ライフサイエンスコーパス: triiodothyronine

1 and T4 were suppressed by administration of triiodothyronine.

2 5'-deiodination of thyroxine to form 3,5,3'-triiodothyronine.

3 roxine (T4) to its active metabolite, 3,5,3'-triiodothyronine.

4 ferase activity by about 90% with or without triiodothyronine.

5 alamus was diminished by coadministration of triiodothyronine.

6 total and free thyroxine, and total and free triiodothyronine.

7 and reversed triiodothyronine prevailed over triiodothyronine.

8 traperitoneal injections of freshly prepared triiodothyronine (0.2 mg x kg(-1) x d(-1)).

9 cultured human epidermal keratinocytes, with triiodothyronine (100 pmol/L) or thyroxine (100 nmol/L).

10 was induced by daily injection of l-3,5, 3'-triiodothyronine (15 ug (100 g)-1) intraperitoneally dai

11 167%) and decreased plasma leptin (-45%) and triiodothyronine (-21%) concentrations.

12 6-dihydroxyindole-2-carboxylic acid, 3,3',5'-triiodothyronine, 3,3',5-triiodothyronine, gentisate, ro

13 ddition, we evaluated the in vitro effect of triiodothyronine, 9-cis-retinoic acid, and the retinoid

14 nal barrier, and (ii) both dexamethasone and triiodothyronine accelerate barrier development.

15 ent with either 10 nM dexamethasone or 10 nM triiodothyronine accelerated SC development and barrier

16 ing concentrations of leptin, thyroxine, and triiodothyronine act coordinately to favor weight regain

17 Triiodothyronine administration prevents decreases in se

18 Triiodothyronine also increased mitochondrial transcript

19 hyronine among women and with total and free triiodothyronine among men in lipid-standardized models.

20 h greater total and free thyroxine and total triiodothyronine among women and with total and free tri

21 as 50 percent, an effect similar to that of triiodothyronine and 9-cis-retinoic acid.

22 orter function is reflected in elevated free triiodothyronine and lowered free thyroxine levels in th

23 terventions described were desmopressin use, triiodothyronine and methylprednisolone replacement, flu

24 for thyroid dysfunction, as serum thyroxine/triiodothyronine and somatic growth were normal.

25 Thyroid hormone triiodothyronine and synthetic thyroid hormone receptor

26 As expected, the serum triiodothyronine and T(4) concentrations decreased in wi

27 However, triiodothyronine and T(4) levels in fasted Car(-/-) mice

28 ormation of (131)I-labeled levothyroxine and triiodothyronine and thereby reduce the protein-bound (1

29 treatment significantly increased uptake of triiodothyronine and thyroxine (4.1- and 4.3-fold, respe

30 Dysregulation of thyroid hormones triiodothyronine and thyroxine (T3/T4) can impact metabo

31 icantly higher circulating concentrations of triiodothyronine and thyroxine at the end of the VLED th

32 could be attributed to decreased circulating triiodothyronine and thyroxine concentrations secondary

33 ssential constituent of the thyroid hormones triiodothyronine and thyroxine, which are required for t

34 differentiation method supplemented with T3 (triiodothyronine) and/or Dex (dexamethasone) during days

35 verts thyroxine to the active hormone 3,5,3'-triiodothyronine, and in the rat is expressed in the bra

36 analysis, changes in skeletal muscle, plasma triiodothyronine, and kidney masses explained 34.9%, 5.3

37 crease in serum levels of thyroxine, reverse triiodothyronine, and thyroid-stimulating hormone and a

38 thyroxine (fT(4)), total T(4) (TT(4)), total triiodothyronine, and thyroid-stimulating hormone concen

39 els of total thyroxine, free thyroxine, free triiodothyronine, and thyroid-stimulating hormone were m

40 '-diiodothyronine, triiodothyronine, reverse triiodothyronine, and thyroxine.

41 cal levels of active thyroid hormone (3,3',5-triiodothyronine) are controlled by the action of activa

42 ropin and the thyroid hormones thyroxine and triiodothyronine) are sometimes used as indicators of io

43 those for the thyroid hormones thyroxine and triiodothyronine, are among the clinical procedures for

44 The total number of triiodothyronine binding sites per nucleus was similar i

45 We observed that the addition of triiodothyronine-bound recombinant TRbeta or a ligand bi

46 D3), which inactivates thyroxine and 3,3', 5-triiodothyronine by 5-deiodination.

47 g in a rapid release of stored thyroxine and triiodothyronine causing an initial thyrotoxic phase, of

48 id produced a more rapid rise in total serum triiodothyronine concentration and a higher total peak s

49 Triiodothyronine concentration in plasma decreases durin

50 concentration and a higher total peak serum triiodothyronine concentration than the other products,

51 GH significantly decreased serum total triiodothyronine concentrations and IGF-I significantly

52 g TRbeta2 to recruit coactivator at limiting triiodothyronine concentrations.

53 the extract of the coactivator function in a triiodothyronine-dependent manner and markedly impaired

54 second included four studies and showed that triiodothyronine did not add hemodynamic benefits versus

55 Thyroxine and reverse triiodothyronine (EC(50) levels approximately 1 nm) were

56 In vitro exposure of immature islets to triiodothyronine enhanced the expression of Mafa, the se

57 -stimulating hormone, total thyroxine, total triiodothyronine, free thyroxine, free triiodothyronine,

58 eptin significantly increased levels of free triiodothyronine, free thyroxine, insulin-like growth fa

59 ulating hormone (TSH) levels and normal free triiodothyronine/free thyroxine (fT(3)/fT(4)) levels.

60 l for the intracellular production of 3,5,3'-triiodothyronine from thyroxine.

61 thyroxine (FT(4)), between BDE-100 and free triiodothyronine (FT(3)), and between anti-Dechlorane Pl

62 At least 11 biomarkers, including free-triiodothyronine (fT3) and free-thyroxine (fT4), showed

63 , thyroid stimulating hormone (TSH) and Free-triiodothyronine (fT3) and Total cholesterol (TChol) con

64 Significant effects on plasma free triiodothyronine (FT3) concentrations occurred with expo

65 DIO1/DIO2), corresponding to low-normal free triiodothyronine (FT3) levels, was causally associated w

66 RL), thyroid stimulating hormone (TSH), free triiodothyronine (fT3), and free thyroxin (fT4) were mea

67 luding soy as an intervention and where free triiodothyronine (fT3), free thyroxine (fT4) and thyroid

68 Cord blood levels of free triiodothyronine (FT3), FT4, TSH, and TPOAb were measure

69 nergy expenditure, percentage body fat, free triiodothyronine (FT3), urinary norepinephrine, and plas

70 SH), free and total thyroxine (fT4, TT4) and triiodothyronine (fT3, TT3), and autoimmunity [thyroid p

71 xylic acid, 3,3',5'-triiodothyronine, 3,3',5-triiodothyronine, gentisate, rosmarinate, and 3-nitrotyr

72 A single large dose of L-triiodothyronine given to hypothyroid rats caused a 4.7-

73 nt elevations in both tetraiodothyronine and triiodothyronine hormones.

74 nuclear in both the absence and presence of triiodothyronine; however, triiodothyronine induced a nu

75 hydrocortisone + placebo group 167 +/- 286; triiodothyronine + hydrocortisone group 466 +/- 495; p =

76 placebo, 3) hydrocortisone + placebo, or 4) triiodothyronine + hydrocortisone.

77 to any changes in plasma cortisol, insulin, triiodothyronine, IGF-1 or glucose.

78 1 nm) were >100-fold more potent than 3,5,3'-triiodothyronine in initiating vesicle binding to actin

79 id inactivation of circulating thyroxine and triiodothyronine in patients with hemangiomas and its bl

80 le for D2 in the generation of plasma 3,5,3'-triiodothyronine in this species.

81 weight loss, whereas serum concentrations of triiodothyronine increased significantly (by approximate

82 Serum concentrations of reverse triiodothyronine increased significantly during and afte

83 Instead, triiodothyronine increased sirtuin-1, fibrillin-1, proli

84 e and presence of triiodothyronine; however, triiodothyronine induced a nuclear reorganization of TRb

85 nd fasting blood biochemistry indexes (total triiodothyronine, insulin, leptin, and ghrelin) as indep

86 by this gene appears to be the transport of triiodothyronine into neurons.

87 thyroxin to the biologically active 3,5, 3'-triiodothyronine, is highly concentrated in a group of s

88 is needed to allow for steady delivery of L-triiodothyronine" (it currently reads "... for steady de

89 5-Triiodothyronine (L-T3) given to intact mice produced a

90 es for muscimol in the presence of 3,3', 5-L-triiodothyronine (L-T3) indicated a noncompetitive inhib

91 erefore examined the effects of TH (L-3,3',5-triiodothyronine, L-T3) given to TH-deprived and to inta

92 Steroids alone and steroids plus triiodothyronine/l-thyroxine also significantly reduced

93 red and, when the latter is normal, the free triiodothyronine level should be obtained.

94 35 pp; -1.91 to -0.80 pp), total or free T3 (triiodothyronine) level testing for hypothyroidism (DiD,

95 here were also no differences in circulating triiodothyronine levels between groups at the end of the

96 Triiodothyronine levels decrease in infants and children

97 Plasma triiodothyronine levels increased to supraphysiological

98 Serum triiodothyronine levels were elevated, but only in anima

99 re low TSH, elevated free-thyroxine and free-triiodothyronine levels, and TSH-receptor autoantibodies

100 -stimulating hormone and a decrease in serum triiodothyronine levels.

101 34 muIU/mL; normal range, 0.35-5.5 muIU/mL), triiodothyronine (<2.5 ng/dL [0.0385 pmol/L]; normal ran

102 4.34 uIU/mL; normal range, 0.35-5.5 uIU/mL), triiodothyronine (<2.5 ng/dL [0.0385 pmol/L]; normal ran

103 e prohormone thyroxine to the active hormone triiodothyronine, modifying the expression of approximat

104 olus and infusions of vasopressin and either triiodothyronine or L-thyroxine.

105 olus and infusions of vasopressin and either triiodothyronine or l-thyroxine.

106 rum concentrations of total thyroxine, total triiodothyronine, or free thyroxine index.

107 there is a perception that adding synthetic triiodothyronine, or liothyronine, to levothyroxine impr

108 total triiodothyronine, free thyroxine, free triiodothyronine, parathyroid hormone, prolactin, N-term

109 mug/kg; placebo + placebo group 208 +/- 392; triiodothyronine + placebo group 501 +/- 370; hydrocorti

110 hour IV infusion of 1) placebo + placebo, 2) triiodothyronine + placebo, 3) hydrocortisone + placebo,

111 Thyroid hormone (T3 or 3,5,3'-triiodothyronine) plays a causative role during amphibia

112 yroxine (47.9-193.6 mug L(-1)), and reversed triiodothyronine prevailed over triiodothyronine.

113 ue reveals a novel mechanism for controlling triiodothyronine production that provides the first exam

114 Cluster analysis of triiodothyronine-regulated gene expression among 19 diff

115 igation and puncture, with or without 3,5,3'-triiodothyronine replacement (3 ng/hr), or sham surgery.

116 hyroid-stimulating hormone (TSH), thyroxine, triiodothyronine resin uptake, and free thyroxine index

117 In previous work, we characterized a 3,5,3'-triiodothyronine response element (T3RE) in acetyl-CoA c

118 siently transfected with plasmids containing triiodothyronine response elements and a minimal promote

119 ganded TR.RXR recruits both complexes to the triiodothyronine-responsive region of growth hormone gen

120 isoform is mainly limited to the pituitary, triiodothyronine-responsive TRH neurons, the developing

121 nsolidation; while activation of TR with T3 (triiodothyronine) resulted in increased memory formation

122 circulating concentrations of thyroxine and triiodothyronine returned to pre-weight-loss levels.

123 ence or absence of cycloheximide or 3,3', 5'-triiodothyronine (reverse T3, rT3) in rat pituitary tumo

124 values of 75-100 microM included 3,3', 5'-l-triiodothyronine (reverse T3; r-T3), 3,3'-diiodo-L-thyro

125 ta: L-triiodothyronine, TH, TH receptor, and triiodothyronine (reverse) were inferred as upstream reg

126 id hormones, including 3,3'-diiodothyronine, triiodothyronine, reverse triiodothyronine, and thyroxin

127 was derived from the T4 metabolite, reverse triiodothyronine (revT3), while functional studies provi

128 to 3,5,3'-triiodothyronine (T3) and 3,3',5'-triiodothyronine (rT3) by the outer- and inner-ring deio

129 d hormones triiodothyronine (T3) and reverse triiodothyronine (rT3) in their deprotonated form was st

130 hormones, triiodothyronine (tT3) and reverse triiodothyronine (rT3), were negatively (tT3) and positi

131 increase in rat HCN2 mRNA is likely due to L-triiodothyronine stimulation of HCN2 gene transcription.

132 ee thyroxine, but not the minimal isoform of triiodothyronine, suggesting that chronic anti-VEGF trea

133 In vivo neonatal triiodothyronine supplementation and TH inhibition, resp

134 3,5,3'-triiodothyronine supplementation appeared to increase la

135 All were conducted with triiodothyronine (T(3)) and a tricyclic antidepressant.

136 f acute illness, namely a decrease in 3,5,3'-triiodothyronine (T(3)) and thyroid-stimulating hormone

137 compared the effectiveness of lithium versus triiodothyronine (T(3)) augmentation as a third-step tre

138 Synthesis of triiodothyronine (T(3)) in the hypothalamus induces mark

139 of pRb and compared the effects of E(2) and triiodothyronine (T(3)) on these phenomena.

140 The thyroid hormones thyroxine (T(4)) and triiodothyronine (T(3)) play key roles in regulating dev

141 n between SREBP-1c, nuclear factor Y, 3,5,3'-triiodothyronine (T(3)) receptors, and co-activators usi

142 on the one hand, and by thyroxine (T(4)) and triiodothyronine (T(3)) replacement on the other.

143 In this study, a high triiodothyronine (T(3)) state was induced in mice by sup

144 Triiodothyronine (T(3)) supplementation may be a useful

145 expression of target genes in the absence of triiodothyronine (T(3)) through the recruitment of the c

146 Administration of 3,5,3'-triiodothyronine (T(3)) to rats resulted in an increase

147 ealthy adults before and after three days of triiodothyronine (T(3)) treatment.

148 T(4) is enzymatically deiodinated to 3,5,3'-triiodothyronine (T(3)), a high-affinity ligand for the

149 g hormone (TSH), free thyroxine (T(4)), free triiodothyronine (T(3)), and leptin concentrations were

150 composition (by hydrostatic weighing), serum triiodothyronine (T(3)), and reverse T(3) (rT(3)).

151 nown, highly potent physiological TR ligand, triiodothyronine (T(3)), and with a synthetic TR antagon

152 effects of administering a primary mitogen, triiodothyronine (T(3)), at the time of 70% partial hepa

153 Triiodothyronine (T(3)), the bioactive form of thyroid h

154 T(4)) to the active form of thyroid hormone, triiodothyronine (T(3)).

155 ear receptors (TRs alpha and beta) that bind triiodothyronine (T(3), 3,5,3'-triiodo-l-thyronine) with

156 n whom T(4) replacement was stopped (without triiodothyronine [T(3)] replacement) in preparation for

157 we evaluated the ability of thyroid hormone (triiodothyronine [T(3)]), a known hepatic mitogen, to st

158 Thyroid hormone (3,5,3'-triiodothyronine; T(3)) is essential for normal developm

159 The effects of triiodothyronine (T3 ), insulin and leptin on beta cell

160 pe 2 deiodinase (D2), which generates 3,5,3'-triiodothyronine (T3 ), the active thyroid hormone.

161 (10 mU ml(-1)), thyroxine (T4) (100 nM), and triiodothyronine (T3) (100 pM) alter intrafollicular mit

162 Ralpha and TRbeta plays an important role in triiodothyronine (T3) action and TR isoform specificity.

163 Triiodothyronine (T3) activates rat liver S14 gene trans

164 e the conversion of thyroxine (T4) to 3,5,3'-triiodothyronine (T3) and 3,3',5'-triiodothyronine (rT3)

165 erapy results in relatively low serum 3,5,3'-triiodothyronine (T3) and high serum thyroxine/T3 (T4/T3

166 ents with a poor outcome had lower levels of triiodothyronine (T3) and higher thyroxine (T4).

167 enous conversion of thyroxine (T4) to 3,5,3'-triiodothyronine (T3) and may not be optimal in some cas

168 se behavior of the isomeric thyroid hormones triiodothyronine (T3) and reverse triiodothyronine (rT3)

169 and cardiac abnormalities were alleviated by triiodothyronine (T3) and T4 administration to pups, by

170 ATP1C1 bound with the active thyroid hormone triiodothyronine (T3) and the prohormone thyroxine (T4)

171 Serum triiodothyronine (T3) and thyroxine (T4) concentrations

172 on the effects of Hg on the thyroid hormones triiodothyronine (T3) and thyroxine (T4) in aquatic wild

173 congeners, total and free thyroid hormones (triiodothyronine (T3) and thyroxine (T4)), thyroid-stimu

174 levels were associated with lower cord total triiodothyronine (T3) and total T4 levels, and maternal

175 imulating hormone) and high concentration of triiodothyronine (T3) and/or free thyroxine (FT4), affec

176 evels of thyroid hormones thyroxine (T4) and triiodothyronine (T3) averaged 46.9 and 64%, respectivel

177 ecruitment in vitro, while preserving normal triiodothyronine (T3) binding and CoR interactions.

178 When triiodothyronine (T3) binds the thyroid hormone receptor

179 upon the prereceptor regulation of supply of triiodothyronine (T3) by deiodinase enzymes.

180 Triiodothyronine (T3) causes a 30-fold increase in trans

181 ly in life, D3KO mice exhibit delayed 3,5,3'-triiodothyronine (T3) clearance, a markedly elevated ser

182 e report that despite a normal plasma 3,5,3'-triiodothyronine (T3) concentration, cold-exposed mice w

183 d during pregnancy decreased thyroid hormone triiodothyronine (T3) concentrations and type 1 iodothyr

184 ate of adrenergic overactivity prevails when triiodothyronine (T3) concentrations become excessive, t

185 changes in gene expression and plasma 3,3',5-triiodothyronine (T3) concentrations in tadpoles treated

186 to all three chemicals decreased whole body triiodothyronine (T3) concentrations, either through inh

187 nd ligand-binding protein to decrease T4 and triiodothyronine (T3) cross-reactivity with the antibody

188 eir wild-type counterparts were treated with triiodothyronine (T3) for 14 days and compared to untrea

189 e of a critical role for the thyroid hormone triiodothyronine (T3) in controlling the maturation and

190 thyroxine (T4) to the active hormone 3,5,3'-triiodothyronine (T3) in peripheral tissues.

191 sor thyroxine (T4) to the active form 3,5,3'-triiodothyronine (T3) in the blood is many times higher

192 Treatment of endothelial cells with L-3,5,3'-triiodothyronine (T3) increased the association of TRalp

193 ed to determine if fetal plasma cortisol and triiodothyronine (T3) influenced the mRNA abundance of U

194 We have discovered that 3,3',5-triiodothyronine (T3) inhibits binding of a PIP-box sequ

195 Thyroidal production of triiodothyronine (T3) is absent in athyreotic patients,

196 3,3',5-Triiodothyronine (T3) is an important diagnostic marker

197 Although 3,5,3'-triiodothyronine (T3) is considered to be the primary bi

198 Plasma cortisol and total triiodothyronine (T3) levels increased towards term, wer

199 ween eGFR, BMI, minimal heart rate, and free triiodothyronine (T3) levels were determined using logis

200 by growth in serum-free medium with varying triiodothyronine (T3) levels.

201 The thyroid hormone l-3,3',5-triiodothyronine (T3) plays an important role during cer

202 atal liver accelerates local thyroid hormone triiodothyronine (T3) production and expression of T3-re

203 Triiodothyronine (T3) regulates key metabolic processes

204 a (ACCalpha) promoter 2 that mediated 3,5,3'-triiodothyronine (T3) regulation of ACCalpha transcripti

205 on of the human N-Tera-2 (NT2) cell line, in triiodothyronine (T3) replete and T3-depleted media.

206 We tested the primary hypothesis that triiodothyronine (T3) repletion is safe in this populati

207 sal silencing of genes that contain positive triiodothyronine (T3) response elements.

208 e expression of target genes upon binding to triiodothyronine (T3) response elements.

209 roteins decreased by 50%, and treatment with triiodothyronine (T3) restored ubiquitination to control

210 Addition of the thyroid hormone 3,5,3'-L-triiodothyronine (T3) resulted in reversal of this repre

211 Triiodothyronine (T3) stimulates a 7-fold increase in tr

212 Triiodothyronine (T3) stimulates a robust increase (>40-

213 yroxine (T4) and supplies most of the 3,5,3'-triiodothyronine (T3) that is essential for brain develo

214 roid hormone in the circulation, into 3,5,3'-triiodothyronine (T3) the major ligand for TRs.

215 nase thought to provide intracellular 3,5,3' triiodothyronine (T3) to a restricted group of tissues.

216 efined sleep of acute restoration of l-3,3'5-triiodothyronine (T3) to a sleep-regulatory brain region

217 rk has shown that acute microinjections of l-triiodothyronine (T3) to the preoptic region significant

218 er cone loss) were used to determine whether triiodothyronine (T3) treatment (stimulating TH signalin

219 me (NTIS), characterized by low serum 3,5,3'-triiodothyronine (T3) with normal l-thyroxine (T4) level

220 this conclusion, inhibiting the synthesis of triiodothyronine (T3) with propylthiouracil rescued pres

221 range and levels of free thyroxine (FT4) and triiodothyronine (T3) within the reference range are com

222 the concentration of active cardiac TH (i.e. triiodothyronine (T3)), and type 1 iodothyronine deiodin

223 Mean TSH, total thyroxine (T4), total triiodothyronine (T3), and free T4 levels were higher (

224 hyroid histology, plasma thyroxine (T4), and triiodothyronine (T3), and hepatic outer ring deiodinati

225 Thyrotropin (TSH), total triiodothyronine (T3), and thyroxine (T4) were measured

226 to the metabolically active molecule 3,5,3'-triiodothyronine (T3), but its global inactivation unexp

227 hed when the normal prepartum rise in plasma triiodothyronine (T3), but not cortisol, was prevented b

228 BDEs were not strongly associated with total triiodothyronine (T3), free T4, or thyroid-stimulating h

229 We measured serum concentrations of triiodothyronine (T3), free thyroxine (FT4), thyroid per

230 condition, antibody response to vaccination, triiodothyronine (T3), hepatic biotransformation (7-etho

231 nstrate a negative role of one such hormone, triiodothyronine (T3), in triggering the differentiation

232 Addition of the ligand, 3,5,3'-triiodothyronine (T3), induces transcriptional repressio

233 s as in other vertebrates: thyroxin (T4) and triiodothyronine (T3), making the zebrafish a very usefu

234 In mouse myocyte cultures, triiodothyronine (T3), norepinephrine (NE) through a bet

235 Thyroid hormones (THs), thyroxine (T4), and triiodothyronine (T3), regulate growth, metabolism, and

236 issues convert the prohormone thyroxine into triiodothyronine (T3), the active ligand for the thyroid

237 (D2) that activates thyroxine (T4) to 3,3',5-triiodothyronine (T3), the disruption of which (Dio2(-/-

238 Here, we show that the thyroid hormone triiodothyronine (T3), through binding to its nuclear re

239 serum thyrotropin-stimulating hormone (TSH), triiodothyronine (T3), thyroxine (T4), and free T4 conce

240 The thyroid hormones triiodothyronine (T3), thyroxine (T4), and thyrotropin (

241 anti-thyroid peroxidase (anti-TPO) antibody, triiodothyronine (T3), thyroxine (T4), thyroid- stimulat

242 ulin), which contain both thyroxine (T4) and triiodothyronine (T3), were the first pharmacologic trea

243 The thyroid hormone triiodothyronine (T3), which antagonizes TRH action in t

244 DIO2 converts thyroxine (T4) to triiodothyronine (T3), which binds to the TH receptor, w

245 (as studied in several laboratories), and by triiodothyronine (T3), which has not been previously exa

246 hich encodes deiodinase 3 (D3) to catabolize triiodothyronine (T3), while a maternally imprinted long

247 ter-transcription factor (COUP-TF) represses triiodothyronine (T3)-dependent transcriptional activati

248 ted dominant negative activity as it blocked triiodothyronine (T3)-mediated transcriptional activity

249 assays showed that TRalpha failed to support triiodothyronine (T3)-stimulated transcription on "inact

250 primarily tetraiodothyronine (T4), and some triiodothyronine (T3).

251 o hepatocytes is stimulated about 30-fold by triiodothyronine (T3).

252 iological effects of thyroid hormone, 3,5,3'-triiodothyronine (T3).

253 rget genes whose expression are decreased by triiodothyronine (T3).

254 ession only in the presence of its ligand, L-triiodothyronine (T3).

255 ts elicited by the thyroid hormone, 3,5,3'-L-triiodothyronine (T3).

256 mmunosensor was constructed to detect 3,3',5-triiodothyronine (T3).

257 rmone by converting thyroxine (T4) to 3,5,3'-triiodothyronine (T3).

258 oxine (T4) to the physiologically active TH, triiodothyronine (T3).

259 Thyroid hormone [triiodothyronine (T3)] positively regulates NADPH cytoch

260 adrenergic receptor blocker, when given with triiodothyronine (T3, a thyroid hormone) accentuates the

261 ial organ donor can include thyroid hormone (triiodothyronine [T3] or levothyroxine [T4]), corticoste

262 yme that inactivates thyroid hormone (3,5,3' triiodothyronine [T3]), is frequently expressed by tumor

263 ter is responsive to thyroid hormone (3,3',5-triiodothyronine, T3) and efficiently repressed by unlig

264 dels with a hyperthyroid-like phenotype, TH (triiodothyronine, T3), in culture and exercise in vivo.

265 localized regulation of levels of active TH (triiodothyronine, T3), through spatiotemporal expression

266 homeostasis in response to thyroid hormone (triiodothyronine, T3).

267 rates with PGC-1alpha to enhance response to triiodothyronine, T3.

268 ial sequence of the thyroid hormone (3,5, 3'-triiodothyronine; T3) receptor.

269 sion of more than 43% of positive T3 (3,3',5-triiodothyronine) targets in hypothyroid mice.

270 and functions were inferred from the data: L-triiodothyronine, TH, TH receptor, and triiodothyronine

271 ned more non-thyroglobulin-associated T4 and triiodothyronine than did those in wild-type mice, indep

272 ne, which undergoes peripheral conversion to triiodothyronine, the active form of thyroid hormone.

273 entration of the active thyroid hormone (TH) triiodothyronine through regioselective deiodination.

274 wing ligands: deoxycholate, 5-leuenkephalin, triiodothyronine, thyronine, dabsyl-L-valine, and N-benz

275 Circulating triiodothyronine, thyroxine, and glucagon concentrations

276 genous and xenobiotic compounds, including L-triiodothyronine, thyroxine, estrone, p-nitrophenol, 2-n

277 on is achieved through the binding of 3,5,3'-triiodothyronine to its nuclear receptor, which results

278 paper, we demonstrate that binding of TH T3 (triiodothyronine) to THRB induces senescence and deoxyri

279 0.0000), the serum level of free thyroxine, triiodothyronine, total hemoglobin, uric acid, low- and

280 enuated by cotransfection of TR alpha 1 plus triiodothyronine treatment.

281 s between BDE-47 and maternal total and free triiodothyronine (TT3 and FT3).

282 ally, we observed that two thyroid hormones, triiodothyronine (tT3) and reverse triiodothyronine (rT3

283 A), thyroid-stimulating hormone (TSH), total triiodothyronine (TT3), and free thyroxine (FT4), and we

284 circulating total thyroxine (TT4) and 3,5,3'-triiodothyronine (TT3), respectively, while TT4 and TT3

285 free thyroxine [TT4 and FT4], total and free triiodothyronine [TT3 and FT3], thyroid-stimulating horm

286 hy men treated for 14 days with 75 microg of triiodothyronine, using 24,000 cDNA element microarrays.

287 Similarly, mean triiodothyronine values ranged from 156.18 to 195.13 ng/

288 ty acid synthase gene by the thyroid hormone triiodothyronine, various constructs of the human fatty

289 accelerating the conversion of thyroxine to triiodothyronine via type 2 deiodinase in mouse skeletal

290 No change in levels of serum thyrotropin or triiodothyronine was detected, although the thyroxine le

291 rast, the decline of TSH by treatment with L-triiodothyronine was severely blunted in SRC-1(-/-) mice

292 Triiodothyronine was significantly higher in the ISP90 g

293 However, removal of cortisol and triiodothyronine was similar to removal of cytokines.

294 ine excretion and in serum concentrations of triiodothyronine were significantly correlated with perc

295 r binding activities, putative receptors for triiodothyronine, were detected after incubation of horm

296 and propionic acid analogs of thyroxine and triiodothyronine, which are inseparable by currently ava

297 id, the prohormone thyroxine is converted to triiodothyronine, which is essential in brain developmen

298 lysates activate thyroxine (T(4)) to 3,5,3'-triiodothyronine with typical characteristics of D2 such

299 ine the safety and efficacy of administering triiodothyronine, with and without hydrocortisone, in a

300 A 24-hour infusion of triiodothyronine, with or without hydrocortisone, in an