1 ociations of rs6822844 with SLE (P = 0.
008),
type 1 DM (P = 0.014), RA (P = 0.019), and primary SS (P
2 erative colitis) (P(meta) = 3.48 x 10(-
12)),
type 1 DM (P(meta) = 5.33 x 10(-5)), and CD (P(meta) = 5
3 /+ mutation, which induces hyperglycemia
and type 1 DM, we show that the compound mutant mice develop
4 of the type 1 diabetes mellitus-
associated (
type 1 DM-associated) autoantigen insulinoma-associated-
5 The association
between type 1 DM and RA is specific for a particular RA subset,
6 t CAD in diabetics are confined to
Caucasian type 1 DM patients.
7 ens clarity in children with well-
controlled type 1 DM and to compare the results obtained with those
8 Studies were included (199
for type 1 DM and 144 for type 2 DM, and 38 from other sourc
9 ong children younger than 10 years, most
had type 1 DM, regardless of race/ethnicity.
10 I(Na), which impacts susceptibility to AF
in type 1 DM.
11 d either alone or in association with Met
in type 1 DM associated with periodontitis.
12 pe 2 DM, can predict islet graft survival
in type 1 DM islet transplant (ITx) recipients.
13 mellitus (DM) is more heterogeneous than
in type 1 DM.
14 ft rejection, we used streptozotocin-
induced type 1 DM (DM1) and transgenic Lep(ob/ob) type 2 DM (DM2
15 78.6%, and 85.3%, and in diabetes
mellitus (
type 1 DM) rates were 75.9%, 69.8%, and 70.5%.
16 hyperglycemia in rat type 1 and 2 and
mouse type 1 DM models.
17 ggressive periodontitis who had a history
of type 1 DM and the outcome of her treatment.
18 justment for HLA genotype, family history
of type 1 DM, ethnicity, and maternal age.
19 In a second model
of type 1 DM, mice treated with streptozotocin (STZ) showed
20 A cases was higher than the US prevalence
of type 1 DM (P < 0.003).
21 The prevalence
of type 1 DM among JIA cases was higher than the US prevale
22 The prevalence
of type 1 DM and type 2 DM was compared between patients wi
23 Our data document the incidence rates
of type 1 DM among youth of all racial/ethnic groups, with
24 The highest rates
of type 1 DM were observed in non-Hispanic white youth (18.
25 First-degree relatives
of type 1 DM individuals were recruited from the Denver met
26 risk (odds ratio [OR]) of developing JIA
or type 1 DM was established (cases compared with controls)
27 familial relationships among cases of JIA
or type 1 DM were established.
28 Records of individuals with JIA
or type 1 DM were probabilistically linked with records in
29 For each case of JIA
or type 1 DM, 10 matched controls or 5 matched controls, re
30 arious levels of familial exposure to JIA
or type 1 DM, one's risk (odds ratio [OR]) of developing JI
31 nce of an association with either RA, MS,
or type 1 DM, were selected for genotyping in UK JIA cases
32 ansplantation into patients with FGS, PC,
or type 1 DM, grafts from LURD are preferred over parental
33 proliferation frequently in HLA-DR4-
positive type 1 DM patients, but rarely in non-HLA-DR4 patients,
34 these changes might have been caused by
the type 1 DM.
35 sposition for type 2 DM can coexist with
the type 1 DM phenotype and is associated with earlier decli
36 the healing epithelia of normal (NL)
versus type 1 DM rat corneas.
37 The children
with type 1 DM had decreased lens clarity and increased LT, e
38 stigated long-term survival in patients
with type 1 DM (T1DM) and type 2 DM (T2DM) following CABG.
39 developing RA later in life in patients
with type 1 DM may be attributed, in part, to the presence of
40 ferred over regular insulin in patients
with type 1 DM since they improve HbA1C and reduce episodes o
41 isk of anti-CCP-positive RA in patients
with type 1 DM to an OR of 5.3 (95% CI 1.5-18.7).
42 Among patients
with type 1 DM with end-stage nephropathy, SPK transplantatio
43 In patients
with type 1 DM, physiologic replacement, with bedtime basal i
44 c) levels were evaluated among patients
with type 1 DM.
45 Even among older youth (> or =10
years),
type 1 DM was frequent among non-Hispanic white, Hispani