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1 e seam of natural B-lattice MTs may act as a trigger point, and potentially a regulation point, for c
2  with either global tenderness or myofascial trigger points, and 92% displayed evidence of impaired m
3                   Providing an array of heat trigger points applicable for different end-users may im
4  to visualize the GON and identify suspected trigger points associated with headache symptoms.
5 istically identified to represent different "trigger points" at which heat-health intervention measur
6 ly, the stimulation of this newly identified trigger point by a single XIP substitution resulted in h
7 tein contacts at a remote site may provide a trigger point for DNA-protein disassembly during telomer
8 immune evasion and, if improperly engaged, a trigger point for various clinical conditions.
9 d temperatures revealed large differences in trigger points for possible interventions related to hea
10 nd show that Mints may help facilitate a key trigger point in SNARE (soluble N-ethylmaleimide-sensiti
11 WE is a reliable method for detecting latent trigger points in MPS, and it can be used for evaluating
12                  Thirty volunteer women with trigger points in the upper part of the trapezius muscle
13 gly suggest that H4 monomethylation is a key triggering point in PARP-1 dependent processes in chroma
14 drodissection technique (UMHT) compared with trigger point injection of lidocaine (TPI) in the patien
15 aluronic acid injection, relaxation therapy, trigger point injection, acetaminophen (with or without
16 f other commonly used FMS therapies, such as trigger point injections, have not been adequately evalu
17 a-articular injections, regional blocks, and trigger-point injections continue to be redefined as we
18 omatic pain, including nonopioid analgesics, trigger-point injections, and physical therapy.
19                                   Myofascial trigger points (MTrPs) are localized contraction knots t
20 ized primarily by the presence of myofascial trigger points (MTrPs).
21 keletal disorder characterized by myofascial trigger points (MTrPs).
22 this paper, we proposed a covariate-adjusted triggering point process to simultaneously model the eff
23         Twenty-five subjects with myofascial trigger point(s) [MTrP(s)] on the low back participated
24 ation (RD 36% (95% CI 31 to 40)), and manual trigger point therapy (RD 32% (29 to 34)).
25 ised jaw exercise and stretching with manual trigger point therapy, and usual care (such as home exer
26 ercise and stretching with or without manual trigger point therapy, and usual care (such as home exer
27 apy, therapist-assisted mobilisation, manual trigger point therapy, supervised postural exercise, sup
28 ature metric, we observed large contrasts in trigger points (up to 22 degrees C) across multiple heal
29                    TFIIB releases after both trigger points with similar kinetics, suggesting the rat