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1 hat Boudicca is actively transcribed in this trematode.
2 sed to isolate E. risticii from the infected trematode.
3  host for at least four species of digenetic trematode.
4 physiological susceptibility of fish to this trematode.
5 ng to their sources: horse blood or infected trematodes.
6 cariae and sporocysts of digenetic virgulate trematodes.
7 arasitic lifestyle intrinsic to cestodes and trematodes.
8  specialized soldier caste yet documented in trematodes.
9 which harbored snails infected with zoonotic trematodes.
10 ratures than either cold- or warm-acclimated trematodes.
11 HDM-1 may be involved in heme homeostasis in trematodes.
12  gastropod immune molecule and resistance to trematodes.
13  a major group of snail pathogens, digenetic trematodes.
14 cts containing encysted N. risticii-infected trematodes.
15  following priming with attenuated digenetic trematodes (acquired resistance) in this model invertebr
16 terest for understanding heme homeostasis in trematodes and as potential targets for the development
17                                           In trematodes and cyclophyllidean cestodes, which display h
18 reams, we quantified the standing biomass of trematodes and free-living organisms at nine sites in th
19 s an obligate intracellular bacterium of the trematodes and mammals.
20              We exposed these communities to trematodes and measured disease outcomes at the communit
21          We document that 3 human-infectious trematodes and their introduced first intermediate host
22 suggest that MF6p/HDMs can transport heme in trematodes and thereby shield the parasite from the harm
23  mikrocytids, Vibrio spp., fungi, Sacculina, trematodes, and haemolymph bacterial loads.
24                                     Digenean trematodes are a large, complex group of parasitic flatw
25                                              Trematodes are long-lived and our analysis quantifies th
26              A new discovery highlights that trematodes are unique in forming different societies at
27 abundance of these often debilitating larval trematodes (atrazine alone accounted for 51%).
28  collectively accounted for 87% of the total trematode biomass within the three ponds.
29  cloned and characterised from the parasitic trematode blood fluke Schistosoma haematobium.
30 sterase (AChE) present on the surface of the trematode blood fluke Schistosoma has been implicated in
31 crease exposure and susceptibility to larval trematodes by augmenting snail intermediate hosts and su
32            We discovered the abundant cattle trematode, Calicophoron sukari, fails to develop in Biom
33 s developing stages (metacercariae) of these trematodes cause reductions in minnow activity, growth a
34 tures above 22 degrees C the snails released trematode cercariae tentatively identified as virgulate
35 cretions was associated with the presence of trematode cercariae.
36                      Functional profiling of trematode, cestode, and a free-living flatworm TRPM(PZQ)
37 asitic roundworms (nematodes) and flatworms (trematodes), collectively known as helminths.
38 se cercariae for 3-4 months a year, the pond trematode communities produced an average of 153 mg m(-2
39 nd is frequently parasitized by the digenean trematode Cryptocotyle lingua.
40                                   Mid-summer trematode dry biomass averaged 0.10 g m(-2), which was e
41 two amphibian parasites (ranaviruses and the trematode Echinoparyphium sp.), we examined the influenc
42           After infection with the digenetic trematode Echinostoma paraensei, hemolymph of the snail
43       Opisthorchis viverrini is a fish-borne trematode endemic in East Asia.
44 abundance (first intermediate host) and thus trematode exposure by increasing mortality of snail pred
45                                              Trematode exposure increased mortality and malformations
46 ile a similar pattern in snail abundance and trematode exposure was observed with triazine herbicides
47 her prospecting and screening of the broader trematode family of peptides for the discovery of novel
48  protein secreted by the parasitic flatworm (trematode) Fasciola hepatica that belongs to a broad fam
49  The aetiological agents of this disease are trematode flatworms (Schistosoma) that live and lay eggs
50  bilharzia--is a parasitic disease caused by trematode flukes of the genus Schistosoma.
51              Water borne cercaria(ae) of the trematode genus Schistosoma rapidly penetrate host skin.
52 e situation that the genomes of cestodes and trematodes have lost the piwi and vasa genes that are ha
53                           Infection with the trematode helminth Schistosoma mansoni results in a para
54                        In infection with the trematode helminth Schistosoma mansoni, the severity of
55 arvae, which are both important intermediate trematode hosts, dominated the dry biomass of free-livin
56   Common parasitic castrators include larval trematodes in snails, and isopod and barnacle parasites
57 e Fasciola MF6p/FhHDM-1 are present in other trematodes, including Clonorchis, Opistorchis, Paragonim
58 cant cataract secondary to uveitis caused by trematode induced anterior chamber granuloma were includ
59 es of cataract surgery in eyes with persumed trematode induced granulametous anterior uveitis are fav
60  Lymnaea stagnalis, a cosmopolitan vector of trematodes infecting diverse vertebrates, whose ancestra
61 4% of the competitive deaths in the guild of trematodes infecting its host snail in its invasive rang
62                              Here we ask how trematode infection affects the expression of boldness i
63 empted to examine the synergistic effects of trematode infection and exposure to chemical contaminant
64 portance of this predator-prey relationship, trematode infection can act as an important, although in
65 nitrogen-poor litter species should decrease trematode infection in tadpoles via density- and trait-m
66 -elevation ponds, even while controlling for trematode infection load.
67 g predictions about whether Chaetogaster and trematode infection of snails correlate negatively ('pro
68 h polyphenolic levels, which should increase trematode infection via trait-mediated effects on tadpol
69 7: wording changed to 'which should increase trematode infection via trait-mediated effects on tadpol
70  to 301 amphibian populations, we found that trematode infection was associated with an average of be
71 s conclusively demonstrated that exposure to trematode infection was required for the development of
72 d laboratory experiments that link increased trematode infection, and increased limb deformities, to
73 une mechanisms to resist different stages of trematode infection, and that each set of mechanisms has
74  broad categories: chemical contaminants and trematode infection.
75 ng effects on tadpole per capita exposure to trematode infection.
76 g this outcome are in need of further study, trematode infections appear to benefit hosts that are ex
77 indirectly increased host susceptibility and trematode infections by (1) increasing time spent in sus
78 l relationship between atrazine and elevated trematode infections in amphibians.
79 romising drug candidate to control neglected trematode infections in human and animal health.
80 urd uses examples of parasitized insects and trematode infections of snails to consider the evolution
81                                              Trematode infections such as schistosomiasis and fasciol
82 known immunomodulators in human nematode and trematode infections, C. elegans is unique as a non-para
83 ng data from a study of Schistosoma mansoni (trematode) infections in Biomphalaria glabrata snails, w
84 s consistent with mechanical perturbation by trematode infestation but not with the effects of retino
85 ti-centennial scale changes in prevalence of trematodes infesting the bivalve host Abra segmentum thr
86                               This parasitic trematode is endemic in sub-Saharan Africa and the Middl
87 elanoides tuberculata and 2 of the fishborne trematodes it transmits (Haplorchis pumilio and Centroce
88 o digenetic trematode parasites, and bind to trematode larval surfaces, suggestive of a role in inter
89 r relative susceptibility of host species to trematodes, leading to cascading effects on the diversit
90  Opisthorchis felineus, a bile duct-dwelling trematode liver fluke is highly endemic.
91 e and snails, and had tadpoles with elevated trematode loads, further supporting a causal relationshi
92  involved in reproductive development in the trematode model Schistosoma mansoni.
93                           Using an amphibian-trematode model, we tested how NaCl influences diversity
94                                              Trematode.net is an independent component of Helminth.ne
95 d user-friendly manner and (ii) we introduce Trematode.net, a site dedicated to the dissemination of
96 ntroduces and presents its new sibling site, Trematode.net.
97  a collection of databases: Nematode.net and Trematode.net.
98                                          The trematodes obtained from each snail were pooled and test
99             Fasciola hepatica is a parasitic trematode of global importance in livestock.
100 ndemic parasitic infection caused by various trematodes of the genus Schistosoma.
101 logenetic relatives of these helminths, also trematodes of the phylum Platyhelminthes and major human
102 asis, a neglected tropical disease caused by trematodes of the Schistosoma genus, affects over 250 mi
103 d Schistosoma indicum are ruminant-infecting trematodes of the Schistosoma indicum group that are wid
104 gate these hidden dynamics for the foodborne trematode Opisthorchis viverrini, which is a primary cau
105                                              Trematodes or tadpoles were independently acclimated to
106 riae) of two of their most common species of trematode, Ornithodiplostomum ptychocheilus and Posthodi
107 in the extremely O2-avid hemoglobin from the trematode Paramphistomum epiclitum have been investigate
108 ion-resistant oxy-myoglobin complex from the trematode Paramphistomum epiclitum, and the residues wer
109     Sequence alignment of hemoglobins of the trematodes Paramphistomum epiclitum and Gastrothylax cru
110 es of growth, development and survival), the trematode parasite (production of the cercaria infective
111 of mortality induced by an endemic, virulent trematode parasite (Ribeiroia ondatrae) on hundreds of a
112 ogs (Hyla regilla) exposed to cercariae of a trematode parasite (Ribeiroia sp.).
113                                          The trematode parasite community had the fifth highest dry b
114 Here, we combined field-derived estimates of trematode parasite infections in aquatic snails with mea
115                                          The trematode parasite Ribeiroia ondatrae sequentially infec
116  frog tadpoles (Lithobates clamitans) by the trematode parasite Ribeiroia ondatrae with fully replica
117            Aquatic larvae (cercariae) of the trematode parasite Schistosoma mansoni rapidly penetrate
118                           Infection with the trematode parasite Schistosoma mansoni results in a dist
119 emarkable activity against the blood-feeding trematode parasite Schistosoma mansoni.
120 e major intermediate hosts for the digenetic trematode parasite Schistosoma mansoni.
121 ish were exposed to a fixed dose of a common trematode parasite.
122 ng cercariae stages of two common freshwater trematode parasites (Plagiorchis spp., Trichobilharzia f
123                      Using a guild of larval trematode parasites (six species) and an amphibian host,
124  ecosystem-level biomass and productivity of trematode parasites alongside the biomass of free-living
125 workers on the role of NO in defense against trematode parasites and of Kanazawa et al. on cestode pa
126  was postulated that components derived from trematode parasites block receptors on the defence cells
127 and replication in their intermediate hosts, trematode parasites down regulate host defence responses
128 ylus variegatus and Aplodactylus punctatus), trematode parasites for a keyhole limpet (Fissurella lat
129 a significant amount of energy moves through trematode parasites in freshwater pond ecosystems, and t
130 uably the most unique biological features of trematode parasites involve their clonal parthenitae and
131               These results demonstrate that trematode parasites play underappreciated roles in the e
132 biomass flow from snails Juga plicifera into trematode parasites relative to aquatic vertebrate preda
133 up-regulated following exposure to digenetic trematode parasites, and bind to trematode larval surfac
134 tebrates because of their role in supporting trematode parasites, the larvae of which have antagonist
135  involving freshwater snails, amphibians and trematode parasites, we conducted a year-long, outdoor e
136 cept - subversion of host cell signalling by trematode parasites.
137 val amphibian exposure and susceptibility to trematode parasites.
138 s), that are secreted by medically important trematode parasites.
139 sible candidates) of the abundance of larval trematodes (parasitic flatworms) in the declining northe
140  will lead to significant intensification of trematode parasitism, suppressed fecundity of common ben
141 velopmental pathway in the life cycle of the trematode pathogen Schistosoma mansoni.
142 parasite 'extended phenotype', consisting of trematode plus castrated host biomass, exceeded the indi
143 uence of the 16S rRNA gene identified in one trematode pool was identical to that of the type strain
144                        Out of a total of 209 trematode pools, 50 pools were found to be positive by P
145 ographic regions and persistent infection of trematode populations with E. risticii during summer and
146 ocestus formosanus, are fishborne intestinal trematodes recognized as being important human pathogens
147 nly consume free-living cercariae (parasitic trematodes) reduced metacercarial infections in tadpoles
148 site transmission mode affected Chaetogaster-trematode relationships using data from 20,759 snails co
149     Comparing the HDMs from several zoonotic trematodes revealed a similar capacity for immune regula
150 nts to test how transmission of the virulent trematode Ribeiroia ondatrae was affected by the diversi
151 ns: wetlands with a greater abundance of the trematode Ribeiroia ondatrae were more likely to have ma
152 trials with P. regilla and the most virulent trematode (Ribeiroia ondatrae), experimental reductions
153 dies has implicated infection by a digenetic trematode--Ribeiroia ondatrae--as an important cause of
154 tal schistosomiasis, caused by the parasitic trematode Schistosoma haematobium, affects over 112 mill
155  and chronic disease caused by the parasitic trematode Schistosoma mansoni after deposition of eggs i
156  while conferring immunity to the intestinal trematode Schistosoma mansoni Here, we report that abrog
157                            The intravascular trematode Schistosoma mansoni is a causative agent of sc
158                                          The trematode Schistosoma mansoni is one of the etiological
159                                The digenetic trematode Schistosoma mansoni that causes the form of sc
160 g lymphoid tissues of mice infected with the trematode Schistosoma mansoni.
161 oots with potent activity against pathogenic trematode Schistosoma mansoni.
162  Biomphalaria glabrata to infection with the trematode Schistosoma mansoni.
163 e a population of neoblast-like cells in the trematode Schistosoma mansoni.
164 tein produced exclusively by the eggs of the trematode Schistosoma mansoni.
165            One representative example is the trematode Schistosoma, which causes schistosomiasis, an
166 new NRs were identified in the Platyhelminth trematode, Schistosoma mansoni.
167                                 Unlike other trematodes, schistosomes exhibit distinct sexes, with eg
168  reports documenting division of labor and a trematode soldier caste have involved soldiers that may
169 soldier, while extending the phenomenon of a trematode soldier caste to freshwater and to an invasive
170 des clear evidence for an obligately sterile trematode soldier, while extending the phenomenon of a t
171           But sexual adults are the focus of trematode species-level taxonomy, partially explaining t
172 bout whether similar cell types exist in any trematode species.
173 ausative agent of Potomac horse fever, using trematode stages collected from Juga yrekaensis snails.
174                    Here, we confirm that the trematode stages infectious to people (metacercariae) co
175 nsume and respond numerically to free-living trematode stages released from infected snails (cercaria
176    Upon exposure to infection with digenetic trematodes such as Echinostoma paraensei, the freshwater
177 ivity of heme, and (ii) MF6p/HDMs from other trematodes, such as Opisthorchis viverrini and Paragonim
178 rphosis) in our tadpole-parasitic cercarial (trematode) system, would be a negative and positive pred
179 es commensally on freshwater snails and nine trematode taxa that infect snails.
180  mature snails were infected with one of six trematode taxa, amounting to a density of 13 infected sn
181 e effects of atrazine were consistent across trematode taxa.
182                             On average, each trematode taxon produced between 14 and 1660 free-swimmi
183 but soldiers readily attacked heterospecific trematodes that coinfect their host.
184  active (i.e. mobile), free-living stages of trematodes that infect snails (miracidia), but not the p
185 etogaster were less likely to be infected by trematodes that rely on active transmission.
186                                              Trematodes that were acclimated to intermediate temperat
187 nal mass of infected hosts that consisted of trematode tissue (M = 31% per snail).
188 thin each infected snail consisted of larval trematode tissue, which collectively accounted for 87% o
189 ived from gene duplication, as are all known trematode TKs.
190  risticii was found to be transmittable from trematodes to mice and was subsequently passaged from in
191 those same fish species in ways conducive to trematode transmission (namely, eating fish unfrozen and
192                     Our results suggest that trematode transmission mode mediates the net outcome of
193       The annual production of free-swimming trematode transmission stages was greater than the combi
194 lighting the risk that additional pathogenic trematodes transmitted by the snail in its native range
195 h the mostly hematophagous but endoparasitic trematodes (Trematoda), rather than sharing a common anc
196 e exhibiting elevated abundance was the same trematode used in the laboratory infection.
197                               The biomass of trematodes was particularly high, being comparable to th
198  of yearly biomass transfer from snails into trematodes were slightly higher than the combined estima
199 ium, five nematodes, three cestodes, and one trematode) were included in the negative specimens.
200 differed in their relative susceptibility to trematodes when exposed to NaCl.
201                   In stark contrast to other trematodes, which do not exhibit the same host-manipulat
202                                          For trematodes with active infectious stages, predatory Chae
203                    Conversely, infections by trematodes with passive infectious stages were positivel
204 sion of an entire guild of parasites (larval trematodes) within 902 amphibian host communities, we sh
205 regulatory peptides exclusively expressed by trematode worms.

 
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