ライフサイエンスコーパス: trihalomethane

1 95th percentile, 30.0 mug/L) for brominated trihalomethanes.

2 with estimates of average lifetime level of trihalomethanes.

3 d carbon media was important for controlling trihalomethanes.

4 D risk, with the highest risk for brominated trihalomethanes.

5 black ashes had lower reactivity in forming trihalomethanes (55%-of-control) and haloacetic acids (6

6 ees C resulted in decreased reactivities for trihalomethane and chloral hydrate formation and diverge

7 ug/L; 95th percentile, 57.8 mug/L) for total trihalomethanes and 2.7 mug/L (IQR, 0.7-11.3 mug/L; 95th

8 of 4 trihalomethanes, including 3 brominated trihalomethanes and chloroform, were calculated using an

9 re-response association was observed between trihalomethanes and CKD risk, with the highest risk for

10 e second and third trimesters for both total trihalomethanes and five haloacetic acids when comparing

11 intiles of exposure to constituents of total trihalomethanes and five haloacetic acids.

12 would be comparable to that associated with trihalomethanes and haloacetic acids at levels of regula

13 beyond the potential to form lower regulated trihalomethanes and haloacetic acids compared to chlorin

14 r-source drinking water treatment plant, the trihalomethanes and haloacetic acids each increased by u

15 Birth weight, ethnicity, and exposure to trihalomethanes and haloacetic acids in drinking water d

16 Trihalomethanes and haloacetic acids monitoring is based

17 Regulated disinfection byproducts (e.g., trihalomethanes and haloacetic acids) in drinking water

18 of regulated disinfection byproducts (DBPs) trihalomethanes and haloacetic acids.

19 nations to reduce the formation of regulated trihalomethanes and haloacetic acids.

20 on nuclei achieved recoveries of 50-100% for trihalomethanes and haloacetonitriles and approximately

21 in commercial PAA solutions, resulted in no trihalomethanes and only low haloacetic acid concentrati

22 998 US pregnancy cohort study (n = 5,104) of trihalomethanes and spontaneous abortion.

23 The volatile trihalomethanes and the nonvolatile haloacetic acids (HA

24 seawater to assess its potential to produce trihalomethanes and to determine the byproducts generate

25 At the 10-L scale, recovery of certain trihalomethanes and trihalogenated haloacetic acids by X

26 of this protocol for dihaloacetonitriles and trihalomethanes and validate a novel Alternative Method

27 The water system was monitored weekly for trihalomethanes and, system-wide, it maintained geograph

28 used in caulking), disinfection byproducts (trihalomethanes), and contaminants present in source wat

29 Disinfection byproducts such as trihalomethanes are commonly found in drinking water.

30 Trihalomethanes are formed upon chlorination of natural

31 advantage over chlorine in that carcinogenic trihalomethanes are not generated.

32 Trihalomethanes are regulated disinfection byproducts as

33 DBPs, including haloacetic acids and trihalomethanes, are possible or probable human carcinog

34 ls (a) direct electrochemical reduction of a trihalomethane at a silver cathode to form halide ions i

35 HRs for CKD risk associated with brominated trihalomethanes at the highest 2 exposure categories (75

36 ternal exposure to drinking water brominated trihalomethanes (BTHM) in relation to MN frequency in ma

37 This work determined the trihalomethanes concentration, both speciated and total,

38 y of California female teachers, exposure to trihalomethane concentrations less than 80 mug/L (US cur

39 tor organic byproducts (haloacetic acids and trihalomethanes) during electrolysis dramatically exceed

40 Animal studies report trihalomethanes, especially brominated compounds, may da

41 that high levels (> or = 70 microg/liter) of trihalomethanes experienced during the second trimester

42 A high average total trihalomethane exposure (> or = 70 microg/liter) during

43 y loss was not associated with high personal trihalomethane exposure (> or =75 micro g/liter and > or

44 Research has suggested that trihalomethane exposures during pregnancy might impair f

45 acetic acids, and haloacetonitriles, whereas trihalomethane formation decreases during chlorine photo

46 regulated THM formation; however, iodinated trihalomethane formation was observed at lower pH.

47 and (O(3)/GAC)-BAF met regulatory levels for trihalomethanes, HAAs, NDMA, and bromate.

48 f the soils heated to 350 degrees C, whereas trihalomethane, haloacetic acid and chloropicrin yields

49 reased risk of pregnancy loss in relation to trihalomethane, haloacetic acid, or total organic halide

50 The class sum concentrations of trihalomethanes, haloacetic acids, and unregulated DBPs,

51 idant exposures, concentrations of regulated trihalomethanes, haloacetic acids, bromate, and chlorite

52 ribute to the formation targeted DBPs (i.e., trihalomethanes, haloacetic acids, haloacetonitriles, ch

53 limits were in the range of 0.3-10 ng/g for trihalomethanes, haloacetic acids, nitrogenous DBPs, and

54 ased chlorine reactivities and formations of trihalomethanes, haloacetonitriles, chloral hydrate, and

55 Rate constants for 37 trihalomethanes, haloacetonitriles, haloacetaldehydes, h

56 Among 26 trihalomethanes, haloacetonitriles, haloacetaldehydes, h

57 greater than those from chlorine, including trihalomethanes, haloaldehydes, haloketones, haloacetoni

58 plication of vacuum to measure six iodinated-trihalomethanes (I-THMs) in drinking water and urine.

59 Iodinated trihalomethanes (I-THMs) reached 63 mug/L in DPR and 25

60 addition revealed that the formation of iodo-trihalomethanes (I-THMs), especially iodoform, was great

61 fection byproducts (DBPs), such as iodinated trihalomethanes (I-THMs), which pose significant health

62 Raising pH to 10 increased trihalomethanes, I-THMs, and dihalogenated haloacetamide

63 fic and pregnancy average exposures to total trihalomethane in drinking water on term low birth weigh

64 e importance of controlling the formation of trihalomethanes in water throughout treatment and distri

65 byproduct (DBP) concentrations, principally trihalomethanes, in drinking water with the expectation

66 dence time-weighted mean concentrations of 4 trihalomethanes, including 3 brominated trihalomethanes

67 ios (HRs) and 95% CIs for CKD by exposure to trihalomethane levels (US maximum contaminant level of 8

68 e, it maintained geographically stable total trihalomethane levels during the study period.

69 torical information from water utilities and trihalomethane levels in recent samples.

70 P concentration and ingested amount and, for trihalomethanes only, bathing/showering and integrated e

71 reactions of laser-ablated uranium atoms and trihalomethanes or carbon tetrafluoride in excess argon.

72 examined whether exposure to the four total trihalomethanes or the five haloacetic acids (two major

73 Ps (N-DBPs) (r = 0.97) and the least with Br-trihalomethanes (r = 0.29) and Br-N-DBPs (r = 0.04).

74 s than a specialty activated carbon used for trihalomethane removal.

75 sachusetts with complete quarterly 1999-2004 trihalomethane (THM) and haloacetic acid (HAA) data.

76 s have used various measures to characterize trihalomethane (THM) exposures, but the relationship of

77 oad estimates and median flow conditions, on trihalomethane (THM) formation and associated risk were

78 Evidence for a relationship between trihalomethane (THM) or haloacetic acid (HAA) exposure a

79 Lifetime exposure to trihalomethanes (THM) has been associated with increased

80 logical studies linking drinking water total trihalomethanes (THM) with pregnancy disorders or bladde

81 Regulations typically use four trihalomethanes (THM4) and five haloacetic acids (HAA5)

82 pically are adopting regulations focusing on trihalomethanes (THM4) as an indicator of overall DBP ex

83 increases in the mass of the four regulated trihalomethanes (THM4) concentrations (due to increased

84 focused on the possible presence of up to 10 trihalomethanes (THMs) and 13 haloacetic acids (HAAs) in

85 toward formation of brominated and iodinated trihalomethanes (THMs) and brominated haloacetonitriles

86 Trihalomethanes (THMs) and haloacetic acids (HAAs) are r

87 carbonaceous disinfection byproducts (DBPs) (trihalomethanes (THMs) and haloacetic acids (HAAs)), and

88 DMA), total N-nitrosamines (TONO), regulated trihalomethanes (THMs) and haloacetic acids (HAAs), 1,1-

89 ble reductions in the formation of regulated trihalomethanes (THMs) and haloacetic acids (HAAs).

90 We derived RPFs for four trihalomethanes (THMs) and nine haloacetic acids (HAAs)

91 abolomic profile associated with exposure to trihalomethanes (THMs) and nitrate in drinking water and

92 e tap, and to evaluate if routinely measured trihalomethanes (THMs) are an appropriate proxy measure

93 Trihalomethanes (THMs) are conditionally carcinogenic co

94 Trihalomethanes (THMs) are water disinfection by-product

95 Trihalomethanes (THMs) are widespread disinfection by-pr

96 loped and evaluated for the determination of trihalomethanes (THMs) at the submicromolar concentratio

97 Citric acid formed trihalomethanes (THMs) broadly across the matrix, genera

98 gen exchange) on the yield and speciation of trihalomethanes (THMs) formed during water chlorination.

99 city evaluation was carried out based on the trihalomethanes (THMs) formed.

100 he generation of haloacetic acids (HAAs) and trihalomethanes (THMs) in simulated wash water.

101 xposure was assessed by estimating levels of trihalomethanes (THMs) in tap water during pregnancy and

102 port, and fate of chloroform and three other trihalomethanes (THMs) in untreated groundwater were rev

103 ons exist between conditionally carcinogenic trihalomethanes (THMs) levels, water temperature (r = 0.

104 ancer and total, chlorinated, and brominated trihalomethanes (THMs) through various exposure routes.

105 active halogens and their potential to form trihalomethanes (THMs) when combined with synthetic sewa

106 ulated iodinated haloacetic acids (HAAs) and trihalomethanes (THMs), haloacetaldehydes, haloketones,

107 reated wastewaters met regulatory levels for trihalomethanes (THMs), haloacetic acids (HAAs), and bro

108 hanisms for eight classes of DBPs, including trihalomethanes (THMs), haloacetic acids (HAAs), haloket

109 Among these are trihalomethanes (THMs), identified as carcinogenic compo

110 to disinfection byproducts (DBPs), including trihalomethanes (THMs), which have been associated with

111 mation of disinfection byproducts, including trihalomethanes (THMs).

112 od samples were quantified for biomarkers of trihalomethanes (THMs): chloroform (TCM), bromodichlorom

113 enated disinfection byproducts (DBPs) (e.g., trihalomethanes, THMs and haloacetic acids, HAAs) during

114 atment combinations were capable of removing trihalomethanes to some degree and trichloramine and dic

115 loyed our methodology to determine the total trihalomethane (TTHM) content in a prepared water sample

116 estimated average nitrate (NO3-N) and total trihalomethane (TTHM) levels and the number of years exc

117 ormation potentials of haloacetonitriles and trihalomethanes under free chlorination and free bromina

118 ed that MX levels were associated with total trihalomethanes, UV-absorbance and pH.

119 Precision and detection limits for selected trihalomethanes were comparable to those of EPA method 5

120 Brominated trihalomethanes were the largest contributor (52.9%) to

121 nd a-bromocinnamaldehyde) and citrate (i.e., trihalomethanes) were generated via a pathway involving

122 oacetic acids (HAAs) was higher than that of trihalomethanes where dichloroacetic acid was dominant i

123 increased CKD risk, particularly brominated trihalomethanes, which are not separately regulated in c

124 The trihalomethane yields of anilines correlated with their