戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1  the keto group at C-8 and hence is a type A trichothecene.
2 about cellular protection mechanisms against trichothecenes.
3 t not in related species that do not produce trichothecenes.
4 sphate, the immediate molecular precursor of trichothecenes.
5 ter (QQQ) for screening of "unknown unknown" trichothecenes.
6 exploited to detect evidence of type A and B trichothecenes.
7 any under-characterized and unknown modified trichothecenes.
8 llected in Hawaii, was a source of three new trichothecenes, 3-hydroxyroridin E (1a), 13'-acetyltrich
9 y the PIS method to a laboratory-synthesized trichothecene, a first step in demonstrating the power o
10                         Occurrence of type A trichothecenes, Alternaria mycotoxins and their conjugat
11 e infested seeds are often contaminated with trichothecene and estrogenic mycotoxins that pose a seri
12 NK/p38 kinases and induction of apoptosis by trichothecenes and anisomycin.
13 ses and induction of apoptosis) of apoptotic trichothecenes and anisomycin.
14 lready reported for legislated mycotoxins as trichothecenes and zearalenone (ZON) separately, we desc
15 otein synthesis inhibitor that competes with trichothecenes (and anisomycin) for ribosome binding, al
16                                              Trichothecenes are isoprenoid mycotoxins produced in whe
17                                              Trichothecenes are phytotoxic sesquiterpenic mycotoxins
18                                              Trichothecenes are terpene-derived secondary metabolites
19                        Some fungal genes for trichothecene biosynthesis (Tri genes) are known to be u
20                                              Trichothecene biosynthetic enzymes accumulate in organiz
21 ium graminearum and F. sporotrichioides have trichothecene biosynthetic genes (TRI) at three loci: a
22        Conserved functional regions of three trichothecene biosynthetic genes (TRI1, TRI8, and TRI13)
23 first metabolic intermediate specific to the trichothecene biosynthetic pathway.
24                          Fluorescence tagged trichothecene biosynthetic proteins co-localize with the
25 n the parental strain treated with different trichothecenes, but not in a petite version of the paren
26 tion of unknown, though structurally similar trichothecenes, by leveraging objective ML techniques.
27 ient mutants, suggesting that elimination of trichothecene-damaged mitochondria by mitophagy improves
28  the occurrence of eighteen mycotoxins, nine trichothecenes (deoxynivalenol, 3-acetyl-deoxynivalenol,
29 revealed the presence of 4 out of 12 studied trichothecenes: DON (deoxynivalenol), 15AcDON (15-acetyl
30 f beer a combined solid phase extraction for trichothecenes, enniatins, beauvericin and zearalenone w
31 the final product is not suitable to reflect trichothecene exposure.
32                                          The trichothecene family of mycotoxins inhibit protein synth
33 einforced the interconvertibility of group B trichothecene forms during bread making and digestion.
34 s and toxin evolution, a 19-kb region of the trichothecene gene cluster was sequenced in 39 strains c
35 ichodiene synthase) and probably maps in the trichothecene gene cluster.
36 nt and evidence of adaptive evolution within trichothecene genes are also reported.
37 ssibility and permeation capacity of group B trichothecenes in bread by different parameters of ferme
38 used as a representative compound for type-B trichothecenes in this detection scheme.
39 l protein synthesis as a novel mechanism for trichothecene-induced cell death.
40 y is a cellular protection mechanism against trichothecene-induced mitochondrial oxidative stress and
41 le and sensitive method for the screening of trichothecenes is important to prevent economic loss and
42                            To understand the trichothecene mechanism of action, we screened the yeast
43               Strain-specific differences in trichothecene metabolite profiles (chemotypes) are not w
44                   To determine the effect of trichothecene metabolites on gene expression, cultures w
45                      In rice cultures, a new trichothecene mycotoxin (named NX-2) was characterized b
46 ivalenol (DON) is a toxicologically relevant trichothecene mycotoxin frequently found in cereal produ
47 nome-wide insight into the mode of action of trichothecene mycotoxins and uncover a critical role for
48                                              Trichothecene mycotoxins are natural contaminants of sma
49 e isolated, which produced none of the known trichothecene mycotoxins despite causing normal disease
50 expensive method for the detection of type-B trichothecene mycotoxins has been developed in our labor
51                                              Trichothecene mycotoxins in animal feed and human food c
52                                              Trichothecene mycotoxins synthesized by Fusarium species
53 ion, scabby grain is often contaminated with trichothecene mycotoxins that act as virulence factors o
54                          The fungus produces trichothecene mycotoxins that render grain unsuitable fo
55 , these findings support the hypothesis that trichothecene mycotoxins, and in particular NIV, have th
56 nzyme A to the C3 hydroxyl moiety of several trichothecene mycotoxins.
57 or both of these enzymes with coenzyme A and trichothecene mycotoxins.
58     When triggered to produce sesquiterpene (trichothecene) mycotoxins, the endoplasmic reticulum (ER
59  total of 12 mycotoxins simultaneously, nine trichothecenes (NIV, DON, FUS-X, DAS, 15-AcDON, 3-AcDON,
60 graminearum, a fungus able to produce type B trichothecenes on cereals, including deoxynivalenol (DON
61 ety in DON is common to virtually all type-B trichothecenes, our approach may be ideal for type-speci
62 uccessfully applied for the determination of trichothecenes, patulin and zearalenone in 182 milled gr
63              The locus governing the type of trichothecene produced (nivalenol or deoxynivalenol) cos
64         Harzianum A (HA) is a non-phytotoxic trichothecene produced by Trichoderma arundinaceum.
65     Isolates of F. graminearum can differ in trichothecene production phenotypes (chemotypes), with i
66               Cotreatment with rapamycin and trichothecenes reduced ROS levels and cytotoxicity in th
67   To identify cellular functions involved in trichothecene resistance, we screened the Saccharomyces
68  oxidative stress and a potential target for trichothecene resistance.
69 gesting that oxidative stress contributes to trichothecene sensitivity.
70                  We have found that selected trichothecenes strongly activate JNK/p38 kinases and ind
71 uce systemic defences indicates that complex trichothecene structures may not be necessary for induci
72 s in activity of these enzymes toward B-type trichothecenes such as deoxynivalenol.
73                                              Trichothecene (TCN) contamination in food and feed is a
74                                              Trichothecenes that inhibit protein synthesis without ac
75                                        Among trichothecenes that strongly activate JNK/p38 kinases, i
76                                        Among trichothecenes that strongly inhibit protein synthesis,
77 esistance to Fusarium species by detoxifying trichothecenes through de-epoxidation.
78  Artificially inoculated grasses accumulated trichothecenes to a much lesser extent than wheat, and n
79           Although the ability of individual trichothecenes to inhibit protein synthesis and activate
80 less than 10(0.7) L/kg(oc) (e.g., all type B trichothecenes) to 10(4.0) L/kg(oc) (positively charged
81 ting a role for fully active mitochondria in trichothecene toxicity.
82                               Loci governing trichothecene toxin amount and type (deoxynivalenol or n
83 rk exists on the detection of commonly known trichothecene toxins, biological, environmental, and oth
84 AG42R in Fusarium sporotrichioides increases trichothecene (TR) mycotoxin production and alters TR ge
85 icity in the parental strain relative to the trichothecene treatment alone, but not in mitophagy defi
86 idated for the simultaneous extraction of 12 trichothecenes (type A and type B) from baby foods, foll
87                              Cytotoxicity of trichothecenes was alleviated after treatment of the par