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1 ne nephropathy (e.g., caused by indinavir or triamterene).
2 was a beta-blocker, 1 was the combination of triamterene and hydrochlorothiazide, and 1 an alpha-bloc
4 rbital, phenytoin, primidone, sulfasalazine, triamterene, and trimethoprim) during the first or secon
5 h include such common drugs as trimethoprim, triamterene, carbamazepine, phenytoin, phenobarbital, an
6 ), and determined the effect of the diuretic triamterene-hydrocholothiazide (T-HCTZ), or anakinra in
8 ermore, inhibition of the TGR5 receptor with triamterene or of deiodinase II downstream of the TGR5 r
9 250 microM phloretin (perfusate); (b) 100 nM triamterene (perfusate); (c) 1 mM ouabain (bath); and (d
10 screening resulted in three promising drugs-triamterene, phenazopyridine, and CRA_1801-with predicte