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1 in a large epidemiologic study (the Missouri Twin Study).
2 ion to dog ownership in adulthood in a large twin study.
3 ed from the Arthritis and Rheumatism Council Twin Study.
4 none in the Environmental Risk Longitudinal Twin Study.
5 Prospective, classic twin study.
6 part of the Environmental Risk Longitudinal Twin Study.
7 nd 606 males) from the Brisbane Longitudinal Twin Study.
8 tic predisposition to CHIP mutations in this twin study.
9 a from a prospective, four-wave longitudinal twin study.
10 ve National Heart, Lung, and Blood Institute Twin Study.
11 t for NHL, identified from the International Twin Study.
12 shown a substantial heritability of AF in a twin study.
13 d heart rate and movement sensing in a large twin study.
14 of human lifespan is 23-33% as evident from twin studies.
15 A demethylation in SLE was confirmed through twin studies.
16 mesial temporal lobe epilepsy has come from twin studies.
17 genotypes, using methods analogous to human twin studies.
18 easons for different outcomes of these large twin studies.
19 is also consistent with the results of adult twin studies.
20 evidence of a genetic basis in familial and twin studies.
21 support for this is derived from family and twin studies.
22 major consequences for the interpretation of twin studies.
23 d by familial aggregation (lambdas = 20) and twin studies.
24 pturing heritability estimates obtained from twin studies.
25 fluences on childhood victimization based on twin studies.
26 These five findings arose primarily from twin studies.
27 0.38 (95% CI, 0.34 to 0.43; I(2) = 12.9%) in twin studies, 0.15 (95% CI, 0.06 to 0.24; I(2) = 0%) in
28 by government agencies, such as in the NASA Twins Study(1), but new commercial spaceflight opportuni
29 Murine quantitative trait loci(6), human twin studies(7) and microbiome genome-wide association s
30 iverse range of missions, including the NASA Twins Study(7), JAXA CFE study(8,9), SpaceX Inspiration4
32 ted from the Environmental-Risk Longitudinal Twin Study, a nationally representative 1994-1995 birth
33 the Environmental Risk (E-Risk) Longitudinal Twin Study, a nationally representative birth cohort of
35 the Environmental Risk (E-Risk) Longitudinal Twin Study, a nationally representative U.K. cohort of 2
36 the Environmental Risk (E-Risk) Longitudinal Twin Study, a nationally representative UK birth cohort
37 nts from the Environmental Risk Longitudinal Twin Study, a population-representative birth-cohort of
38 ata from the Environmental Risk Longitudinal Twin Study, a representative British sample of 1116 twin
39 mbers of the Environmental Risk Longitudinal Twin Study, a representative cohort of British twins (n
40 ar models to analyze data from a Children of Twins Study, a quasiexperimental design, to explore the
46 alian families in the Tasmanian and Brisbane twin studies and from 661 participants in the 20-year fo
48 lizing liability than previously detected in twin studies and indicates that sibling rather than pare
53 es of migraine, large-scale population-based twin studies, and linkage and association studies of fam
54 imitation and mindreading, the relevance of twin studies, and the meaning of cross-cultural differen
55 e Study in Tasmania, the Brisbane Adolescent Twin Study, and the Western Australian Pregnancy Cohort
56 mission, JAXA (Cell-Free Epigenome) mission, Twins study, and spleens from mice on the International
64 etween ASD and ADHD symptoms is supported by twin studies, but the genetic overlap between clinically
65 from the social environment of rearing, and twin studies can often give clues to the importance of b
67 xamination II of the Kaiser Permanente Women Twins Study carried out in 1989-1990 in Oakland, Califor
69 y has been indicated by familial clustering, twin studies, complex segregation analyses and human leu
73 es; (b) cross-sectional human studies; (c) a twin study; (d) studies of changes in cognition with abs
74 compiled from the astronaut cohort and NASA Twin Study data, indicating mitochondrial stress as a co
78 nalysis of 2301 twin pairs confirms that the twin study design results in a very low estimate of shar
83 we combine the power of a within-monozygotic twin study design with longitudinal neuroimaging methods
84 nnings of these topological features using a twin study design, examining the heritability of such st
85 We estimated the heritability of GGT using a twin-study design and examined the genetic covariance be
86 and colleagues combine structural MRI and a twin-study design to investigate the influence of geneti
87 We address this debate by reviewing digital twin studies designed to reverse engineer the learning a
88 (MZ) twins were selected from an adolescent twin study designed to investigate the interplay of gene
91 oyed prospective, cross-sectional, and intra-twin study designs to characterise the relationship of X
94 athic stone formers remains considerable and twin studies estimate a heritability of >45% for nephrol
98 ctors for DR, including heritability scores, twin studies, family studies, candidate gene studies, li
124 uggesting a genetic contribution to EoE, and twin studies have suggested a powerful contribution from
125 studies have suggested a genetic component, twin studies have suggested that little genetic contribu
134 s of depression has been well established in twin studies, identification of genome-wide significant
136 cal debates and reinterpretation of adoption/twin studies in the 1980s regarding intelligence suggest
137 nts participated in the Child and Adolescent Twin Study in Sweden (CATSS) at twin age 9 or 12 years.
138 win participants in the Child and Adolescent Twin Study in Sweden (CATSS) followed up from ages 18 to
139 s cohort study used the Child and Adolescent Twin Study in Sweden (CATSS), in combination with inpati
141 ch 2022, within the Roots of Autism and ADHD Twin Study in Sweden (RATSS) during a 2.5-day participan
142 d genetic data from the Child and Adolescent Twin Study in Sweden and included individuals with Inter
143 t study is based on the Child and Adolescent Twin Study in Sweden with questionnaire data reported by
144 n aged 9 or 12 from the Child and Adolescent Twin Study in Sweden, using results from an independent
149 G, AND PARTICIPANTS: This nationwide Swedish twin study includes 16 951 twin pairs born between 1992
155 on, and severity of this disease, family and twin studies indicate that genetic variation also influe
158 These DNA results confirm the results of twin studies indicating strong genetic stability but inc
159 data from astronaut participants in the NASA Twin Study, Inspiration4, and JAXA missions, we reveal t
161 l genome-wide molecular genetic, family, and twin studies investigating familial/genetic influences o
162 CIPANTS: The Environmental-Risk Longitudinal Twin Study is a population-based cohort study of 2232 ch
165 sing genetically informative designs such as twin studies, it is possible to show that the causes of
167 detailed response to several defenses of the twin study methodology; and in (4) I conclude with sever
168 Using data from the Colorado Longitudinal Twin study (N = 1,098), we examined the stability of GCA
169 UK Environmental Risk (E-Risk) Longitudinal Twin Study (N = 1448, age 18), to investigate associatio
170 years), and Environmental Risk Longitudinal Twin Study (N = 1631, age 18 years), and results were co
174 otic pairs) enrolled in the Older Australian Twins Study (OATS) and explored corresponding gene expre
175 the Environmental Risk (E-Risk) Longitudinal Twin Study observed 2232 participants born during the pe
178 esent study are comparable to those found in twin studies of adult substance use and substance use di
179 transmission, gene-environment interaction, twin studies of anxiety and eating disorders, the so-cal
183 family environment, special issues raised by twin studies of drug use and abuse, and gene-environment
185 opments in twin studies, recent results from twin studies of new phenotypes and recent insights into
186 ducted meta-analyses of data from family and twin studies of panic disorder, generalized anxiety diso
188 hese meta-analytic results from 12 published twin studies of schizophrenia are consistent with a view
191 We analysed data from the Swedish Adoption/Twin Study of Aging (SATSA): a cohort with 3175 assessme
192 men of European ancestry in the Vietnam Era Twin Study of Aging (VETSA): 7% with amnestic MCI (aMCI)
193 asions spanning 28 y in the Swedish Adoption/Twin Study of Aging and parental social class based on t
194 ained in a separate twin sample (Vietnam Era Twin Study of Aging), but birth weight in the child samp
197 children (N=2,620 children) from the Swedish Twin Study of Child and Adolescent Development reported
198 in Sweden) (n = 2309) and the TCHAD (Swedish Twin Study of Child and Adolescent Development) (n = 124
204 Pairs were selected from the Virginia Adult Twin Study of Psychiatric and Substance Use Disorders fo
205 ale and female twins from the Virginia Adult Twin Study of Psychiatric and Substance Use Disorders, w
206 f Schizophrenia Franz Kallmann's influential twin study of schizophrenia in 691 twin pairs was the la
208 TCHAD study and in the Child and Adolescent Twin Study of Sweden (CATSS) (n = 1771), the association
210 autoantibodies were determined in a classic twin study of twin pairs discordant for type 1 diabetes
211 , and ADHD and ASD longitudinally in a large twin study of young adults to investigate the stability
212 ast cancer susceptibility genes arising from twin studies, pedigree analysis and studies of phenotype
215 and guilt) (1) ; heritability estimated from twin studies ranges from 30 to 50% (2) , and SNP-based h
219 Comparing GCTA results to the results of twin studies reveals important insights into the genetic
228 amily studies in clinical samples as well as twin studies suggest a familial liability and consequent
230 defects account for up to 2-6% of cases, but twin studies suggest a hereditary component in 35%.
232 is not fully understood, but population and twin studies suggest a large heritable component to the
248 ion of SUDs has familial contributions, with twin studies suggesting a strong contribution of additiv
253 e basis of a review of family, adoption, and twin studies that met specific inclusion criteria, the a
254 dications from epidemiological and identical-twin studies that MS is infectious, no virus or other in
255 CD Twin Study is a semi-longitudinal classic twin study that collected repeated dietary, anthropometr
258 in independent datasets, including the NASA Twins Study, the I4 skin spatial transcriptomics, and 81
259 ets available on NASA GeneLab, from the NASA Twins Study, the JAXA CFE study, and the Inspiration4 mi
272 Burden of Atherosclerotic Lesions) classical twin study, we analyzed twin pairs without known coronar
278 th erythrocyte zinc levels in the Australian Twin Study were used as instrumental variables for level
279 ere from the Environmental Risk Longitudinal Twin Study, which follows 2,232 twin children and their
280 IGN, SETTING, AND PARTICIPANTS: Multivariate twin study with BPD criteria assessed by personal interv