ライフサイエンスコーパス: triphosphatase

1 n proposed for other AAA+ ATPases (adenosine triphosphatases).

2 -SNPH coupling inhibited the motor adenosine triphosphatase.

3 were shown to be activated by Ran guanosine triphosphatase.

4 he level of the vacuolar-type H(+)-adenosine triphosphatase.

5 pumping enzyme vacuolar-type H(+)-adenosine triphosphatase.

6 hich encodes a copper-transporting adenosine triphosphatase.

7 is tunnel and into the active site of the CE triphosphatase.

8 ral large structural proteins and nucleoside triphosphatases.

9 C is activated by Rho family small guanosine triphosphatases.

10 f cell surface-associated vacuolar adenosine triphosphatases.

11 sensitivity and drug efficacy in nucleotide triphosphatases.

12 nd physically interacts with small guanosine triphosphatases.

13 horylation of plasma membrane H(+)-adenosine triphosphatase 2 at Ser(899), mediating the inhibition o

14 a critical proteasome subunit, non-adenosine triphosphatase 4 (PSMD4), was reduced in old mice.

15 s involved in the purine metabolism (inosine triphosphatase, 5'-nucleotidase cytosolic-II, purine nuc

16 at the Insert B domain of the Dnm1 guanosine triphosphatase (a DRP) contains a novel motif required f

17 es (AIs) that contain the cellular adenosine triphosphatase ABCE1 (ATP-binding cassette protein E1).

18 ing protein 1 (ELMOD1), a guanine nucleoside triphosphatase activating protein (GAP) for ARF6, as the

19 rsed at the lagging pole where the guanosine triphosphatase activating protein MglB disrupts the MglA

20 ce that neurofibromin, via its Ras guanosine triphosphatase -activating activity, controls ERK1/2-dep

21 -CYK4 centralspindlin complex is a guanosine triphosphatase-activating protein (GAP) for Rac1 and not

22 y binds to the IQ motif-containing guanosine triphosphatase-activating protein 1 (IQGAP1) scaffold pr

23 binds simultaneously to the Cdc42p guanosine triphosphatase-activating protein Bem3p.

24 ure events, independent of the Rho guanosine triphosphatase-activating protein domain.

25 es small ARF GAP1 (SMAP1), an ARF6 guanosine triphosphatase-activating protein that functions in clat

26 lating the expression level of Ras guanosine triphosphatase-activating protein.

27 vity for p120RasGAP (RASA1), a Ras guanosine triphosphatase-activating protein.

28 2/Bub2/Cdc16 domain-containing Rab guanosine triphosphatase-activating proteins (GAPs) and identified

29 Two Rab guanosine triphosphatases-activating proteins (GAPs) have been imp

30 signaling to engage small GTPase (guanosine triphosphatase) activation and AMPAR synaptic delivery d

31 Both nuclease and dNTP triphosphatase activities of SAMHD1 are associated with

32 r has ATPase and, more generally, nucleoside triphosphatase activities that are indistinguishable fro

33 dependent protease, RNA helicase, and 5'-RNA triphosphatase activities.

34 the N-terminal part exhibits NTPase and RNA triphosphatase activity and is proposed to have helicase

35 s altered by inhibition of dynamin guanosine triphosphatase activity and is temporally distinct from

36 Both Rab18 guanosine triphosphatase activity and isoprenylation are required

37 Here, we show that the RNA triphosphatase activity of DUSP11 promotes the RNA silen

38 es contractility by decreasing the adenosine triphosphatase activity of the cardiac myosin heavy chai

39 , suppression of PDGF-induced Rac1 guanosine triphosphatase activity, and Akt and extracellular signa

40 this protein has been shown to possess dNTP triphosphatase activity, which is proposed to inhibit HI

41 d N7 positions, and also displays nucleotide triphosphatase activity.

42 its double-stranded DNA-dependent adenosine triphosphatase activity.

43 ng predominantly determined by its guanosine triphosphatase activity.

44 MDCK) cells and identified the rho-guanosine triphosphatase adaptor and scaffolding protein IRSp53 as

45 The loss of the small guanosine triphosphatase ADP-ribosylation factor 1 (Arf1) or its e

46 , inhibition of filamentous septin guanosine triphosphatases alleviates constriction defects in Auror

47 the Na(+)/K(+) exchanger ATPalpha (adenosine triphosphatase alpha) in glia may be modulated by seroto

48 ses expression of the gastric H, K-adenosine triphosphatase alpha-subunit (HKalpha), which could cont

49 Recently, human dCTPase (deoxycytidine triphosphatase), also known as DCTPP1 (human all-alpha d

50 e folate pathway-related genes, deoxyuridine triphosphatase and dihydrofolate reductase, the silencin

51 We identified Arf6 guanosine triphosphatase and its activators, cytohesins, as previo

52 23, Lys-148, and Arg-321 uncoupled adenosine triphosphatase and Rca activity, implicating them in the

53 by overexpression of the capping enzymes RNA triphosphatase and RNA guanylyltransferase.

54 strate- and product-bound mammalian thiamine triphosphatase and with previously reported structures o

55 oluble and stable, did not bind to guanosine triphosphatases and bound more tightly to the PAK1 kinas

56 ns, including interferon-inducible guanosine triphosphatases and the antimicrobial cathelicidin, asse

57 e and activates mTORC1 via the Rag guanosine triphosphatases and their regulators GATOR1 and GATOR2.

58 ally distinct enzymes (a kinase, a guanosine triphosphatase, and a ubiquitin protein hydrolase) that

59 tein levels of MIG-2, a Rho family guanosine triphosphatase, and/or down-regulate INA-1, an integrin

60 ility signaling proteins (kinases, guanosine triphosphatases, and guanine exchange factors) controlle

61 gella when exogenous ubiquitin and adenosine triphosphatase are added, suggesting that the ubiquitin

62 bers of the RAS subfamily of small guanosine triphosphatases are found in > 30% of all human cancers.

63 We found that HIV-1 Nef and the guanosine triphosphatase Arf1 induced trimerization and activation

64 Rafiq et al. reveal that the small guanosine triphosphatase ARF1, a well-known orchestrator of membra

65 Here, using helicase and adenosine triphosphatase assays we show that a complex containing

66 served members of the AAA+ family (adenosine triphosphatases associated with diverse cellular activit

67 6 (which carries activators of Rho guanosine triphosphatase) at the cell cortex using total internal

68 an ER network is dependent on the guanosine triphosphatase atlastin (ATL).

69 clohexylurea group, which binds to adenosine triphosphatase (ATP)-sensitive K(+) (K(ATP)) channels fo

70 retion by the nongastric H(+)/K(+) adenosine triphosphatase (ATP12A) acidified airway surface liquid,

71 We found that the vacuolar H(+)-adenosine triphosphatase ATPase (v-ATPase) is necessary for amino

72 The Cdc48 adenosine triphosphatase (ATPase) (p97 or valosin-containing prote

73 The vesicular adenosine triphosphatase (ATPase) acidifies intracellular compartm

74 ed of the Mre11 nuclease and Rad50 adenosine triphosphatase (ATPase) active sites dimerizes through a

75 tive orientation of the N-terminal adenosine triphosphatase (ATPase) and C-terminal nuclease domains.

76 nd 130 nM in microtubule-dependent adenosine triphosphatase (ATPase) and cell-based cytotoxicity assa

77 Here we present evidence that the adenosine triphosphatase (ATPase) cycle of the SF1 helicase Upf1 i

78 ent in the catalytic space of gp17-adenosine triphosphatase (ATPase) determines the rate at which the

79 nally characterized the DExD/H box adenosine triphosphatase (ATPase) Dhh1, a critical regulator of th

80 the conserved Microrchidia (MORC) adenosine triphosphatase (ATPase) family, which are predicted to c

81 xamined whether ITP can be used by adenosine triphosphatase (ATPase) in human erythrocytes or recombi

82 species and cell types, Na(+),K(+)-adenosine triphosphatase (ATPase) is arguably the most powerful co

83 As a ring-shaped adenosine triphosphatase (ATPase) machine, cohesin organizes the e

84 t of the system is the proteasomal adenosine triphosphatase (ATPase) Mpa, which captures, unfolds, an

85 otein (Prp5p) is an RNA-stimulated adenosine triphosphatase (ATPase) required for prespliceosome form

86 tructures portraying the hexameric adenosine triphosphatase (ATPase) rho on a pathway to terminating

87 p97 is a hexameric AAA+ adenosine triphosphatase (ATPase) that is an attractive target for

88 for packaging in such viruses: the adenosine triphosphatase (ATPase) that powers DNA translocation an

89 Here, we found that the orphan P5A-adenosine triphosphatase (ATPase) transporter ATP13A1 (Spf1 in yea

90 acilitates both recruitment of the adenosine triphosphatase (ATPase)-activating cochaperone Aha1 and,

91 symporter, and hydrogen potassium adenosine triphosphatase [ATPase]) showed reduced expression or mi

92 croscopy structure reveals how the adenosine triphosphatases (ATPases) form a closed spiral staircase

93 In eukaryotes, P-type adenosine triphosphatases (ATPases) generate the plasma membrane p

94 m ring-shaped hexameric AAA-family adenosine triphosphatases (ATPases), dynein's large size and compl

95 the microrchidia (MORC) family of adenosine triphosphatases (ATPases), has been shown to be involved

96 es belonging to the AAA+ family of adenosine triphosphatases (ATPases).

97 Here we show that rotary adenosine triphosphatases (ATPases)/synthases from Thermus thermop

98 the BAG1 and bradyzoite-specific nucleoside triphosphatase (B-NTPase) promoters.

99 Protein release factor 3 (RF3), a guanosine triphosphatase, binds to ribosome after release of the n

100 witch by sensing mitochondrial Rho guanosine triphosphatase-Ca(2+) and as a brake by anchoring mitoch

101 e cleaved from a2 isoform vacuolar adenosine triphosphatase called a2NTD.

102 at in living cells the cytoplasmic adenosine triphosphatase called ClpV specifically recognizes the c

103 re, we examine the function of Rho guanosine triphosphatase CDC-42 in AJ formation and regulation dur

104 PAR proteins (including the small guanosine triphosphatase CDC-42) have an active role in regulating

105 zation of the conserved Rho-family guanosine triphosphatase, Cdc42, to the cortical region destined t

106 etween the decoding center and the guanosine triphosphatase center of EF-Tu.

107 embrane by the p97/Cdc48-Ufd1-Npl4 adenosine triphosphatase complex is essential for mitochondria-ass

108 s a chromosome-bound, multisubunit adenosine triphosphatase complex.

109 neuron-specific proton pump and V0 adenosine triphosphatase component V100.

110 of BRG1-also known as SMARCA4, an adenosine triphosphatase-containing chromatin remodeler-and SMAD3

111 The Rab subfamily of small guanosine triphosphatases controls these processes by acting as a

112 It is caused by mutations in the adenosine triphosphatase copper transporting beta gene (ATP7B), wh

113 s on membranes and suppress futile adenosine triphosphatase cycles.

114 how that genetic variants leading to inosine triphosphatase deficiency, a condition not thought to be

115 ed Trio-dependent Rac1 activation, adenosine triphosphatase-deficient Hsc70 (D10N) abrogated Trio Rac

116 n a Rag-, Ragulator-, and vacuolar adenosine triphosphatase-dependent fashion, the translocation of m

117 the TASCC was amino acid- and Rag guanosine triphosphatase-dependent, and disruption of mTOR localiz

118 d assembly of some dynamin-related guanosine triphosphatases depends on adaptor proteins restricted t

119 iral replication on an intact nucleotide/RNA triphosphatase domain and an N-terminal cluster of basic

120 conserved Swc2/YL1 subunit and the adenosine triphosphatase domain of Swr1 are mainly responsible for

121 ns within Hebo: a TUDOR domain, an adenosine triphosphatase domain, and a new domain, HEBO, specifica

122 ositions (p.G488R, p.A495V) in the guanosine triphosphatase domain, each segregating with affected in

123 a C-terminal guanylyltransferase and RNA 5'-triphosphatase domain.

124 For example, RNA capping enzymes possess triphosphatase domains that remove the gamma phosphates

125 The dynamin-related guanosine triphosphatase Drp1 mediates the division of mitochondri

126 Here we demonstrate that the RNA triphosphatase dual-specificity phosphatase 11 (DUSP11)

127 The large guanosine 5'-triphosphatase dynamin (Dyn) plays an important role in

128 we provide evidence that the large guanosine triphosphatase dynamin2 and its partner, endothelial nit

129 actor Tif6 by the translocase-like guanosine triphosphatase Efl1 is a critical late maturation step.

130 ltistep process facilitated by the guanosine triphosphatase elongation factor (EF)-Tu.

131 o tRNAs, nascent polypeptides, the guanosine triphosphatase elongation factors mtEF-Tu and mtEF-G1, a

132 42 (Cdc42) is a member of the Rho guanosine triphosphatase family and has pivotal functions in actin

133 ppears to load/unload RuvBL AAA(+) adenosine triphosphatase from pre-snoRNPs; and (d) a potential mec

134 mutations in a copper-transporting adenosine triphosphatase gene, ATP7A.

135 tional antiterminator, an inosine/xanthosine triphosphatase, GidA, a methyl-accepting chemotaxis prot

136 e factor (GEF) for Tem1, the small guanosine triphosphatase governing activity of the Saccharomyces c

137 Rho family guanosine triphosphatase (GTPase) 3 (Rnd3), a member of the small

138 1 (mTORC1) protein kinase via its guanosine triphosphatase (GTPase) activating protein (GAP) activit

139 SRP and SR, which stimulates their guanosine triphosphatase (GTPase) activities, leading to dissociat

140 onstitutively active because their guanosine triphosphatase (GTPase) activity is disabled.

141 this study, we show that the small guanosine triphosphatase (GTPase) adenosine diphosphate ribosylati

142 kinase 2 (LRRK2) protein has both guanosine triphosphatase (GTPase) and kinase activities, and mutat

143 LRRK2 has guanosine triphosphatase (GTPase) and kinase activities, and mutat

144 , ciliary trafficking of the small guanosine triphosphatase (GTPase) Arl13b, loss of which causes cys

145 SKIP binds to the small guanosine triphosphatase (GTPase) ARL8 on the lysosomal membrane t

146 The Rho guanosine triphosphatase (GTPase) Cdc42 regulates sequential molec

147 itochondrial division, the mechano-guanosine triphosphatase (GTPase) dynamin-related protein (Drp1) s

148 the exocyst complex, a common Ral guanosine triphosphatase (GTPase) effector.

149 The guanosine triphosphatase (GTPase) elongation factor G (EF-G) catal

150 t rotation and is catalyzed by the guanosine triphosphatase (GTPase) elongation factor G (EF-G).

151 Protein synthesis requires several guanosine triphosphatase (GTPase) factors, including elongation fa

152 by amino acids is mediated by Rag guanosine triphosphatase (GTPase) heterodimers on the lysosome.

153 g KRAS(G12C), a mutant form of the guanosine triphosphatase (GTPase) KRAS, are a promising new class

154 ion in the gene encoding the small guanosine triphosphatase (GTPase) NRAS.

155 infection was a large set encoding guanosine triphosphatase (GTPase) of immunity-associated proteins

156 Deregulation of Rho guanosine triphosphatase (GTPase) pathways plays an important role

157 The Cdc42 guanosine triphosphatase (GTPase) plays a central role in polarity

158 The small guanine triphosphatase (GTPase) proteins RhoA and RhoC are essen

159 pulating the activity of the small guanosine triphosphatase (GTPase) Rab1.

160 The guanosine triphosphatase (GTPase) Rab32 coordinates a cell-intrins

161 The small guanosine triphosphatase (GTPase) Rab7 has been implicated in the

162 cell migration, whereas the small guanosine triphosphatase (GTPase) Rac1 mediates cell migration.

163 bioprobe), we revealed that Cdc42 guanosine triphosphatase (GTPase) remains inactive within Drosophi

164 and animal cytokinesis, the small guanosine triphosphatase (GTPase) Rho1/RhoA has an established rol

165 rotrimeric G proteins to the small guanosine triphosphatase (GTPase) RhoA, enabling Galpha(q/11)-coup

166 Etd1 activates the guanosine triphosphatase (GTPase) Spg1 to trigger signaling throug

167 of a 43-member IFN-gamma-inducible guanosine triphosphatase (GTPase) superfamily in mouse and human g

168 Elongation factor G (EF-G) is a guanosine triphosphatase (GTPase) that plays a crucial role in the

169 (PKG), protein kinase A (PKA), Rho guanosine triphosphatase (GTPase), and MLC phosphatase was monitor

170 sis, elongation factor G (EF-G), a guanosine triphosphatase (GTPase), binds to the ribosomal PRE-tran

171 ion, we identified the RAB35 small guanosine triphosphatase (GTPase)-a protein previously implicated

172 tor subunit interacts with the Rag guanosine triphosphatase (GTPase)-Ragulator complex.

173 les mediated by the atlastin (ATL) guanosine triphosphatase (GTPase).

174 ase-associated RAS subfamily small guanosine triphosphatase (GTPase).

175 membrane-associated ROPs [Rho-like guanosine triphosphatases (GTPase) from plants], leading to change

176 witch region I of immunity-related guanosine triphosphatases (GTPases) (IRGs), a family of host defen

177 The Rag guanosine triphosphatases (GTPases) activate mTORC1 in response to

178 vated on lysosomes by Rag and Rheb guanosine triphosphatases (GTPases) and drives biosynthetic proces

179 rotein that interacts with the Rag guanosine triphosphatases (GTPases) and Ragulator in an amino acid

180 s recruited to the lysosome by Rag guanosine triphosphatases (GTPases) and regulates anabolic pathway

181 ositol 4-kinases (PI4Ks) and small guanosine triphosphatases (GTPases) are essential for processes th

182 Rho guanosine triphosphatases (GTPases) are implicated in TEM, but the

183 Rho guanosine triphosphatases (GTPases) are master regulators of cell

184 The guanosine triphosphatases (GTPases) Arf, Sar1, and dynamin are cor

185 tivation and inactivation of small guanosine triphosphatases (GTPases) by their specific guanine exch

186 rocess is mediated by dynamin-like guanosine triphosphatases (GTPases) called atlastins (ATLs), which

187 Rab guanosine triphosphatases (GTPases) control cellular trafficking p

188 Rho guanosine triphosphatases (GTPases) control the cytoskeletal dynam

189 uring the cellular activity of small guanine triphosphatases (GTPases) in response to a specific stim

190 The Rho family of guanosine triphosphatases (GTPases) is composed of members of the

191 The guanosine triphosphatases (GTPases) of the immunity-associated pro

192 he activity of the recycling small guanosine triphosphatases (GTPases) Rab4 or Rab11 was sufficient t

193 The Rag guanosine triphosphatases (GTPases) recruit the master kinase mTOR

194 signaling pathways involving small guanosine triphosphatases (GTPases) regulate cell polarization.

195 Ras guanosine triphosphatases (GTPases) regulate signaling pathways on

196 Septins are filamentous guanine triphosphatases (GTPases) that associate with MTs, but t

197 o acids, which act through the Rag guanosine triphosphatases (GTPases) to promote mTORC1 translocatio

198 Nutrients signal via the Rag guanosine triphosphatases (GTPases) to promote the localization of

199 Activation of Rho guanosine triphosphatases (GTPases) to the guanine triphosphate (G

200 feron (IFN)-inducible subfamily of guanosine triphosphatases (GTPases) with well-established activity

201 the Ragulator complex and the Rag guanosine triphosphatases (GTPases), causing release of the inacti

202 proteins, the Rho family of small guanosine triphosphatases (GTPases), is critical for this actin an

203 to other members of the Ras small guanosine triphosphatases (GTPases), mutations affecting RIT1 are

204 Many of those factors are guanosine triphosphatases (GTPases), proteins that catalyze the hy

205 t to be dependent on the Rag small guanosine triphosphatases (GTPases), the Ragulator complex, and th

206 esses all involve Rho family small guanosine triphosphatases (GTPases), which are regulated by the op

207 Amino acids activate the Rag guanosine triphosphatases (GTPases), which promote the translocati

208 vation of Ras homolog (Rho) family guanosine triphosphatases (GTPases), which regulate the formation

209 s signal to mTORC1 through the Rag guanosine triphosphatases (GTPases).

210 he surface of lysosomes by the Rag guanosine triphosphatases (GTPases).

211 Activation of the small guanosine triphosphatase H-Ras by the exchange factor Son of Seven

212 e coiled coil emerging from Smc3's adenosine triphosphatase head.

213 e through the action of Na(+)/K(+) adenosine triphosphatases in an integrated in vitro lipid bilayer

214 ocus on the role of the Rho family guanosine triphosphatases in endothelial function and vasoconstric

215 hagy, we silenced VCP or expressed adenosine triphosphatase-inactive VCP.

216 hydrazide (Dynasore), a dynamin guanosine 5'-triphosphatase inhibitor, protected stromal cells agains

217 Proton pump (H(+)/K(+)-adenosine triphosphatase) inhibitors (PPIs) are widely used to tre

218 42 (Cdc42Hs) is a small, Rho-type guanosine triphosphatase involved in multiple cellular processes t

219 The Cdc42 guanosine triphosphatase is essential for cell polarization in sev

220 The a2 isoform vacuolar adenosine triphosphatase is found on the surface on many solid tum

221 hat the a3-subunit of the vacuolar adenosine triphosphatase is not only responsible for the acidifica

222 ubunit of the vacuolar-type (H(+))-adenosine triphosphatase is required for establishing a luminal pH

223 ally encoded, photoactivatable Rac guanosine triphosphatase is sufficient to direct migration of neut

224 The Rab family of small guanosine triphosphatases is evolutionarily conserved and mediates

225 ow that dynamin1 (Dyn1), the large guanosine triphosphatase, is an interacting partner of IRSp53 thro

226 Cdc42, a Rho guanosine triphosphatase, is thought to orchestrate critical actin

227 Genetic variation of inosine triphosphatase (ITPA) causing an accumulation of inosine

228 Two functional variants in the inosine triphosphatase (ITPA) gene causing inosine triphosphatas

229 Inosine triphosphatase (ITPA) variants causing ITPase deficiency

230 e triphosphatase (ITPA) gene causing inosine triphosphatase (ITPase) deficiency protect against ribav

231 nctional variants in ITPA that cause inosine triphosphatase (ITPase) deficiency were shown to protect

232 owth factor receptor (EGFR) or the guanosine triphosphatase KRAS.

233 nhibition of Cdc42 and related Rho guanosine triphosphatases may be a general feature of cytokinesis

234 An intrinsic guanosine triphosphatase mediates a contact between the head and c

235 The mammalian dynamin-related guanosine triphosphatases Mfn1,2 and Opa1 are required for mitocho

236 t the mitochondrial outer membrane guanosine triphosphatase mitofusin (Mfn) 2 mediates Parkin recruit

237 RSC is delineated into the adenosine triphosphatase motor, the actin-related protein module,

238 reported that the sodium potassium adenosine triphosphatase (Na/K-ATPase) can effect the amplificatio

239 alpha1 subunit of sodium potassium adenosine triphosphatase (Na/K-ATPase), acts as a receptor and an

240 nits and possesses 40S-stimulated nucleoside triphosphatase (NTPase) activity.

241 Here, by nucleoside triphosphatase (NTPase) assay and comparisons of six hig

242 its monomer-associated nucleoside and 5' RNA triphosphatase (NTPase/RTPase) activities that are media

243 ed as putative nucleotide binding nucleoside triphosphatases (NTPases) or nucleoside triphosphate (NT

244 lytic cleavage of the dynamin-like guanosine triphosphatase OPA1 in mitochondria is emerging as a cen

245 c oxidase, inhibition of F(1)F(0) adenosine triphosphatase, or replacement of all mtDNA-encoded gene

246 1, its partner Sel1, the cytosolic adenosine triphosphatase p97, and degradation by the proteasome.

247 Cdc42, a member of Rho GTPases (guanosine triphosphatases), participates in cytokine- and growth f

248 tructures through a unidirectional adenosine triphosphatase polymerization, primed with a single PrgJ

249 However, constitutively active Rag guanosine triphosphatases prevented TFEB translocation during mito

250 the sarcoplasmic reticulum Ca(2+)-adenosine triphosphatase pump and by augmented levels of the Na(+)

251 The small guanosine triphosphatase Rab13 functions in exocytic vesicle traff

252 tide exchange factor for the small guanosine triphosphatase Rab8, to promote recruitment of Rab8 and

253 downstream effectors of the small guanosine triphosphatases Rac and Cdc42, biochemically couple lead

254 mics and the activity of the small guanosine triphosphatases Rac and Cdc42.

255 jor effectors of the Rho guanosine 5'\x{2011}triphosphatase, Rac, and Cdc42.

256 creasing the activity of the small guanosine triphosphatase Rac1 and Cdc42.

257 hanced by signaling from the small guanosine triphosphatase, Rac1.

258 iated by chromosomes and the small guanosine triphosphatase Ran in a process requiring ~16 hours.

259 The small guanosine triphosphatase Rap1 regulates LFA-1 adhesiveness through

260 reduction by activating the small guanosine triphosphatase Ras homolog family member J by releasing

261 ficantly regulated by the enzymes' adenosine triphosphatase regions.

262 , such as RPGR (retinitis pigmentosa guanine triphosphatase regulator) that are expressed in both rod

263 B1 (encoding beta-catenin) and RHO guanosine triphosphatase [RHO GTPase, RHO], two signaling pathways

264 In many organisms, the small guanosine triphosphatase RhoA controls assembly and contraction of

265 ase c-Src and stimulated the small guanosine triphosphatase RhoA, consequently inhibiting cell spread

266 owth response 3 (EGR3) and the Rho guanosine triphosphatase RhoA.

267 a mimic of the human Ras-like Rho guanosine triphosphatase RhoG.

268 mouse embryos that lack the small guanosine triphosphatase RSG1 die at embryonic day 12.5, with deve

269 The guanosine triphosphatase Sar1 controls the assembly and fission of

270 lum (SR) and reuptake by the Ca(2+)adenosine triphosphatase SERCA.

271 asmic/endoplasmic reticulum Ca(2+) adenosine triphosphatase (SERCA)2a, a critical regulator of calciu

272 rcoplasmic reticulum Ca(2+) uptake adenosine triphosphatase (SERCA2a), phospholamban (PLB), and incre

273 eIF2 is a guanosine triphosphatase that becomes activated by eIF2B, a two-fo

274 receptor for DRP1, the cytoplasmic guanosine triphosphatase that catalyzes mitochondrial fission.

275 Mtr4 is an essential RNA-dependent adenosine triphosphatase that is required for all of the nuclear f

276 ated protein 1 (Drp1), a cytosolic guanosine triphosphatase that polymerizes and constricts the organ

277 ncodes a multitransmembrane domain adenosine triphosphatase that traffics from the trans-Golgi networ

278 the activity of Rho and Rap small guanosine triphosphatases that control integrin activation by chem

279 , a member of the septin family of guanosine triphosphatases that form a diffusion barrier in budding

280 Rabs are monomeric guanosine triphosphatases that regulate membrane trafficking and a

281 Fzo1 and Mgm1 are conserved guanosine triphosphatases that reside in the outer and inner membr

282 Competitive binding of small guanosine triphosphatases to the IS domain disrupts the autoinhibi

283 4 (EF4/LepA) is a highly conserved guanosine triphosphatase translation factor.

284 oil, akin to the way AAA+ ATPases (adenosine triphosphatases) unfold peptides.

285 ogether with reduced vacuolar H(+)-adenosine triphosphatase (V-ATPase) activity, accounts for the red

286 leading to the removal of vacuolar adenosine triphosphatase (V-ATPase) and the neutralization of lyso

287 iched for subunits of the vacuolar adenosine triphosphatase (V-ATPase) complex, a proton pump require

288 gh inhibition of the vacuolar H(+)-adenosine triphosphatase (V-ATPase) increased the luminal concentr

289 The vesicular adenosine triphosphatase (v-ATPase) is a proton pump that acidifie

290 0 domain of the vacuolar-type H(+)-adenosine triphosphatase (V-ATPase) is directly implicated in secr

291 s never recycle vacuolar-type H(+)-adenosine triphosphatase (V-ATPase) or neutralize to form postlyso

292 tor complex, and the vacuolar H(+)-adenosine triphosphatase (v-ATPase).

293 Vesicular- or vacuolar-type adenosine triphosphatases (V-ATPases) are ATP-driven proton pumps

294 umping vesicular- or vacuolar-type adenosine triphosphatases (V-ATPases).

295 at the neuronal vacuolar-type H(+)-adenosine triphosphatase V0 subunit a1 (V100) can regulate the for

296 Binding to the AAA adenosine triphosphatase valosin-containing protein (VCP)/p97 is t

297 The AAA-adenosine triphosphatase Vps4 disassembles and recycles the ESCRT-

298 pressorium by means of four septin guanosine triphosphatases, which polymerize into a dynamic, hetero

299 ter clusters may be universal for nucleotide triphosphatases with conserved active sites, such as myo

300 The activation of Rho guanosine triphosphatases within the extending growth cone facilit