ライフサイエンスコーパス: trigeminal nerve

1 reas innervated by the third division of the trigeminal nerve.

2 n unconditioned stimulus (US) applied to the trigeminal nerve.

3 ds on the head of the fish innervated by the trigeminal nerve.

4 peptide (CGRP)-immunoreactive fibers of the trigeminal nerve.

5 ith the transmission of headache pain by the trigeminal nerve.

6 ntegral role for the neuropeptide-containing trigeminal nerve.

7 tina, that have no direct connections to the trigeminal nerve.

8 y unilateral or bilateral stimulation of the trigeminal nerve.

9 t changes in ADCs in either direction in the trigeminal nerve.

10 stimulus (US) was applied to the ipsilateral trigeminal nerve.

11 d cerebral, is innervated by fibers from the trigeminal nerve.

12 y branches of the ophthalmic division of the trigeminal nerve.

13 ed exposure to odors that also stimulate the trigeminal nerve.

14 stopping and the branching of the mandibular trigeminal nerve.

15 timulation of the supraorbital branch of the trigeminal nerve.

16 ffecting the ophthalmic division (V1) of the trigeminal nerve.

17 lized to the ophthalmic (V1) division of the trigeminal nerve.

18 ck injections to the sensory branches of the trigeminal nerve.

19 or microvascular decompression (MVD) of the trigeminal nerve.

20 ing primary afferent fibers chiefly from the trigeminal nerve.

21 -sensitive (nociceptive) nerve fibers of the trigeminal nerve.

22 l facial pain within areas innervated by the trigeminal nerve.

23 argets along extracellular components of the trigeminal nerve.

24 alization of DPANs in all 3 divisions of the trigeminal nerve.

25 tion, to ablate the ophthalmic branch of the trigeminal nerve.

26 essure, as induced by occlusal loads, on the trigeminal nerve.

27 ocoagulation of the ophthalmic branch of the trigeminal nerve.

28 erior colliculus to the motor nucleus of the trigeminal nerve.

29 TSE) to destroy the ophthalmic branch of the trigeminal nerve.

30 a significant contribution to the ophthalmic trigeminal nerve.

31 al and bilateral), arm and third division of trigeminal nerve.

32 imaged its localization within the brain and trigeminal nerves.

33 whisker formation and also for branching of trigeminal nerves.

34 lie in the trajectories of the olfactory and trigeminal nerves.

35 higher (80.0%) compared to the asymptomatic trigeminal nerves (56.9%).

36 al area 14/16 (86.7%), SRS to the SPG and/or trigeminal nerve 7/9 (77.8%) and MVD 56/73 (76.7%).

37 f the principal sensory nucleus (PrV) of the trigeminal nerve, a major relay station for somatotopic

38 distribution of the ophthalmic branch of the trigeminal nerve, abnormal capillary venous vessels in t

39 a of distribution of the first branch of the trigeminal nerve, accompanied by cranial autonomic sympt

40 Following trigeminal nerve activation, afferent release of CGRP ca

41 We propose that following trigeminal nerve activation, CGRP secretion from neurona

42 nnervation of the nasal mucosa by monitoring trigeminal nerve activity in patients with IR and health

43 Disordered sensory input from trigeminal nerve afferents, such as aberrant feedback fr

44 The trigeminal nerve, along with the cervical nerve roots, s

45 ture branching of the major divisions of the trigeminal nerve and a failure to correctly innervate wh

46 thalmic and maxillary divisions of the right trigeminal nerve and cervical spinal nerve afferents.

47 applied to the nasal epithelium activate the trigeminal nerve and evoke changes in respiratory rate.

48 of afferents from the three branches of the trigeminal nerve and from the lingual branch of the hypo

49 that spry3 is expressed specifically in the trigeminal nerve and in spinal motor and sensory neurons

50 leus, A1-5 and C1-3 nuclei, subnuclei of the trigeminal nerve and nucleus tractus solitarius.

51 ons such as the mesencephalic nucleus of the trigeminal nerve and the interomedial lateral cell colum

52 TSE was confirmed by gross histology of the trigeminal nerve and was considered effective if the cor

53 FD patients have small trigeminal nerves and impaired facial pain and temperatu

54 Activation of trigeminal nerves and release of neuropeptides that prom

55 cquisitions of the cisternal segments of the trigeminal nerves and vessels.

56 ommissure and the mesencephalic tract of the trigeminal nerve) and one ventral (the mammillotegmental

57 erves C1-C8, branches of C2, branches of the trigeminal nerve, and hindlimb nerves.

58 enhancement of the cauda equina nerve roots, trigeminal nerve, and pachymeninges.

59 uclei, motor and mesencephalic nuclei of the trigeminal nerve, and some motor neurons in the spinal c

60 halon and pons, the principal nucleus of the trigeminal nerve, and the dorsal raphe.

61 Two patients with unilateral trigeminal nerve anesthesia-one following basal skull fr

62 e activated by antidromic stimulation of the trigeminal nerve, as well as by acute immobilization str

63 nal fluid (CSF) through direct olfactory and trigeminal nerve-associated pathways originating in the

64 s of partial infraorbital transection of the trigeminal nerve at the cellular level.

65 the diameter and cross-sectional area of the trigeminal nerves at 5 mm from the entry point of the ne

66 The results indicate that trigeminal nerve atrophy can be depicted noninvasively i

67 tering the response to A-fiber inputs to the trigeminal nerve because all stimuli were too weak to ac

68 e, the ophthalmic, maxillary, and mandibular trigeminal nerve branches maintain a somatotopic segrega

69 ists of a thin membrane, innervated by three trigeminal nerve branches that project to a specific nuc

70 al tracers to identified pit-organ-supplying trigeminal nerve branches.

71 reas innervated by cervical nerves or by the trigeminal nerve but do not cross a border defined by th

72 ated by the first and second division of the trigeminal nerve but in some cases also included areas i

73 ed by the first (ophthalmic) division of the trigeminal nerve, but also the back of the head, innerva

74 eted VR1 mRNA from the spinal nucleus of the trigeminal nerve, but not from other areas such as the i

75 es between the peripheral electrosensory and trigeminal nerves, but these senses remain separate in t

76 ed by damage to the ophthalmic branch of the trigeminal nerve by conditions such as herpes simplex or

77 Macrophages, recruited to sciatic/trigeminal nerves by IL-1beta from Schwann cells, increa

78 Injury to branches of the trigeminal nerve can sometimes result in persistent dysa

79 at stimulation of another cranial nerve, the trigeminal nerve, can also cause cortical and thalamic d

80 he lingual nerve, a peripheral branch of the trigeminal nerve, can be damaged during the surgical rem

81 Divergent trigeminal nerve (CN V) differentiation and altered trig

82 t prefigure disrupted differentiation of the trigeminal nerve (CN V), a cranial nerve essential for s

83 Trigeminal nerves collecting sensory information from th

84 nopus embryos growth cones of the mandibular trigeminal nerve contact cells located in the posterior

85 al ganglion nor the ophthalmic branch of the trigeminal nerve contained cholinergic elements.

86 The trigeminal nerve (cranial nerve V), along with other cra

87 nerves (n = 47 patients, n = 50 symptomatic trigeminal nerves) demonstrated ipsilateral neurovascula

88 nella gastroenteritis, transfusion reaction, trigeminal nerve disorder, and urinary tract infection).

89 Trigeminal nerves displayed straight parallel courses at

90 We present a rare case of trigeminal nerve-distributed hyperpigmentation following

91 rative skin hyperpigmentation localized to a trigeminal nerve distribution has not been documented in

92 ly 100% of patients experience trauma to the trigeminal nerve during orthognathic surgery, impairing

93 tro evidence that Robo-Slit signaling guides trigeminal nerves during cornea innervation.

94 Nasal trigeminal nerve endings are particularly sensitive to o

95 uropeptides and their release from meningeal trigeminal nerve endings in the mechanism of migraine, b

96 timulation of the supraorbital branch of the trigeminal nerve evoked relatively larger amplitude blin

97 Detection of such irritants by the trigeminal nerve evokes protective reflexes, including s

98 SCCs are innervated by peptidergic trigeminal nerve fibers but it is currently unknown if i

99 ndence of SCCs on innervation by eliminating trigeminal nerve fibers during development with neurogen

100 cleus also showed substantial innervation by trigeminal nerve fibers immunoreactive for calcitonin ge

101 outlier, with more than twice the number of trigeminal nerve fibers than any other species.

102 ese proteins might be involved in supporting trigeminal nerve fibers that innervate the dental pulp.

103 e to direct depolarization of acid-sensitive trigeminal nerve fibers, for example, polymodal nocicept

104 reversible subcutaneous block of peripheral trigeminal nerve fibers.

105 each case, infrared signals are detected by trigeminal nerve fibres that innervate specialized pit o

106 underwent microvascular decompression of the trigeminal nerve for medically intractable trigeminal ne

107 marked reduction in the calibre of the left trigeminal nerve from the nerve root exit zone in the po

108 The linear lesions extended along the trigeminal nerve, from the root entry zone (REZ) (57.8%,

109 Gem was also observed in developing trigeminal nerve ganglia in 12.5 day mouse embryos, demo

110 ry nerves are all derived as branches of the trigeminal nerve/ganglion similar to the situation encou

111 Effects of polySia removal on trigeminal nerve growth behavior were determined in vivo

112 Effects of purified GAGs on trigeminal nerve growth cone behavior were tested using

113 Sensory trigeminal nerve growth cones innervate the cornea in a

114 suggest that GAGs may direct the movement of trigeminal nerve growth cones innervating the cornea.

115 During cornea development, chick trigeminal nerve growth cones reach the cornea margin at

116 The trigeminal nerve has been suggested as another route for

117 e and the upper end of the size range in the trigeminal nerve (i.e., > 5 microm).

118 fields of each of the three divisions of the trigeminal nerve in healthy volunteers.

119 n involving the maxillary region (V2) of the trigeminal nerve in patients with spontaneous pain and e

120 n the principal sensory nucleus (PrV) of the trigeminal nerve in the brainstem.

121 timulation of the supraorbital branch of the trigeminal nerve in the dark and in the light.

122 the emerging fibers of the motor root of the trigeminal nerve in the mouse, which we have called the

123 ffusion coefficients (ADCs) of the optic and trigeminal nerves in 2-10-week-old rats were measured wi

124 The sizes of the trigeminal nerves in 31 patients (18 men and 13 women; m

125 There were differences between somatic and trigeminal nerves in VGSC subtypes underlying action pot

126 Stimulation of trigeminal nerve induces pressor response and improves c

127 The mechanism behind the site specificity of trigeminal nerve injury is still unclear.

128 o peripheral cold hypersensitivity following trigeminal nerve injury, and Kv4.3 activators may be cli

129 investigate a potential mechanism underlying trigeminal nerve injury-induced orofacial hypersensitivi

130 development of ectopic activity at a site of trigeminal nerve injury.

131 et for the treatment of pain associated with trigeminal nerve injury.

132 Although the trigeminal nerve innervates the meninges and participate

133 lforhodamine 101, peripheral hypoglossal and trigeminal nerves involved with tongue and jaw movements

134 s an inflammatory demyelination mechanism of trigeminal nerve involvement in MS.

135 We aim to determine the prevalence of trigeminal nerve involvement in patients with MS in a co

136 Although trigeminal nerve involvement is a characteristic of mult

137 In this 7T MRI cohort, the prevalence of trigeminal nerve involvement was 15.8%.

138 The area innervated by the contralateral trigeminal nerve is represented in an 8-mm mediolateral

139 Tissue phantoms made of normal fixed mouse trigeminal nerves juxtaposed with and without gel were e

140 y to the infraorbital nerve, a branch of the trigeminal nerves, led to synaptic ultrastructural chang

141 Trigeminal nerve lesions at differing levels can result

142 marily targeted by the lingual branch of the trigeminal nerve (LV).

143 rovascular canals, that include parts of the trigeminal nerve; many branches of this complex terminat

144 lts indicate that viral dissemination in the trigeminal nerve may occur both within the axon and in t

145 Injury to the trigeminal nerves may cause maladaptive changes in synap

146 nt stress, local constriction, and injury in trigeminal nerves may contribute to the pathogenesis of

147 ry nerve-mediated and combined olfactory and trigeminal nerve-mediated odorants activate different re

148 of this study showed that the stimulation of trigeminal nerve modulates both sympathetic and parasymp

149 n restricted to the region innervated by the trigeminal nerves (n = 37); (2) pain in the trigeminal d

150 MRI of the trigeminal nerves (n = 47 patients, n = 50 symptomatic t

151 sensory cortex, in the representation of the trigeminal nerve, near cells with a tactile representati

152 o reported in the vertex, second division of trigeminal nerve, neck, nose, jaw, parietal region, ear,

153 nuclei, cranial nerve motor nuclei, sensory trigeminal nerve nuclei, inferior and superior colliculi

154 nucleus (SpM), the principle nucleus of the trigeminal nerve, nucleus laminaris and scattered cell g

155 Trigeminal nerve number was by far the largest and also

156 The present study examined the trigeminal nerve of a teleost fish, the rainbow trout (O

157 t C. pneumoniae can infect the olfactory and trigeminal nerves, olfactory bulb and brain within 72 h

158 The mean diameter of the trigeminal nerve on the symptomatic side was significant

159 infraorbital nerve (a major component of the trigeminal nerve) on protein expression was examined.

160 driven orofacial pain, acute activity of the trigeminal nerve, or TMJ tissue degeneration and/or dama

161 ion of somatotopic organization, we compared trigeminal nerve organization in turtle, chick, and mous

162 s innervated by the ophthalmic branch of the trigeminal nerve originate contralaterally from insular

163 erent regions of the brain via olfactory and trigeminal nerve pathways.

164 timulation of low threshold afferents in the trigeminal nerve produced a clear SAI (P < 0.05) when th

165 Odorants that also stimulated the trigeminal nerve produced additional cingulate, temporal

166 otine to the various epithelia served by the trigeminal nerve produces irritation and/or pain by acti

167 ipsilateral principal sensory nucleus of the trigeminal nerve (PrV) correspond to the whiskers.

168 The principal sensory nucleus of the trigeminal nerve (PrV) relays the facial sensations to t

169 target, the principal sensory nucleus of the trigeminal nerve (PSN).

170 Continuous unilateral stimulation of the trigeminal nerve reduced electrographic seizure activity

171 ges in subjects with neuropathic pain of the trigeminal nerve resulting in most cases (20 of 23) from

172 , because the local block of Na(V)1.1 in the trigeminal nerve reverses nerve injury-induced mechanica

173 developed the foramen lacerum impingement of trigeminal nerve root (FLIT) model of human trigeminal n

174 d altered white matter microstructure at the trigeminal nerve root (n = 53), including reduced fracti

175 In most cases, the trigeminal nerve root demyelination involves the proxima

176 ffusivity measures and the PET signal at the trigeminal nerve root, in addition to the brainstem func

177 Examination of trigeminal nerve roots from patients with compression of

178 We report a patient with a trigeminal nerve section who continued to have attacks.

179 timulation of the supraorbital branch of the trigeminal nerve (SO) ipsilateral to the upper eyelid wi

180 muli (HFS) to the supraorbital branch of the trigeminal nerve (SO) modified subsequent reflex blinks

181 anscranial magnetic stimulation and external trigeminal nerve stimulation (all with regulatory cleara

182 External Trigeminal Nerve Stimulation (e-TNS) is a non-pharmacolo

183 ial magnetic stimulation (low SOE), external trigeminal nerve stimulation (low SOE), and noninvasive

184 ble of increasing cerebral perfusion, making trigeminal nerve stimulation (TNS) a promising strategy

185 Trigeminal nerve stimulation also decreased systemic inf

186 s such as deep brain stimulation, vagus, and trigeminal nerve stimulation are effective only in a fra

187 Trigeminal nerve stimulation elicited strong synergistic

188 Furthermore, trigeminal nerve stimulation generated sympathetically m

189 Trigeminal nerve stimulation had been reported to activa

190 Trigeminal nerve stimulation is currently being evaluate

191 These findings suggest that trigeminal nerve stimulation is safe in awake rats and s

192 e results demonstrate that seizure-triggered trigeminal nerve stimulation is technically feasible and

193 The effects of trigeminal nerve stimulation on survival rate, autonomic

194 t volume expansion with fluid resuscitation, trigeminal nerve stimulation significantly attenuated sy

195 Trigeminal nerve stimulation significantly increased the

196 The survival rate at 60 minutes was 90% in trigeminal nerve stimulation treatment group whereas 0%

197 omly assigned to either control, vehicle, or trigeminal nerve stimulation treatment groups.

198 Trigeminal nerve stimulation was explored as a novel res

199 nks and reflex blinks evoked by supraorbital trigeminal nerve stimulation.

200 arch examining the therapeutic mechanisms of trigeminal nerve stimulation.

201 rval (ISI) between the onset of amygdala and trigeminal nerve stimulation.

202 ectopic activity in adjacent branches of the trigeminal nerve suggest that the fibre types or anatomi

203 y, the selective upregulation of Na(V)1.1 in trigeminal nerves suggests a novel therapeutic target fo

204 tion through a critical interaction with the trigeminal nerve, supporting the concept that the integr

205 cal anesthetics and electric ablation of the trigeminal nerve suppressed the pollen shell-stimulated

206 inputs, it is hypothesized that auditory and trigeminal nerve synaptic inputs onto abducens motor neu

207 use this novel in vivo assay to isolate the trigeminal nerve target-recognition molecule(s).

208 that human OSCC tumors sensitize peripheral trigeminal nerve terminals, providing a unique opportuni

209 ctional MRI response to innocuous ophthalmic trigeminal nerve territory stimulation.

210 timulation of the infraorbital branch of the trigeminal nerve that enables future research examining

211 ntia nigra, the mesencephalic nucleus of the trigeminal nerve, the cochlear nucleus and the superior

212 ral and nasal papillae are innervated by the trigeminal nerve, the gill pore papillae are innervated

213 ssociated with sensory-nerve branches of the trigeminal nerve, the orbital vasculature, and the perio

214 ity properties in the root entry zone of the trigeminal nerve, the spinal trigeminal tract, or the ve

215 terization of the infraorbital branch of the trigeminal nerve, the tactile map in the granule cell la

216 Electrical stimulation of the trigeminal nerve (TNS) plays a pivotal role in modulatin

217 ensitive fibers of the lingual branch of the trigeminal nerve to cooling from 35 degrees to 10 degree

218 f injury to the maxillary branch (V2) of the trigeminal nerve to produce constant and long-lasting pr

219 showed direct projections from auditory and trigeminal nerves to abducens motor neurons.

220 The overall response of trigeminal nerves to peripheral inflammatory stimuli inv

221 ferior alveolar nerve, another branch of the trigeminal nerve, to determine whether differences in ne

222 tating substances leads to activation of the trigeminal nerve, triggering protective reflexes that in

223 interface for the infraorbital branch of the trigeminal nerve utilizing a thin film (TF) nerve cuff c

224 n at sites within the three divisions of the trigeminal nerve (V1, V2, and V3) and also to the ipsila

225 istribution of the maxillary division of the trigeminal nerve (V2), with normal motor and sensory fun

226 Motor function of the left trigeminal nerve was preserved.

227 ke rats while the infraorbital branch of the trigeminal nerve was stimulated via a chronically implan

228 of neurovascular contact on the symptomatic trigeminal nerves was higher (80.0%) compared to the asy

229 nvestigate potential clinical effects on the trigeminal nerve, we further performed quantitative sens

230 esentation of body regions innervated by the trigeminal nerve were elucidated in monkey cerebral cort

231 otal of 165 symptomatic and 153 asymptomatic trigeminal nerves were analysed.

232 nd odorants that stimulate the olfactory and trigeminal nerves were compared by using repetitive imag

233 The sensory fibers of the hypoglossal and trigeminal nerves were found projecting to the Purkinje

234 verse and longitudinal sections of optic and trigeminal nerves were studied with electron microscopy.

235 ajor organs, along with the nasal tissue and trigeminal nerve, were harvested to assess the biodistri

236 lation included face areas innervated by the trigeminal nerve, whereas somatic fields of 89% of neuro

237 KS was localized in the path of trigeminal nerves, whereas DS and CSA/C-rich areas were

238 tional interaction between the occipital and trigeminal nerves which has been proposed in neurophysio

239 c reflex (TCR) occurs upon excitation of the trigeminal nerve with a resulting bradycardia and hypote

240 ct blockade of CGRP release by inhibition of trigeminal nerves would be similarly effective in both m