戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 ional SR surface facing the surface membrane/transverse tubule.
2 cose transporters to the plasma membrane and transverse tubules.
3 lin is highly enriched in the sarcolemma and transverse tubules.
4 communication with the voltage sensor in the transverse tubules.
5 )1.6 plus beta1 and/or beta3 subunits in the transverse tubules.
6  Na(v)1.3, and Na(v)1.6 are localized in the transverse tubules.
7 ivation of multiple Ca2+ release sites along transverse-tubules.
8 s are organized within the dyad, linking the transverse tubule and sarcoplasmic reticulum and ensurin
9 ) coupling in cat atrial myocytes which lack transverse tubules and contain both subsarcolemmal junct
10                     Cat atrial myocytes lack transverse tubules and contain sarcoplasmic reticulum (S
11             A vacuolar myopathy with dilated transverse tubules and disruption of the triad junctions
12  colocalizes with L-type calcium channels in transverse tubules and is associated with L-type calcium
13  formed adjacent to Z-lines by apposition of transverse tubules and junctional sarcoplasmic reticulum
14 dic junctions (i.e., the close apposition of transverse tubules and sarcoplasmic reticulum [SR]), fac
15  sorcin localizes to the dyadic junctions of transverse tubules and sarcoplasmic reticulum and coimmu
16 ed triad organization with visibly malformed transverse tubules and sarcoplasmic reticulum, suggestin
17 number of gold particles associated with the transverse tubules and the sarcolemma by three-fold.
18 two novel systems of internal membranes, the transverse tubules and the sarcoplasmic reticulum (SR).
19                         The surface membrane/transverse tubules and the SR form functional units (cal
20 lut4 was highly enriched in membranes of the transverse tubules and the terminal cisternae of the tri
21                                          The transverse tubules and vacuoles displayed distinct Ca(2+
22 sulted in defective structural maturation of transverse-tubules and mitochondria.
23 pose that these membranes, consisting of the transverse-tubules and sarcoplasmic reticulum, are dynam
24 ies of junctional sarcoplasmic reticulum and transverse tubules, and (4) attenuated mitochondrial abn
25                                          The transverse tubules are a system of membrane invagination
26 otoxin (TTX)-sensitive brain isoforms in the transverse tubules are small and are detectable only aft
27 D hearts displayed a marked reduction in the transverse tubule area (59% of sham) and the surface are
28 duces the targeting of these proteins to the transverse tubules as well as reducing overall protein l
29 d without affecting dysferlin trafficking to transverse tubules, but Dysf-DeltaC2A fails to support n
30 nsport is mediated by Glut4 localized in the transverse tubules; (c) insulin increases the apparent s
31 myocytes, from both rodents and humans, this transverse tubule coupling between LTCC and beta(2)AR wa
32  to disruption of E-C coupling, plasmalemmal transverse tubule degeneration, abnormal Ca(2+) homeosta
33 teract with the carboxy-terminal tail of the transverse tubule dihydropyridine receptor (DHPR).
34             Additional studies revealed that transverse-tubule disruption precedes the development of
35  loss of SPEG phosphorylation of JPH2 led to transverse-tubule disruption, a precursor of HF developm
36 g in an increase of the channel activity and transverse-tubule expression.
37 e length, mitochondrial density, networks of transverse tubules), force-frequency and force-length re
38 tional sarcoplasmic reticulum positioning or transverse tubule formation.
39 re mature electrophysiology, and evidence of transverse-tubule formation.
40 and glycerol exposure promotes detachment of transverse tubules from the sarcoplasmic reticulum.
41 ial independence of electrical events in the transverse tubules from those responsible for the rapid
42 he importance of dysferlin and myoferlin for transverse tubule function and in the genesis of muscula
43               Atrial myocytes generally lack transverse tubules; however, during ECC Ca2+ release is
44 calized with beta1 and beta3 subunits in the transverse tubules, identified by immunostaining of alph
45                         Calcium (Ca(2+)) and transverse-tubule imaging of ventricular myocytes from M
46 ole for brain sodium channel isoforms in the transverse tubules in coupling electrical excitation to
47 plasmic reticulum and ectopic and misaligned transverse tubules in FER skeletal muscle.
48 se that the molecular switch for the loss of transverse tubules in HF and their restoration following
49 ctional sarcoplasmic reticulum proteins, and transverse tubules in mature cardiomyocytes.
50 sulin increases the apparent surface area of transverse tubules in skeletal muscle; and (d) insulin c
51  colocalized with the L-type Ca2+ channel in transverse tubules in wild-type skeletal muscle and reta
52 partmentalized translocation of GLUT4 to the transverse-tubules in skeletal muscle.
53 ng RV cardiomyocyte and nuclear hypertrophy, transverse tubule integrity, and connexin-43 localizatio
54   The subcellular targeting of alpha2 to the transverse tubules is important for this role.
55    Localization of dysferlin variants to the transverse tubules is not sufficient to support normal C
56 ed AKAP100 localization at the region of the transverse tubule/junctional sarcoplasmic reticulum.
57 ect sparks on an inhomogeneous background of transverse tubule-labeled rat ventricular cells.
58 ardiac atrial cells lack a regular system of transverse tubules like that in cardiac ventricular cell
59 except C2B regulate Ca(2+) signalling; (iii) transverse tubule localization is insufficient for norma
60                           The deeper regular transverse tubules located at each A-I boundary of the s
61 ment improves contractile function, reverses transverse tubule loss, restores calcium transient ampli
62 use hearts causes JPH2 dephosphorylation and transverse-tubule loss associated with downstream Ca(2+)
63 sensing dihydropyridine receptors (DHPRs) in transverse tubule membrane and Ca(2+) release channel ry
64 l muscle dihydropyridine receptor transduces transverse tubule membrane depolarization into release o
65 d by Ca(2+) influx across the sarcolemmal or transverse tubule membrane neighboring the cluster, the
66 e that interacts in vivo with the sarcolemma/transverse tubule membrane system, whereas CaM binds wit
67 rom ventricle because atrial myocytes lack a transverse tubule membrane system: Ca(2+) release starts
68 uscle fiber restores a small fraction of the transverse tubule membrane voltage sensors from the inac
69 18c to block GLUT4-EGFP translocation to the transverse-tubule membrane but not the sarcolemma membra
70 Munc18c inhibited GLUT4 translocation to the transverse-tubule membrane without affecting translocati
71 antially reduced levels of syntaxin 4 in the transverse-tubule membrane.
72 es and intramembrane charge movements in the transverse tubule membranes (T-system) of frog fast twit
73 lization of the AHNAKs in close proximity to transverse tubule membranes and Z-band regions of cardia
74 rlin is located at the intercalated disc and transverse tubule membranes.
75 d L-type channels from detergent-solubilized transverse tubule membranes.
76 ted translocation to both the sarcolemma and transverse-tubule membranes.
77 he proximity of the channel (<350 nm) in the transverse tubule microdomain.
78                                Disruption of transverse tubule morphology by in vitro glycerol shock
79  the form of disorganization and loss of the transverse tubule network.
80                      The average diameter of transverse tubules observed in longitudinal sections inc
81 ocalizes with CaV1.2 channels and PKA in the transverse tubules of isolated ventricular myocytes.
82 ized by immunofluorescence microscopy to the transverse tubules of murine cardiac myocytes.
83      ABSTRACT: Dysferlin concentrates in the transverse tubules of skeletal muscle and stabilizes Ca(
84 erlin is an integral membrane protein of the transverse tubules of skeletal muscle that is mutated or
85 C2 domains, C2A through C2G, concentrates in transverse tubules of skeletal muscle, where it stabiliz
86 phy 2B and Miyoshi myopathy, concentrates in transverse tubules of skeletal muscle, where it stabiliz
87 se, as well as in targeting dysferlin to the transverse tubules of skeletal muscle.
88 re present and functionally important in the transverse tubules of ventricular myocytes.
89 plasma membrane forms tubular invaginations (transverse tubules or T-tubules) that function in depola
90 uction in adult animals results in disrupted transverse tubule organization and calcium handling.
91 n function, which may include maintenance of transverse tubule organization and intracellular Ca(2+)
92 cement with S157A/S161A telethonin disrupted transverse tubule organization and prolonged the time to
93 ignificantly improved Ca(2+) homeostasis and transverse tubule organization with significantly attenu
94 d post-myocardial infarction, resulting from transverse tubule remodeling, leading to distorted beta(
95 distributed on the cell surface membrane and transverse tubules, resulting in a striated pattern.
96 se function, ie, Ca(2+) spikes at individual transverse tubule-sarcoplasmic reticulum (T-SR) junction
97 n initial segments, specialized sites at the transverse tubule/sarcoplasmic reticulum in cardiomyocyt
98 scle dihydropyridine receptor complex to the transverse tubule/sarcoplasmic reticulum junction.
99 inositol trisphosphate (InsP(3)) receptor at transverse-tubule/sarcoplasmic reticulum sites in cardio
100                                    Disrupted transverse tubule structure may contribute to the decrea
101 d take up Ca(2+) from the cytoplasm but only transverse tubules supported store-operated Ca(2+) entry
102 A critical cardiac membrane organelle is the transverse tubule system (called the t-tubule system) wh
103 to study global and localized regions of the transverse tubule system (T-system).
104       In many species atrial myocytes lack a transverse tubule system, dividing the sarcoplasmic reti
105 natal skeletal muscle lacks a well developed transverse tubule system, the RyR3 reinforcement of CICR
106 taining SH3-BAR domains and regulates muscle transverse tubule (T-tubule) formation in flies.
107                               Despite normal transverse tubule (T-tubule) formation, GLT myotubes lac
108 2) tether the sarcoplasmic reticulum (SR) to transverse tubule (T-tubule) membranes, generating stabl
109 ldtype skeletal muscle EHD1 localizes to the transverse tubule (T-tubule), and loss of EHD1 results i
110                                              Transverse tubules (t-tubules) are uniquely-adapted memb
111                                              Transverse tubules (t-tubules) form gradually in the dev
112                                          The transverse tubules (t-tubules) of mammalian cardiac vent
113 eases, especially in cells lacking organized transverse tubules (T-tubules) such as atrial myocytes (
114 -selective dye Di-8-ANEPPS demonstrated that transverse tubules (t-tubules) were absent in atrial cel
115 o lateral sarcolemma, intercalated discs and transverse tubules (t-tubules), while NHE1 is absent fro
116 Ca(2+) alternans in cardiac myocytes lacking transverse tubules (t-tubules).
117 opy revealed their colocalization in cardiac transverse tubules (T-tubules).
118 ovide evidence for ET-1 receptors in cardiac transverse tubules (T-tubules).
119 g integrator 1 (cBIN1 or BIN1+13+17) creates transverse-tubule (t-tubule) membrane microfolds, which
120 aborate, unique surface topography including transverse-tubule (T-tubule) openings leading into a cel
121                                          The transverse-tubule (T-tubule) system of ventricular myocy
122 ranched from the SSTN, indicating individual transverse tubules that form triads are continuous with,
123 brane (comprising the surface sarcolemma and transverse-tubules), the intracellular calcium store (th
124                             In cells lacking transverse tubules, the same Ca influx initiates regener
125 ailure, beta2ARs were redistributed from the transverse tubules to the cell crest, which led to diffu
126 rial myocyte (AM) is characterized by sparse transverse tubule (TT) invaginations and slow intracellu
127  sides of a flattened vesicle derived from a transverse tubule (TT).
128  surface and membrane invaginations known as transverse tubules (TT).
129 c reticulum (SR) cisternae and extensions of transverse-tubules (TT) that increase co-localization of
130 tubular system (T-system), which consists of transverse tubules (TTs) that align with sarcomeres and
131 ease and increased spatial dispersion of the transverse tubules (TTs).
132  between the sarcoplasmic reticulum (SR) and transverse-tubules (TTs).
133 r-protein kinase D1-Rem1 signaling increases transverse-tubule VLCC expression that results in increa
134 n via targeted delivery of ryanodine through transverse tubules, we achieve in-preparation isolation
135 entrate on tubular PM invaginations known as transverse tubules, we hypothesize that PM curvature pla
136 ponsible for the flattened appearance of the transverse tubules when viewed in cross-section.
137 ack alpha2 protein and have no alpha2 in the transverse tubules, where its expression is normally enh
138 ignals are localized exclusively to the deep transverse tubules, whereas functional beta1ARs are dist
139 rain of as little as 5% affects the shape of transverse tubules, which has important implications for

 
Page Top