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1 t triacylglycerol lipase 3 and human adipose triglyceride lipase.
2 equilibrium parameters is demonstrated for a triglyceride lipase.
3 ats is dependent on the action of pancreatic triglyceride lipase.
4 atglistatin, an inhibitor of adipose tissue triglyceride lipase.
5 ssion lowered the LD localization of adipose triglyceride lipase.
6 f insulin signaling and mice lacking adipose triglyceride lipase.
7 ytes by inhibiting the expression of adipose triglyceride lipase.
8 inflammation, and an upregulation of adipose triglyceride lipase.
9 acellular triacylglycerol hydrolase, adipose triglyceride lipase.
10 olysis of triglyceride by activating adipose triglyceride lipase.
11 activity and the protein content of adipose triglyceride lipase, acetyl-CoA carboxylase 2 and AMP-ac
12 inhibitor of all known iPLA2s, inhibits the triglyceride lipase activity of each of the three isofor
13 IPRP2 lid domain or beta5-loop had decreased triglyceride lipase activity similar to that of PNLIPRP2
18 erived from females in which we silenced the triglyceride lipase AgTL2 or the lipid storage droplet A
19 ctivities, including coactivation of adipose triglyceride lipase and acylation of lysophosphatidic ac
20 -containing 2 (PNPLA2) family that possesses triglyceride lipase and acylglycerol transacylase activi
21 e lipase activation without altering adipose triglyceride lipase and CGI-58 expression in adipocytes.
23 sue cultures increased expression of adipose triglyceride lipase and genes regulated by FFA, and incr
28 and lipolytic mechanism of human pancreatic triglyceride lipase and of colipase, another pancreatic
29 ability of endothelial cells to secrete the triglyceride lipase and phospholipase activities charact
30 we used protein homology modeling, measured triglyceride lipase and phospholipase activities in cell
31 D membranes, to prevent lipolysis by adipose triglyceride lipase and promote cellular LD accumulation
32 eatic protein that interacts with pancreatic triglyceride lipase and that is required for lipase acti
33 testine by the pancreatic enzyme, pancreatic triglyceride lipase, and intestinal brush border enzyme,
34 The expression of OXPAT/perilipin-5, adipose triglyceride lipase, and stearoyl-CoA desaturase protein
35 ta-oxidation via activation of the conserved triglyceride lipase ATGL-1, triggers a feedback transcri
37 this process through repressing the adipose triglyceride lipase (ATGL) activity in neutrophils in pr
38 apillary lumen, and the discovery of adipose triglyceride lipase (ATGL) and comparative gene identifi
39 levels of the key lipolytic enzymes adipose triglyceride lipase (ATGL) and hormone sensitive lipase
40 ment of two major metabolic lipases, adipose triglyceride lipase (ATGL) and hormone-sensitive lipase
41 via activation of cytosolic lipases adipose triglyceride lipase (ATGL) and hormone-sensitive lipase
42 This study examined the role that adipose triglyceride lipase (ATGL) and hormone-sensitive lipase
45 n of TXNIP is largely dependent upon adipose triglyceride lipase (ATGL) and not hormone-sensitive lip
48 work designated as the "lipolysome." Adipose triglyceride lipase (Atgl) catalyzes the initiating step
52 step in triacylglycerol hydrolysis, adipose triglyceride lipase (ATGL) has been proposed to influenc
56 aboratory and others have shown that adipose triglyceride lipase (ATGL) increases the activity of the
59 sent investigation demonstrates that adipose triglyceride lipase (Atgl) is one of the enzymes involve
62 The impaired lipolysis in global adipose triglyceride lipase (ATGL) knockout mice reduced free PA
65 at (VF) was attributable to elevated adipose triglyceride lipase (Atgl) protein expression localized
66 ILPDA) protein, which down-regulated adipose triglyceride lipase (ATGL) protein levels, in turn leadi
67 Inhibition of the lipolysis enzyme adipose triglyceride lipase (ATGL) resulted in large cytoplasmic
70 creases the colocalization of adipose tissue triglyceride lipase (Atgl) with its coactivator, Abhd5.
71 ctions as an endogenous inhibitor of adipose triglyceride lipase (ATGL), a key enzyme in intracellula
72 ipin proteins control lipolysis by adipocyte triglyceride lipase (ATGL), a key lipase in adipocytes a
73 tch gene 2 (G0S2) as an inhibitor of adipose triglyceride lipase (ATGL), a key mediator of intracellu
74 found that hepatic levels of SRA and adipose triglyceride lipase (ATGL), a major hepatic triacylglyce
75 ation and proteasomal degradation of adipose triglyceride lipase (ATGL), a rate limiting enzyme of TA
76 a lipid droplet scaffold, and adipose tissue triglyceride lipase (Atgl), a triglyceride-specific lipa
77 re, we identified that expression of adipose triglyceride lipase (ATGL), an enzyme that controls lipi
78 sferase 1 (DGAT1), overexpression of adipose triglyceride lipase (ATGL), and epistasis tests together
79 bly proline dehydrogenase (POX), and adipose triglyceride lipase (ATGL), as well as markedly reduced
80 gulate fat metabolism by attenuating adipose triglyceride lipase (ATGL), but repression of oncogene-i
81 the rate-limiting lipolytic enzyme, adipose triglyceride lipase (ATGL), has two FoxO1-binding sites,
82 gnaling pathway on the expression of adipose triglyceride lipase (ATGL), hormone-sensitive lipase (HS
83 inhibit the principal TAG hydrolase adipose triglyceride lipase (ATGL), in the regulation of cardiac
85 fectors hormone-sensitive lipase and adipose triglyceride lipase (ATGL), suggesting a link between ad
86 -modulates LD catabolism mediated by adipose triglyceride lipase (ATGL), the key enzyme for intracell
87 LDs interferes with the activity of adipose triglyceride lipase (ATGL), the key lipolytic enzyme in
88 n on triglyceride (TG) hydrolysis by adipose triglyceride lipase (ATGL), the major lipase in the live
89 e cell models, which express neither adipose triglyceride lipase (ATGL), the rate-limiting enzyme for
90 2 (G0S2) is a specific inhibitor of adipose triglyceride lipase (ATGL), the rate-limiting enzyme for
91 D size by inhibiting the activity of adipose triglyceride lipase (ATGL), the rate-limiting enzyme in
92 genic 1 (COP1) regulates turnover of Adipose Triglyceride Lipase (ATGL), the rate-limiting lipolytic
93 t hormone-sensitive lipase (HSL) and adipose triglyceride lipase (ATGL), two enzymes critical for lip
94 stimulates the enzymatic activity of adipose triglyceride lipase (ATGL), which catalyzes the hydrolys
95 kinase A (PKA) signaling to enhance adipose triglyceride lipase (ATGL)-dependent intrahepatic lipoly
96 cally, Wnt activation stimulates the Adipose Triglyceride Lipase (ATGL)-mediated lipolysis pathway, a
97 , is a highly conserved regulator of adipose triglyceride lipase (ATGL)-mediated lipolysis that plays
99 was concomitant to an increase in adipocyte triglyceride lipase (ATGL)-mediated triglyceride breakdo
104 ting disease progression, inhibiting adipose triglyceride lipase (ATGL)/patatin-like phospholipase do
105 ulated lipolysis by interacting with adipose triglyceride lipase (ATGL, also called desnutrin or PNPL
107 gree, but it did not affect adipose or liver triglyceride lipase (ATGL, known also as Pnpla2) mRNA in
108 ion of two key metabolic regulators, adipose triglyceride lipase (ATGL/bmm) and transcriptional cofac
110 we hypothesized that the absence of adipose triglyceride lipase (ATGL/PNPLA2)-the main enzyme for in
111 verexpressing desnutrin (also called adipose triglyceride lipase [ATGL]) in adipocytes (aP2-desnutrin
113 cated that CGI-58 may stimulate an epidermal triglyceride lipase beyond ATGL required for the adequat
115 reakdown in both sexes identified a role for triglyceride lipase brummer (bmm) in the regulation of s
116 t a level much lower than that of pancreatic triglyceride lipase, but close to that of carboxyl ester
117 th receptors enforced suppression of adipose triglyceride lipase, but each receptor also demonstrated
119 osis and lethal heart dysfunction in adipose triglyceride lipase deficiency, Plin5-Tg mice do not dev
120 lipolytic signals activate p53 in an adipose triglyceride lipase-dependent manner, and pharmacologic
121 -chain saturated fatty acids, but pancreatic triglyceride lipase did not appear to have a role in the
122 essions of hormone-sensitive lipase, adipose triglyceride lipase enzymes, leptin, adiponectin and glu
124 ex vivo activity of 11beta-HSD1 and adipose triglyceride lipase expression in subcutaneous fat biops
127 e molecular cloning of a novel member of the triglyceride lipase family, a 2.4-kb cDNA encoding human
128 tightly regulated by several members of the triglyceride lipase family, including endothelial lipase
129 d ablated expression of adiponectin, adipose triglyceride lipase, fatty acid synthase, perilipin 1, o
131 ely related enzymes among the members of the triglyceride lipase gene family with regard to primary s
132 lipase, a relatively recent addition to the triglyceride lipase gene family, is a major determinant
133 dothelial lipase (EL) is a new member of the triglyceride lipase gene family, which includes lipoprot
135 in the C-terminal domain of human pancreatic triglyceride lipase (hPTL) makes a major contribution in
136 nges in lipoprotein lipase (LpL) and hepatic triglyceride lipase (HTGL) similar to those found in IDD
137 at endothelial lipase is distinct from other triglyceride lipases in showing the highest activity on
138 pin RNA-mediated silencing of adipose tissue triglyceride lipase inhibited both forskolin-stimulated
139 corticosterone-infused rats with an adipose triglyceride lipase inhibitor blocked corticosterone-ind
140 r screen discovered the repeated loss of the triglyceride lipase inhibitor PNLIPRP1, suggesting enhan
141 y increasing the activity of hepatic adipose triglyceride lipase, intrahepatic lipolysis, hepatic ace
143 d coculture systems, we confirm that adipose triglyceride lipase is required for inflammation-induced
144 The amphipathic, helical lid found in other triglyceride lipases is truncated in the structure of ch
145 lar characterization of a homologous ADIPOSE TRIGLYCERIDE LIPASE-LIKE gene, previously referred to as
146 deficient mice, and concomitantly, adipocyte triglycerides lipase mRNA levels were downregulated.
147 us by placental-specific knockout of adipose triglyceride lipase not only reduced fetal beta-cell are
152 ABHD5) is best known as a coactivator of the triglyceride lipase PNPLA2, facilitating intracellular l
153 lipases, including the rate limiting adipose triglyceride lipase (Pnpla2/ATGL), is critical to the ad
154 l chemical sympathectomy and loss of adipose triglyceride lipase protect mice from GDF15-induced weig
155 Defective lipolysis in mice lacking adipose triglyceride lipase provokes severe cardiac steatosis an
156 op, the lid, controls activity of pancreatic triglyceride lipase (PTL) by moving from a position that
157 f carboxyl ester lipase (CEL) and pancreatic triglyceride lipase (PTL) in lipid nutrient absorption.
163 also named desnutrin, iPLA2zeta, and adipose triglyceride lipase), resulting in increased fatty acid
164 enetic ablation of the rate-limiting adipose triglyceride lipase specifically from adipocytes (Adipoq
166 tween Parkin and lipoprotein lipase (LPL), a triglyceride lipase that is widely expressed in the brai
167 -containing 2 (PNPLA2; also known as adipose triglyceride lipase) the rate-limiting enzyme for TAG hy
169 ncoding hormone-sensitive lipase and adipose triglyceride lipase, two enzymes involved in lipolysis a
170 intracellular translocation of ATGL (adipose triglyceride lipase) under fatty acid stimulation, inhib
171 regulates the enzymatic activity of adipose triglyceride lipase via interacting with its activator,
172 orage by decreasing transcription of adipose triglyceride lipase via the mTORC1-mediated pathway, alt
173 equence, have distinct lipolytic properties (triglyceride lipase vs. phospholipase) as well as differ