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1 resented the highest fluorescence quenching (turn-off).
2 during the first min after the treadmill was turned off).
3 participates in G protein-mediated signaling turn-off.
4 n DNA intercalation, and Eu-DOTA-Phen, which turns off.
5 expression of the Nrg1-ntfbeta transgene was turned off.
6 ks of daily pacing at a time when pacing was turned off.
7 mechanism that engages when poleward flux is turned off.
8 events expansion of slp stripes after Eve is turned off.
9 s that was reversed after the high light was turned off.
10 voltaic effect to be reversed in polarity or turned off.
11 ein is expressed and the wild-type Calpha is turned off.
12 oving through the x-ray beam or the beam was turned off.
13 ing persisted even after the stimulation was turned off.
14 process is reversed, and KLF2 expression is turned off.
15 rom the place cells after the landmarks were turned off.
16 re to light that persists after the light is turned off.
17 from solution and the conjugate addition is turned OFF.
18 oA/ROCK is turned on, endothelial cells when turned off.
19 es' as a travelling wave when the tweezer is turned off.
20 pray emitters when the ionization voltage is turned off.
21 upstream DNA damage checkpoint signaling is turned off.
22 ermanently in G2 phase when Ku expression is turned off.
23 part the spindle poles when poleward flux is turned off.
24 iomarkers be nonexistent, the signal remains turned-off.
25 nd mRNA levels is transient and begins to be turned off 3-6 h after growth stimulation of cultured ce
29 desensitization is a universal mechanism to turn off a biological response; in this process, the abi
30 Depletion of amino acids from the medium turns off a signaling pathway involving the Ragulator co
33 sient rise in chlorophyll fluorescence after turning off actinic light reflects nonphotochemical redu
35 he protein RibR counteracts the FMN-induced "turn-off" activities of both FMN riboswitches in Bacillu
36 nel where the voltage across the nanopore is turned off after a user-defined number of single molecul
41 ow this response gets turned on, how it gets turned off again when no longer needed is just as import
43 ells from being fully activated, CTLA4Ig can turn off already activated effector T cells by an NO/reg
44 ion; the silencing persisted after p15AS was turned off, although methylation and heterochromatin inh
48 ADP and AMP, we have developed fluorescence turn-off and turn-on assays that monitor the catalytic a
50 kinetochores come under tension, the SAC is turned off and APC/C(Cdc20) degrades cyclin B and securi
52 c supramolecular receptors for fullerenes is turned off and on by changing the solvent, whereas the s
53 amphiphilicity of the copolymer that can be turned off and on by varying the polarity of the solvent
54 e alphaHL channel, the DC driving voltage is turned off and the conductance of the channel is monitor
55 ever, the mechanism of how the pEMT phase is turned off and the redifferentiation phase is initiated
57 ment was maintained when the stimulation was turned off and was accompanied by a higher number of tyr
59 x and cornea-transparency-related genes were turned off, and a repertoire of macrophage genes were sw
60 rent which is shown to be enhanced, reduced, turned off, and even inverted due to the change of the a
61 spontaneously when calcineurin activity was turned off, and expression of fetal genes reverted to ba
62 s, can be stable for hours after the beam is turned off, and ions that screen charges on the filament
63 -anaphase transition, the spindle checkpoint turns off, and MCC disassembles to allow anaphase onset.
65 ndifferentiated embryonic stem cells but was turned off as these cells differentiate in vitro and in
66 When attractive van der Waals forces are turned off as well, the dewetting in the interdomain reg
68 tein phosphatase type 5 (PP5), which acts to turn off ASK-1/MKK-4/JNK signaling via two mechanisms.
69 signal transducer, a real-time fluorescence turn-off assay for the enzyme alkaline phosphatase (ALP)
71 mining gene Sox9 in mutant Sertoli cells was turned off at embryonic day 14.5 after Wt1 ablation, sug
73 specifically seen in progenitor cells and is turned off at the onset of neuronal differentiation.
74 When transgenic human RTN3 expression was turned off at young age, formation of RIDNs was essentia
75 was found that reserpine oxidation could be "turned off" at flow rates as low as 2.5 microL/min as we
76 spo0A cells, yloA expression was completely turned off because of upregulation of AbrB throughout gr
77 s was dependent on whether the transgene was turned off before or after the formation of RTN3 aggrega
80 its fully reduced 4Cu(I) state, the cluster turns off binding of stronger donor ligands while enabli
82 ined stable even when the electric field was turned off but readily redissolved when counterions were
85 system and allows the dynamic exchange to be turned off by addition of 18-crown-6, which binds the eq
87 ealthy cells, the SR-BI apoptotic pathway is turned off by eNOS and HDL which prevents inappropriate
91 ism by which NF-kappaB-mediated signaling is turned off by its removal from the DNA in the presence o
93 the triple-layer complex and then completely turned off by reforming it through the abstraction of Cl
96 nthesis in both asexual and sexual spores is turned off by the NF-kB like fungal regulators VosA and
99 tually leads to release from DNA damage thus turning off catalytic activity, although the effects of
103 PDL pancreas were impaired when ERalpha was turned off chemically or genetically (ERalpha(-/-)), whe
104 ects the expression of the DprA protein that turns off competence through interaction with phosphoryl
108 to study the effect of continued CRT versus turning off CRT pacing following continuous flow LVAD im
109 nging the catalyst to a Echavarren's gold(I) turned off cyclization to seven member ring and turned o
111 ease of the signal from the individual MNPs (turn-off detection strategy), whereas the techniques pro
112 n immature CD4- CD8- CD44+ CD25- thymocytes, turned off during further stages of thymocyte developmen
114 nt study was to investigate how autophagy is turned off during prolonged oxidative stress, to avoid o
115 ng through, even when the signal is actually turned off during the interruption-an effect known as th
116 aive host, the activity of the M promoter is turned off early, preventing excessive synthesis of meth
118 and autophagy in response to low ATP, while turning off energy consumption mediated by mammalian tar
119 eroidal antiestrogens selectively turn on or turn off estrogen target tissues throughout the body.
120 s transduce light information to immediately turn off ethylene signaling for de-etiolation initiation
122 nt with doxycycline for 2 months effectively turned off expression of the human RTN3 transgene, confi
125 etermining transcription factors to promptly turn off extrinsic inhibitory Wnt signaling mediated by
128 ts a highly selective, sensitive and instant turn-off fluorescence response to Fe(3+) over other meta
129 -metal interactions lead to both turn-on and turn-off fluorescence sensing mechanisms, enhancing the
133 o test the efficacy of the dyes as probes, a turn-off fluoride ion probe was prepared from 3C, which
136 nical status when deep brain stimulation was turned off for 2 days, suggesting that deep brain stimul
142 (GPCRs) by GPCR kinases (GRKs) functions to turn off G-protein signaling and turn on arrestin-mediat
152 ation: miR-211 is a molecular switch that is turned off in melanoma cells, raising the hope that in t
153 emonstrate that (independent of EBV) TET2 is turned off in normal and malignant germinal center (GC)
154 wnstream target of the GTPase Rheb, which is turned off in response to AMPK via the tuberous sclerosi
155 side by the mid-gastrula stage, and is then turned off in the dorsal ectoderm, the neural plate, at
156 xpression of episome maintenance proteins is turned off in the predominant long-term latency reservoi
158 MCC that takes place when the checkpoint is turned off involves the conversion of C-Mad2 back to O-M
160 del, the transcription factor UNC-55/COUP-TF turns off IRX-1, thus maintaining high levels of OIG-1 t
162 dation is necessary for the yeast to quickly turn off its hypoxic response when oxygen is restored.
163 ts reveal a mechanism that enables CCaMK to 'turn off' its function through autophosphorylation.
164 re also demonstrate that hysteresis and slow turn off kinetics are the only two system properties ass
165 e LTi developmental pathway, but needs to be turned off later to avoid diversion to the T cell fate.
167 in which all interspecific interactions are turned off leads to analytical results that are in agree
169 st participants mentioned curtailment (e.g., turning off lights, driving less) rather than efficiency
170 urthermore, ligated Notch-2 also efficiently turns off LMP1 expression from the ED-L1 promoter in LCL
174 on pump in mitochondria to spatiotemporally "turn off" mitochondrial function through PMF dissipation
177 ransition indicates that osteoblasts neither turn off nor slow down their matrix secreting genes--a w
179 over time causes autophagy deficiency, which turns off Nrf2-mediated defense while switching on an Nr
180 s of a ligand at T3beta, but not at T3alpha, turns off O(2) reactivity, indicating that T3beta and T2
181 proteins) are responsible for the subsecond turn off of G protein signaling and are inhibitors of si
182 G protein signaling (RGS proteins) speed the turn-off of G protein signals and inhibit signal transdu
183 tly more robust than the analysis based on a turn-off of the signal due to free magnetic nanobeads us
185 n to their well-known role in regulating the turning off of Rh*, can also modulate the decay of light
186 e of RhoA activation is insensitive to rapid turning off of the receptor and is required for maintain
193 -spin ions such as Cu and Ni can be entirely turned off or on, respectively, whereas the magnetic con
194 hanisms whereby inflammation can be reduced, turned off or perhaps prevented, both in the context of
195 osphorylation-sensitive molecular switch for turning off or on RP mRNA translation and subsequent rib
199 Either receptors reach full methylation and turn off, or receptors become saturated and cease to res
202 that Nkx2.2 functions as a major 'switch' to turn off Pdgfra signaling in OPCs and initiate the intri
203 ), showing that in Schizosaccharomyces pombe turning off peroxide defenses preserves the pool of redu
204 Hh stimulation, thus maximizing signaling by turning off PKA activity and hence Gli3 repressor format
205 in cellular grafts after NT-3 expression was turned off possibly via reduction in L1 and laminin expr
209 n just clathrin adaptor proteins involved in turning off receptor signals; they are actually capable
210 cience under an evolutionary framework risks turning off researchers who have their own theoretical p
211 hereas the Rh5 photoreceptors switch fate by turning off Rh5 and then turning on Rh6 expression.
213 it displays a ratiometric response, while a "turn-off" sensing is observed specifically in the presen
214 discovery that MBBI can serve not only as a turn-off sensor via the simultaneous inclusion of a Trp
215 Taking advantage of this phenomenon, a "turn-off" sensor was developed, which is easy to control
218 i subunits of heterotrimeric G-proteins that turns off signaling by G-protein coupled receptors.
221 H(+) work arises because that chemical clock turns off sooner than the H2D(+) clock, but both results
222 e that inactivates Lck in the TCR signalling turn-off stage, leading to suppression of T-cell-mediate
223 e), but typically are more luminous than the turn-off stars and therefore long ago should have evolve
225 nt behavior that occurs after the forcing is turned off suggests that the double knocks of Campephilu
227 acity in adipocytes and participates in the "turn-off" switch that limits adipocyte fat accumulation
234 ibited by GABA(A) receptors is sufficient to turn off the brainstem REM sleep-generating network; the
236 transdetermine to another fate, they fail to turn off the cell cycle and continue dividing long after
237 w many attached microtubules are required to turn off the checkpoint, and how the kinetochore monitor
240 much-sought-after feature is the ability to turn off the dynamic exchange of the system, in order to
241 osa has evolved tight regulatory networks to turn off the energy-expensive T3SS when striving for sur
243 vary: some centers continue CRT while others turn off the left ventricular (LV) lead at LVAD implant.
244 ressively attacks other coral-reef fish, can turn off the mimetic colours that disguise it as the ben
245 en at a particular time, and, in some cases, turn off the protein as development progresses or the an
247 Moreover, this challenge simultaneously turned off the IgE enhancers among the Vgamma1(+) cells.
249 ntiangiogenic switch that is responsible for turning off the antiangiogenic activity of the native se
252 assembly factors (CAFs), Asf1 and CAF-1, in turning off the DNA damage checkpoint in budding yeast.
254 hat LCUFAs inhibit PhoQ autokinase activity, turning off the expression of the PhoP-dependent regulon
256 (PTEN), a phosphatase critically involved in turning off the insulin signal transduction cascade, to
260 ge also destroys the tension responsible for turning off the SAC, cells must possess a mechanism to p
261 otecting cohesin, plays an important role in turning off the SAC, in promoting the congression and bi
262 Therefore, these residues may play a role in turning off the signal transmitter domain of the recepto
266 d curtailment (e.g., taking shorter showers, turning off the water while brushing teeth) rather than
267 segment morphology is not achieved merely by turning off the wing and mesothoracic development progra
269 d by a combination of gene repression, which turns off the genes associated with most possible lineag
272 Light beam illumination on a phototransistor turns off the trap and releases the trapped cell, which
274 acilitates stabilization of YAP1 protein and turning "off" the Hippo pathway through ubiquitination o
275 e, we show that larval zebrafish essentially turn off their visual system at night when they are not
276 er nTregs can convert into effector cells by turning off their Foxp3 expression and, if so, whether T
277 ploited and it is shown how to spontaneously turn off these dipolar capillary interactions by exceedi
280 o turn on anion transport, which can then be turned off through addition of the blocker dodecyl sulfa
281 eins is known, still obscure is how ORAI1 is turned off through Ca(2+)-dependent inactivation (CDI),
285 tual tasks and spontaneous activity are also turned off together as a unit, with interneurons serving
288 nderstood how genes in the first wave become turned off transcriptionally and how their precise timin
292 (ryanodine receptors or RyRs) can be made to turn off when their activity is sustained by the Ca2+ re
294 d by the integrated fluorophores, which are "turned off" when the particles become destructed and dye
295 nificantly declined when NT-3 expression was turned off, whereas continued NT-3 expression sustained
296 ngth, remain on in the dark for minutes, and turned off with millisecond long pulses at the other wav
297 apped pristine SWCNTs as the internal either turn-off (with sodium cholate (SC)) or reference (with c
298 subsequent peeling performance when AFF was turned off, with reductions in average force, SD of forc