戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1  between human HaCaT keratinocytes and mouse trigeminal neurons.
2 n of sulfated GAGs canceled their effects on trigeminal neurons.
3  adult corneas, whole embryonic corneas, and trigeminal neurons.
4 dedicated population of about 50 specialized trigeminal neurons.
5 muli to cause acute or chronic excitation of trigeminal neurons.
6  as well as a subset of chemosensitive mouse trigeminal neurons.
7 umably represents activation of second-order trigeminal neurons.
8 teractions between the dental pulp cells and trigeminal neurons.
9  the presence of adenosine A(1) receptors on trigeminal neurons.
10  influence axon growth patterns of embryonic trigeminal neurons.
11  (PKC) was determined in acutely dissociated trigeminal neurons.
12 odontitis upregulates TLR4 expression in the trigeminal neurons.
13  We investigated bursting behavior in rodent trigeminal neurons.
14 ripherally derived survival factor for early trigeminal neurons.
15 essing of nociceptive information by central trigeminal neurons.
16 nsity of calcium responses in cultured mouse trigeminal neurons.
17 ve (5-HT1) receptors on postsynaptic central trigeminal neurones.
18 lts showed that EP2 and EP3 are expressed in trigeminal neurons (58% and 53% of total neurons, respec
19 and axon degeneration in spontaneously dying trigeminal neurons: although pieces of Wld(S)-expressing
20 afferents by using primary cultures of mouse trigeminal neurons and an organotypic model of cultured
21                                           In trigeminal neurons and CHO cells, the manipulation of ce
22 ed currents of different kinetics in corneal trigeminal neurons and contributes to transduction of me
23                                        Mouse trigeminal neurons and human pulp explants were pretreat
24 deleterious feedback loop in migraine at the trigeminal neurons and provide a general mechanism by wh
25                            Postnatally, more trigeminal neurons and types of mystacial pad innervatio
26 is study, we evaluated whether LPS activates trigeminal neurons, and sensitizes TRPV1 responses via T
27 s that LPS is capable of directly activating trigeminal neurons, and sensitizing TRPV1 via a TLR4-med
28 sensor GCaMP3, we show that TRPV1-expressing trigeminal neurons are activated by heat at behaviorally
29 ings are consistent with the hypothesis that trigeminal neurons are capable of detecting pathogenic b
30 visually foraging bird, the majority of duck trigeminal neurons are mechanoreceptors that express the
31                                              Trigeminal neurons are the major source of cerebrovascul
32  brain, ascending projections from medullary trigeminal neurons arrive at taste neurons in the parabr
33  in the axons and cell bodies of adult mouse trigeminal neurons, as well as in the processes and cell
34 F-dependent nodose neurons and NGF-dependent trigeminal neurons at stages during and after the period
35                  Knockdown of Piezo2 in duck trigeminal neurons attenuates mechano current with inter
36  demonstration of inhibition of second order trigeminal neurons by direct local application of 5HT1B/
37 trolling the survival and differentiation of trigeminal neurons by regulating expression of each of t
38 nin gene-related peptide (CGRP) release from trigeminal neurons by the serotonergic antimigraine drug
39                                              Trigeminal neurons convey somatosensory information from
40 YFP fluorescence in wholemounted corneas and trigeminal neuron cultures was determined.
41 mmunohistochemical analyses of human and rat trigeminal neurons demonstrated that a capsaicin-sensiti
42 in defined without NGF whereas BAX-deficient trigeminal neurones died in the absence of NGF.
43 is required for the in vivo survival of many trigeminal neurons during the early stages of target fie
44                            However, Elp1 CKO trigeminal neurons exhibit abnormal axon outgrowth and d
45                    In vitro, 22% of cultured trigeminal neurons exhibited YFP neurofluorescence.
46                                              Trigeminal neuron firing rate increases with airspeed, i
47 nal death in the trigeminal ganglion at E14, trigeminal neurons from bcl-2(-/-) embryos initially sur
48 caused PAR(2)-dependent hyperexcitability of trigeminal neurons from WT female mice.
49  of such cells in zebrafish--Rohon-Beard and trigeminal neurons--have served as models for neural dev
50 , TRPV1, and ASIC3 are often co-expressed in trigeminal neurons, implying the formation of functional
51                   Its release from adult rat trigeminal neurons in culture was shown to be markedly i
52 ophysiological recordings from ChR2-positive trigeminal neurons in intact fish revealed that these ce
53 lected from animals exposed to CSD activates trigeminal neurons in naive mice in part by CSF-borne ca
54  show that RBs are molecularly distinct from trigeminal neurons in zebrafish.
55  occurs, in part due to molecular changes in trigeminal neurons, including Robo1 downregulation, thus
56                                              Trigeminal neurons innervating gingivomucosa were identi
57                       Our data revealed that trigeminal neurons innervating the cheek exhibited compl
58  neurons, and (3) NRM neurons inhibit spinal trigeminal neurons involved in reflex blink circuits.
59 togenesis, the cell count of radial glia and trigeminal neurons is reduced in some mutants of the spa
60 GRP can also be secreted from cell bodies of trigeminal neurons located within the ganglion, the func
61 order to determine whether the mesencephalic trigeminal neurons may receive a direct hypothalamic ore
62 igeminal motoneurons (Mo5) and mesencephalic trigeminal neurons (Me5) are important constituents of t
63  Trigeminal motoneurons (Mo5), mesencephalic trigeminal neurons (Me5), and supratrigeminal (Su5) and
64 ations and burst generation in mesencephalic trigeminal neurons (Mes V).
65 ns in vivo but surprisingly fail to activate trigeminal neuron monocultures.
66 y was used to indicate whether activation of trigeminal neurons occurs in voluntarily diving rats.
67 s deleted established latency in only 10% of trigeminal neurons (P < 0.00001), and these mice were im
68 re were nearly equal proportions of premotor-trigeminal neurons (pre-mV) in the IRt and PCRt.
69      In the present study, we labeled spinal trigeminal neurons projecting to the cochlear nucleus us
70 gradely regulate transcription in developing trigeminal neurons, providing a mechanism of integrating
71  boundary (MHB) domain, the neural crest and trigeminal neurons, raising the possibility that iro1 an
72 ore, the CXCR2 antagonist SB225002 prevented trigeminal neuron sensitization to capsaicin induced by
73 timulation identified that wild-type central trigeminal neurons showed diverse responses to oral cool
74 raveling toward and contacting mesencephalic trigeminal neurons, some of which were multipolar.
75               We recently found in mice that trigeminal neurons supplying craniofacial somatosensatio
76                                        Mouse trigeminal neurons survive independently of neurotrophin
77 rring neuronal death at E18, Bcl-2-deficient trigeminal neurons survived with NGF as well as wild-typ
78 ular Ca(2+) changes from primary cultures of trigeminal neurons (TGNs).
79 2) specifically activates a subpopulation of trigeminal neurons that express TRPA1, a mustard oil- an
80 le-unit recordings were obtained from spinal trigeminal neurons that proved to received convergent in
81 cant but incomplete overlap between afferent trigeminal neurons that respond to oral thermal stimulat
82                 When DPC are cocultured with trigeminal neurons, they promote survival and a specific
83 ontrols the differential gene expressions in trigeminal neurons through both Smad4-independent and Sm
84                        We have used cultured trigeminal neurons to demonstrate that sumatriptan can d
85 e chromatin immunoprecipitation in embryonic trigeminal neurons to show that Brn3a is a direct repres
86             We have used primary cultures of trigeminal neurons under conditions simulating migraine
87                At E14 in vivo, the number of trigeminal neurons undergoing apoptosis was significantl
88 dence for NMDA receptor subunits in neonatal trigeminal neurons used in oral-motor circuitry and sugg
89  demonstrated that LPS binds to receptors in trigeminal neurons using competitive binding.
90                                     When E10 trigeminal neurons were cultured on different substrates
91                                       Spinal trigeminal neurons were distributed primarily in pars ca
92 l PC targets: PC synapses onto mesencephalic trigeminal neurons were not observed even though these c
93                            By contrast, when trigeminal neurons were seeded onto cryosections of E10
94 and elaborate neurite outgrowth pattern from trigeminal neurons, whereas skin fibroblasts do not prov
95 dy aims to morphometrically characterize rat trigeminal neurons, which express TLR4, and to investiga
96 an excitatory postsynaptic current (EPSC) in trigeminal neurones with a latency of 1.8 +/- 0.1 ms, ji