ライフサイエンスコーパス: trinitrobenzene

1 l chemical inputs, namely chloride anion and trinitrobenzene.

2 with related nitroaromatic compounds (1,3,5 trinitrobenzene, 1,3 dinitrobenzene, and 2,4 dinitrotolu

3 ,4-dinitrotoluene, 1,3-dinitrobenzene, 1,3,5-trinitrobenzene, 2-amino-4,6-dinitrotoluene, 4-amino-2,6

4 says: TNT (4.3 +/- 2.6) x 10(8) M(-1); 1,3,5-trinitrobenzene (5.1 +/- 3.3) x 10(7) M(-1); picric acid

5 nd detection were selective for TNT and TNB (trinitrobenzene) but not RDX (hexahydro-1,3,5-trinitro-1

6 ene, 2,4-dinitrotoluene, 2,6-dinitrotoluene, trinitrobenzene, dinitrobenzene, or 1,3,5-trinitro-1,3,5

7 The substrate analog trinitrobenzene does not induce a substrate-like type I

8 ition behavior of TATB (1,3,5-triamino-2,4,6-trinitrobenzene) is a major focus in energetic materials

9 ncentrations as low as parts per billion for trinitrobenzene, methylphosphonic acid, cysteine, and gl

10 ective in mucosal inflammation as modeled by trinitrobenzene sulfonate (TNBS) colitis.

11 s model, enteral administration of immunogen trinitrobenzene sulfonate (TNBS) in 10-d-old mouse pups

12 ced in mice by intra-colonic instillation of Trinitrobenzene Sulfonate (TNBS).

13 administration of dextran sodium sulfate or trinitrobenzene sulfonate than mice with lower mucosal l

14 tran sodium sulfate or rectal application of trinitrobenzene sulfonate.

15 Trinitrobenzene sulfonic acid (TNBS) administration was

16 3p were tested in NCM460-NK-1R cells and the trinitrobenzene sulfonic acid (TNBS) and dextran sodium

17 s induced by dextran sulfate sodium (DSS) or trinitrobenzene sulfonic acid (TNBS) as well as nonstero

18 characterized by (i) loss of available 2,4,6-trinitrobenzene sulfonic acid (TNBS) binding to primary

19 itis and partially resistant to induction of trinitrobenzene sulfonic acid (TNBS) colitis and (2) mic

20 After induction of trinitrobenzene sulfonic acid (TNBS) colitis in SJL/J mi

21 The dextran sodium sulfate (DSS) and trinitrobenzene sulfonic acid (TNBS) colitis models were

22 necrosis factor-alpha (TNFalpha)] in murine trinitrobenzene sulfonic acid (TNBS) colitis, most likel

23 Chronic trinitrobenzene sulfonic acid (TNBS) colitis-associated

24 Mice administered intrarectal 2,4,6-trinitrobenzene sulfonic acid (TNBS) develop inflammatio

25 Colitis was induced via a 2.5% trinitrobenzene sulfonic acid (TNBS) enema.

26 ntal chronic colitis where administration of trinitrobenzene sulfonic acid (TNBS) in 50% ethanol indu

27 150 g-250 g) received 20 mg/rat intracolonic trinitrobenzene sulfonic acid (TNBS) in 50% ethanol or s

28 Colitis, induced by trinitrobenzene sulfonic acid (TNBS) in guinea pig, lead

29 y role in the T-cell helper 1 (Th1)-mediated trinitrobenzene sulfonic acid (TNBS) model of colitis.

30 age larvae, were given intrarectal saline or trinitrobenzene sulfonic acid (TNBS) on day 10 postinfec

31 n promoters; colitis was induced using 2,4,6-trinitrobenzene sulfonic acid (TNBS) or dextran sodium s

32 re compared to that found from the classical trinitrobenzene sulfonic acid (TNBS) procedure.

33 tes received dextran sulfate sodium (DSS) or trinitrobenzene sulfonic acid (TNBS) to induce intestina

34 In contrast, oral administration of trinitrobenzene sulfonic acid (TNBS) to Peyer's patch-nu

35 Foxo4-null mice were subjected to trinitrobenzene sulfonic acid (TNBS) treatment.

36 varia of 6-7-week-old mice administered with trinitrobenzene sulfonic acid (TNBS) with or without neu

37 Dextran sodium sulfate (DSS), 2,4,6-trinitrobenzene sulfonic acid (TNBS), and Salmonella typ

38 6 weeks posttransplantation by injection of trinitrobenzene sulfonic acid (TNBS), and tissues were a

39 f the Th1-mediated inflammation occurring in trinitrobenzene sulfonic acid (TNBS)-colitis.

40 e models of intestinal inflammation, we used trinitrobenzene sulfonic acid (TNBS)-hapten to induce in

41 of treating BALB/c mice with oxazolone- and trinitrobenzene sulfonic acid (TNBS)-induced colitides b

42 el disease and whether IL-16 participates in trinitrobenzene sulfonic acid (TNBS)-induced colitis in

43 Trinitrobenzene sulfonic acid (TNBS)-induced colitis in

44 Trinitrobenzene sulfonic acid (TNBS)-induced colitis is

45 ic regions associated with susceptibility to trinitrobenzene sulfonic acid (TNBS)-induced colitis wer

46 known about the effect of baicalein on 2,4,6-trinitrobenzene sulfonic acid (TNBS)-induced colitis, wh

47 FS1 in healthy and health-compromised, 2,4,6-trinitrobenzene sulfonic acid (TNBS)-treated mice.

48 tablished 6 days following administration of trinitrobenzene sulfonic acid (TNBS).

49 s induced by the haptenating reagent, 2,4, 6-trinitrobenzene sulfonic acid (TNBS).

50 inistration of the haptenizing reagent 2,4,6-trinitrobenzene sulfonic acid (TNBS).

51 nduced by intracolonic instillation of 2,4,6-trinitrobenzene sulfonic acid (TNBS).

52 r dextran sulfate sodium salt (DSS) or 2,4,6-trinitrobenzene sulfonic acid (TNBS).

53 was induced using dextran sodium sulfate or trinitrobenzene sulfonic acid (TNBS).

54 uced by intracolonic administration of 2,4,6 trinitrobenzene sulfonic acid (TNBS).

55 shed by weekly intrarectal administration of trinitrobenzene sulfonic acid (TNBS).

56 ter induction of ileitis by means of 2, 4, 6-trinitrobenzene sulfonic acid (TNBS)/ethanol installatio

57 following the intrarectal administration of trinitrobenzene sulfonic acid (TNBS-colitis), as well as

58 -mediated disease, the hapten reagent (2,4,6-trinitrobenzene sulfonic acid [TNBS])-induced colitis.

59 Using both the trinitrobenzene sulfonic acid and dextran sodium sulfate

60 fector phase exhibited a reduced severity of trinitrobenzene sulfonic acid colitis in association wit

61 These studies show that in chronic 2,4,6-trinitrobenzene sulfonic acid colitis, fibrosis is depen

62 T helper cell (Th)1-mediated inflammation in trinitrobenzene sulfonic acid colitis.

63 Six weekly 2,4,6-trinitrobenzene sulfonic acid enemas were given to estab

64 Female Lewis rats underwent weekly trinitrobenzene sulfonic acid enemas yielding models of

65 histologic scores, reduced serum anti-2,4,6-trinitrobenzene sulfonic acid IgG, decreased leukocyte i

66 Intracolonic administration of trinitrobenzene sulfonic acid in mice causes inflammatio

67 CP was induced by intraductal injection of trinitrobenzene sulfonic acid in rats.

68 In the murine trinitrobenzene sulfonic acid model of colitis, inhibiti

69 Colitis was induced with trinitrobenzene sulfonic acid or dextran sulfate sodium,

70 iven intrarectal hREG3A and exposed to 2,4,6-trinitrobenzene sulfonic acid showed less colon damage a

71 induced by dextran sulfate sodium (DSS) and trinitrobenzene sulfonic acid showed that TG FVB/6 mice

72 go spontaneous resolution, despite continued trinitrobenzene sulfonic acid stimulation.

73 11b(+) cells before colitis was induced with trinitrobenzene sulfonic acid to monitor the effects of

74 FVB/6 mice were more susceptible to DSS and trinitrobenzene sulfonic acid treatment than wild-type F

75 grity in tissues from distal colons of rats; trinitrobenzene sulfonic acid was used to induce inflamm

76 of PEG adducts by an indirect colorimetric (trinitrobenzene sulfonic acid) reaction, which detects l

77 ed 3 mouse models of IBD: colitis induced by trinitrobenzene sulfonic acid, colitis induced by microf

78 colonic inflammation after exposure to 2,4,6-trinitrobenzene sulfonic acid, compared with control mic

79 to membrane impermeable reagents, like 2,4,6-trinitrobenzene sulfonic acid, is also reduced severalfo

80 A membrane impermeant quencher, 2,4,6-trinitrobenzene sulfonic acid, or lipid-based quenchers

81 llenge in mice sensitized with hapten (2,4,6-trinitrobenzene sulfonic acid, TNBS).

82 ial regulation of ion transport in mice with trinitrobenzene sulfonic acid- or dextran sodium sulfate

83 hy, edema, ulceration, and necrosis in 2,4,6-trinitrobenzene sulfonic acid-exposed mice.

84 elope prevented and treated a model of acute trinitrobenzene sulfonic acid-induced (TNBS-induced) col

85 ial function in vivo reduced the severity of trinitrobenzene sulfonic acid-induced colitis and associ

86 Anti-CD70 Ab treatment also suppressed trinitrobenzene sulfonic acid-induced colitis in SJL/J m

87 disease and ulcerative colitis, and in 2,4,6-trinitrobenzene sulfonic acid-induced colitis mice.

88 We studied this phenomenon in a chronic trinitrobenzene sulfonic acid-induced colitis model, an

89 In a rat trinitrobenzene sulfonic acid-induced colitis model, ora

90 (gliadin) stimulation and ameliorated acute trinitrobenzene sulfonic acid-induced colitis secondary

91 In contrast, trinitrobenzene sulfonic acid-induced colitis, a predomi

92 y of tissue-specific PPAR-gamma null mice to trinitrobenzene sulfonic acid-induced colitis.

93 o protected against the development of 2,4,6-trinitrobenzene sulfonic acid-induced colitis.

94 significantly attenuated both DSS- and 2,4,6-trinitrobenzene sulfonic acid-induced colonic inflammati

95 erlying chronic inflammation and fibrosis in trinitrobenzene sulfonic acid-induced murine colitis.

96 , transfer (i.v.) of TGF-beta2-treated 2,4,6-trinitrobenzene sulfonic acid-pulsed bone marrow-derived

97 Mice that received 2,4,6-trinitrobenzene sulfonic acid-sensitized cells and chall

98 vatus shifted the immune cell populations in trinitrobenzene sulfonic acid-treated mice and up-regula

99 e the degree of digestion is determined with trinitrobenzene sulfonic acid.

100 /C mice after intracolonic administration of trinitrobenzene sulfonic acid.

101 awley rats by an intraductal injection of 2% trinitrobenzene sulfonic acid.

102 1-7 days after inflammation was induced with trinitrobenzene sulfonic acid.

103 uated colonic inflammatory pain induced with trinitrobenzene sulfonic acid.

104 igs and those treated 6 days previously with trinitrobenzene sulfonic acid.

105 4 T cells into rag2(-/-) recipients and (ii) trinitrobenzene sulfonic acid.

106 ministered weekly doses of intrarectal 2,4,6-trinitrobenzene sulfonic acid.

107 induced in rats by intraductal injection of trinitrobenzene sulfonic acid.

108 administration of dextran sodium sulfate or trinitrobenzene sulfonic acid.

109 navailable to chemical modification by 2,4,6-trinitrobenzene sulfonic acid.

110 nic insult by intraluminal administration of trinitrobenzene sulfonic acid; controls received saline.

111 trol and hREG3A-TG mice by administration of trinitrobenzene sulfonic acid; some mice were given intr

112 Colitis was induced in mice using trinitrobenzene sulfonic acid; symptoms were monitored a

113 ) mice by administration of ethanol; 2, 4, 6-trinitrobenzene sulfonic-acid (TNBS); or infection with

114 g) mice to induction of colitis with 2, 4, 6-trinitrobenzene-sulfonic-acid (TNBS), and the course of

115 olitis induced by dextran sulfate sodium and trinitrobenzene sulfuric acid, KO mice were more toleran

116 C induced by dextran sulfate sodium (DSS) or trinitrobenzene sulphonic acid (TNBS) in mice.

117 atients with inflamed colonic mucosa, and in trinitrobenzene sulphonic acid (TNBS) induced colitis in

118 We have previously demonstrated that trinitrobenzene sulphonic acid (TNBS)-induced colitis in

119 facilitation in the myenteric plexus of the trinitrobenzene sulphonic acid-inflamed guinea-pig dista

120 nzene (TCTNB), 1:1 DADP/1,3,5-tribromo-2,4,6-trinitrobenzene (TBTNB), and 1:1 DADP/1,3,5-triiodo-2,4,

121 e explosives: 1:1 DADP/1,3,5-trichloro-2,4,6-trinitrobenzene (TCTNB), 1:1 DADP/1,3,5-tribromo-2,4,6-t

122 ne (TBTNB), and 1:1 DADP/1,3,5-triiodo-2,4,6-trinitrobenzene (TITNB).

123 g an Affi-Gel resin derivatized with a 2,4,6-trinitrobenzene (TNB) moiety and a fluorophore-labeled a

124 ol (TNP), 2,4,6-trinitrotoluene (TNT), 1,3,5-trinitrobenzene (TNB), and 2,4-dinitrotoluene (DNT).

125 sives, 2,4,6-trinitrotoluene (TNT) and 1,3,5-trinitrobenzene (TNB), are often encountered together as

126 Upon exposure to trinitrobenzene (TNB), the self-assembled structures for

127 Concentrations of 1 ppm of trinitrobenzene (TNB), trinitrotoluene (TNT), dinitroben

128 of a soluble analyte, fluorescently labeled trinitrobenzene, to surface-immobilized monoclonal anti-

129 rotoluenes, dinitrobenzene, trinitrotoluene, trinitrobenzene, two aminodinitrotoluenes, three nitroes

130 ethanol as a precursor, available from 1,3,5-trinitrobenzene, two further nitro-containing molecules

131 1-Cyano-2,4,6-trinitrobenzene was found to be the most reactive and th