ライフサイエンスコーパス: triclosan

1 als treated with the lowest doses of MPB and triclosan.

2 combination of rifampicin, sparfloxacin and triclosan.

3 e of different concentrations of the biocide Triclosan.

4 LR ligands in the presence and/or absence of triclosan.

5 DEET, carbamazepine, dilantin, naproxen, and triclosan.

6 55, which is abrogated by pre-treatment with triclosan.

7 and exposure history alter the influence of triclosan.

8 rnal concentrations followed by tonalide and triclosan.

9 CI: 0.97, 1.4) per log(10) unit increase in triclosan.

10 phthalate, and 8,000-24,000 ppm (range) for triclosan.

11 thpaste with polymer and fluoride containing triclosan.

12 to those achieved with toothpaste containing triclosan.

13 containing chlorophenol side-chains such as triclosan.

14 , ketorolac, and Total toothpaste containing triclosan.

15 e oxidation of either mono- or dichlorinated triclosan.

16 inical strains to survive in the presence of triclosan.

17 is the target of the antibacterial compound, triclosan.

18 , ethionamide, ofloxacin, thiolactomycin and triclosan.

19 oxylated biphenyls in comparison to that for triclosan.

20 edict the environmental toxicity behavior of triclosan.

21 ent inducers of the M6 operon in addition to triclosan.

22 n cells were exposed to P25 and 15.625 mug/L triclosan.

23 nd propyl parabens), 2,5-dichlorophenol, and triclosan.

24 children was also positively associated with triclosan.

25 nd identified sociodemographic predictors of triclosan.

26 nol, ibuprofen, diclofenac, bisphenol A, and triclosan.

27 on balloons were inflated with a solution of triclosan (10 g/L), the catheters drained freely for at

28 none-3 (2-hydroxy-4-metoxybenzophenone); and triclosan (2,4,4'-trichloro-2'-hydroxyphenyl ether).

29 o two groups: (1) toothpaste containing 0.3% triclosan + 2.0% PVM/MA copolymer + 1450 ppm fluoride (t

30 analytes studied (gemfibrozil, nonylphenol, triclosan, 2,4,6-trichlorophenol, and naphthenic acids),

31 We detected benzophenone-3, triclosan, 2,4-dichlorophenol, 2,5- dichlorophenol, and

32 available positive control dentifrice (0.30% triclosan/2.0% Gantrez copolymer).

33 ct concentrations (LOAECs) were 1.04 muM for triclosan, 3.17 muM for triclocarban, 0.372 muM for bis-

34 at were amended with lower concentrations of triclosan (5 mg/kg and 50 mg/kg) initially diverged and

35 Triclosan [5-chloro-2-(2,4-dichlorophenoxy)phenol; TCS]

36 inary inhibition studies, we have shown that triclosan, 5-ethyl-2-phenoxyphenol (EPP), and 5-chloro-2

37 ng on the availability of vanadate salts and triclosan, a component of PIA.

38 s chlorination experiments were performed on triclosan, a previously studied environmental contaminan

39 This enzyme is also sensitive to triclosan, a synthetic antimicrobial diphenyl ether.

40 rophenol, pyraclostrobin, sulfasalazine, and triclosan, achieving detection limits of 0.07, 0.24, 10.

41 -Xpress to identify responses to the biocide triclosan across a range of concentrations.

42 , triclosan dispersion by increased biomass, triclosan adherency on algal exudates, and triclosan ads

43 , triclosan adherency on algal exudates, and triclosan adsorption site reduction on algae surface owi

44 ased concomitantly with the disappearance of triclosan after a 15 min reaction, the sustained toxicit

45 At the end of the experiment, all triclosan-amended Archaea communities had diverged from

46 Triclosan amendment caused all of the Bacteria and Archa

47 ficantly increased with the level of urinary triclosan among male subjects (odds ratio for third vs f

48 Exposure to triclosan, an antimicrobial used in many consumer produc

49 The x-ray crystal structures of five triclosan analogs, in addition to that of the isoniazid-

50 In this paper, new coumarin-based triclosan analogues are reported and their biological pr

51 hod to calculate the affinity of a series of triclosan analogues for the E. coli enoyl reductase FabI

52 ly reduce the teratogenicity of bisphenol A, triclosan and 17alpha-ethinyl estradiol without generati

53 ditional evidence for an association between triclosan and allergy.

54 eactions with free chlorine alone, while for triclosan and bisphenol A the free iodine mediated trans

55 Using triclosan and chlorophene as probe compounds, pseudofirs

56 It is clear, however, that triclosan and CTDs are the dominant source of these cong

57 ing one between the ubiquitous antimicrobial triclosan and erm(X), a 23S rRNA methyltransferase impli

58 A nonmonotonic association was observed for triclosan and girls' adiposity; however, it was due to e

59 dren and exposure to the antibacterial agent triclosan and having filaggrin (FLG) loss-of-function mu

60 persive SPE cleanup procedure for extracting triclosan and methyltriclosan from fish roe and surimi s

61 TiO(2), precursor-complex formation between triclosan and MnO(2) was much slower and likely became t

62 r placental weight retained four biomarkers: triclosan and monocarboxy-isononyl phthalate (MCNP), whi

63 ical for the high quantum yield measured for triclosan and necessary for the photodegradation mechani

64 trend toward an inverse association between triclosan and paraben exposure and allergic sensitizatio

65 association between prenatal and early-life triclosan and paraben exposure and childhood allergic di

66 However, levels of the antimicrobial EDCs triclosan and parabens were significantly associated wit

67 In cross-sectional studies triclosan and parabens, ubiquitous ingredients in person

68 ol and phenanthrene) and additive chemicals (Triclosan and PBDE-47).

69 d children, we examined associations between triclosan and personal-care product use.

70 to increase tolerance of the M6- strains to triclosan and plumbagin by effectively ablating the chan

71 Triclosan and plumbagin effectively altered the intracel

72 sed with the level of the antimicrobial EDCs triclosan and propyl and butyl parabens (P </= .04).

73 samples spiked with the model "unknown" EDCs triclosan and tetrabromobisphenol-A.

74 results in markedly increased resistance to triclosan and the purified enzyme is much more resistant

75 ewed research, this statement concludes that triclosan and triclocarban are environmentally persisten

76 The polychlorinated aromatic antimicrobials triclosan and triclocarban are in widespread use for kil

77 The Florence Statement on Triclosan and Triclocarban documents a consensus of more

78 other hazards to humans and ecosystems from triclosan and triclocarban is presented along with recom

79 Triclosan and triclocarban were found in the extracts of

80 Two common (triclosan and triclocarban) and four emerging (2,4,5 tri

81 rds, among others transformation products of triclosan and triclocarban.

82 of demonstrated benefit from common uses of triclosan and triclocarban.

83 urinary chemical concentrations of parabens, triclosan and triclorcarban in relation to TTP; chemical

84 and analyzed the interaction of saFabI with triclosan and two related diphenyl ether inhibitors.

85 nols (e.g., bisphenol A, benzophenone-3, and triclosan) and parabens is widespread in the population.

86 ting chemicals such as phthalates, parabens, triclosan, and benzophenone-3 (BP-3) for adolescent girl

87 f the phenolic compounds 2,4-dichlorophenol, triclosan, and bisphenol A in chlorinated waters contain

88 nalyzed for phthalate metabolites, parabens, triclosan, and BP-3 using high-performance liquid chroma

89 labeled to be free of phthalates, parabens, triclosan, and BP-3, can reduce personal exposure to pos

90 g10 concentrations of widely monitored HHCB, triclosan, and carbamazepine explained 71-82% of the var

91 nilides, 2,6-dinitroanilines, chlorophenols, triclosan, and guanidines significantly increased in bot

92 e been relatively well studied, bisphenol A, triclosan, and ibuprofen.

93 m of the bacteriostatic agents triclocarban, triclosan, and its transformation product methyl triclos

94 Copper, permethrin, chromium, triclosan, and lead were also important.

95 ntaminants as malathion, glyphosate, diuron, triclosan, and nonylphenol were assessed.

96 ons and urinary bisphenol A, benzophenone-3, triclosan, and paraben levels in male and female childre

97 scopy results indicate that phenol, aniline, triclosan, and related byproducts are associated with th

98 ), bisphenols, benzophenone-type UV filters, triclosan, and triclocarban are used in a variety of con

99 l phthalate (DEP), methyl paraben (MPB), and triclosan] and urine and serum concentrations of their r

100 n urinary concentrations of triclocarban and triclosan, another commonly used antibacterial agent.

101 n northern MN, the four dioxins derived from triclosan are present prior to the patent of triclosan,

102 om 9 phenols (4 parabens, 2 dichlorophenols, triclosan, benzophenone-3, bisphenol A) and 11 phthalate

103 associations between other compounds such as triclosan, benzophenone-3, MCNP, and MCOP and both place

104 ther exposures (total parabens, bisphenol A, triclosan, benzophenone-3, total phthalates, and 2,4-DCP

105 ination Survey in which urinary bisphenol A; triclosan; benzophenone-3; propyl, methyl, butyl, and et

106 scence binding experiments, we conclude that triclosan binds to the enzyme-NAD(+) product complex whi

107 strate specificity that encompasses not only triclosan but the detergent SDS, which are often used to

108 0156-PA0157-PA0158 pump, which only effluxed triclosan, but not closely related compounds, antibiotic

109 ption, biodegradation, and photocatalysis of triclosan by algae and P25, triclosan dispersion by incr

110 explain the observed adsorption behavior of triclosan by the binary oxide mixtures and single oxides

111 to strong adsorption and slow desorption of triclosan by TiO(2), precursor-complex formation between

112 at the sensitivity and fragility of algae to triclosan can be amplified with time extension.

113 y showed that wetland efficiency at removing triclosan can theoretically be increased by limiting DOC

114 High doses of triclosan caused irreversible damage on algae, and envir

115 Previously exposed (to triclosan) communities were amended with 5, 50, and 500

116 when P25 interacted with 62.5 and 250 mug/L triclosan compared with triclosan-only exposure.

117 We show that the AcpP-triclosan complex demonstrates nM binding, inhibits in v

118 m the control communities, regardless of the triclosan concentration added.

119 h percentile, and 4-fold higher than maximum triclosan concentration that has been detected in U.S. b

120 zation among those in the fourth quartile of triclosan concentration was 2.0 [95% confidence interval

121 JK) Delta(mexXY) strain PAO509 to increasing triclosan concentrations and derived a resistant strain,

122 he results were different based on different triclosan concentrations and measurement end points.

123 Triclosan concentrations decreased by 35.7% (95% CI: -53

124 Our results suggest that urinary triclosan concentrations have modest stability over week

125 erns, variability, and predictors of urinary triclosan concentrations in 389 mother-child pairs enrol

126 Triclosan concentrations were associated with allergic s

127 All eight triclosan conjugates identified in the cell cultures wer

128 aste regimen: control dentifrice (n = 21) or triclosan-containing dentifrice ([CT] n = 18).

129 The triclosan-containing dentifrices are effective in contro

130 tracts of intact carrot plants cultivated on triclosan contaminated soils.

131 frice in reducing gingivitis compared to the triclosan/copolymer control in this partially supervised

132 L and root caries versus the sodium fluoride/triclosan/copolymer control in xerostomic patients.

133 This study evaluates the effect of triclosan/copolymer dentifrice on the 6-month clinical r

134 the limits of the present study, the use of triclosan/copolymer dentifrice promoted additional clini

135 n = 15) subjected to FMUD and daily use of a triclosan/copolymer dentifrice.

136 taphosphate dentifrice to a positive control triclosan/copolymer dentifrice.

137 ve control dentifrice (sodium fluoride/0.30% triclosan/copolymer) in 440 medication-induced xerostomi

138 es were amended with 5, 50, and 500 mg/kg of triclosan, corresponding to the median, 95th percentile,

139 This research suggests that triclosan could inhibit methane production in anaerobic

140 he accumulation rates and temporal trends of triclosan, CTDs, and dioxins in aquatic sediments were f

141 ources recorded increasing concentrations of triclosan, CTDs, and their derived dioxins since the pat

142 of waters to these compounds, the levels of triclosan, CTDs, and their derived dioxins were determin

143 Overall, concentrations of triclosan, CTDs, and their dioxins were higher in small-

144 2) dentifrice relative to a positive control triclosan dentifrice for prevention of clinical attachme

145 icrobial agent in handsoaps, and chlorinated triclosan derivatives (CTDs, formed during disinfection

146 hotocatalysis of triclosan by algae and P25, triclosan dispersion by increased biomass, triclosan adh

147 positive bacteria are naturally resistant to triclosan due to the presence of the triclosan-resistant

148 mains of TriA and TriB differentially affect triclosan efflux and assembly of the complex.

149 , but not OprM, for assembly of a functional triclosan efflux pump.

150 he function, composition and assembly of the triclosan efflux transporter TriABC-OpmH from Pseudomona

151 ded parabens, phthalates, bisphenol A (BPA), triclosan, ethanolamines, alkylphenols, fragrances, glyc

152 Controls for the MPB and triclosan experiments were animals treated with olive oi

153 Novel genes identified as being sensitive to triclosan exposure include those involved in barrier fun

154 We investigated whether triclosan exposure was associated with allergic sensitiz

155 e) and one personal care product ingredient (triclosan) for the oligochaete Lumbriculus variegatus.

156 Uptake of Triclosan from PVC diminished the ability of worms to en

157 thyl paraben, butyl paraben, diclofenac, and triclosan) from exposed mussels on the basis of the Vers

158 y methylparaben, propylparaben, bisphenol A, triclosan, galaxolide, and 4- methylbenzilidene camphor

159 stic actions, and the personal care products triclosan, galaxolide, and nonylphenol are representativ

160 ESR increased at a slower rate in the triclosan group (P approximately 0.06) and was less like

161 te than either patients with diabetes in the triclosan group or patients without diabetes in each gro

162 atins but not anti-inflammatory drugs in the triclosan group versus the placebo group.

163 of </=5%, bisphenol A, caffeine, NP, OP, and triclosan had false positive rates >15%.

164 The phototransformation of triclosan has been a matter of longstanding interest due

165 analysis of the inhibition of the enzyme by triclosan has been conducted showing that triclosan is a

166 crobial agents such as stannous fluoride and triclosan have been incorporated into dentifrice formula

167 expressing FabI(Y147H) was also resistant to triclosan; however, the strain expressing FabI(M99T) was

168 very little information, however, about how triclosan impacts microbial communities in anaerobic dig

169 We investigated how triclosan impacts the community structure, function and

170 Triclosan-impregnated sutures did not statistically sign

171 : 0.21-5.43; P = 0.93; I = 44%) CONCLUSIONS: Triclosan-impregnated sutures do not decrease the rate o

172 mized controlled trials (RCTs) investigating triclosan-impregnated sutures were conducted.

173 decrease the rates of SSIs may be the use of triclosan-impregnated sutures.

174 We detected triclosan in >70% of urine samples.

175 nd their derived dioxins since the patent of triclosan in 1964.

176 We quantified triclosan in 3 urine samples collected from women betwee

177 The toxic mechanism of triclosan in a wide range of concentrations was analyzed

178 se oxide [MnOx(s)] with phenol, aniline, and triclosan in batch experiments using X-ray photoelectron

179 Degradation of triclosan in near-surface sunlit waters was relatively f

180 rs of diethyl phthalate, methyl paraben, and triclosan in rats.

181 tylmethoxydibenzoylmethane, triclocarban and triclosan in the aquatic environment.

182 Cyperin mimicked the binding of triclosan in the binding pocket of ENR.

183 and oxidative stress at 1000 and 4000 mug/L triclosan in the presence of P25.

184 This study explored the toxicity of triclosan in the presence of TiO(2) P25 to the green alg

185 yl paraben, benzophenone-3, bisphenol A, and triclosan in urine collected during 1997-2006 from 50 pa

186 of alkyl esters of p-hydroxybenzoic acid and triclosan in vitro on the basis of steady-state fluoresc

187 s, benzophenone-3, BPA, two dichlorophenols, triclosan) in spot urine samples collected during pregna

188 by triclosan has been conducted showing that triclosan is a competitive inhibitor with respect to NAD

189 Triclosan is a frequently detected and toxic antimicrobi

190 Triclosan is a high-production volume chemical, which ha

191 Triclosan is a widespread antimicrobial agent that accum

192 Triclosan is endemic in modern life, but there is uncert

193 Triclosan is known to cause severe oxidative stress, and

194 phthalates and phenols such as parabens and triclosan, is ubiquitous within the U.S. general populat

195 nts was assessed to exceed the amount of the triclosan itself by a factor of 5.

196 unexposed communities were exposed to 500 mg triclosan/kg, function was maintained, but the abundance

197 Triclocarban and methyl triclosan lack a phenolic group and remained unaltered i

198 as a significant interaction between sex and triclosan level, with male subjects being more likely to

199 trogenic activity of CPC, BAK, rotenone, and triclosan may be related to their mitochondrial inhibito

200 Mass ratios of DCC-to-TCC and of methyl-triclosan (MeTCS)-to-TCS, serving as indicators of trans

201 Triclosan, methyl paraben, and propyl paraben concentrat

202 vious studies: 1,735 (DEP), 1,050 (MPB), 50 (triclosan) mg/kg/day.

203 y where the enzyme contained either a single triclosan molecule, in a configuration typical of other

204 al ENR:triclosan structures, or harbored two triclosan molecules bound to the active site.

205 f synthetic musk fragrance compounds, methyl triclosan (MTCS), polychlorinated biphenyls, organochlor

206 tal, Brisbane, Australia, were randomized to triclosan (n = 193) or placebo (n = 190) groups and asse

207 long-term impacts of a pulse disturbance of triclosan on five nontarget green algae in Lake Erie.

208 ical effects of a toothpaste containing 0.3% triclosan on the periodontal parameters of subjects that

209 rmine effects of the anti-inflammatory agent triclosan on the TLR-hyper-inflammatory phenotype, prima

210 Whereas cyperin was much less potent than triclosan on this target site, their ability to cause li

211 h 62.5 and 250 mug/L triclosan compared with triclosan-only exposure.

212 In the lake with no wastewater input, no triclosan or CTDs were detected.

213 association between prenatal and early-life triclosan or paraben concentrations and childhood asthma

214 sociations of maternal plasma or child urine triclosan or paraben concentrations with asthma or recur

215 The differential effects of triclosan or paraben exposure on allergic sensitization

216 her organic chemicals (i.e., benzophenone-3, triclosan, parabens) in human urine, milk, and serum to

217 OC concentration (11 mg/L(-)) would maximize triclosan phototransformation rate while minimizing the

218 tration did not show a significant effect on triclosan phototransformation rate.

219 oncentration that would maximize the rate of triclosan phototransformation while minimizing the accum

220 The chronic toxicity of triclosan presented more distinguishable variations amon

221 ations of antimicrobial chemicals, which for triclosan ranged from 0.5 to 1970 ng/g dust.

222 but only the association in males exposed to triclosan remained statistically significant after corre

223 ss correlation coefficients (ICCs) to assess triclosan reproducibility and identified sociodemographi

224 Triclosan reproducibility was fair to good during pregna

225 ed, but the abundance of a gene encoding for triclosan resistance (mexB) increased.

226 A number of new loci were also implicated in Triclosan resistance and the predicted phenotypes for a

227 those previously reported to have a role in triclosan resistance as well as a new set of genes, incl

228 aeruginosa achieves high-level (MIC>1 mg/ml) triclosan resistance either by constitutive expression o

229 AlbaTraDIS identified all well characterised Triclosan resistance genes, including the primary target

230 this bacterium's high intrinsic and acquired triclosan resistance.

231 tant to triclosan due to the presence of the triclosan-resistant enoyl-ACP reductase isoforms, FabK a

232 A triclosan-resistant target enzyme and perhaps other mech

233 In this study, photolysis of triclosan resulted in several primary photoproducts incl

234 f InhA with the broad spectrum antimicrobial triclosan revealed a unique stoichiometry where the enzy

235 Importantly, triclosan's anti-inflammatory property is effective in a

236 ents of our optimization program to decouple triclosan's specific FabI effect from its nonspecific cy

237 ferent MFPs, TriA and TriB, are required for triclosan/SDS resistance mediated by TriABC-OpmH.

238 the increased tetracycline, penicillin, and triclosan sensitivities resulted from repression of the

239 Triclosan showed long-term effects on biochemical compon

240 e ability to improve the in vitro potency of triclosan significantly by replacing the suboptimal 5-ch

241 configuration typical of other bacterial ENR:triclosan structures, or harbored two triclosan molecule

242 triclosan are present prior to the patent of triclosan, suggesting a secondary source.

243 tional calculations of the excited states of triclosan support the involvement of a radical intermedi

244 in P. aeruginosa shown to efficiently efflux triclosan, supporting the notion that efflux is the prim

245 ic domains, and its inhibition by drugs like triclosan (TCL) can increase cytotoxicity and decrease d

246 Triclosan (TCN) is a long-standing, proven antibacterial

247 rapeutic broad-spectrum antimicrobial agents triclosan (TCS) and benzalkonium chloride (BC) can contr

248 Triclosan (TCS) and triclocarban (TCC) are antimicrobial

249 Removal of triclocarban (TCC) and triclosan (TCS) from wastewater is a function of adsorpt

250 Triclosan (TCS) is a broad-spectrum antimicrobial compou

251 Triclosan (TCS) is a synthetic antibacterial chemical wi

252 Triclosan (TCS) was treated under hydrothermal condition

253 Triclosan (TCS), a broad-spectrum antimicrobial, is used

254 Triclosan (TCS), a high volume chemical widely used in c

255 Triclosan (TCS), a high-production-volume chemical used

256 Triclosan (TCS), an antimicrobial chemical with potentia

257 of bisphenol A (BPA), benzophenone-3 (BP-3), triclosan (TCS), and parabens with serum total testoster

258 ), benzophenone-3 (BP-3), bisphenol A (BPA), triclosan (TCS), butyl paraben (B-PB), methyl paraben (M

259 Triclosan (TCS), employed as an antiseptic and disinfect

260 Triclocarban (TCC) and triclosan (TCS), two of the most commonly used antimicro

261 LISA) for the detection of the antimicrobial triclosan (TCS; 2,4,4'-trichloro-2'-hydroxydiphenyl ethe

262 After dichotomizing SDQ scores, triclosan tended to be positively associated with emotio

263 re/coexposure to other phenolic xenobiotics (triclosan, tetrabromobisphenol A, and bisphenol S) in li

264 he purified enzyme is much more resistant to triclosan than is E. coli FabI.

265 nto surface waters via wastewater effluents, triclosan, the antimicrobial agent in handsoaps, and chl

266 ver weeks to months; children are exposed to triclosan through the use of some personal-care products

267 type mice treated with the SULT1E1 inhibitor triclosan to determine the effect of pharmacologic inhib

268 n of the slow-onset, tight-binding inhibitor triclosan to explore the interactions between the type I

269 The study explored the chronic toxicity of triclosan to green microalga Chlorococcum sp. under mult

270 Triclosan toothpaste is effective in controlling plaque

271 Triclosan toothpaste led to a greater decrease in TC (P

272 These data suggest that triclosan toothpaste may influence some inflammatory bio

273 for a systematic and complete assessment of triclosan toxicity in natural waters and the development

274 evaluation and risk management of real-world triclosan toxicity.

275 This triclosan transformation product was determined to have

276 dations intended to prevent future harm from triclosan, triclocarban, and antimicrobial substances wi

277 stance genes and the antimicrobial chemicals triclosan, triclocarban, and methyl-, ethyl-, propyl-, a

278 of BASL4 overpredicted the concentrations of triclosan, triclocarban, and miconazole in root and shoo

279 DPU predicted concentrations of triclosan, triclocarban, miconazole, carbamazepine, and

280 Earlier research showing that triclosan underwent chlorine addition to form mono- and

281 trends were directly attributed to increased triclosan use, local improvements in treatment, and chan

282 ure to the synthetic antimicrobial chemical, triclosan, used in personal care products, has been hypo

283 Median maternal triclosan varied across pregnancy from 17 to 11 ng/mL, w

284 ch as pentachlorophenol, phenolic acids, and triclosan via surface-catalyzed oligomerization.

285 n), and the aOR per log(10) unit increase in triclosan was 1.2 (95% CI: 1.0, 1.4).

286 Triclosan was 66% (95% CI: 29-113) higher in 8 year olds

287 l in the MnOx(s) surface after reaction with triclosan was detected by XPS survey scans, while no Cl

288 losan, and its transformation product methyl triclosan was investigated after their uptake into carro

289 A toothpaste containing 0.3% triclosan was more effective than a regular fluoride too

290 A fast metabolization of triclosan was observed and eight so far unknown phase II

291 Triclosan was positively associated with adiposity only

292 (2) reactivity only when a limited amount of triclosan was present.

293 Triclosan was used to inhibit specifically Block A, wher

294 Urinary concentrations of triclosan were measured in one first morning void from 6

295 this study, lower chlorinated derivatives of triclosan were synthesized to investigate the impact the

296 a phenolic ring (parabens, bisphenol A, and triclosan) were completely transformed on this anode at

297 and 5-chloro-2-(2,4-dichlorophenoxy)phenol (triclosan)) were measured for 15 sorbents: fresh and car

298 is was associated with the highest levels of triclosan, whereas no association was seen for current a

299 berculosis drug isoniazid and the antiseptic triclosan, which are inhibitors of fatty acid biosynthes

300 rd to the structure-activity relationship of triclosan while affording gains against cultured parasit

301 Smaller cells had higher sensitivity to triclosan, while larger cells had a wider size variation

302 s show a concentration-dependent response to triclosan with different genes important in survival bet

303 hese substituents, the environmental fate of triclosan would be markedly different.