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1 hway enables nicotinamide acquisition by the tubercle bacilli.
2 olic acids for the long-term survival of the tubercle bacilli.
3 vely multiplying and nonactively multiplying tubercle bacilli.
4 compared with phagosomes encompassing killed tubercle bacilli.
5 inducing nonreplicating persistence (NRP) of tubercle bacilli.
6 the proof of concept for genetic exchange in tubercle bacilli.
7 emergence of multidrug resistance strains of tubercle bacilli.
8 with nanomolar bactericidal activity against tubercle bacilli.
9 at NAD(+) starvation is a cidal event in the tubercle bacilli and confirms that enzymes common to the
10 serves a necessary biological function(s) in tubercle bacilli and may contribute to the M. tuberculos
11 o oral regimen further lowers lung burden of tubercle bacilli and significantly more rapidly achieves
12 C (an enzyme naturally expressed/secreted by tubercle bacilli) as a marker and the design of BlaC-spe
13 ric oxide-dependent killing of intracellular tubercle bacilli, but in human monocytes and alveolar ma
14 s for beta-lactamase, an enzyme expressed by tubercle bacilli, but not by their eukaryotic hosts, to
15                      Incomplete clearance of tubercle bacilli frequently results in disease relapse,
16                                  Recovery of tubercle bacilli from sputum, tissue, or body fluid is t
17                     In liquefied caseum, the tubercle bacilli grow extracellularly for the first time
18 ow growth rate and genetic intractability of tubercle bacilli has hindered progress toward understand
19 ta imply that multiplying and nonreplicating tubercle bacilli have different antigen compositions.
20                                              Tubercle bacilli have found a novel way to coat themselv
21 able to control the accumulation of virulent tubercle bacilli in cocultured syngeneic peritoneal macr
22 produced by aerosolized virulent bovine-type tubercle bacilli in commercially available New Zealand w
23 report describes a model based on culture of tubercle bacilli in deep liquid medium with very gentle
24 m bovis BCG were able to limit the growth of tubercle bacilli in the lung and spleen after a virulent
25  differences in the growth rates of virulent tubercle bacilli in the lungs.
26 e diversity and transmission dynamics of the tubercle bacilli in the Republic of Korea.
27  for screening drugs for the ability to kill tubercle bacilli in their different stages of nonreplica
28 eamides, combined with their ability to kill tubercle bacilli, indicates great potential for translat
29 d other members of the animal lineage of the tubercle bacilli is very rare.
30 ylogenetic markers in a larger collection of tubercle bacilli (n = 125), (ii) by evaluating additiona
31 evidence of chromosomal DNA transfer between tubercle bacilli of the early-branching Mycobacterium ca
32                             The evolution of tubercle bacilli parallels a route from environmental My
33 eum, a critical lesion site where persisting tubercle bacilli reside.
34 the rate of decrease in the concentration of tubercle bacilli sputum during the initial days of thera
35                 It was determined that prior tubercle bacilli test results were negative, and thus th
36 rved in all virulent laboratory and clinical tubercle bacilli tested and was deleted only from substr
37  develop tests based on products secreted by tubercle bacilli that are strictly associated with viabi
38 s bacterial enzyme probe to detect and image tubercle bacilli that demonstrates REF is likely to be u
39 tations and granulomatous lesions containing tubercle bacilli throughout the meninges, all of which w
40 ts that it may play a role in the ability of tubercle bacilli to adapt to host defenses and persist d
41    The MHC class I presentation requires the tubercle bacilli to be viable, and it is dependent upon
42 ng persistence associated with adaptation of tubercle bacilli to hypoxia in vitro.
43 rsistence, is responsible for the ability of tubercle bacilli to lie dormant in the host for long per
44 sponse to nutrient starvation, thus enabling tubercle bacilli to restrict growth and shut down metabo
45 ients suggests that the hypoxic shiftdown of tubercle bacilli to the NRP state that occurs in vitro,
46 he data indicate that antigen composition of tubercle bacilli varies with stage of infection and that
47                     In contrast to controls, tubercle bacilli were not conveyed to draining lymph nod
48 croglia were the principal cells infected by tubercle bacilli, which elicited robust amounts of sever
49 and the proximal cause of cellular damage in tubercle bacilli will make it applicable to other pathog