ライフサイエンスコーパス: tubular

1 d by low molecular weight proteinuria, renal tubular acidosis (RTA), aminoaciduria, and hypercalciuri

2 enic kidney stone disorders, including renal tubular acidosis with deafness, Bartter syndrome, primar

3 s been implicated in tumor metastasis, renal tubular acidosis, and osteoporosis.

4 rum pH, consistent with a diagnosis of renal tubular acidosis.

5 leading to mild metabolic alkalosis ("renal tubular alkalosis").

6 eloping collecting ducts results in proximal tubular and collecting duct cysts, respectively.

7 Tubular and glomerular functional parameters were not si

8 ulates viral protein synthesis and exists as tubular and non-tubular forms in infected cells, but how

9 While flat, tubular, and hollow nontubular engineered organs have al

10 f follow-up, including 1 in a patient with a tubular aortic diameter of 50 mm, but none in patients w

11 Sbf localizes to tubular, apical surfaces and associates with Rab35, wher

12 duct-on-a-chip that phenocopies not only the tubular architecture of the bile duct in three dimension

13 ume per glomerulus, and mean cross-sectional tubular area to measure nephron size; and calculations f

14 rejection (67.4%); interstitial fibrosis and tubular atrophy (14.4%); BK virus nephropathy (BKVAN) 9.

15 y luminal stenosis and interstitial fibrosis/tubular atrophy (IF/TA) of the cortex.

16 limeter, and degree of interstitial fibrosis/tubular atrophy (IFTA) were independently associated wit

17 l glomerulosclerosis, interstitial fibrosis, tubular atrophy and arteriolosclerosis.

18 glomerulopathy, fibrous intimal thickening, tubular atrophy, and interstitial fibrosis scores were a

19 al glomerulosclerosis, interstitial fibrosis/tubular atrophy, artery luminal stenosis, and arteriolar

20 aracteristics included interstitial fibrosis/tubular atrophy, larger cortical nephron size (but not n

21 lic nanotubes, which subsequently arrange as tubular bilayers to form giant nanosheets in the mesosca

22 ct delta(34)S and delta(56)Fe values in: (a) tubular biogenic structures and trails (delta(34)S ~ +40

23 age in VLPs and blocks conversion of VLPs to tubular CA assemblies, (15)N and (13)C ssNMR chemical sh

24 compartment on syntaxin-6-positive vesicular/tubular carriers that depend on Rab10 for their fusion w

25 etwork (TGN) has been shown to occur through tubular carriers that emanate from the TGN or through ve

26 es tubule-macrophage crosstalk by supporting tubular CCL2 expression and regulating macrophage phagoc

27 tem cell marker TRA-1-60 and TNFR2, but only tubular CD133(+) cells express proximal tubular markers

28 ice, deletion of Tnfr1 significantly reduced tubular CD44 and annexin two expression, as well as infl

29 odifying factor (Bmf)-induced renal proximal tubular cell (RPTC) apoptosis and loss in diabetic mice.

30 I group demonstrated significantly increased tubular cell apoptosis and caspase-9 expression, whereas

31 In contrast, the CI+Txp group had tubular cell apoptosis associated with expression of cas

32 ith its receptor, integrin-beta1, to inhibit tubular cell cycle arrest and apoptosis in in vivo and i

33 the pathways involved in the development of tubular cell injury and death before and after transplan

34 ltures of cilia-deficient or STAT3-deficient tubular cell lines.

35 04 significantly reduced proteinuria-induced tubular cell mitochondrial damage, suggesting that impro

36 lesions were largely prevented and proximal-tubular-cell turnover was normalized.

37 ir RPTCs and immortalized rat renal proximal tubular cells (IRPTCs) were also studied.

38 bonucleoprotein F (Hnrnpf) in renal proximal tubular cells (RPTCs) suppresses angiotensinogen (Agt) e

39 ng therapeutic strategy for protecting renal tubular cells against cisplatin-induced AKI by enhancing

40 Primary tubular cells and macrophages from SerpinB2 knockout and

41 vitro, SCFAs modulated inflammation in renal tubular cells and podocytes under hyperglycemic conditio

42 In mice treated with SS1P, proximal tubular cells are damaged and albumin in the urine is in

43 e reciprocal interactions between immune and tubular cells are not well characterized.

44 Mouse proximal tubular cells exposed to high glucose showed significant

45 jury model, and primary cultures of isolated tubular cells in a hypoxia-reoxygenation model.

46 associated with a beneficial effect on renal tubular cells in AKI.

47 to reduce phosphate uptake in human proximal tubular cells in vitro.

48 tochondrial function, we used human proximal tubular cells in vitro.

49 ty for autophagy in both podocytes and renal tubular cells is markedly impaired in type 2 diabetes, a

50 n of floxed megalin/LRP2 alleles in proximal tubular cells of cystinotic mice was achieved by a Cre-L

51 ctin-1 (Gal-1), which is highly expressed in tubular cells of kidneys of type 1 and type 2 diabetic m

52 e kidney followed by degradation in proximal tubular cells of the kidney.

53 hese results suggest that DsbA-L in proximal tubular cells promotes TIF via activation of the Hsp90 /

54 SerpinB2 knockout tubular cells showed significantly reduced expression of

55 jor role in the crosstalk between immune and tubular cells that shapes disease expression.

56 response signature and fibrotic signature in tubular cells were each associated with failure to respo

57 bumin induced features of ER stress in renal tubular cells with ATF3/ATF4 activation.

58 ough the glomerulus, is taken up by proximal tubular cells, and transferred to lysosomes.

59 In this process, tubular cells, in coordination with macrophages, overgre

60 Cultured senescent tubular cells, kidneys of aged mice, and renal stress mo

61 ors might modulate glucose influx into renal tubular cells, thereby regulating the metabolic conditio

62 In cultured murine proximal tubular cells, treatment with PCI34051 or specific HDAC8

63 ins could be a source of cystine in proximal tubular cells, we used a mouse model of cystinosis in wh

64 ormations and crystal deposition in proximal tubular cells.

65 nd CXCL10 in polycystic kidneys and cultured tubular cells.

66 ale morphology of the primary cilium, a tiny tubular cellular structure (~2-6 mum long and 200-300 nm

67 fast proton permeation to ~5- angstrom-wide tubular channels that perforate micas' crystal structure

68 This suggests that tubular clearance of secretory solutes provides addition

69 nd faster analyte dispersion across the open-tubular column (OTC) i.d..

70 The picoLC employs a 2-mum-i.d. open tubular column to reduce the sample input needed to grea

71 For an open tubular column, the permissible illuminated length can b

72 delivered into gas chromatography (GC) open-tubular columns (OTC, 0.18 mm i.d., 20 m long, ~0.2 mum

73 ured under flow had more mature podocyte and tubular compartments with enhanced cellular polarity and

74 Actin-based tubular connections between cells have been observed in

75 Furthermore, the generation of uniform tubular constructs remains a challenge.

76 celerate cystic progression and investigated tubular crystal deposition.

77 perturbations in Notch signaling cause renal tubular cysts remains unclear.

78 methyl analog attenuates proteinuria-induced tubular damage by modulating mitochondrial function.

79 reatment prominently ameliorated acute renal tubular damage in mice exposed to cisplatin insult, asso

80 can protect tubules from proteinuria-induced tubular damage via anti-inflammatory and antioxidative s

81 eresting for chemical speciation of abnormal tubular deposits and calcification.

82 We also observed evidence of proximal tubular dextran reuptake.

83 extraction revealed interstitial expansion, tubular dilation and atrophy, and glomerular size variab

84 19 develop a wide spectrum of glomerular and tubular diseases.

85 Reticulon (Rtn) proteins shape tubular domains of the endoplasmic reticulum (ER), and i

86 Mitochondria are tubular double-membrane organelles essential for eukaryo

87 However, the role and regulation of proximal tubular DsbA-L for renal tubulointerstitial fibrosis (TI

88 int was to assess the prevalence of proximal tubular dysfunction and the correlation between elevated

89 ombined with aminoaciduria suggests proximal tubular dysfunction.

90 Tubular ectasia (TE) was shown to be associated with UOC

91 Endogenous WDR44 labels a subset of tubular endosomes, which are closely aligned with the ER

92 Tubular epithelia are a basic building block of organs a

93 Analysis of isolated tubular epithelia by FACS from bigenic SLC34a1-CreERt2;

94 53 and subsequent pro-apoptotic signaling in tubular epithelia of cisplatin-treated mice, leading to

95 culature, alongside renal tubules comprising tubular epithelia of different nephron segments.

96 r patterns of lesion growth were observed in tubular epithelia of the liver and lung; this finding id

97 ormal kidneys, JAK2 expression is limited to tubular epithelial and vascular cells with lesser staini

98 ubulointerstitium; dh404 markedly suppressed tubular epithelial cell damage in the renal interstitium

99 Renal tubular epithelial cells (RTECs) perform the essential f

100 otoxicity, and inflammatory insults to renal tubular epithelial cells (RTECs), resulting in the onset

101 survival and proliferation, is expressed by tubular epithelial cells (TECs), and binds to the cFMS r

102 ithelial-specific genetic ablation of Myc in tubular epithelial cells ameliorated fibrosis and restor

103 Cell proliferation of both tubular epithelial cells and interstitial cells is reduc

104 UUO), HDAC8 was primarily expressed in renal tubular epithelial cells and time-dependently upregulate

105 1 treatment also reduced the number of renal tubular epithelial cells arrested at the G2/M phase of t

106 r 2 (TNFR2) is strongly upregulated on renal tubular epithelial cells by acute cell-mediated rejectio

107 Tubular epithelial cells in normal kidney organ cultures

108 ripts for the IL-27RA and the IL-17RA in the tubular epithelial cells of patients with renal fibrosis

109 Tubular epithelial cells showed strong production of per

110 In vitro exposure of human renal proximal tubular epithelial cells to C5a led to altered mitochond

111 Murine renal tubular epithelial cells were studied in response to in

112 dneys undergoing ACR represent proliferating tubular epithelial cells with TNFR2-induced stem cell ma

113 injury (AKI) with adaptive proliferation of tubular epithelial cells, but repair can also lead to fi

114 ndMT, resulted in increased Myc abundance in tubular epithelial cells, enhanced glycolysis, and suppr

115 ey injury induces cell cycle arrest in renal tubular epithelial cells, resulting in the secretion of

116 ed protein p62/SQSTM1 in cystinotic proximal tubular epithelial cells, we performed a high-throughput

117 The smallest crystals were endocytosed by tubular epithelial cells.

118 stress-responsive protective gene in kidney tubular epithelial cells.

119 ury by enhancing autophagy in renal proximal tubular epithelial cells.

120 expression and promotes cell cycle entry of tubular epithelial cells.

121 ct of oxidative DNA damage) in podocytes and tubular epithelial cells.

122 nterstitium and in TNFalpha-treated proximal tubular epithelial cells.

123 nd shRNA knockdown strains in renal proximal tubular epithelial cells.

124 Hypoxia significantly increased HE4 in renal tubular epithelial cells.

125 akage, reduced tissue hypoxia, and preserved tubular epithelial health and function.

126 jury (AKI) is characterized by injury to the tubular epithelium that leads to the sudden loss of rena

127 The presence of viral particles in the renal tubular epithelium that were morphologically identical t

128 fetal kidney mesenchyme differentiates into tubular epithelium will improve our understanding of the

129 ys induces accumulation of fibrinogen within tubular epithelium.

130 three-way junctions can be stabilized and a tubular ER network can be maintained.

131 lve from dysfunctions of pre-autophagosomes, tubular ER, mature autophagosomes, and the ubiquitin pro

132 whereas flat areas of columnar mucosa in the tubular esophagus can be treated with mucosal ablation.

133 is an absence of columnar epithelium in the tubular esophagus on high-definition white-light endosco

134 (without residual columnar epithelium in the tubular esophagus) should not warrant additional ablatio

135 We further show that proximal tubular expression of PIgR becomes widespread during kid

136 ohoop constituents, which self-assemble in a tubular fashion via a combination of arene-pefluoroarene

137 IgA levels also correlated to the degree of tubular fibrosis, plasma creatinine, and urea levels.

138 different positions along the length of the tubular flow crystallizer correspond to different reacti

139 , and chemistry agnostic computer-controlled tubular flow reactor.

140 itro luciferase assays show that the NS1 non-tubular form upregulates BTV mRNA translation, whereas z

141 vents kidney atrophy in UUO kidneys, retains tubular formation, and reduces TRPM7 expression to norma

142 The two tubular forms are different helical assemblies of the sa

143 tein synthesis and exists as tubular and non-tubular forms in infected cells, but how tubules assembl

144 lesser tubular gluco-toxicity, may preserve tubular function and glomerular filtration rate in the l

145 y be influenced by changes in glomerular and tubular function in varying patterns, which would limit

146 tial for normal kidney development and renal tubular function.

147 Incineration experiments using a tubular furnace and subsequent ICP-MS (ICP Mass Spectrom

148 cement of the hitchhiking initiation rate in tubular geometries such as those found in fungal hyphae.

149 It was observed that the tubular geometry had a strong influence on the photocurr

150 hanges in transformed cells and the existing tubular geometry.

151 cal oxygenation, which, together with lesser tubular gluco-toxicity, may preserve tubular function an

152 nce of FITC-inulin to examine the effects of tubular glucose on NO generation at the macula densa, TG

153 ssed at the macula densa; in the presence of tubular glucose, SGLT1 inhibits TGF and NO generation, b

154 Tubular growth is associated with the development of a s

155 or of FoxM1 because GSK3 inhibition or renal tubular GSK3beta gene deletion significantly increased F

156 rular filtration, while assessment of kidney tubular health has notably been absent.

157 we highlight promising biomarkers of kidney tubular health that have strong underpinnings in the pat

158 a and hemosiderinuria associated with marked tubular hemosiderin deposition and HO-1 induction, while

159 tive oxygen species in human kidney proximal tubular (HK-2) cells that were exposed to recombinant su

160 ng fibrosis, mesangial matrix expansion, and tubular hypertrophy were observed in 0-copy and A71915-t

161 he identified high prevalence of subclinical tubular impairment also highlights the importance of exp

162 acute kidney injury (AKI) by promoting renal tubular inflammation after ischemia and reperfusion (IR)

163 ulointerstitial lesions were frequent: acute tubular injury (94%), tubulitis (82%), tubular rupture (

164 Features of acute tubular injury (ATI) in KTX biopsy may be informative bu

165 lites were measured as well as biomarkers of tubular injury (kidney injury molecule-1 [KIM-1] and neu

166 Biomarkers of tubular injury (such as KIM-1), repair (such as YKL-40),

167 of TNF as these changes are absent in acute tubular injury .

168 A dehydrogenase)], and a marker of proximal tubular injury [KIM-1 (kidney injury molecule-1)/Hepatit

169 rols, whereas knockout mice showed increased tubular injury and deteriorated renal function.

170 Deficiency of Gdf15 exacerbated acute tubular injury and enhanced inflammatory responses.

171 there was a consistent pattern of increased tubular injury and oxidative stress over time, which hav

172 ogical mechanisms, to reverse glomerular and tubular injury at the cellular and gene expression level

173 mization WRF was unrelated to baseline renal tubular injury biomarkers ( r=0.14; P=0.17).

174 Increase in renal tubular injury biomarkers was associated with a higher i

175 tration in situations in which some level of tubular injury is inevitable, such as following a kidney

176 s indicate that MIOX's upregulation in acute tubular injury is mediated by oxidant and ER stress.

177 cisplatin-induced DNA damage and exacerbates tubular injury through the upregulation of p53-dependent

178 However, the degree of acute tubular injury was often less severe than predicted for

179 days after transient renal pedicle clamping, tubular injury was similar to wild type, although accumu

180 ft biopsy samples with ACR compared to acute tubular injury without rejection or pretransplant "norma

181 d phthalates on measures of kidney function, tubular injury, and oxidative stress over time in a coho

182 he clinical and molecular subtyping of acute tubular injury, and the role of non-invasive biomarkers

183 al frequent findings included renal proximal tubular injury, focal pancreatitis, adrenocortical hyper

184 tem, we assayed plasma biomarkers related to tubular injury, inflammation, and fibrosis (KIM-1, TNFR-

185 disease, characterized by crystal deposits, tubular injury, inflammation, and fibrosis.

186 AKI is characterized by tubular injury, inflammation, and vascular impairment.

187 ficantly reducing serum creatinine and urea, tubular injury, neutrophil and macrophage infiltration,

188 exacerbated cisplatin-induced DNA damage and tubular injury, thereby increasing mortality.

189 autopsy histologic evaluation revealed acute tubular injury, which was typically mild relative to the

190 sufficient to prevent the cisplatin-induced tubular injury.

191 is, and 4 were diagnosed with isolated acute tubular injury.

192 inant finding correlating with AKI was acute tubular injury.

193 ted rejection, cortical infarction, or acute tubular injury.

194 ugments growth, maturation, and longevity of tubular intestinal enteroids.

195 Tubular isometric vacuolization observed with light micr

196 promoting mutual proliferation, formation of tubular-like structures in the former and maintenance of

197 endothelial cell migration and formation of tubular-like structures.

198 portant in limiting persistent inflammation, tubular loss, and renal fibrosis after ischemic acute ki

199 ent the backflow of reabsorbed sodium to the tubular lumen along a steep electrochemical gradient.

200 kflow of reabsorbed solutes and water to the tubular lumen, as well as in coupling paracellular and t

201 ria within the glomerular capsular space and tubular lumens.

202 rystals into the interstitium, restoring the tubular luminal patency; this was followed by degradatio

203 docin mRNA:creatinine ratio: UPPod:CR) and a tubular marker (Urinary pellet aquaporin 2:creatinine ra

204 h tenofovir alafenamide (TAF) improves renal tubular markers in HIV-infected individuals but the impa

205 only tubular CD133(+) cells express proximal tubular markers megalin and aquaporin-1.

206 peripheral glomeruli, and decreased proximal tubular mass in the outer cortex.

207 e-by membranes, early endocytic vesicles, or tubular membrane structures.

208 NAD(+)-dependent maintenance of renal tubular metabolic health may also attenuate long-term pr

209 A tubular micromotor with spatially resolved compartments

210 ationally model the squeezing of the largely tubular mitochondrion and show that proteins and conical

211 they retain stable separation performance in tubular modules.

212 Morphotype 2 displays more elongated, tubular morphology.

213 Urinary pellet podocyte and tubular mRNA markers were increased in all diabetic grou

214 glomerulotubular balance, such that proximal tubular Na+ reabsorption varies proportionally to the si

215 BK virus nephropathy (BKVAN) 9.9%; and acute tubular necrosis (ATN with i-INT) in 5.9% of cases.

216 diagnostic groups-SAGN, primary IgAN, acute tubular necrosis (ATN) and normal kidney (baseline trans

217 MR, acute cellular rejection (ACR), or acute tubular necrosis (ATN).

218 ed with severe COVID-19 in China found acute tubular necrosis in the kidney, a few patient reports ha

219 od-perfused kidneys had vastly reduced acute tubular necrosis scores and degrees of terminal deoxynuc

220 opsy samples showed varying degrees of acute tubular necrosis, and one patient had associated widespr

221 Acute tubular necrosis, apoptosis, urinary kidney damage marke

222 emic AKI with significantly attenuated renal tubular necrosis, inflammation, and apoptosis when compa

223 zed patients with AKI and COVID-19 was acute tubular necrosis.

224 SYT7 localizes to an internal membrane tubular network within the peri-active zone, but does no

225 lnp2-1 mutant cells, the ER becomes a dense tubular network.

226 embrane organelles often form highly dynamic tubular networks interacting with many cellular structur

227 talluria due to urinary acidification caused tubular obstruction, inflammation, and interstitial fibr

228 However, whether the ER becomes more tubular or more cisternal during mitosis is controversia

229 which range from shallow domes to elongated tubular or pearl-like structures.

230 tablishing and maintaining crista junctions, tubular or slit-like structures that connect the cristae

231 sive search on planar networks, motivated by tubular organelle networks in cell biology that contain

232 1,1'-biphenyl)]porphyrin affords a porphyrin tubular organic cage, PTC-1(2H).

233 lishment and maintenance are fundamental for tubular organs physiological functions.

234 t recapitulate the anatomy and physiology of tubular organs.

235 her, our studies suggest that renal proximal tubular P2X4 activation exacerbates ischemic AKI and pro

236 The tubular penetration of rhodamine B-combined CPNE7 was co

237 This is reminiscent of the well-known "tubular pinch effect," which arises from inertial effect

238 is located near domains that interact with T-tubular plasma membrane, where LTCCs are housed.

239 Microtubules are tubular polymers with essential roles in numerous cellul

240 samples containing mixtures of spherical and tubular polymersomes, or pure samples of tubesomes, of p

241 These characteristics together with tubular presence of low molecular mass dextran provide c

242 The nanochannels possess a tubular profile 70 nm in nominal diameter and fabricated

243 Previously described markers of renal tubular progenitor cells were analyzed using immunohisto

244 elongated within members of a collection of tubular protein nanoreactors, which contained cysteine r

245 ar filtration rate, glomerular protein leak, tubular protein uptake via endocytosis and PT water reab

246 model to explain how loss of OCRL results in tubular proteinuria as well as the other commonly observ

247 A second class of tubular protrusion, which we term cell-substrate protrus

248 aging reveals McTNs are dynamic, CD44-coated tubular protrusions containing microtubules and actin fi

249 We find tubular protrusions, which we classify as TMTs, in a pan

250 Melamine, which induces proximal tubular (PT) cell damage has a greater nephrotoxic effec

251 ury markers, and the activation of the intra-tubular RAS even in normotensive young adults.

252 c cystinosis had significantly lower percent tubular reabsorption of phosphate and fibroblast growth

253 two significant loci possibly related to its tubular reabsorption, SLC6A19, and its production, ERO1A

254 rier, albuminuria, and the oxygen demand for tubular reabsorption.

255 emonstrate that FoxM1 is important for renal tubular regeneration following AKI and that GSK3beta sup

256 factors and cell proliferation in vitro, and tubular regeneration in mouse kidneys after AKI.

257 Proper tubular regeneration is essential to prevent progression

258 lated, non-reactive RE pupil, clear lens and tubular remnant of HA containing blood cells in its lume

259 Chronic exposure of tubular renal cells to high glucose contributes to tubul

260 he activation of both the systemic and intra-tubular renin angiotensin systems (RAS), which are in tu

261 ad box family member transcription factor in tubular repair after AKI.

262 tly increased FoxM1 expression, and improved tubular repair and renal function.

263 n following AKI and that GSK3beta suppresses tubular repair by inhibiting FoxM1.

264 hiostrepton treatment abolished the improved tubular repair in GSK3beta knockout mice following AKI.

265 Impaired tubular reuptake of glycation free adducts by lysine and

266 acute tubular injury (94%), tubulitis (82%), tubular rupture (62%), giant cell reaction (60%), and co

267 Renal tubular secretion is an active efflux pathway for the ki

268 Although proximal tubular secretion is the primary mechanism of kidney dru

269 ur results suggest that mIBG undergoes renal tubular secretion mediated by hOCT2 and hMATE1/2-K, and

270 ed pharmacokinetic study to compare GFR with tubular secretory clearance for predicting kidney drug e

271 ever, tight linkage between GFR and proximal tubular secretory clearance in stable outpatients provid

272 al significance of the kidney's clearance of tubular secretory solutes is uncertain.

273 nd eventually differentiate into the various tubular segments of the nephron.

274 The heart of rnf2 mutants shows a tubular shaped morphology and to further understand the

275 This cell bridge was tubular shaped with an average length and width of 700 a

276 ated with interesting optical properties and tubular-shaped cavities are able to recognize geometric

277 minent scientist Albert von Kolliker named a tubular-shaped piece of tissue found in the brains of al

278 However, the proximal tubular site of action of the sodium-glucose cotransport

279 um homeostasis is a major determinant of the tubular site of sodium retention in nephrotic mice.

280 did not increase renal medullary blood flow, tubular sodium reabsorption was not downregulated, and p

281 ple repressor of PPM1A, as conditional renal tubular-specific induction of TGF-beta1 in mice dramatic

282 igenic SLC34a1-CreERt2; R26tdTomato proximal tubular-specific reporter mice revealed that KU55933 upr

283 d the safety and efficacy of a biodegradable tubular stent (BTS) for preventing a pancreaticojejunost

284 This tubular strategy for viral spread and biparental transmi

285 which may be more appropriately termed the "tubular striatum" (TuS), is a neural system in which sen

286 w medulla, mineralized tubules, and sandwich-tubular structure) are created, and the role of its stru

287 d fiber spatial organization (spider web and tubular structure).

288 and boron nanodroplets incorporate into the tubular structure.

289 and RUSC2 colocalize with EGFR in vesicular/tubular structures and at the Golgi compartment.

290 The experimental evidence suggests that the tubular structures are a consequence of the self-scrolli

291 y on the functionality of the glomerular and tubular structures in these tissues.

292 predominantly of variably dilated, branching tubular structures with pseudo-apocrine snouts, and eith

293 an alternative way for fabricating polymeric tubular structures, and will expand the toolbox for the

294 n phospholipid recognition and biogenesis of tubular structures.

295 ubunit (CTB) enabled the ordered assembly of tubular supra-molecular complexes.

296 own, is able to form complex interconnected tubular systems in vitro, reminiscent of t-tubule system

297 odes for cubilin, an intestinal and proximal tubular uptake receptor containing 27 CUB domains for li

298 e contributions of glomerular filtration and tubular uptake to urinary protein excretion, we develope

299 We recently proposed that the tubular-vesicular recycling endosome membranes were a co

300 anges in the presence of PSP-2, with thicker tubular walls and an overall decreased meshes area.