戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (1語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 ation of acinar cells to the nestin-positive transitional cell.
2  superficial endocardial layer of atrial and transitional cells.
3 artment, enriched for CXCL13-responsive late transitional cells.
4 of large-sized cells somewhat similar to the transitional cells.
5  band of well-polarized but poorly excitable transitional cells.
6 lls with AT2 and AT1 cell features, known as transitional cells.
7 er cells: Core Pacemaker, Sinus Venosus, and Transitional Cells.
8 d promoting the accumulation of VSMC-derived transitional cells.
9 ore readily proliferate and form KRT8/CLDN4+ transitional cells.
10 of integrated stress response (ISR) genes in transitional cells.
11 had reduced intercalated cells and increased transitional cells.
12 is higher than that of mesothelial cells and transitional cells.
13 iated with the appearance of nestin-positive transitional cells.
14 ls cause fibrosis, whether keratins regulate transitional cell accumulation and fibrosis, and why tra
15 events AT1 differentiation and leads to KRT8 transitional cell accumulation in progressive pulmonary
16 osis in a K8-dependent manner; in mice, most transitional cells and fibrosis resolved, whereas in hum
17 venting the uncontrolled accumulation of AT2 transitional cells and promoting AT1 differentiation.
18 native pathway, the survival of neonatal B-1 transitional cells and their maturation into B-1 B cells
19 of gene regulatory programs leading to Krt8+ transitional cells and their terminal differentiation to
20 lar B cells in splenic development, immature transitional cells are an essential component for unders
21                                     However, transitional cells are larger and have an overall less d
22                                         Some transitional cells are supporting cells participating in
23              High numbers of non-HLA-binding transitional cells associated with B-suppressed interfer
24  Fifteen patients with recurrent superficial transitional cell bladder carcinoma who experienced prio
25 ammary and prostate cancers, the majority of transitional cell bladder tumors showed Id1 protein expr
26  exhibits significant activity in metastatic transitional cell cancer with minimal toxicity, but surv
27 ificant risk factor for EN and its attendant transitional cell cancer.
28 ed EN, (ii) these adducts can be detected in transitional cell cancers, and (iii) A:T --> T:A transve
29 onstrated absent or greatly reduced pinin in transitional cell carcinoma (TCC) and RCC tumors.
30 y culture (P0) and after passage in vitro of transitional cell carcinoma (TCC) biopsies that represen
31 ghly tumorigenic and highly metastatic human transitional cell carcinoma (TCC) cell line 253J B-V ove
32 ltetrazolium bromide (MTT) assays in bladder transitional cell carcinoma (TCC) cell lines RT4 and SW7
33 and protein are observed among several human transitional cell carcinoma (TCC) cell lines, suggesting
34 GF) accumulates in the nucleus in aggressive transitional cell carcinoma (TCC) cells and this histolo
35                                        Human transitional cell carcinoma (TCC) cells are labeled with
36 ptor (BMP-R) types IA, IB, and II in bladder transitional cell carcinoma (TCC) cells.
37  IFN-alpha therapy for clinical treatment of transitional cell carcinoma (TCC) has prompted us to inv
38                                              Transitional cell carcinoma (TCC) is a pan-urothelial di
39 therapy surgery for patients with metastatic transitional cell carcinoma (TCC) is controversial.
40                                    Prostatic transitional cell carcinoma (TCC) is present in up to 48
41                                              Transitional cell carcinoma (TCC) of the bladder compris
42  of noninvasive methods for the diagnosis of transitional cell carcinoma (TCC) of the bladder remains
43                We have screened 193 cases of transitional cell carcinoma (TCC) of the bladder using 3
44 invasive method for monitoring patients with transitional cell carcinoma (TCC) of the bladder would b
45 q is the most frequent genetic alteration in transitional cell carcinoma (TCC) of the bladder, implic
46 e identify a potential role for proHB-EGF in transitional cell carcinoma (TCC) of the bladder.
47 ured biopsies of superficial and myoinvasive transitional cell carcinoma (TCC) of the bladder.
48 vement in the development and progression of transitional cell carcinoma (TCC) of the bladder.
49  9 are the most common genetic alteration in transitional cell carcinoma (TCC) of the bladder.
50 mine the relative levels of MSI and EMAST in transitional cell carcinoma (TCC) of the upper and lower
51 eleted in many human malignancies, including transitional cell carcinoma (TCC) of the urinary bladder
52 ations are known to be associated with human transitional cell carcinoma (TCC) of the urinary bladder
53            Our microRNA profiling of bladder transitional cell carcinoma (TCC) patient samples reveal
54  orifice positive for a high-grade papillary transitional cell carcinoma (TCC) with muscularis propri
55 on forms of bladder tumors: 69 of 97 (71.1%) transitional cell carcinoma (TCC), 38 of 53 (71.6%) squa
56 ividuals currently face a high prevalence of transitional cell carcinoma (TCC), a form of bladder can
57                                              Transitional cell carcinoma (TCC), a neoplasm of urinary
58  remains the standard of care for metastatic transitional cell carcinoma (TCC), but its limitations i
59  predominant component in the development of transitional cell carcinoma (TCC), but the particular pa
60                                              Transitional cell carcinoma (TCC), the most common cance
61  potential as an immunotherapeutic target in transitional cell carcinoma (TCC).
62 dies demonstrated chromosome 3p deletions in transitional cell carcinoma (TCC).
63  be well tolerated in patients with advanced transitional cell carcinoma (TCC).
64 proven modality for treatment of upper-tract transitional cell carcinoma (TCC).
65 invasive or high-grade recurrent superficial transitional cell carcinoma (TCC; lymph node-negative, 1
66    In the SCID-beige mice, T24 human bladder transitional cell carcinoma also was used as the tumor i
67 he current fund of knowledge about prostatic transitional cell carcinoma and the implications for dia
68 ntly it has been shown to be a human bladder transitional cell carcinoma cell line.
69 n of p27(Kip1) and cyclin E in human bladder transitional cell carcinoma cells correlates with advanc
70 chemotherapy in advanced upper urinary tract transitional cell carcinoma continue to evolve and remai
71 F-7, pancreatic carcinoma (CRL 1420, bladder transitional cell carcinoma EJ, and melanoma LOX) but no
72         The operative management of invasive transitional cell carcinoma has advanced significantly i
73 onsidered related to the study drug (bladder transitional cell carcinoma in the ozanezumab group and
74 ase 2 trial, we enrolled patients with T2-4a transitional cell carcinoma of the bladder at 24 medical
75       Patterns of RT112 cells derived from a transitional cell carcinoma of the bladder could be depo
76 Rb) expression was evaluated in 185 cases of transitional cell carcinoma of the bladder from patients
77                                              Transitional cell carcinoma of the bladder is a common m
78                                              Transitional cell carcinoma of the bladder is comprised
79 erin and interferon are clinically active in transitional cell carcinoma of the bladder, but their me
80 ir derived cell lines from two patients with transitional cell carcinoma of the bladder.
81 nti-Yo antibody response in association with transitional cell carcinoma of the bladder.
82 n a wide variety of human cancers, including transitional cell carcinoma of the bladder.
83 27(Kip1) and cyclin E, in the progression of transitional cell carcinoma of the bladder.
84 s the standard treatment for muscle-invasive transitional cell carcinoma of the bladder.
85 th bacille Calmette-Guerin (BCG) -refractory transitional cell carcinoma of the bladder.
86                                              Transitional cell carcinoma of the renal pelvis or dista
87                            In a patient with transitional cell carcinoma of the renal pelvis, hydrone
88                     We present four cases of transitional cell carcinoma of the transplant ureter (TC
89 rectomy has been the treatment of choice for transitional cell carcinoma of the upper urinary tract.
90 lear cell) renal carcinoma (11 of 11 cases), transitional cell carcinoma of the urinary bladder (6 of
91 ssue sections of canine spontaneous invasive transitional cell carcinoma of the urinary bladder (a mo
92 s accrued from patients with muscle-invasive transitional cell carcinoma of the urinary bladder (TCC-
93 COX-1 and COX-2 expression in human invasive transitional cell carcinoma of the urinary bladder by im
94 of chromosome 3 was conducted in 72 cases of transitional cell carcinoma of the urinary bladder using
95 n pet dogs with naturally occurring invasive transitional cell carcinoma of the urinary bladder.
96 oving the precision of the estimated risk of transitional cell carcinoma of the urinary tract associa
97 n of the bladder tumor and biopsy identified transitional cell carcinoma or urothelial carcinoma inva
98                                              Transitional cell carcinoma should be considered in pati
99                         Treatment of T24 and transitional cell carcinoma TCC-SUP bladder cancer cells
100                               Using a murine transitional cell carcinoma tumor model, MB49, which nat
101           We measured DNA methylation in 342 transitional cell carcinoma tumors at BCL2, PTGS2 (COX2)
102                                Additionally, transitional cell carcinoma was observed in approximatel
103 % of the K5.COX-2 transgenic mice; no TCH or transitional cell carcinoma was observed in wild-type bl
104 of 31 consecutive patients with renal pelvic transitional cell carcinoma were evaluated.
105      Bladder cancer cell lines (J82, T24 and transitional cell carcinoma) significantly restored hepa
106 ickening in four patients (three of whom had transitional cell carcinoma), a renal abscess, a coloves
107 ses, 24 were renal cell carcinoma, one was a transitional cell carcinoma, and one was an angiomyolipo
108                     Forty-three patients had transitional cell carcinoma, and six had squamous cell c
109 sing an early-stage renal cell carcinoma and transitional cell carcinoma, chronic lymphocytic leukemi
110 ancer, testis cancer and upper urinary tract transitional cell carcinoma, no recently published study
111 sic fibroblast growth factor (bFGF) in human transitional cell carcinoma, reduce its angiogenesis, an
112  identified as a region of non-random LOH in transitional cell carcinoma, suggesting the presence of
113                                           In transitional cell carcinoma, the most common form of bla
114 st described in 1991 for upper urinary tract transitional cell carcinoma, with long-term data now eme
115 inoma, adult polycystic kidney disease or in transitional cell carcinoma.
116 atment of organ-confined upper urinary tract transitional cell carcinoma.
117                             Of patients with transitional-cell carcinoma (n = 776), LVI status was av
118 ed as an intravesical agent in patients with transitional-cell carcinoma (TCC) of the bladder.
119 d primarily patients with non-organ-confined transitional-cell carcinoma (TCC) of the bladder.
120 nd carboplatin for the treatment of advanced transitional-cell carcinoma (TCC) of the urothelium has
121 in previously treated patients with advanced transitional-cell carcinoma (TCC) of the urothelium.
122 patients with locally advanced or metastatic transitional-cell carcinoma (TCC) of the urothelium.
123  previously untreated patients with advanced transitional-cell carcinoma (TCC) to assess its efficacy
124 eported survival of patients with metastatic transitional-cell carcinoma (TCC) treated with systemic
125 icacy and toxicity in patients with advanced transitional-cell carcinoma (TCC) who had failed to resp
126 ymphadenectomy, with the intent to cure, for transitional-cell carcinoma of the bladder between July
127   Patients with measurable, locally advanced transitional-cell carcinoma of the bladder who were not
128 STT1, GSTM3, and GSTP1 in 1150 patients with transitional-cell carcinoma of the urinary bladder and 1
129 al urotoxicity, including the development of transitional-cell carcinoma of the urinary bladder.
130                Seven patients (5%) developed transitional-cell carcinoma of the urinary bladder.
131 ond-line treatment of patients with advanced transitional-cell carcinoma of the urothelial tract.
132  overall survival for patients with advanced transitional-cell carcinoma of the urothelium.
133  in patients with unresectable or metastatic transitional-cell carcinoma.
134 line therapy and emerging drugs for advanced transitional-cell carcinoma.
135 tment has an antitumor effect in superficial transitional-cell carcinoma.
136 y included 59 well-characterized superficial transitional cell carcinomas (Ta, n = 28; T1, n = 31) fo
137  pure squamous cell carcinomas (SCC), and in transitional cell carcinomas (TCC) of the bladder with S
138 five sarcomatoid RCC, two oncocytomas, three transitional cell carcinomas (TCC) of the renal pelvis a
139                FGFR3 coding sequence from 63 transitional cell carcinomas (TCC) of various stages and
140 elial carcinomas, including 28 pTa low-grade transitional cell carcinomas (TCC), 31 pTa high-grade TC
141 b and p53 pathway alterations in 12 invasive transitional cell carcinomas (TCCs) and addressed the fu
142                               A survey of 50 transitional cell carcinomas (TCCs) revealed a UP-II pol
143  and nature of FGFR3 mutations in a panel of transitional cell carcinomas and cell lines and studied
144 PDE5 was overexpressed in human squamous and transitional cell carcinomas compared with normal urothe
145 ylation of four CpG islands in human urinary transitional cell carcinomas of different stages and gra
146 ed that around one-third (33/101) of primary transitional cell carcinomas of the bladder overexpress
147                             More than 50% of transitional cell carcinomas of the bladder show loss of
148 ation of these genes by analyzing 69 primary transitional cell carcinomas of the bladder with a panel
149  but was detected in 25 of 29 (86%) invasive transitional cell carcinomas of the urinary bladder and
150 ity, we have studied a cohort of superficial transitional cell carcinomas of the urinary bladder by i
151 ed the pattern of allelic imbalance in human transitional cell carcinomas of the urinary bladder incl
152 NU plus H202 or H202 alone formed high-grade transitional cell carcinomas when injected into nude mic
153 g water and the incidence of urinary bladder transitional cell carcinomas.
154 microsatellite loci in 20 grade III invasive transitional cell carcinomas.
155 adder proliferative lesions, including three transitional cell carcinomas.
156 ssues and normal mammary tissues and bladder transitional cell carcinomas.
157                     However, whether and how transitional cells cause fibrosis, whether keratins regu
158                                              Transitional cells, coexpressing hair cell and supportin
159 as defined by expression of alpha-SM actin) "transitional" cells, coexpressing both endothelial and S
160                                             "Transitional" cells, coexpressing both endothelial marke
161    identify ICAM1+NKX2-1+ progenitors as key transitional cells conserved in the developing human and
162                    Putative ER(+)PR(+)CK5(+) transitional cells could be seen only in colonies or tum
163 ed >80 stem, progenitor, immune, stromal and transitional cells defined by distinctive surface marker
164 AEC1s without fibrosis in human ARDS and why transitional cells differentiate into AEC1s during physi
165 gulating AT2 cell self-renewal and promoting transitional cell differentiation into AT1 cells.
166 ly Walthard cell nests represent urothelial (transitional cell) differentiation.
167 d thymocytes paradoxically appear as CD4+8lo transitional cells during their differentiation into CD8
168  specimens, as well as 11 cases of low-grade transitional cell dysplasia, 21 cases of carcinoma in si
169                        It is unknown whether transitional cells emerge and differentiate into AEC1s w
170                                          The transitional cells exhibit a similar apical-basal polari
171  mRNA was observed in proliferating oval and transitional cells, forming duct-like structures of cyto
172                                      TACI(+) transitional cells from BAFF-transgenic mice are charact
173                            Patients with non-transitional-cell histology, poor performance status, an
174 a keratin 5 promoter, is sufficient to cause transitional cell hyperplasia (TCH) in 17% and 75% of th
175 hocytes are thought to develop from immature transitional cells in a BCR-dependent manner.
176 een proposed that these oval cells represent transitional cells in a nonhepatocytic liver facultative
177 the risk allele, due to an expansion of late transitional cells in a stage targeted by selection mech
178 eviously associated with dysplastic alveolar transitional cells in fibrotic lungs.
179 /or single-cell RNA sequencing revealed that transitional cells in mouse models of physiological rege
180  markers, suggest that CK19+/NCAM+ cells are transitional cells in the biliary lineage and that rare
181 ether, these results identify progenitor and transitional cells in the brown adipose lineage and defi
182  that are negative for all three markers are transitional cells in the hepatocytic lineage.
183 tinguished tumor populations from normal and transitional cells, including acinar-to-ductal metaplasi
184 layers, significantly increase the number of transitional cells, induce clustering of mitochondria an
185 onstrate that B-1 B cells are generated from transitional cell intermediates that emerge in a distinc
186  fatal early ARDS, AEC1 differentiation from transitional cells is incomplete, underlying persistent
187  but ongoing without fibrosis; senescence of transitional cells may be associated with pulmonary fibr
188 Akap12-/- prostate lobes suggests that these transitional cells may be the source of the lymph node m
189  Severe B-cell deficiency affects mature and transitional cells, mimicking the action of rituximab.
190 or, proHB-EGF, localizes to the cytoplasm of transitional cells of the human bladder urothelium and t
191 hway and allowing circus movement within the transitional cells of the posterior AV nodal connection.
192 w that, in contrast to the dependence of B-2 transitional cells on the alternative pathway, the survi
193 cells, suggesting that these newly generated transitional cells play a critical role in PD-1-based im
194                                            A transitional cell population, defined by the first downr
195 r ~2 wk after birth and then declines as B-2 transitional cells predominate.
196 gest that hFLMPCs are mesenchymal-epithelial transitional cells, probably derived from mesendoderm.
197 distinct treatment naive tumors, a papillary transitional cell renal cell carcinoma, a duodenal carci
198             Cases included 76 renal cell, 24 transitional cell renal pelvis and ureter, and 22 other
199                                              Transitional cells represent a crucial step in the diffe
200 ed, among the oval cells, a subpopulation of transitional cells showing features of maturing hepatocy
201 on programs that evolve through at least one transitional cell stage.
202 nto type-1 cells acquire pre-alveolar type-1 transitional cell state (PATS) en route to terminal matu
203 otes AT1 differentiation through action in a transitional cell state analogous to a transient interme
204 egulation of recently described iAT2-derived transitional cell state genetic markers.
205 d-type and mutant retinal cells, we reveal a transitional cell state of retinal progenitor cells (RPC
206                                              Transitional cell states are at the crossroads of crucia
207                                              Transitional cell states are critical in lung injury rep
208                               A continuum of transitional cell states exists between these discrete s
209  fate conversion is mediated via distinctive transitional cell states of damage-associated transient
210 iment, MC offers a robust way to assay rare, transitional cell states that are pertinent to human dev
211 that the presence of unspliced mRNA reflects transitional cell states, informative for studies of dyn
212 e bacterial QIRs were harbored in underlying transitional cells, stimulation of epithelial turnover t
213 isthmal) zone of the gastric unit and become transitional cells (TCs) with molecular and ultrastructu
214 ated disks and gap junctions of Purkinje and transitional cells, telocytes, macrophages, endothelium,
215 clinically relevant but poorly characterized transitional cells that bridge the CCS and surrounding m
216 auses loss of parietal cells, development of transitional cells that express markers of mucus neck an
217 wly formed human B cells from bone marrow as transitional cells, they diverge along two developmental
218 ntative sample of normal and malignant human transitional cell tissues.
219 s of carcinoma in situ (CIS), 38 superficial transitional cell tumors (STCC, stages T(a)-T(1)), 65 mu
220 NA expression in two of five muscle-invasive transitional cell tumors when compared with normal sampl
221  that SHPCs may represent an intermediate or transitional cell type between oval cells and mature hep
222                                            A transitional cell type from the submesothelial cell phen
223                                              Transitional cells undergo extensive stretching during d
224 is regimen in patients with both TCC and non-transitional-cell urothelial tumors is ongoing.
225  renal failure and a strong association with transitional cell (urothelial) carcinoma of the upper ur
226 lear outer sulcus cells (OSC) and vestibular transitional cells (VTC) are part of the parasensory epi
227                                              Transitional cells were negative for principal cell and
228 acterized by a preponderance of immature and transitional cells, whose persistence was associated wit
229 n controls, and an increase in the number of transitional cells with a CD4+CD8low phenotype.
230 clustering structure for both pure cells and transitional cells with soft memberships.
231 mplete but ongoing AEC1 differentiation from transitional cells without fibrosis may underlie persist
232 g of postmortem ARDS lungs revealed abundant transitional cells without fibrosis.

 
Page Top