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1                              We propose that trophoblastic ACKR2 is responsible for ensuring chemokin
2 action produces soluble endoglin, we treated trophoblastic BeWo cells with either a broad-spectrum MM
3 d immunosorbent assay in the supernatants of trophoblastic cell cultures.
4 e first demonstrated that alpha(2)M* induced trophoblastic cell fusion.
5 gnalling molecules and mechanisms regulating trophoblastic cell fusion.
6 f giant syncytia, and that fusion of a human trophoblastic cell line expressing endogenous syncytin c
7         HIP supports attachment of the human trophoblastic cell line, JAR, in a HS-dependent fashion.
8 ellular Ca handling was investigated using a trophoblastic cell line, Rcho-1, derived from a rat chor
9 tokine production by endothelial cells and a trophoblastic cell line.
10  blastocysts to uterine epithelium and human trophoblastic cell lines to uterine epithelial cell line
11 he rat choriocarcinoma cell line Rcho-1 as a trophoblastic cell model system.
12 tate 1 (STEAP1), delta-like ligand 3 (DLL3), trophoblastic cell-surface antigen 2 (TROP2), prostate s
13 n that mediates apical cell adhesion between trophoblastic cells and luminal epithelial cells of the
14  This study showed that CORIN is secreted by trophoblastic cells and that high levels of soluble CORI
15                            Activation of the trophoblastic cells by anticardiolipin occurs through To
16 n of single chain urokinase binding to human trophoblastic cells by long chain fatty acids was dose-d
17 Our results demonstrate that transduction of trophoblastic cells by the herpes simplex virus vector,
18 4% of cases, whereas the normal extravillous trophoblastic cells did not.
19  high basal expression of CYP27B1 and VDR in trophoblastic cells from the placenta, we hypothesized t
20 pression and enhanced lymphocyte adhesion to trophoblastic cells in experimental systems.
21 nal albumin endocytosis, was associated with trophoblastic cells in the interphase labyrinth.
22                                  In general, trophoblastic cells of HIV-positive placentas expressed
23 h levels were observed only in proliferating trophoblastic cells of the placenta and in adrenal gland
24 mpound had any effect on normal extravillous trophoblastic cells or JEG-3 cells.
25 ransfer of the lacZ reporter gene into human trophoblastic cells using herpes simplex virus and adeno
26 t inefficient transduction of differentiated trophoblastic cells with HSV.CMVlac is because of dimini
27 ed (mRNA and protein levels) and secreted by trophoblastic cells, mostly by the syncytiotrophoblast.
28 d that alpha(2)M mRNA is highly expressed in trophoblastic cells, whereas it is not expressed in the
29 )M* may also activate signalling pathways in trophoblastic cells, which, in turn, may promote cell fu
30 IV gag transcripts of HIV-positive placental trophoblastic cells.
31 opin hormone (CSH) produced by the placental trophoblastic cells.
32 arcinoma JEG-3 cells and normal extravillous trophoblastic cells.
33 se to proliferative stem cells with enhanced trophoblastic characteristics.
34  vivo studies using placentas -/- for fetal (trophoblastic) Cyp27b1 or VDR.
35 e placental syncytiotrophoblast and deported trophoblastic debris serve as sources of fetal mHAgs.
36 (TS) cell self-renewal, and promotes further trophoblastic differentiation both upstream and independ
37                                  Gestational trophoblastic disease (GTD) is a spectrum of rare neopla
38 noma, the most malignant form of gestational trophoblastic disease (GTD).
39 otherapy to eliminate persistent gestational trophoblastic disease (pGTD) was required in three of 19
40 ise strategies for management of gestational trophoblastic disease and address some of the controvers
41 astic tumours are a rare form of gestational trophoblastic disease and consequently information about
42                                  Gestational trophoblastic disease encompasses a range of pregnancy-r
43  of 631 patients who underwent treatment for trophoblastic disease had or developed evidence of brain
44 5 550 women were registered with gestational trophoblastic disease in the UK (1976-2006), of whom 62
45  Indications for chemotherapy in gestational trophoblastic disease include raised human chorionic gon
46                                  Gestational trophoblastic disease metastatic to the brain is curable
47 resents the malignant end of the gestational trophoblastic disease spectrum.
48 ly lower levels of expression in gestational trophoblastic disease tissues, particularly in complete
49 ics, diagnosis, and treatment of gestational trophoblastic disease.
50                                  Gestational trophoblastic diseases (GTDs) are a group of pregnancy-r
51                                  Gestational trophoblastic diseases comprise a spectrum of interrelat
52 , and International Society for the Study of Trophoblastic Diseases) delegated 53 expert GTD clinicia
53  (European Organisation for the Treatment of Trophoblastic Diseases, European Society of Gynecologic
54 rtant role in the development of gestational trophoblastic diseases.
55                                The placental trophoblastic epithelium functions to transport nutrient
56 itive to estrogen and progesterone, and that trophoblastic EVs stimulate their release in both recept
57  EECs exposed to estrogen, progesterone, and trophoblastic EVs to gain deeper insights into its poten
58 uch as binding, fusion or internalization of trophoblastic EVs within EECs.
59 trial epithelial cells (EECs) in response to trophoblastic EVs.
60 on can divert ES cells toward extraembryonic trophoblastic fates and implicate Ras-MAPK signaling in
61 established a role for PLA2G6 in attenuating trophoblastic ferroptosis, and provided mechanistic insi
62 ing the differentiation of Rcho-1 cells into trophoblastic giant cells supports the importance of CaB
63 cidual cells cause a paracrine inhibition of trophoblastic HPGD expression, increasing PGE(2)/PGF(2al
64 aracrine mechanism that stimulates levels of trophoblastic HPGD, whose function is to inactivate labo
65 thologic tissue-remodeling processes such as trophoblastic implantation, wound healing, and tumor inv
66 ression of SRC-3, which impairs extravillous trophoblastic invasion and migration by directly interac
67 ogy of preeclampsia originates from impaired trophoblastic invasion of the placental resulting in mal
68 trium, whereas syncytiotrophoblasts covering trophoblastic lacunae or newly formed chorionic villi re
69 o remove the amniotic epithelium and chorion trophoblastic layer, which resulted in exposing the base
70                                          The trophoblastic layers of sos1(-/-) embryos were poorly de
71 nes remained intact while the epithelial and trophoblastic layers were entirely removed without consi
72 ons, such as histiocytic intervillositis and trophoblastic necrosis, in addition to fetal tissue dama
73 ife-threatening complications of gestational trophoblastic neoplasia (four of 15 vs none of 36; p=0.0
74 logy and Obstetrics score >/= 7) gestational trophoblastic neoplasia (GTN) frequently receive etoposi
75          Treatment of persistent gestational trophoblastic neoplasia (GTN) has been one of the succes
76 All women should be screened for gestational trophoblastic neoplasia after termination of pregnancy.
77      Although most patients with gestational trophoblastic neoplasia are cured by chemotherapy and tu
78 eed, most patients with low-risk gestational trophoblastic neoplasia are treated with single agent ch
79 with a pathological diagnosis of gestational trophoblastic neoplasia at the time of their pregnancy t
80 Until the fundamental biology of gestational trophoblastic neoplasia becomes more clearly understood,
81                                  Gestational trophoblastic neoplasia comprises a unique group of huma
82                         Low-risk gestational trophoblastic neoplasia including choriocarcinoma is oft
83                     Prognosis in gestational trophoblastic neoplasia is now excellent following treat
84 evelopments in the management of gestational trophoblastic neoplasia over the past few years.
85  Approximately 60% of women with gestational trophoblastic neoplasia presenting with a FIGO risk scor
86  we identified 431 patients with gestational trophoblastic neoplasia presenting with a FIGO risk scor
87 e, cohort study of patients with gestational trophoblastic neoplasia presenting with a FIGO score of
88  who received treatment at three gestational trophoblastic neoplasia reference centres in the UK, Bra
89                                  Gestational trophoblastic neoplasia represents the malignant end of
90                    Patients with gestational trophoblastic neoplasia who have an International Federa
91  and molar placentas, as well as gestational trophoblastic neoplasia, and assessed the effects of a n
92   Of 5025 patients with low-risk gestational trophoblastic neoplasia, we identified 431 patients with
93 or metastatic and chemoresistant gestational trophoblastic neoplasia.
94 utic targets to treat metastatic gestational trophoblastic neoplasia.
95 enetic changes that give rise to gestational trophoblastic neoplasia.
96 often not routinely examined for gestational trophoblastic neoplasia.
97  is expressed in the majority of gestational trophoblastic neoplasias, and is essential for choriocar
98  studies, but direct molecular evidence of a trophoblastic origin has not been established.
99 is in an attempt to confirm their presumable trophoblastic origin.
100 ithelioid trophoblastic tumors are of fetal (trophoblastic) origin.
101                                              Trophoblastic proliferation and invasion regulated by AC
102 tified gene regulatory networks for a set of trophoblastic proteins found to be differentially expres
103 ophinin, tastin, and cytokeratins in a human trophoblastic teratocarcinoma cell, HT-H.
104  trophoblast, IDO is not expressed in murine trophoblastic tissue but instead is found in stromal cel
105 te hydatidiform mole in which extraembryonic trophoblastic tissue develops but the embryo itself suff
106  in the invasive trophoblasts of gestational trophoblastic tissue.
107 n neoplastic diseases that derive from fetal trophoblastic tissues and represent semiallografts in pa
108                               Placental site trophoblastic tumor (PSTT) is a gestational neoplasm der
109                       Women with gestational trophoblastic tumors (GTT) resistant to single-agent che
110 en treated with chemotherapy for gestational trophoblastic tumors (GTT).
111 c tumors (n = 20), and 60% of placental site trophoblastic tumors (n = 10) exhibited FASN positivity.
112 horiocarcinomas (n = 33), 90% of epithelioid trophoblastic tumors (n = 20), and 60% of placental site
113   Genetic analysis identified nongestational trophoblastic tumors (nGTTs).
114                               Placental site trophoblastic tumors and epithelioid trophoblastic tumor
115 first molecular evidence that placental site trophoblastic tumors and epithelioid trophoblastic tumor
116 al site trophoblastic tumors and epithelioid trophoblastic tumors are of fetal (trophoblastic) origin
117 al site trophoblastic tumors and epithelioid trophoblastic tumors are thought to arise from intermedi
118                                              Trophoblastic tumors represent a unique group of human n
119                                   All of the trophoblastic tumors were heterozygous in at least 1 of
120 ncer risks for patients cured of gestational trophoblastic tumors with modern chemotherapy appear sim
121  composed of choriocarcinoma, placental-site trophoblastic tumour, and epithelioid trophoblastic tumo
122 l-site trophoblastic tumour, and epithelioid trophoblastic tumour, and many forms are derived from th
123 choriocarcinoma, and the rare placental-site trophoblastic tumour.
124 f whom 62 were diagnosed with placental-site trophoblastic tumours and included, retrospectively, in
125                               Placental-site trophoblastic tumours are a rare form of gestational tro
126 These malignant forms are termed gestational trophoblastic tumours or neoplasia.
127 ffectiveness of treatment for placental-site trophoblastic tumours.
128 ression were observed with placentas -/- for trophoblastic VDR.
129 these separated blastomeres can become small trophoblastic vesicles, which in turn induce decidual re
130 and immunohistochemistry localized CYP2J2 in trophoblastic villi and deciduas at 12 weeks and term.
131              These data indicate that fetal (trophoblastic) vitamin D plays a pivotal role in control

 
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