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1  and UBD in glomerulus; SNOR14B and MUC13 in tubulointerstitium).
2 y (glomeruli), as well as T-cell activation (tubulointerstitium).
3 rythropoietin-producing cells located in the tubulointerstitium.
4 ssion of CSE, localized to glomeruli and the tubulointerstitium.
5 lammation, and scarring in glomeruli and the tubulointerstitium.
6 c abnormalities of the kidney glomerulus and tubulointerstitium.
7 ls from apoptotic death in the glomeruli and tubulointerstitium.
8  membrane of the glomerulus and in the renal tubulointerstitium.
9 scents and in infiltrating leukocytes in the tubulointerstitium.
10 as well as by infiltrating leukocytes in the tubulointerstitium.
11 t a dose of 5 mg/kg did not affect the renal tubulointerstitium.
12 tion, affecting the microcirculation and the tubulointerstitium.
13  HIF-target genes with eGFR in glomeruli and tubulointerstitium.
14 f complement is rapidly activated within the tubulointerstitium after renal ischemia/reperfusion (I/R
15  an ameliorated inflammatory infiltration in tubulointerstitium and a favored M2-skewed macrophage po
16 ation, and ADAMTS5(+) cells were seen in the tubulointerstitium and glomeruli.
17 h high dose CsA showed increased Nox2 in the tubulointerstitium and greater Nox2, alpha-SMA, phosphor
18 appaB pathway were evaluated in the cortical tubulointerstitium and in cellular infiltrates using dig
19 zyme, was significantly increased in the AMR tubulointerstitium and in TNFalpha-treated proximal tubu
20 Upon SNx, extracellular TG2 deposited in the tubulointerstitium and peri-glomerulus via binding to he
21 the loss of nephron function by damaging the tubulointerstitium and that prevention of C5b-9 formatio
22 tubule, but also in a subset of cells in the tubulointerstitium and the glomerulus.
23  Matrix deposition was present mostly in the tubulointerstitium and vessels in accordance with the FK
24 gen matrix deposition within the glomerulus, tubulointerstitium, and arterial walls (all with P < 0.0
25  deposition within the glomerulus, the renal tubulointerstitium, and the posterior pole of the eye.
26              DCs were present throughout the tubulointerstitium but not in glomeruli.
27 terized by damage to both the glomerulus and tubulointerstitium, but relatively little is known about
28 the downstream effects of proteinuria on the tubulointerstitium by negatively modulating TRPV5.
29 nd non-HLA antigens in the glomeruli and the tubulointerstitium cause AMR while inflammatory cytokine
30 bardoxolone methyl analog, in protecting the tubulointerstitium; dh404 markedly suppressed tubular ep
31 her patterns of IF/TA or inflammation in the tubulointerstitium have prognostic importance beyond per
32 er-captured and microdissected glomeruli and tubulointerstitium identified early ECM remodeling, whic
33  in extracellular TG2 and TG activity in the tubulointerstitium in both models.
34 he same pathways that are upregulated in the tubulointerstitium in human diabetic nephropathy.
35 zed the expression of genes in glomeruli and tubulointerstitium in kidney biopsies from diabetic neph
36                                          The tubulointerstitium in kidneys with findings of chronic r
37 expression was dramatically decreased in the tubulointerstitium in obstructive and aristolochic acid
38 een shown to minimize fibrosis of the kidney tubulointerstitium in several diseases.
39 nd strongly support an important role of the tubulointerstitium in the role of renal impairment.
40                       In contrast, the renal tubulointerstitium is the domain of local synthesis of c
41           WWP2 expression is elevated in the tubulointerstitium of fibrotic kidneys and contributes t
42 nce Nox2 and alpha-SMA were increased in the tubulointerstitium of kidneys from 15 liver transplant r
43 2000-2015, and morphometrically analyzed the tubulointerstitium of the cortex for percentage IF/TA, I
44 dex, chronicity index, and assessment of the tubulointerstitium of the kidney.
45 ophages) were found in glomeruli but not the tubulointerstitium of the macaques inoculated with SIVma
46 , apoptotic cells were identified within the tubulointerstitium on day 3 and progressively increased
47 and Col4a3;Tsp1 DKO mice exhibited a widened tubulointerstitium, predominant lesions in Col4a3 KO kid
48          Since loss of PPM1A and PTEN in the tubulointerstitium promotes fibrogenesis, defining molec
49  contributes to the fibrotic response in the tubulointerstitium (TI) after unilateral ureteral obstru
50                                       In the tubulointerstitium, tubular degeneration and macrophage
51 ntrast to the severe glomerular lesions, the tubulointerstitium was not involved in the disease proce
52 er-captured and microdissected glomeruli and tubulointerstitium was performed on 30 for-cause kidney
53                     Expression of HE4 in the tubulointerstitium was positively correlated with tubulo
54     Renal inflammation in both glomeruli and tubulointerstitium was significantly attenuated, associa
55 nt transcriptional changes in microdissected tubulointerstitium were also observed in an independent
56 1(+) T cells were localized primarily to the tubulointerstitium, whereas TGF-beta1(-)FoxP3(+)CD25(+)
57 4- T cells formed small neighborhoods in the tubulointerstitium, with frequency that predicted progre