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1 resented the highest fluorescence quenching (turn-off).
2 during the first min after the treadmill was turned off).
3 participates in G protein-mediated signaling turn-off.
4 n DNA intercalation, and Eu-DOTA-Phen, which turns off.
5 expression of the Nrg1-ntfbeta transgene was turned off.
6 ks of daily pacing at a time when pacing was turned off.
7 mechanism that engages when poleward flux is turned off.
8 events expansion of slp stripes after Eve is turned off.
9 s that was reversed after the high light was turned off.
10 voltaic effect to be reversed in polarity or turned off.
11 ein is expressed and the wild-type Calpha is turned off.
12 oving through the x-ray beam or the beam was turned off.
13 ing persisted even after the stimulation was turned off.
14  process is reversed, and KLF2 expression is turned off.
15 rom the place cells after the landmarks were turned off.
16 re to light that persists after the light is turned off.
17  from solution and the conjugate addition is turned OFF.
18 oA/ROCK is turned on, endothelial cells when turned off.
19 es' as a travelling wave when the tweezer is turned off.
20 pray emitters when the ionization voltage is turned off.
21  upstream DNA damage checkpoint signaling is turned off.
22 ermanently in G2 phase when Ku expression is turned off.
23 part the spindle poles when poleward flux is turned off.
24 iomarkers be nonexistent, the signal remains turned-off.
25 nd mRNA levels is transient and begins to be turned off 3-6 h after growth stimulation of cultured ce
26                        The fast active sites turned off ~40-fold quicker than the slow ones and exhib
27 ene silencing remains stable even if Xist is turned off(8).
28                Hence, rather than serving to turn off a biological response, reversible desensitizati
29  desensitization is a universal mechanism to turn off a biological response; in this process, the abi
30     Depletion of amino acids from the medium turns off a signaling pathway involving the Ragulator co
31 t in this process by acting as a switch that turns off a somatic lineage pathway.
32                                              Turning off ABA response requires turnover and/or inacti
33 sient rise in chlorophyll fluorescence after turning off actinic light reflects nonphotochemical redu
34 dent chlorophyll fluorescence increase after turning off actinic light.
35 he protein RibR counteracts the FMN-induced "turn-off" activities of both FMN riboswitches in Bacillu
36 nel where the voltage across the nanopore is turned off after a user-defined number of single molecul
37            In contrast, how transcription is turned off after activation is unclear.
38  occurs only in subconfluent cultures and is turned-off after a polarized epithelium has formed.
39                However, the GO emission was "turn-off" after the formation of AuNPs on GO surface due
40 or; the activator-bound gene may not readily turn off again.
41 ow this response gets turned on, how it gets turned off again when no longer needed is just as import
42 fides it receives from 2-Cys Prx to APS1 and turns off AGPase.
43 ells from being fully activated, CTLA4Ig can turn off already activated effector T cells by an NO/reg
44 ion; the silencing persisted after p15AS was turned off, although methylation and heterochromatin inh
45                             When STN DBS was turned off, an initial fast but partial loss of benefit
46 phosphorylates key regulatory substrates and turns off anabolic biosynthetic pathways.
47          These results reveal a potential to turn off and turn on the motor by attaching or removing,
48  ADP and AMP, we have developed fluorescence turn-off and turn-on assays that monitor the catalytic a
49                                          The turn-off and turn-on detection strategies-based on capac
50  kinetochores come under tension, the SAC is turned off and APC/C(Cdc20) degrades cyclin B and securi
51 rment was ameliorated when the transgene was turned off and D2R levels were normalized.
52 c supramolecular receptors for fullerenes is turned off and on by changing the solvent, whereas the s
53  amphiphilicity of the copolymer that can be turned off and on by varying the polarity of the solvent
54 e alphaHL channel, the DC driving voltage is turned off and the conductance of the channel is monitor
55 ever, the mechanism of how the pEMT phase is turned off and the redifferentiation phase is initiated
56                      Second, the pressure is turned off and the voltage is applied to reverse the mig
57 ment was maintained when the stimulation was turned off and was accompanied by a higher number of tyr
58 rate that genes turn on, the rate that genes turn off, and the rate of transcription.
59 x and cornea-transparency-related genes were turned off, and a repertoire of macrophage genes were sw
60 rent which is shown to be enhanced, reduced, turned off, and even inverted due to the change of the a
61  spontaneously when calcineurin activity was turned off, and expression of fetal genes reverted to ba
62 s, can be stable for hours after the beam is turned off, and ions that screen charges on the filament
63 -anaphase transition, the spindle checkpoint turns off, and MCC disassembles to allow anaphase onset.
64 those associated with body column tissue are turned off as the tentacle zone expands.
65 ndifferentiated embryonic stem cells but was turned off as these cells differentiate in vitro and in
66     When attractive van der Waals forces are turned off as well, the dewetting in the interdomain reg
67        Furthermore, when P3H2 expression was turned off, as seen naturally in cultured SAOS-2 osteosa
68 tein phosphatase type 5 (PP5), which acts to turn off ASK-1/MKK-4/JNK signaling via two mechanisms.
69  signal transducer, a real-time fluorescence turn-off assay for the enzyme alkaline phosphatase (ALP)
70                 The fluorescence turn-on and turn-off assays monitor the forward and reverse transpho
71 mining gene Sox9 in mutant Sertoli cells was turned off at embryonic day 14.5 after Wt1 ablation, sug
72 F(o) complex explains how ATPase activity is turned off at night by a redox switch.
73 specifically seen in progenitor cells and is turned off at the onset of neuronal differentiation.
74    When transgenic human RTN3 expression was turned off at young age, formation of RIDNs was essentia
75 was found that reserpine oxidation could be "turned off" at flow rates as low as 2.5 microL/min as we
76  spo0A cells, yloA expression was completely turned off because of upregulation of AbrB throughout gr
77 s was dependent on whether the transgene was turned off before or after the formation of RTN3 aggrega
78 ll mutants were similar to wild type showing turned-off behaviors.
79             This sheath or coaxial fluid was turned off between analysis of cells and on during cell
80  its fully reduced 4Cu(I) state, the cluster turns off binding of stronger donor ligands while enabli
81 andcuffing") between pairs of replicons that turn off both origins.
82 ined stable even when the electric field was turned off but readily redissolved when counterions were
83  fire full Ca2+ spikes, which are eventually turned off by a D1/D5-Ca2+-dependent PKC mechanism.
84 by a brief light pulse at one wavelength and turned off by a pulse at a second wavelength.
85 system and allows the dynamic exchange to be turned off by addition of 18-crown-6, which binds the eq
86 TP but, unlike the E. coli enzyme, cannot be turned off by combinations of ATP and dGTP/dTTP.
87 ealthy cells, the SR-BI apoptotic pathway is turned off by eNOS and HDL which prevents inappropriate
88                             The surge can be turned off by fixing the rate of fluid flow through the
89                          Galpha subunits are turned off by GTP hydrolysis.
90                                              Turned off by inhibited anion mobility, that is, proton
91 ism by which NF-kappaB-mediated signaling is turned off by its removal from the DNA in the presence o
92                                       Rh* is turned off by phosphorylation by rhodopsin kinase [G-pro
93 the triple-layer complex and then completely turned off by reforming it through the abstraction of Cl
94 sporters require PIP2 to function and can be turned off by signaling pathways that deplete PIP2.
95                         Signaling is instead turned off by the endosomal retromer complex.
96 nthesis in both asexual and sexual spores is turned off by the NF-kB like fungal regulators VosA and
97 istinct points in the cycle, one of which is turned off by the presence of P(i).
98 turned on" by acute Gq-protein signaling or "turned off" by sustained Gq-protein signaling.
99 tually leads to release from DNA damage thus turning off catalytic activity, although the effects of
100                 In contrast, SpTm ultimately turns off Cdc12p-mediated elongation by "trapping" Cdc12
101 f cyclin B is the major known mechanism that turns off Cdk1 during mitotic exit.
102 n localizes Evi5 to centriolar appendages to turn off centrosomal Rab11 activity.
103  PDL pancreas were impaired when ERalpha was turned off chemically or genetically (ERalpha(-/-)), whe
104 ects the expression of the DprA protein that turns off competence through interaction with phosphoryl
105 esence of doxycycline, and the gene could be turned off completely by removal of doxycycline.
106 s information is essential to initiate or to turn off complex signaling pathways.
107                      Our study suggests that turning off CRT pacing after LVAD implantation in patien
108  to study the effect of continued CRT versus turning off CRT pacing following continuous flow LVAD im
109 nging the catalyst to a Echavarren's gold(I) turned off cyclization to seven member ring and turned o
110  used to monitor cytoplasmic partitioning to turn off cytokinesis signaling.
111 ease of the signal from the individual MNPs (turn-off detection strategy), whereas the techniques pro
112 n immature CD4- CD8- CD44+ CD25- thymocytes, turned off during further stages of thymocyte developmen
113                          When the current is turned off during phases of the domain wall motion when
114 nt study was to investigate how autophagy is turned off during prolonged oxidative stress, to avoid o
115 ng through, even when the signal is actually turned off during the interruption-an effect known as th
116 aive host, the activity of the M promoter is turned off early, preventing excessive synthesis of meth
117                  After the electric field is turned off, electrical current is measured and observed
118  and autophagy in response to low ATP, while turning off energy consumption mediated by mammalian tar
119 eroidal antiestrogens selectively turn on or turn off estrogen target tissues throughout the body.
120 s transduce light information to immediately turn off ethylene signaling for de-etiolation initiation
121 e was sufficient for fructose and glucose to turn off expression of the corresponding protein.
122 nt with doxycycline for 2 months effectively turned off expression of the human RTN3 transgene, confi
123 pertrophic markers, which were normalized on turning off expression of the mutant protein.
124 p unfolds, allowing for mRNA degradation and turning off expression.
125 etermining transcription factors to promptly turn off extrinsic inhibitory Wnt signaling mediated by
126 ing serine 381, a covalent modification that turns off F-actin bundling activity.
127 and density functional theory (DFT)) showing turn off fluorescence behavior.
128 ts a highly selective, sensitive and instant turn-off fluorescence response to Fe(3+) over other meta
129 -metal interactions lead to both turn-on and turn-off fluorescence sensing mechanisms, enhancing the
130 ating the real-world application of FH as a "turn off" fluorescence sensor.
131                                       Thus, "turn-off" fluorescence method was applied for ultrasensi
132                              Herein, a novel turn-off fluorescent aptasensor was developed for select
133 o test the efficacy of the dyes as probes, a turn-off fluoride ion probe was prepared from 3C, which
134  NMDAR-induced FMRP dephosphorylation, which turns off FMRP suppression of Kv4.2.
135          Accordingly, miR-150 repression was turned off following treatment with the DNMT inhibitor,
136 nical status when deep brain stimulation was turned off for 2 days, suggesting that deep brain stimul
137                 When the smoke evacuator was turned off for 30 seconds, there was a more than 26-fold
138 from this "memory" after the illumination is turned off for an extended period of time.
139                       When the transgene was turned off from conception to 3 months of age, no phenot
140 ished in the host, these virulence genes are turned off, fully attenuating the vaccine strain.
141                           Are "turn-on" and "turn-off" functions in protein-protein interaction netwo
142  (GPCRs) by GPCR kinases (GRKs) functions to turn off G-protein signaling and turn on arrestin-mediat
143 otein signaling 4 (RGS4), a protein known to turn off G-protein signaling.
144 ls may acquire differentiated status as they turn off Gli3 expression during neurogenesis.
145                             When spikes were turned off GOM recoveries returned to 60 +/- 19% of perm
146                    Because MSD refolding may turn off GPIb-IX's mechanosensory signals, our results p
147 ibed, but the pathway whereby the levator is turned off have not.
148         Thus, TTP plays an important role in turning off IFN-gamma expression at the appropriate time
149 the STAT3 target Socs3, which is required to turn off IFNAR signaling.
150   In adults, Brg1 (also known as Smarca4) is turned off in cardiomyocytes.
151 g antisense agents that can be turned on and turned off in mammalian tissue culture.
152 ation: miR-211 is a molecular switch that is turned off in melanoma cells, raising the hope that in t
153 emonstrate that (independent of EBV) TET2 is turned off in normal and malignant germinal center (GC)
154 wnstream target of the GTPase Rheb, which is turned off in response to AMPK via the tuberous sclerosi
155  side by the mid-gastrula stage, and is then turned off in the dorsal ectoderm, the neural plate, at
156 xpression of episome maintenance proteins is turned off in the predominant long-term latency reservoi
157                          The current rapidly turns off in the dark as the PTL relaxes thermally back
158  MCC that takes place when the checkpoint is turned off involves the conversion of C-Mad2 back to O-M
159                       The addition of ligand turns off IRF3-dependent transcription and activates ERK
160 del, the transcription factor UNC-55/COUP-TF turns off IRX-1, thus maintaining high levels of OIG-1 t
161 rsed, if expression of the mutant protein is turned off, is unknown.
162 dation is necessary for the yeast to quickly turn off its hypoxic response when oxygen is restored.
163 ts reveal a mechanism that enables CCaMK to 'turn off' its function through autophosphorylation.
164 re also demonstrate that hysteresis and slow turn off kinetics are the only two system properties ass
165 e LTi developmental pathway, but needs to be turned off later to avoid diversion to the T cell fate.
166  a leading kinetochore abruptly disengage or turn off, leading to a direction reversal.
167  in which all interspecific interactions are turned off leads to analytical results that are in agree
168                   The coupling field is then turned off, leaving the atoms in the first condensate in
169 st participants mentioned curtailment (e.g., turning off lights, driving less) rather than efficiency
170 urthermore, ligated Notch-2 also efficiently turns off LMP1 expression from the ED-L1 promoter in LCL
171  H3K9me3 demethylation, and fast feedback to turn off Lsd1.
172                When the checkpoint signal is turned off, MCC is disassembled and the checkpoint is in
173                When the checkpoint signal is turned off, MCC is disassembled, a process required for
174 on pump in mitochondria to spatiotemporally "turn off" mitochondrial function through PMF dissipation
175                           When the light was turned off motoneuron firing and locomotor frequency bot
176       We showed that bacteria that failed to turn off MSHA biosynthesis were unable to colonize the i
177 ransition indicates that osteoblasts neither turn off nor slow down their matrix secreting genes--a w
178 differentiation at least partly because they turn off Notch3.
179 over time causes autophagy deficiency, which turns off Nrf2-mediated defense while switching on an Nr
180 s of a ligand at T3beta, but not at T3alpha, turns off O(2) reactivity, indicating that T3beta and T2
181  proteins) are responsible for the subsecond turn off of G protein signaling and are inhibitors of si
182 G protein signaling (RGS proteins) speed the turn-off of G protein signals and inhibit signal transdu
183 tly more robust than the analysis based on a turn-off of the signal due to free magnetic nanobeads us
184 acute restraint stress followed by a delayed turn-off of the stress response.
185 n to their well-known role in regulating the turning off of Rh*, can also modulate the decay of light
186 e of RhoA activation is insensitive to rapid turning off of the receptor and is required for maintain
187 context-contamination" leads to voluntarily "turn off" of negative states.
188                        How the SAC signal is turned off once all the chromosomes have successfully at
189                     Mammalian X inactivation turns off one female X chromosome to enact dosage compen
190 rformed in real-time and without the need to turn off operating room lights.
191 ut how the tectum's saccade-related activity turns off OPNs is not known.
192 r phosphatase activity and is monitored as a turn-off or turn-on signal, respectively.
193 -spin ions such as Cu and Ni can be entirely turned off or on, respectively, whereas the magnetic con
194 hanisms whereby inflammation can be reduced, turned off or perhaps prevented, both in the context of
195 osphorylation-sensitive molecular switch for turning off or on RP mRNA translation and subsequent rib
196 ion wavelength resulting in the fluorescence turning OFF or ON, respectively.
197 istinct lineage-specific gene networks while turning off or rewiring pluripotency networks.
198 g the AP axis, using transgenes that rapidly turn "off" or "on" BMP signaling.
199  Either receptors reach full methylation and turn off, or receptors become saturated and cease to res
200 on-bound replication initiator (Rep) protein turns off origin function.
201  signals and pathways that turn on ospC also turn off ospA.
202 that Nkx2.2 functions as a major 'switch' to turn off Pdgfra signaling in OPCs and initiate the intri
203 ), showing that in Schizosaccharomyces pombe turning off peroxide defenses preserves the pool of redu
204 Hh stimulation, thus maximizing signaling by turning off PKA activity and hence Gli3 repressor format
205 in cellular grafts after NT-3 expression was turned off possibly via reduction in L1 and laminin expr
206                              A fluorescence "turn-off" probe has been designed and successfully appli
207                                         This turns off quenching, increases emission intensity, and s
208          Ethers and chloride salts dampen or turn off reactivity, leading to three distinct catalyst
209 n just clathrin adaptor proteins involved in turning off receptor signals; they are actually capable
210 cience under an evolutionary framework risks turning off researchers who have their own theoretical p
211 hereas the Rh5 photoreceptors switch fate by turning off Rh5 and then turning on Rh6 expression.
212 izes kinetochore microtubule attachments and turns off SAC activity.
213 it displays a ratiometric response, while a "turn-off" sensing is observed specifically in the presen
214  discovery that MBBI can serve not only as a turn-off sensor via the simultaneous inclusion of a Trp
215      Taking advantage of this phenomenon, a "turn-off" sensor was developed, which is easy to control
216 expression), induce Shh expression, and then turn off Shh responsiveness.
217 asome-mediated degradation through ERK1/2 to turn off signaling.
218 i subunits of heterotrimeric G-proteins that turns off signaling by G-protein coupled receptors.
219 othing is known about Smad phosphatases that turn off Smad activity.
220                 When the electrorotation was turned off, so that the load was again high, stator unit
221 H(+) work arises because that chemical clock turns off sooner than the H2D(+) clock, but both results
222 e that inactivates Lck in the TCR signalling turn-off stage, leading to suppression of T-cell-mediate
223 e), but typically are more luminous than the turn-off stars and therefore long ago should have evolve
224 umor-suppressive gene-expression program and turning off STAT3's oncogenic program.
225 nt behavior that occurs after the forcing is turned off suggests that the double knocks of Campephilu
226                           Here, we report a "turn-off" surface enhanced Raman scattering (SERS)-based
227 acity in adipocytes and participates in the "turn-off" switch that limits adipocyte fat accumulation
228                                           By turning off SynCAM 1 overexpression in transgenic brains
229                                       In the turn-off system enzyme-catalyzed peptide hydrolysis is s
230                                    MicroRNAs turn off target gene expression within cells by binding
231 l cells, by downregulating 14-3-3sigma, thus turning off TGF-beta's tumor suppression function.
232 ules that turn on the enzyme (activator) and turn off the activation (blocker).
233          Small molecules that can be used to turn off the activities of specific cellular proteins ar
234 ibited by GABA(A) receptors is sufficient to turn off the brainstem REM sleep-generating network; the
235 Once activated, the events that subsequently turn off the CARMA1 signalosome are unknown.
236 transdetermine to another fate, they fail to turn off the cell cycle and continue dividing long after
237 w many attached microtubules are required to turn off the checkpoint, and how the kinetochore monitor
238 uring pneumonia-derived sepsis but failed to turn off the competent state in mice.
239                 Thus Blimp1 probably acts to turn off the default pathway that allows epiblast cells
240  much-sought-after feature is the ability to turn off the dynamic exchange of the system, in order to
241 osa has evolved tight regulatory networks to turn off the energy-expensive T3SS when striving for sur
242 n of the yeast poly(A) binding protein would turn off the expression of the PAB1 gene.
243 vary: some centers continue CRT while others turn off the left ventricular (LV) lead at LVAD implant.
244 ressively attacks other coral-reef fish, can turn off the mimetic colours that disguise it as the ben
245 en at a particular time, and, in some cases, turn off the protein as development progresses or the an
246               Doxycycline administration can turn off the transgene expression allowing the restorati
247      Moreover, this challenge simultaneously turned off the IgE enhancers among the Vgamma1(+) cells.
248  with the Set8 induction being important for turning off the activation of Smad2.
249 ntiangiogenic switch that is responsible for turning off the antiangiogenic activity of the native se
250 progression (a process called "recovery") by turning off the checkpoint.
251 om the anode for at least 2 days, even after turning off the current.
252  assembly factors (CAFs), Asf1 and CAF-1, in turning off the DNA damage checkpoint in budding yeast.
253 ed genes associated with cytotoxic stress by turning off the expression of IFNbeta.
254 hat LCUFAs inhibit PhoQ autokinase activity, turning off the expression of the PhoP-dependent regulon
255                  We show that in addition to turning off the immune/eIF2alpha phosphorylation-depende
256 (PTEN), a phosphatase critically involved in turning off the insulin signal transduction cascade, to
257 ns, the second enzyme outcompetes the first, turning off the latter's response.
258                                   By rapidly turning off the mitochondria using depolarization of the
259             Obstructive apnea was induced by turning off the respirator during end expiration for 2 m
260 ge also destroys the tension responsible for turning off the SAC, cells must possess a mechanism to p
261 otecting cohesin, plays an important role in turning off the SAC, in promoting the congression and bi
262 Therefore, these residues may play a role in turning off the signal transmitter domain of the recepto
263 e integrated alongside policies directed at 'turning off the tap'.
264                  Surprisingly, we found that turning off the transgene expression before the onset of
265                                              Turning off the transgene expression for 3 weeks reverse
266 d curtailment (e.g., taking shorter showers, turning off the water while brushing teeth) rather than
267 segment morphology is not achieved merely by turning off the wing and mesothoracic development progra
268  bulb in a series of Christmas lights, which turns off the entire string.
269 d by a combination of gene repression, which turns off the genes associated with most possible lineag
270                                    This step turns off the IKK/NF-kappaB/SOD2 antioxidant pathway.
271                            This substitution turns off the intrinsic TL-dependent hydrolytic activity
272 Light beam illumination on a phototransistor turns off the trap and releases the trapped cell, which
273  against a series of metal cations to first "turn-off" the fluorescence.
274 acilitates stabilization of YAP1 protein and turning "off" the Hippo pathway through ubiquitination o
275 e, we show that larval zebrafish essentially turn off their visual system at night when they are not
276 er nTregs can convert into effector cells by turning off their Foxp3 expression and, if so, whether T
277 ploited and it is shown how to spontaneously turn off these dipolar capillary interactions by exceedi
278                               Mechanisms for turning off this enzyme once released, preventing unwant
279                             A point mutation turns off this motor's contribution to phagocytosis, sug
280 o turn on anion transport, which can then be turned off through addition of the blocker dodecyl sulfa
281 eins is known, still obscure is how ORAI1 is turned off through Ca(2+)-dependent inactivation (CDI),
282                               "Turn-ON" and "turn-OFF" time course experiments were performed with th
283 apter) by Act1/CIKS (IL17R adapter), thereby turning off TLR2 signaling to restore homeostasis.
284                           The ISC process is turned off to deactivate PDT ability, when the PS is tra
285 tual tasks and spontaneous activity are also turned off together as a unit, with interneurons serving
286 ce a DNA-bound holo-CueR with apo-CueR, thus turning off transcription promptly and facilely.
287                                              Turning off transcription promptly is important, however
288 nderstood how genes in the first wave become turned off transcriptionally and how their precise timin
289  0.001 to 100000ng/mL (R(2) = 0.9339) by the turn off-turn on principle.
290               Importantly, in distant sites, turning off Twist1 to allow reversion of EMT is essentia
291                  A reproducible shift in the turn-off voltage of the semiconducting SWNT FETs was see
292 (ryanodine receptors or RyRs) can be made to turn off when their activity is sustained by the Ca2+ re
293                 However, how this pathway is turned off when hepatocyte proliferation is no longer re
294 d by the integrated fluorophores, which are "turned off" when the particles become destructed and dye
295 nificantly declined when NT-3 expression was turned off, whereas continued NT-3 expression sustained
296 ngth, remain on in the dark for minutes, and turned off with millisecond long pulses at the other wav
297 apped pristine SWCNTs as the internal either turn-off (with sodium cholate (SC)) or reference (with c
298  subsequent peeling performance when AFF was turned off, with reductions in average force, SD of forc
299 ts expressed in resting T cells were rapidly turned off within 30 min after activation.
300 ng the ability of limiting levels of Axin to turn off Wnt signaling.

 
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