ライフサイエンスコーパス: truss

1 ally actuated by the rotation of interlinked trusses.

2 t, a COOH-terminal-deletion mutant of TRUSS (TRUSS(1-723)) was found to inhibit NF-kappaB activation

3 biquitous scaffolding and signaling protein (TRUSS), a 90.1 kDa TNF-R1-associated scaffolding protein

4 biquitous scaffolding and signaling protein (TRUSS), a novel TNF-R1-interacting protein of 90.7 kDa.

5 Transient overexpression of TRUSS activated NF-kappaB and increased NF-kappaB activa

6 ography, we produced and characterized micro-truss and -shell structures made from alumina-polymer co

7 s of 6 nanometers, and complex high-strength trusses and lattice structures with minimum feature size

8 glected aspects of hernia management such as trusses, antibiotic cover, return to work and activity,

9 th enforced physical constraints, to predict truss architectures with fully tailored anisotropic stif

10 8) and central (residues 249-440) regions of TRUSS are required to form a docking interface that supp

11 c stress within the metamaterial compared to truss-based morphologies, leading to the earlier activat

12 TRPC4AP/TRUSS binds specifically to the Myc C terminus and promo

13 This structure was called the "octet truss" by Buckminster Fuller.

14 co-immunoprecipitation assays revealed that TRUSS can interact with TRADD, TRAF2, and components of

15 terface, and (iii) the assembly of homomeric TRUSS complexes may contribute to its role in TNF-R1 sig

16 the successful and steady enrichment of the truss design space, the inverse design has remained a ch

17 Finally, we found that TRPC4AP/TRUSS expression is strongly down-regulated in most canc

18 Via extensive simulations based on a dynamic truss-frame model, this study shows that an origami stru

19 e entire N-terminal half (residues 1-440) of TRUSS, (ii) the binding interface for TNF-R1 is closely

20 ce, highly branched inflorescences and fruit trusses, indeterminate shoots in place of determinate fl

21 eletion mutagenesis of TNF-R1 indicated that TRUSS interacts with both the membrane-proximal region a

22 by crystallography, the periodic assembly of trusses into architected materials has enjoyed popularit

23 rmous design space of topologically distinct truss lattices and accurately identifies architectures m

24 ed as nodes and beams to construct nanosized truss lattices.

25 We find a truss-like arrangement of cadherins that resembles the c

26 The stretchability was enabled by a truss-like microstructure that was self-assembled under

27 These findings suggest that TRUSS may serve as a scaffolding protein that interacts

28 Through the open and close of truss meshes, this large scale biocrystal microstructure

29 roperties, using machine learning to enhance truss metamaterials.

30 We demonstrate this principle on a truss modular robot platform.

31 TRUSS mRNA was ubiquitously expressed in mouse tissues b

32 Previous work has focused on truss networks and anisotropic honeycombs, neither of wh

33 olumetric transformations, Variable Geometry Trusses offer rich morphing capabilities by coordinating

34 optimization, heat transfer enhancement and truss optimization.

35 s well as known structures such as the octet-truss or the Kelvin cell and show how crystallographic s

36 ined a challenge: While predicting effective truss properties is now commonplace, efficiently identif

37 In addition, the N-terminal region of TRUSS (residues 1 to 440) contains sequences that permit

38 Previous work introduced the metatruss, a truss robot that simplifies control by grouping actuator

39 ak allele of the florigen gene SINGLE FLOWER TRUSS (SFT) and two mutations affecting a bZIP transcrip

40 cription factors by activating SINGLE FLOWER TRUSS (SFT) in the leaves and the MADS-Box gene APETALA1

41 of the florigen-encoding gene SINGLE FLOWER TRUSS (SFT) in the leaves.

42 and how the FLOWERING LOCUS T/SINGLE FLOWER TRUSS (SFT)-like and TERMINAL FLOWER1/SELF-PRUNING (SP)-

43 a 2D triangular lattice and a 3D octahedral truss, showing excellent agreement between experimental

44 we firstly introduce the constraints that a truss structure is simultaneously compatible at multiple

45 the DNA-guided assemblies by using a simple truss-structure model.

46 hen, we solve for the design of multi-stable truss structures, named multi-compatible structures in t

47 By assembling TCO-based truss structures, we experimentally verify the tunable n

48 esistance such as in bridges, buildings, and truss structures.

49 TRPC4AP/TRUSS suppresses Myc-mediated transactivation and transf

50 ystematically investigate (i) the regions of TRUSS that interact with TNF-R1 and TRAF2 and (ii) the a

51 own about the mechanism of interaction among TRUSS, TNF-R1, and TRAF2.

52 ith TNF-R1 and TRAF2 and (ii) the ability of TRUSS to self-associate to form higher-order complexes.

53 he FE simulation was built using a series of trusses to reconstruct the muscles.

54 contrast, a COOH-terminal-deletion mutant of TRUSS (TRUSS(1-723)) was found to inhibit NF-kappaB acti

55 l, subfamily C, member 4-associated protein)/TRUSS (tumor necrosis factor receptor-associated ubiquit

56 -immunoprecipitation experiments showed that TRUSS was constitutively associated with unligated TNF-R

57 on repeating unit cells composed of webs or trusses, when made from materials of high elastic stiffn

58 with beam count, limiting implementations to trusses with only a handful of beams or to designs where