells transfected with chromosome

162 uman chronic lymphocytic leukemia cells, and transfectant HEK293 cells.

163 144E acted as dominant-negative mutations in transfectants, homozygosity for A144E in mice resulted i

164 s of micrometastases in the brain as control transfectants; however, the Her-2 transfectants yielded

165 tants produced 3-fold more HYAL1 than vector transfectants, HYAL1-AS transfectants showed approximate

166 ontaneous lymph node metastasis in all Hyal1 transfectant-implanted mice, and node burden increased a

167 me profiling of wild-type and variant CARD14 transfectants in keratinocytes differentiated probably p

168 COX-2 stable transfectants in LN-18 (LN-18-COX2) also induced a Tr1 r

169 roximately 50% less invasive than the vector transfectants in vitro.

170 protein expression were determined in stable transfectants in vivo.

171 have generated and characterized stable DT40 transfectants in which both topo 2 genes have been in si

172 ultiple proinflammatory genes relative to WT transfectants, including genes for pentraxin 3, granuloc

173 studied in VPAC2-low D10G4.1 model Th2 cell transfectants individually expressing the respective typ

174 d osteolysis, whereas DU145 MT1-MMP-silenced transfectants induced osteogenic changes.

175 CA2 expression in Skp2-overexpressing stable transfectants inhibited the migratory and growth propert

176 human CD40L-transfected fibroblasts or mock transfectants into human skin on SCID mice.

177 says indicated that the delayed entry of RII transfectants into phase was associated with markedly re

178 T cell transfectants maintained exquisite hemoglobin peptide sp

179 enhanced tumor growth as compared to control transfectants (mean tumor volumes, day 16: control, 56.8

180 We rescued transfectant mutant viruses by reverse genetics and exam

181 f LdASNA could not be obtained as the double transfectant mutants showed aneuploidy.

182 l expression occurring at about 36 h; stable transfectants normally can be generated within three or

183 rved in the lower metastatic antisense Ezrin transfectant of a murine osteosarcoma model system, conf

184 ematopoietic diseases, we generated a stable transfectant of Ba/F3 cells expressing EpoR and analyzed

185 t assay, which revealed a decrease to 40% in transfectant of HepG2, but no significant change in Hep3

186 was confirmed by induction of a stable Mirk transfectant of Mv1Lu cells, which blocked cell migratio

187 In this study, we characterized stable transfectants of A2780 ovarian carcinoma cells.

188 Stable transfectants of baby hamster kidney (BHK) epithelial ce

189 Both BHMT transfectants of HepG2 cells and primary mouse hepatocyt

190 sted that HCV core gene expression in stable transfectants of Huh-7 cells resulted in a basal up-regu

191 n in vivo tumorigenesis, we generated stable transfectants of Huh7 cells overexpressing either MAT1A

192 Using stable transfectants of human bronchial epithelial cells, RNA e

193 anscription factor TBX2, we generated stable transfectants of human embryonic kidney cells (293) that

194 We then isolated stable transfectants of Hut102/6TG cells that express the T1 pe

195 breast cancer in humans, we generated stable transfectants of MCF7 breast cancer cells negative for e

196 in human monocytic THP-1 cells and in stable transfectants of mouse J774A.1 macrophages.

197 1 was observed in either stable or transient transfectants of nm23-H1 in MDA-MB-435 human breast carc

198 Finally, the transfectants of truncated FasL showed strong anchorage-

199 AS were less tumorigenic than vector control transfectants on orthotopic injection into mice.

200 A-expressing bacteria and DEC-205-expressing transfectants or alveolar MPhis could be inhibited by an

201 sorted T cell clones were reactive with CD1d transfectants or proliferated/secreted cytokine in respo

202 ailed to stimulate either the growth of ebp1 transfectants or transcription of AR-regulated reporter

203 xes containing MVLBG2 are significantly more transfectant over the entire composition range in mouse

204 e endoplasmic reticulum was increased in CHO transfectants overexpressing Slc35c2.

205 ncreased angiogenesis in vivo from the V659E transfectants paralleled increased angiogenic potential

206 n) and protein (>or=43% down) or vector-only transfectants (PC-3V) were characterized.

207 Stable shRNA transfectants (PLK1-PLK5) that express significantly red

208 rexpression of Nek3 in Chinese hamster ovary transfectants potentiated cytoskeletal re-organization i

209 HYAL1-S transfectants produced < or = 42 milliunits (moderate ov

210 HYAL1-AS transfectants produced <10% hyaluronidase activity when

211 Whereas HYAL1-S transfectants produced 3-fold more HYAL1 than vector tra

212 Whereas HAS1-S transfectants produced approximately 1.7-fold more HA th

213 creased cell growth was observed in wt-ACVR2 transfectants relative to ACVR2-deficient vector-transfe

214 n of cyclin D3 (2-2.5 fold) in Jurkat T cell transfectants renders them resistant to lower doses (1-1

215 wn of PEA15 expression in OVCAR-3 stable E1A transfectants resulted in a nuclear accumulation of the

216 r IIB (CD32B), or coincubation with CD32B(+) transfectants, resulted in robust T cell activation.

217 Studies of HEK293 transfectants revealed that expression of human TLR2 was

218 WT and SD VPAC2 Th2 cell transfectants secreted equal amounts of VIP.

219 T cells in intracerebral xenografts of tumor transfectants secreting MCP-1.

220 e vector was transfected into CHO cells, and transfectants secreting Stx2-specific antibody were scre

221 ol transfectant, whereas MSH2 and MLH1 siRNA transfectants show 6-TG resistance as expected.

222 Compared with nontransformed controls, v-Jun transfectants show enhanced ability of anchorage-indepen

223 Moreover, RSK1 transfectants show mislocalization of p27 to cytoplasm,

224 HAS1-AS transfectants showed a approximately 4-fold decrease in

225 re HYAL1 than vector transfectants, HYAL1-AS transfectants showed approximately 90% reduction in HYAL

226 Furthermore, nuclear extract from the SM22 transfectants showed diminished c-Fos binding to this mo

227 Moreover, Pg-treated PR transfectants showed significant morphologic change, app

228 In athymic mice, SCC-S2 transfectants showed significantly enhanced tumor growth

229 Compared with mock transfectants, the HS6ST1 transfectants showed significantly increased binding of

230 In contrast, SULF2 transfectants showed significantly reduced FGF2 binding

231 Sprouty-2 transfectants showed strong upregulation of c-Met protei

232 Dilution cloning of P. falciparum transfectants showed that individual clones possess as m

233 AL-E and anti-PV-1 antibodies, whereas NRP-1 transfectants stain with anti-NRP-1 antibodies in flow c

234 Knocking out PTTG in STAT3-C HCT116 stable transfectants strongly decreased tumor metastases in nud

235 milar studies were conducted in two pairs of transfectant sublines established from the Ku86-deficien

236 The proteomic profiles of antisense transfectants suggest the involvement of peptidyl-prolyl

237 ace expression of A2BR chimeric molecules in transfectants suggested that A2BR does not have the domi

238 CR-ABL kinase in K562 cells and BaF3/BCR-ABL transfectants, suggesting a mechanism for the generation

239 Akt at serine-473 was detected in N-cadherin transfectants, suggestive of N-cadherin-mediated Akt act

240 G expression by siRNA in STAT3 HCT116 stable transfectants suppressed cell growth and colony formatio

241 e experiments demonstrated that the LmexCht1 transfectants survived significantly better in human mac

242 Uptake of Hg(2+) was twofold greater in the transfectants than in wild-type cells.

243 aggregate production, even in wild type SOD1 transfectants that do not readily form inclusions, sugge

244 tric MS cases by flow cytometric labeling of transfectants that expressed different myelin proteins.

245 cell line, WM35, we have established stable transfectants that overexpress RhoC (called WM35RhoC).

246 and DHFR amplification proceeded in v-K-ras transfectants that possess a functional p53 in the absen

247 The IL8-S transfectants that secreted IL-8 at levels similar to th

248 60 cells and their WT or mutant c-Cbl stable transfectants, the c-Cbl/Vav/Slp-76 complex is also foun

249 Compared with mock transfectants, the HS6ST1 transfectants showed significa

250 ctions from both mouse thymocytes and ALST-1 transfectants, the PTP activity of CD45 was found to be

251 tive HYAL1 protein when compared with vector transfectants, their conditioned medium had 4-fold less

252 anchorage-independent growth of Galpha(12)QL-transfectants, thereby establishing the critical role of

253 on completely abolished the ability of these transfectants to grow in poly(A)-containing medium demon

254 id A induced NF-kappaB activity in wild-type transfectants under the identical transfection condition

255 th S and AS plasmids for selection of stable transfectants using Geneticin, and the presence of plasm

256 , we compared gain-of-function (WNT5A stable transfectants) versus loss-of-function (siRNA knockdown)

257 tated and chimeric NAs could be rescued into transfectant viruses.

258 Ag stimulation of Lyn unique domain transfectants was accompanied by enhanced phosphorylatio

259 igration through cell monolayers of endoglin transfectants was clearly enhanced in the presence of en

260 ng Geneticin, and the presence of plasmid in transfectants was confirmed by polymerase chain reaction

261 The increased apoptosis in HYAL1-v1 transfectants was due to the extrinsic pathway involving

262 a membrane-structural order parameter of the transfectants was measured by labeling cells with Laurda

263 on cultured cells such as K562 and CHO-CD32A transfectants was not affected by PMA.

264 ia spp. lose virulence during the raising of transfectants, we developed a method to label live Leish

265 ing of stable AID-yellow fluorescent protein transfectants, we now demonstrate that AID-yellow fluore

266 ssay for the inhibition of MGAT1 in MGAT4D-L transfectants, we performed site-directed mutagenesis to

267 HYAL1-S transfectants were 30% to 44% more invasive, and HYAL1-A

268 These transfectants were also 5- to 10-fold more apoptotic due

269 Such transfectants were also evaluated for their infectivity

270 The 13271 transfectants were also used as targets for clonal and p

271 were 30% to 44% more invasive, and HYAL1-AS transfectants were approximately 50% less invasive than

272 HAS1-AS transfectants were blocked in G(2)-M phase of the cell c

273 cies with IL-1R, IRAK-1, and MyD88 in HEK293 transfectants were compared.

274 NKRP1 molecules expressed on NK cells or transfectants were down-regulated by cross-linking with

275 DcR3 promotes tumor growth, CT26-DcR3 stable transfectants were established.

276 nontransformed lung epithelial cells, stable transfectants were generated in RL-65 cells, a spontaneo

277 HYAL1-AS transfectants were not generated for PC-3 ML because it

278 ro experiments with chemokine receptor CXCR3 transfectants were performed to confirm binding of gliad

279 Doubly sensitized L. braziliensis transfectants were photo-inactivated before (Strategy #1

280 Transfectants were selected and analyzed for GPx-1 enzym

281 Stable transfectants were selected for overexpression of Hyal1

282 cell line by transfection, and the resulting transfectants were tested for susceptibility to Lkt-indu

283 form stable dimers in binding-competent 293T transfectants when assessed using bioluminescent resonan

284 r levels of 6-TG cytotoxicity as the control transfectant, whereas MSH2 and MLH1 siRNA transfectants

285 ponse compared with the empty-vector control transfectant, whereas no significant increase resulted f

286 This was confirmed in CHO alphaIIbbeta3-H723 transfectants, which also exhibited increased PAC-1 bind

287 d CD44 expression and cell growth in HAS1-AS transfectants, which could be blocked by CD44 siRNA.

288 egy used previously produced L. braziliensis transfectants, which gave the same phenotype of aminolev

289 ppa B (NF-kappaB) activation in HEK293T/TLR5 transfectants, which was blocked by cotransfection with

290 ulation of the 189 isoform of VEGF in maspin transfectants, while a fivefold induction of Smad4 mRNA

291 a BRCA1 mutated cell line (HCC1937) and its transfectant with a wild-type BRCA1 cDNA.

292 Using a stable transfectant with high expression of PCFT, physiologic l

293 Using COS transfectants with different truncated forms of MARCO, a

294 Incubation of N-cadherin transfectants with either PI3 kinase or Akt inhibitors r

295 Stable transfectants with expression of small interfering RNA f

296 h amyloid of several mutants produced [PSI+] transfectants with similar efficiency as did reference s

297 15-induced proliferation than wild-type (WT) transfectants with similar levels of IL-15Ralpha express

298 ation of human embryonic kidney 293/CD14/MD2 transfectants with wild-type (WT) or mutant yellow fluor

299 DU-145 maspin cells, compared to DU-145 neo-transfectants without a significant effect on cell migra

300 as control transfectants; however, the Her-2 transfectants yielded 3-fold greater large metastases (>