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1 spects of antisocial adjustment appear to be treatment responsive.
2 ve symptoms of schizophrenia are enduring or treatment-responsive.
3 haryngeal squamous cell carcinoma (OPSCC) is treatment-responsive.
4 stic biomarker that can discriminate between treatment responsive and resistant tumor models.
5                        Compared to controls, treatment responsive and treatment-resistant participant
6  (1 year later), when 35 were categorized at treatment-responsive and 15 as treatment-resistant.
7 roduction, were compared in 16 patients with treatment-responsive and 16 with treatment-resistant Lym
8 bolic response to a neuroleptic challenge in treatment-responsive and nonresponsive schizophrenic pat
9 t mechanisms underlie the pathophysiology of treatment-responsive and treatment-resistant schizophren
10 ociate with LE that is often paraneoplastic, treatment responsive, and has a tendency to relapse.
11 ral prefrontal cortex when compared to those treatment-responsive, and with bilateral dorsolateral pr
12 pes), whereas only 5 of the 16 patients with treatment-responsive arthritis had reactivity with these
13 in A (OspA), but TCL from four patients with treatment-responsive arthritis only rarely recognized th
14 etween patients with treatment-resistant and treatment-responsive arthritis was in reactivity with N-
15 andidiasis were paired with controls who had treatment-responsive candidiasis and who had been observ
16 sentation were independently associated with treatment-responsive causes of RPD after controlling for
17 mized to recognize patients with potentially treatment-responsive causes of RPD early in the diagnost
18 , and sTREM2 identified the 44 patients with treatment-responsive causes of RPD with 89% accuracy.
19 y and to recognize patients with potentially treatment-responsive causes of RPD.
20 eliably identified patients with potentially treatment-responsive causes of RPD.
21  of whom 86 patients (55.5%) had potentially treatment-responsive causes.
22 ate that AAG patients may also show signs of treatment-responsive cognitive impairment.
23 es significantly associated with lower-stage/treatment-responsive CTCL versus higher-stage/treatment-
24 tions, and most often manifests with milder, treatment-responsive disease.
25 eceptor (AMPAR) encephalitis is a severe but treatment-responsive disorder with prominent short-term
26  described a severe, potentially lethal, but treatment-responsive encephalitis that associates with a
27 ers of the NMDAR associate with a severe but treatment-responsive encephalitis.
28 d controlled trial, was profiled to reveal a treatment-responsive gene signature.
29                    We demonstrated how these treatment-responsive genes could be translated into a pr
30                      Most of the rest of the treatment-responsive genes we categorized as mediators o
31              We discovered approximately 200 treatment-responsive genes, many of them known mediators
32 sting to confirm benefit in this potentially treatment-responsive group is needed.
33 ere compared between treatment-resistant and treatment-responsive groups and related to glutamate.
34 ion over time of patients with complex, less treatment-responsive illnesses.
35 plication, indicating that HIV infection was treatment responsive in the DRAGA mice.
36 DG-PET biomarker strategy to identify mGluR2 treatment-responsive individuals.
37 ent-resistant depression (TRD) compared with treatment-responsive major depressive disorder (trMDD).
38 TRD vs. non-major depressive disorder (MDD), treatment responsive MDD (trMDD) vs. non-MDD, and TRD vs
39                Results indicate that HCO3(-) treatment responsive metabolomic changes depend on the H
40                                              Treatment-responsive Mtm1 p.R69C gastrocnemius muscles d
41 tment response: treatment resistant (n=5) or treatment responsive (n=4).
42 tiation (N=45), patients whose symptoms were treatment responsive (N=40), and patients whose symptoms
43 d with RPD were characterized as potentially treatment-responsive or non-responsive, referencing clin
44 cerbations, those whose asthma might be less treatment responsive, or both might guide treatment sele
45                                              Treatment-responsive participants showed a negative rela
46                                              Treatment-responsive participants showed significant sig
47                              The mean age of treatment-responsive patients (group 1) was 45.88 +/- 18
48  differences between treatment-resistant and treatment-responsive patients include reductions in grey
49 apacity and prefrontal grey matter volume in treatment-responsive patients with psychosis, and compar
50 management of a non-negligible proportion of treatment-responsive patients.
51 esults suggest that treatment-predictive and treatment-responsive regions may be anatomically differe
52                   Sixteen clinically silent, treatment-responsive rejection episodes occurred in 10 r
53  PI3K-alpha (P = 0.04) were downregulated in treatment responsive residual tumors, when compared with
54 AM(3) P), captured 82 of 86 (95.3%) cases of treatment-responsive RPD.
55 in such individuals compared with those with treatment responsive schizophrenia, and healthy controls
56  hypothesis to indicate it can be applied to treatment-responsive schizophrenia only.
57 iagnosis (past or present), individuals with treatment-responsive schizophrenia taking nonclozapine a
58 nt-resistant schizophrenia, 21 patients with treatment-responsive schizophrenia, and 18 healthy contr
59 ant schizophrenia lie along a continuum with treatment-responsive schizophrenia, whereas other differ
60 rived subphenotyping algorithms may identify treatment-responsive subgroups of critically ill patient
61  in retrospective studies, and that the same treatment-responsive subpopulations may cross multiple c
62 ntly higher than the RVI among patients with treatment-responsive symptoms (mean=0.30 [SD=0.02]).
63 h SZ (n=25); NC, treatment-resistant SZ, and treatment-responsive SZ; and 3) NC and SZ treated with t
64 g response profiles in TCF3-HLF-positive and treatment-responsive TCF3-PBX1-positive ALL.
65                                  Compared to treatment-responsive, treatment-resistant participants s
66                                           On-treatment, responsive tumours contained abundant granzym