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1 ion of this procedure for children born with tricuspid atresia.
2  suggest a genetic basis for the syndrome of tricuspid atresia.
3 yndrome, 10 had pulmonary atresia, and 1 had tricuspid atresia.
4 ies: tetralogy of Fallot (15 patients, 25%), tricuspid atresia (12 patients, 20%), Ebstein's anomaly
5 (95% confidence interval, 45.8 to 64.4); and tricuspid atresia, 74.6% (95% confidence interval, 62.4
6        We examined a total of 53 hearts with tricuspid atresia and 58 normal hearts matched for age.
7  was previously identified in a patient with tricuspid atresia and large secundum atrial septal defec
8 ct ventricular septum, single ventricle, and tricuspid atresia born in 1996 to 2003 were identified f
9 lar septal defects, tetralogy of Fallot, and tricuspid atresia, defects that resemble those associate
10 with the nonfenestrated Fontan procedure for tricuspid atresia has been gratifying, with most survivo
11                The myocardium of hearts with tricuspid atresia is consistently more fibrotic than nor
12 (n = 16) or secondary (n = 26) palliation of tricuspid atresia (n = 13), hypoplastic left heart syndr
13 ere hypoplastic left heart syndrome (n=346), tricuspid atresia (n=103), tetralogy of Fallot (n=127),
14 and2 endocardial deletion (H2CKO) results in tricuspid atresia or double inlet left ventricle with ac
15                                              Tricuspid atresia (TA) is a common form of congenital he
16 tive heart failure at E13 with a syndrome of tricuspid atresia that includes an absent tricuspid valv
17         Initially designed for patients with tricuspid atresia, this procedure is now offered for a v
18                        Morphology other than tricuspid atresia was associated with increased risk of
19 ultivariable analysis, morphology other than tricuspid atresia was the only significant predictor of
20 present clinical status of 216 patients with tricuspid atresia who had a nonfenestrated Fontan proced
21 l Wnt signaling in the myocardium results in tricuspid atresia with hypoplastic right ventricle assoc
22 s with double-inlet left ventricle (DILV) or tricuspid atresia with transposed great arteries (TA-TGA
23 t ventricle in 10, unbalanced AV canal in 5, tricuspid atresia with transposition of the great vessel