戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 ys induces accumulation of fibrinogen within tubular epithelium.
2 ular endothelium but A-subtypes II-III/IV on tubular epithelium.
3  accumulate in and damage the proximal renal tubular epithelium.
4  cytoskeleton and junction remodeling in the tubular epithelium.
5 ered to Bowman's capsule and infiltrated the tubular epithelium.
6 docytes and the brush border of the proximal tubular epithelium.
7 l component C3, principally expressed by the tubular epithelium.
8 g the structural and functional integrity of tubular epithelium.
9 s and might facilitate the recovery of renal tubular epithelium.
10 nd were intimately associated with the renal tubular epithelium.
11 y of nonhematologic tissues, including renal tubular epithelium.
12 rafts is isometric vacuolization of proximal tubular epithelium.
13 n and proliferate to re-establish the normal tubular epithelium.
14 ion is predominantly found in the collecting tubular epithelium.
15 14% that of wild-type mice and is limited to tubular epithelium.
16  graft functional elements such as the renal tubular epithelium.
17 coronary and intrarenal vessels and in renal tubular epithelium.
18  primitive inner ear cells, and seminiferous tubular epithelium.
19  increased in renal glomeruli and induced in tubular epithelium.
20 pression remains in most, if not all, of the tubular epithelium.
21 ithrombin was found only within the proximal tubular epithelium.
22 and fewer cells than normal are converted to tubular epithelium.
23 s and on the apical surface of the trachea's tubular epithelium.
24 3p localized to the endothelium and proximal tubular epithelium.
25  epithelium (11 of 11 cases), proximal renal tubular epithelium (5 of 5 cases), colonic ganglion cell
26                                 In the renal tubular epithelium, A(1), A(2A), and A(3) receptors have
27 unctionally significant replenishment of the tubular epithelium after ischemia.
28 xclusively in the degenerated, dilated renal tubular epithelium after unilateral ureteral obstruction
29 esults suggest that TGFbeta signaling in the tubular epithelium alone is sufficient to cause acute tu
30 odel, activation of TGFbeta signaling in the tubular epithelium alone was sufficient to cause AKI cha
31 ion reveals increased Nox4 expression in the tubular epithelium also during obstructive nephropathy.
32 f pFhit immunoreactivity was confined to the tubular epithelium and absent in the glomeruli.
33 dextran-labeled cells were restricted to the tubular epithelium and excluded from the interstitium.
34 normal transition of induced mesenchyme into tubular epithelium and full growth and branching of the
35 aining was diminished in the nuclei of renal tubular epithelium and interstitium after obstructive in
36 butes to the maintenance and repair of renal tubular epithelium and may be a novel therapeutic target
37 hed podocytes crawling on the surface of the tubular epithelium and occasionally, in contact with per
38 tegration and necrosis of the renal proximal tubular epithelium and of the intestinal mucosal epithel
39                                    Damage to tubular epithelium and peritubular capillary endothelium
40 in mediating cell dedifferentiation of renal tubular epithelium and suggest that EMT is a multistep p
41 tes to renal IRI by a direct effect on renal tubular epithelium and that this effect is independent o
42 e mAb prevented the deposition of C3b on the tubular epithelium and the generation of systemic C3a af
43 chanistically interesting features in kidney tubular epithelium, and that somatic mutation may play a
44 asts, a loss of functional microvilli on the tubular epithelium, and varying degrees of chronic inter
45 ression of a complement inhibitor within the tubular epithelium appears to be a critical factor permi
46 lows for selective propagation of the mature tubular epithelium, as immature cells, stroma, and undes
47 cells in crescents, parietal epithelium, and tubular epithelium, as well as by infiltrating leukocyte
48 erentiation of the condensed mesenchyme into tubular epithelium, as well as the rate of growth and br
49 pha production specifically within the renal tubular epithelium attenuated the AKI and the increase i
50 in is expressed in the human embryonic renal tubular epithelium beginning on approximately day 75 to
51 2 activation and its nuclear accumulation in tubular epithelium, but it restored SnoN protein abundan
52 n DNA fragmentation and cell death in kidney tubular epithelium, but the endonucleases responsible fo
53 n of HIV-1-specific proviral DNA and mRNA in tubular epithelium cells, argue strongly for localized r
54 ry dickkopf-3 (DKK3), a stress-induced renal tubular epithelium-derived glycoprotein, has been identi
55 e with a targeted deletion for Alk3 in their tubular epithelium did not respond to therapy with THR-1
56 t of CD8 effectors that infiltrate the graft tubular epithelium during clinical rejection episodes, p
57 , the EOR rapidly develops into a convoluted tubular epithelium ending in a distal dilated tip.
58 ndings of increased synthesis of C3 in human tubular epithelium exposed to high concentrations of pro
59  was due, at least in part, to the rescue of tubular epithelium from lethal injury.
60                                     In renal tubular epithelium, glutaminase produces ammonia to buff
61 ased in rejected kidney, particularly in the tubular epithelium; however, enzymatic activity was sign
62  low expression of PAI-1 mRNA and protein in tubular epithelium in beta6(-/-) UUO kidneys, with incre
63 ta-catenin is induced predominantly in renal tubular epithelium in CKD, surprisingly, depletion of tu
64 of proliferating cell nuclear antigen in the tubular epithelium, inflammatory cell infiltrate, and ne
65                                    The renal tubular epithelium is a target for many toxicants.
66                                          The tubular epithelium is identified as a site capable of su
67                     However, its role in the tubular epithelium is not known.
68 Recently we have established that the kidney tubular epithelium is repaired by surviving epithelial c
69 ent morphogenesis and differentiation of the tubular epithelium lead to the establishment of a functi
70       We also found that its deletion in the tubular epithelium leads to enhanced TGF-beta1-Smad fami
71 intercalation of cells into the plane of the tubular epithelium maintained the normal tubular morphol
72 olycystin-1 from its basolateral location in tubular epithelium may alter critical pathways controlli
73 nzymatic conversion of tryptophan to NAD+ in tubular epithelium may contribute to adverse cellular an
74 ed that NRP mediates attachment to the renal tubular epithelium of Ca stone crystals through an elect
75 hat of mediating the attachment to the renal tubular epithelium of calcium stone crystals.
76 mmunostaining for nitrotyrosine localized to tubular epithelium of chronically rejected human renal a
77 ion of clusterin mRNA occurred mostly in the tubular epithelium of dilated, convoluted proximal tubul
78 Nlrp3 and Asc were highly expressed in renal tubular epithelium of humans and mice, and the absence o
79 in the renal vasculature and in the cortical tubular epithelium of kidneys exposed to I/R.
80 pression by podocytes (GEC) and the proximal tubular epithelium of rat kidney was confirmed.
81 tion induced Shh, predominantly in the renal tubular epithelium of the fibrotic kidneys.
82                                          The tubular epithelium of the kidney is susceptible to injur
83 ly for the formation of the undifferentiated tubular epithelium of the nascent pancreatic rudiment an
84 teopontin expression could be located to the tubular epithelium of the renal cortex.
85 t receptor 1-related protein y (Crry) in the tubular epithelium preceded activation of the alternativ
86 w that complement inhibitors targeted to the tubular epithelium protect against tubulointerstitial in
87 transgene expression was localized to kidney tubular epithelium rather than vascular endothelial cell
88        We propose a 2D mechanical model of a tubular epithelium resembling the early Drosophila embry
89         Conditional loss of Dnajb11 in renal tubular epithelium results in PC1 dosage-dependent kidne
90  borne by albumin into endocytosing proximal tubular epithelium results in the synthesis and release
91  PKD2 have been previously observed in renal tubular epithelium (RTE).
92  promoting proliferation within the proximal tubular epithelium supports therapeutic targeting of HIF
93 jury (AKI) is characterized by injury to the tubular epithelium that leads to the sudden loss of rena
94 The presence of viral particles in the renal tubular epithelium that were morphologically identical t
95  gastrointestinal mucosal and renal proximal tubular epithelium, the Gb3 receptor glycolipid of the r
96                    Increased exposure of the tubular epithelium to filtered protein is a proposed mec
97 cused on the transformation of the organized tubular epithelium to the myofibroblastic phenotype, a p
98  processed by urokinase, released from renal tubular epithelium, to generate active plasmin.
99 ion of RANTES was localized primarily to the tubular epithelium, underscoring a role for tubular cell
100 he transition of nascent epithelial cells to tubular epithelium was derived, helping to reconcile in
101                             The renal distal tubular epithelium was spared.
102 esponse in PVAN is partly coordinated by the tubular epithelium, whereas in TCMR, this may be control
103 seen by a profound increase in proliferating tubular epithelium, which coincided with increased hepat
104 t-AKI, and activates RA signaling in injured tubular epithelium, which in turn promotes alternatively
105                    In contrast, normal renal tubular epithelium, which poorly expresses DR3, is minim
106  fetal kidney mesenchyme differentiates into tubular epithelium will improve our understanding of the

 
Page Top