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1 by the addition of 2.5% phenylephrine and 1% tropicamide.
2 raction after cycloplegia with 2 drops of 1% tropicamide.
3 inducement of cycloplegia with 2 drops of 1% tropicamide.
4 octramine but not to 4-DAMP, pirenzepine, or tropicamide.
5 EK autorefractor with administration of 0.5% tropicamide.
6 ropicamide, or 1% hydroxyamphetamine / 0.25% tropicamide.
7 rect MSNs mediate the antiakinetic action of tropicamide.
9 illary mydriasis with phenylephrine 2.5% and tropicamide 1% administered via standard eye drops compa
10 re randomized to either cyclopentolate 1% or tropicamide 1% in the first visit with autorefraction me
11 .In all groups oxybuprocain 0.4%, cocain 4%, tropicamide 1%, phenylephrine 10%, diclophenac 0.1% alon
13 rinic antagonists atropine, scopolamine, and tropicamide (1-2 microM) caused substantial decreases of
19 rated that an ophthalmic spray containing 1% tropicamide and 2.5% phenylephrine was non-inferior to t
20 Provocative testing with mydriatic agents (tropicamide and atropine 1%) caused significant increase
23 eye followed 1 minute later by 1 drop of 1% tropicamide and then a second drop of 1% tropicamide 4 t
24 images were ungradable, reflex dilation (1% tropicamide) and mydriatic photography were performed fo
28 topical, ocular, ophthalmic, phenylephrine, tropicamide, cardiovascular effect, side effect, blood p
34 ll subjects underwent pupil dilation with 1% tropicamide eye drops at the baseline visit, before any
35 SE after instillation of cyclopentolate and tropicamide in both eyes was 1.07+/-5.2 and 0.96+/-5.1,
37 lts from the open field assay indicated that tropicamide increases activity in both the WT and KO mic
39 , 8.1) > hexahydrosiladiphenidol (M3, 8.0) > tropicamide (M4, 6.4) > pirenzepine (M1, 6.1) > methoctr
41 upil dilation with 2.5% phenylephrine and 1% tropicamide, ocular alignment was remeasured in primary
42 nstillation of either 2.5% phenylephrine, 1% tropicamide, or 1% hydroxyamphetamine / 0.25% tropicamid
44 mal human volunteers underwent dilation with tropicamide, phenylephrine, and a combination of the two
45 M1 and M4 mAChR antagonists telenzepine and tropicamide, respectively, were tested in the same model
46 than that of pirenzepine, methoctramine, and tropicamide to inhibit the contractile response to musca
47 ss this hypothesis we used an M4 antagonist, tropicamide, to reduce the activity through the M4 mAChR
48 rom the marble-burying assay have shown that tropicamide treatment resulted in a decreased number of
50 erence between DeltaSE of cyclopentolate and tropicamide was found statistically significant at 0.11+
51 The greatest effect of cyclopentolate and tropicamide was in hyperopic eyes with DeltaSE of 1.54+/
54 and its effects were compared with those of tropicamide, with and without dapiprazole in a double-ma