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1 bo, 170 [91%] on memantine, and 168 [90%] on trazodone).
2 euptake inhibitors, and 1.2 (1.0 to 1.4) for trazodone.
3 ribing included sertraline, mirtazapine, and trazodone.
4 ) did not differ significantly from that for trazodone.
5 ospital and 36 (15.3/1,000) total deaths for trazodone.
6 spital and 256 (15.8/1,000) total deaths for trazodone.
7 dementia might be preferentially prescribed trazodone.
8 stage two interim analysis for memantine and trazodone.
11 ants were randomised (1:1:1) to receive oral trazodone 200 mg once a day, oral memantine 20 mg once a
12 tonin-reuptake inhibitors (612 subjects), or trazodone (304 subjects) or nonusers of antidepressants
14 vitro and animal studies have suggested that trazodone, a licensed antidepressant, may protect agains
15 or donepezil, sertraline, norsertraline, and trazodone across each of these early life stages of brow
17 Here we show that a chronic treatment with trazodone, an antidepressant with positive effects on sl
22 luoxetine, its metabolite norfluoxetine, and trazodone and its active metabolite m-chlorophenylpipera
24 r to the chemically related drug vilazodone, trazodone and nefazodone are allosteric ligands: trazodo
26 odone and nefazodone are allosteric ligands: trazodone and nefazodone inhibit uptake by and transport
29 LSFRS-R was -0.650 for memantine, -0.625 for trazodone, and -0.655 for placebo (memantine versus plac
30 ated memantine, 175 (33%) had been allocated trazodone, and 180 (34%) had been allocated placebo.
32 iveness of a combination of serotoninergics, trazodone, and L-tryptophan, in our animal model of OSAH
33 nzodiazepines, daridorexant, suvorexant, and trazodone) can be effective in the acute treatment of in
35 to describe trends in zolpidem and low-dose trazodone dispensing among adults with employer-sponsore
36 18) were significantly increased relative to trazodone; findings were similar with exclusion of overd
37 lopram, citalopram, mirtazapine, duloxetine, trazodone, fluoxetine, bupropion, paroxetine, and venlaf
39 ths in the memantine group, 52 deaths in the trazodone group, and 48 deaths in the placebo group.
42 pressants (B = 0.046; SE = 0.013; P < .001), trazodone hydrochloride (B = 0.033; SE = 0.010; P < .001
44 rperazine (HR, 0.47; 95% CI, 0.33-0.68), and trazodone hydrochloride vs. paroxetine (HR, 0.49; 95% CI
48 ese results suggest that the clinical use of trazodone is not associated with a reduced risk of demen
54 tly powered to warrant no further testing of trazodone or memantine in motor neuron disease at the do
58 the pathway-including by the licensed drug, trazodone-restores global protein synthesis rates with p
59 tamol and triflusal, but not dimethadione or trazodone, significantly elevated FGF20 levels in the ni
60 nzodiazepine hypnotics, z-drugs, or low-dose trazodone, study hypnotics were not associated with mort
61 ezil, sertraline, norsertraline, citalopram, trazodone, telmisartan, and mirtazapine in eggs and yolk
62 ights into the neuroprotective mechanisms of trazodone through reversal of this toxicity, relevant fo
64 dementia among patients who were prescribed trazodone to that of patients with similar baseline char
67 ains of prion-diseased mice with and without trazodone treatment, in both whole hippocampus and cell-
68 lth records, we found no association between trazodone use and a reduced risk of dementia compared wi
69 cords study assessed the association between trazodone use and the risk of developing dementia in cli
71 90% CI lower level -0.085; one-sided p=0.36; trazodone vs placebo: 0.065, -0.051; one-sided p=0.24).
72 to dementia diagnosis for people prescribed trazodone was 1.8 years (interquartile range [IQR] = 0.5
73 Incidence of dementia among patients taking trazodone was higher than in matched users of other anti