ライフサイエンスコーパス: type B

1 type A) and F. tularensis subsp. holarctica (type B).

2 (monolayers of type A) and 38 +/- 4 degrees (type B).

3 in strain PMSS1 (hpEurope/type-B and cagPAI type-B).

4 novel virus, designated PhV1 type A and PhV1 type B.

5 l disorders, including peeling skin syndrome type B.

6 d booster, except for Haemophilus influenzae type b.

7 gainst H3N2, H1N1pdm09, H1N1 (pre-2009), and type B.

8 greater than the IIV3 effectiveness against type B.

9 of both type A viruses and both lineages of type B.

10 sponded to type C, and 18.3% corresponded to type B.

11 versus 18%, P=0.06); 44% of dissections were type B.

12 pEurope into two groups: hpEurope/type-A and type-B.

13 multi-cagA genotype strains displayed cagPAI type-B.

14 ay whereby membrane-bound scavenger receptor type B-1 (SR-B1) in parent cells becomes incorporated in

15 Scavenger receptor type B-1 (SR-B1), found in lipid rafts, is a receptor fo

16 PD-L2 mAb blockade of wild-type B-1 cells in culture significantly increased CD138

17 and was rescued by adoptive transfer of wild-type B-1 cells.

18 y but had abnormally low osmotic thresholds (type B); 44% had normal copeptin concentrations independ

19 5.6 cm] versus 3.3 cm [2.9-3.7 cm], P<0.001; type B: 5.0 cm [3.9-6.0 cm] versus 4.0 cm [3.5-4.8 cm],

20 coccus serogroup W (45.5%) and H. influenzae type b (54.5%), respectively.

21 )=44.4) for H3N2, 54% (46-61; I(2)=61.3) for type B, 61% (57-65; I(2)=0.0) for H1N1pdm09, and 67% (29

22 rm groups, except for Haemophilus influenzae type b (88.1%).

23 on of resurgent A(H1N1)pdm09 and late-season type B activity.

24 fore, in mainland China, safety for TEVAR of type B AD appeared better between 2008 and 2015 than in

25 estern data (2006-2013) on acute complicated type B AD, stroke, paraplegia, in-hospital mortality and

26 53.9 years, and acute (70%) or chronic (30%) type B AD.

27 target (type A ADR) or an unintended target (type B ADR).

28 Immunologically mediated type B ADRs, such as drug hypersensitivity syndrome, dru

29 lations at risk for immunologically mediated type B ADRs.

30 plied in Type A electrospray system, and for Type B, an additional ring electrode is included.

31 luenza, and 7200 from Haemophilus influenzae type b and 24,700 diarrheal deaths from rotavirus occurr

32 g that, like beta toxin, TpeL contributes to type B and C infections in hosts with decreased trypsin

33 ines during diseases caused by TpeL-positive type B and C strains, as a toxin whose cytotoxicity decr

34 th groups received the combined H influenzae type b and capsular group C Neisseria meningitidis tetan

35 in is a Cl(-)/HCO3(-) exchanger expressed in type B and non-A, non-B intercalated cells in the distal

36 nt Cl(-)/HCO3(-) exchanger that localizes to type B and non-A, non-B intercalated cells, which are ex

37 ation in patients with peeling skin syndrome type B and other diseases related to epidermal barrier d

38 invasive pathogen as Haemophilus influenzae type b and pneumococcal vaccine use in Mali has diminish

39 m was defined as time-signal intensity curve type B and showed a significant association with incompl

40 nd subretinal hyperreflective material, then type B and type C fibroglial lesion (FGL; 17/47 eyes); 2

41 ulti-cagA genotype in strain PMSS1 (hpEurope/type-B and cagPAI type-B).

42 ion networks of the five most relevant HIV-1 types (B and circulating recombinant forms [CRFs] CRF01_

43 ates diagnosed with MoCD (11 type A and five type B) and continued in eight type A patients for up to

44 sis; hepatitis B; and Haemophilus influenzae type b) and pneumococcal vaccine.

45 ensis subsp. holarctica (also referred to as type B), and F. tularensis subsp. mediasiatica, as well

46 2)=17.6) for H3N2, 63% (33-79; I(2)=0.0) for type B, and 62% (36-78; I(2)=0.0) for H1N1pdm09.

47 licy, candidates with a CPRA>20%, with blood type B, and aged 18-49 years were more likely to undergo

48 , tetanus, pertussis, Haemophilus influenzae type b, and hepatitis B) at 6, 10, and 14 weeks of age.

49 roup B Streptococcus, Haemophilus influenzae type b, and meningococcus vaccines.

50 tion against H1N1pdm09, H1N1 (pre-2009), and type B, and reduced protection against H3N2.

51 is lower for H3N2 relative to H1N1pdm09 and type B, and this is not entirely explained by antigenic

52 in mice intranasally challenged with a wild-type B anthracis strain or with an isogenic mutant defic

53 Anti-H influenzae type b anti-polyribosylribitol phosphate IgG geometric m

54 The outcome of patients with acute type B aortic dissection (ABAD) is strongly related to t

55 racic endovascular aortic repair (TEVAR) for type B aortic dissection (AD).

56 he endovascular era, the management of acute type B aortic dissection (ATBAD) is undergoing dramatic

57 ld standard for treating acute uncomplicated type B aortic dissection (TBAD) has been aggressive medi

58 identify clinical parameters associated with type B aortic dissection and to develop a risk model to

59 be surgically repaired immediately, whereas type B aortic dissection can be treated medically.

60 In the risk model, the 10-year occurrence of type B aortic dissection in low-, moderate-, and high-ri

61 ction and to develop a risk model to predict type B aortic dissection in patients with Marfan syndrom

62 The risk for type B aortic dissection in the same population was 0.5

63 The risk of type B aortic dissection is close to the remaining risk

64 High IFM in chronic Type B aortic dissection is linked to improved aortic re

65 IVUS assessment of IFM in chronic Type B aortic dissection might be helpful in identifying

66 uary 1, 2007, through December 31, 2013, for type B aortic dissection were analyzed.

67 Independent variables associated with type B aortic dissection were prior prophylactic aortic

68 AS: 125 had type A aortic dissection, 53 had type B aortic dissection, 35 had intramural aortic hemat

69 pe B dissection or the combined end point of type B aortic dissection, distal aortic surgery, and dea

70 c aortic surgery are at substantial risk for type B aortic dissection, even when the descending aorta

71 on aortic remodelling after TEVAR in chronic Type B aortic dissection.

72 or blocker therapy was associated with fewer type B aortic dissections (hazard ratio: 0.3; 95% confid

73 Data on thoracic aortic aneurysms (TAA), type B aortic dissections (TBAD), and traumatic aortic i

74 ute, 2 had chronic type A, and 3 had chronic type B aortic dissections before surgery.

75 Between 1998 and 2013, 54 type B aortic dissections occurred in 600 patients with

76 erapy may be protective in the prevention of type B aortic dissections.

77 s in patients who underwent acute or chronic type B aortic dissections.

78 nt of initially uncomplicated acute Stanford type-B aortic dissection is associated with a high rate

79 3 patients with acute uncomplicated Stanford type-B aortic dissection, followed over a median of 850

80 erse events after an initially uncomplicated type-B aortic dissection.

81 ne KINASE2 (AHK2) and AHK3 receptors and the type B Arabidopsis response regulator1 (ARR1) and ARR12.

82 the transcriptional network initiated by the type-B ARABIDOPSIS RESPONSE REGULATORs (ARRs) that media

83 ulating the cytokinin response, mechanism of type-B ARR activation, and basis by which cytokinin regu

84 Three type-B ARR DNA-binding motifs, determined by use of prot

85 shed light on the physiological role of the type-B ARRs in regulating the cytokinin response, mechan

86 investigated using Candida antarctica lipase type B as biocatalyst.

87 tween 83.0% and 100%, Haemophilus influenzae type b between 34.7% and 46.2% (40.6% among all preterm

88 Botulinum neurotoxin type B (BoNT/B) recognizes nerve terminals by binding to

89 While wild-type B. bronchiseptica was shed from colonized mice and

90 ure-detectable spirochetemia induced by wild-type B. burgdorferi (WT), indicating that VlsE was likel

91 same conditions, the swimming speed of wild-type B. burgdorferi slowed by approximately 15%, with on

92 was significantly higher than parental wild-type B. burgdorferi strains, suggesting that OspC has an

93 In wild-type B. burgdorferi, bb0449 transcript and BB0449 protei

94 rogressively smaller excitatory responses in type B but not type A neurons.

95 rmidium had 100% nucleotide identity to PhV1 type B, but this region was absent from PhV1 type A.

96 ating blood type A2 and A2B kidneys to blood type B candidates.

97 -matrix ratio (p < 0.001), an 8% decrease of type B carbonate substitution (p < 0.001), and a 2% incr

98 Nasopharyngeal H influenzae type b carriage was detected in one (0.2%) of 623 childr

99 Type B cases presented an increased survival rate compar

100 iet but then relapsed despite a strict diet (type B cases).

101 M_E1 are at the interface between type A and type B CBMs.

102 able identification of three lymphocyte cell types (B, CD4+ T, and CD8+ T cells) with high sensitivit

103 R1 antagonist enhanced CXCL13-triggered wild-type B cell migration.

104 Peptidomes of human HLA-DP-typed B cell lines were analyzed with mass spectrometry

105 of the NF-kappaB inhibitor, JSH-23, to wild-type B cells 15 h after LPS plus IL-4 stimulation select

106 p40, IL-10, TNF-alpha, and IgM than did wild-type B cells after TLR stimulation.

107 led heterogeneous expression of Ifnb in wild-type B cells and distinct gene expression patterns assoc

108 anced the proliferation of B1- as well as B2-type B cells and promoted the production of IgM, IgG1, I

109 Enzymatic desialylation of wild-type B cells blocks their migration into lymph nodes, in

110 ation of T cells in vitro compared with wild-type B cells from the same tumor.

111 CD40 ligation during LPS stimulation of wild-type B cells is sufficient to inhibit PC generation.

112 Type B cells mediate Cl(-) absorption and HCO3(-) secret

113 peak firing during the CS-US interval, while type B cells presented a second peak during US presentat

114 include radial glia comparable to Type E and Type B cells, and a neuronal-like population of cerebros

115 nced in Abp1(-/-) B cells compared with wild-type B cells, including Ca(2+) flux and phosphorylation

116 sponses to anti-IgM as mCD22-expressing wild-type B cells.

117 ion and insulin resistance, compared to wild type B cells.

118 g and cytokine production compared with wild-type B cells.

119 al immune responses by antigen-specific wild-type B cells.

120 n and degradation dynamics of different cell types (B cells, T cells, naive, memory) is governed by a

121 ediated translocations in two different cell types, B cells and mouse embryonic fibroblasts (MEFs).

122 Similarly, wild-type B. cellulosilyticus DSM 14838, but not a close rela

123 ussis-inactived polio-Haemophilus influenzae type b combined vaccine (DTaP-IPV-Hib) at 2, 3, and 4 mo

124 vaccine (PsACWY); or Haemophilus influenzae type b conjugate vaccine (Hib-TT).

125 Three types of Salmonella antibodies (type B, D and E) were mixed separately with the cross-li

126 type A (progressive myoclonus epilepsy) and type B (dementia with motor disturbance) Kufs disease wa

127 ar aortic repair may be optimal for treating type B (descending aorta) AAS.

128 The incidence of invasive H influenzae type b disease in children younger than 5 years declined

129 -14), only one case of invasive H influenzae type b disease was detected in a child younger than 5 ye

130 sustained reduction in invasive H influenzae type b disease.

131 emical or clinical response in patients with type B disease.

132 Only hpEurope/type-B displayed the multi-cagA genotype.

133 typing was developed to predict the risk for type B dissection in patients with Marfan syndrome.

134 or a median of 6 years for the occurrence of type B dissection or the combined end point of type B ao

135 Type B dissection was strongly associated with previous

136 nt in patients treated for acute and chronic type B dissection, and when the endograft is significant

137 Four patients had type A and nine had type B dissection.

138 .9%; 95% CI, 2.5-6.3) for surgical repair of type B dissection.

139 Four type b dissections were observed without further sequela

140 ing aorta have been classified as type A and type B dissections, respectively.

141 lves a region of the protein that contains a Type B dsRBD.

142 BDs interact with dsRNA, while non-canonical Type B dsRBDs lack RNA-binding residues and instead inte

143 he protein/protein interaction properties of Type B dsRBDs.

144 patitis B, polio, and Haemophilus influenzae type b (DTaP-IPV-Hib) and pneumococcal vaccination among

145 chemokine receptor 2 (CXCR2), is involved in type B EAE development, and type B EAE is ameliorated by

146 Remission was minimal in type B EAE due to neuronal damages induced by semaphorin

147 , is involved in type B EAE development, and type B EAE is ameliorated by antagonizing these receptor

148 erferon-beta (IFNbeta)-resistant EAE (termed type B EAE), whereas EAE induced by weak activation of i

149 EDN3), its receptor (the endothelin receptor type B [EDNRB]), and the transcription factors SRY-box 1

150 charge, which is the fundamental reason for Type B electrospray system to perform better.

151 Type B enhancement was most common, with a prevalence of

152 eptor type A (ET(A)) and endothelin receptor type B (ET(B)) - with equal affinity, whereas ET-3 has a

153 e cells highly expressed endothelin receptor type B (ETB(R)) and Jagged1, a Notch1 receptor ligand.

154 The endothelin receptor type B (ETBR) regulates water and electrolyte balance an

155 tor type A (ETAR) and/or endothelin receptor type B (ETBR).

156 r detection of antibodies from patients with type B F. tularensis infections and that these can be us

157 A (macrophage-like) synoviocytes and CD44(+) type B (fibroblast-like) synoviocytes.

158 ional role of glycosyltransferases modifying type B flagellin in the 023 and 027 hypervirulent C. dif

159 in neonates diagnosed with MoCD (type A and type B) following a standardised protocol.

160 lysed sterile site cultures for H influenzae type b from children (aged </=12 years) admitted to the

161 extraction of 12 trichothecenes (type A and type B) from baby foods, followed by gas chromatography-

162 gamma-Aminobutyric acid type B (GABAB) receptor autoantibodies were identified i

163 utoantibodies to the gamma-aminobutyric acid type B (GABAB) receptor have recently been identified as

164 The gamma-aminobutyric acid type B (GABAB) receptor undergoes splicing and is an alc

165 ition is mediated by gamma-aminobutyric acid type B (GABAB) receptors, which are heterodimeric G-prot

166 e present the functional characterization of type B Ggamma subunit (SlGGB1) in tomato (Solanum lycope

167 We conclude that the type B Ggamma subunit SlGGB1 mediates auxin and ABA sign

168 Type B Ggamma subunits, lacking a carboxyl-terminal isop

169 C. intestinalis (formerly Ciona intestinalis type B), globally distributed and sympatric in Europe.

170 ents (amino-terminal pro-natriuretic peptide type B >8500 ng/L) had lower response rates (42%, >/= VG

171 nant CHO cells stably expressing hCAR or the type B HBGA alone did not support ReCV infection.

172 ever, CHO cells expressing both hCAR and the type B HBGA were susceptible to ReCV infection.

173 y replacing the major antigenic sites of the type B hemagglutinin with corresponding sequences from e

174 ctivated poliomyelitis-Haemophilus influenza type b-hepatitis B combination vaccine were given at 2,

175 Of 9 cases of H. influenzae, 8 were type b (Hib) and 1 was type f.

176 te vaccine containing Haemophilus influenzae type b (Hib) and group C meningococcal polysaccharides,

177 Encapsulated H. influenzae type b (Hib) and type f (Hif) are the most common seroty

178 (S. pneumoniae), and Haemophilus influenzae type b (Hib) are three most common pathogens accounting

179 coccus pneumoniae and Haemophilus influenzae type b (Hib) between 2000 and 2015.

180 Haemophilus influenzae type b (Hib) conjugate vaccine, delivered as a three-dos

181 incidence of invasive Haemophilus influenzae type b (Hib) disease has significantly decreased since t

182 Haemophilus influenzae type b (Hib) vaccine and the 13-valent pneumococcal conj

183 valent MMR vaccine and Haemophilus influenza type B (HiB) vaccine.

184 H. influenzae type b (Hib) was historically responsible for the majori

185 arides extracted from Haemophilus influenzae type b (Hib), and the corresponding glycoconjugate made

186 (HR = 7.1), and time-signal intensity curve type B (HR = 4.3) were associated with earlier recurrenc

187 ase was more prominent in type A ICs than in type B ICs.

188 in early life were enriched in cells bearing type B IELp TCR usage.

189 In young mice, both type A and type B IELps had an S1PR1+ and alpha4beta7+ emigration-

190 as type A if located underneath the RPE, as type B if located above the RPE, and as type C if the re

191 The development of FID in the acute phase of type B IMH has a poor prognosis owing to the high risk o

192 ere were 107 consecutive patients with acute type B IMH were included prospectively in a multicenter

193 evolution of medically treated patients with type B IMH with and without FID.

194 The QAV was type A in 32% and type B in 32% (Hurwitz and Roberts classification).

195 he BDNF receptor tropomyosin-receptor-kinase type B in rats and mice, we observed that chronic opiate

196 including one with motor neuron disease, 27 type B including 21 with motor neuron disease, eight typ

197 Immune tolerance to alpha-Gal in blood type B individuals might reduce the risk to this allergy

198 type C infections and is also implicated in type B infections, but little is known about the CPB str

199 gs also suggest that prior exposure to H1 or type B influenza may differentially affect cross-reactiv

200 ed from human postvaccination sera with only type B influenza preexposure consistently showed good co

201 those derived from sera with neither H1 nor type B influenza preexposure, and the correlation lessen

202 those derived from sera with neither H1 nor type B influenza preexposure.

203 Type B (inherited) uncertainties are usually insignifica

204 rs) other medications (eg, monoamine oxidase type B inhibitors [MAOBIs], amantadine, anticholinergics

205 R607H knockin did not affect type B intercalated cells.

206 sruption (FID) has been described in >20% of type B intramural hematomas (IMH), with unclear prognosi

207 lmonella samples were spiked with Salmonella type B, introduced into the biosensor via the sample inl

208 e discovered that EDNRB (Endothelin receptor type B) is a candidate gene involved in HA adaptation.

209 sis type IIIB (MPS IIIB, Sanfilippo syndrome type B) is a lysosomal storage disease characterized by

210 Type B Kufs is usually associated with recessive CTSF pa

211 irus infection who all presented with severe type B lactic acidosis shortly after starting treatment

212 r-paritaprevir-ritonavir-dasabuvir may cause type B lactic acidosis.

213 ) of 39 HU +/- 13 in the arterial phase, and type B lesions had a difference of -58 HU +/- 26 in the

214 tion than the thyroid in the arterial phase, type B lesions were not higher in attenuation than the t

215 imates of vaccine effectiveness against both type B lineages were similar (overall, 58%; 95% CI, 35-7

216 The Type B live vaccine strain was also 50% less capable of

217 o different F. tularensis subsp. holarctica (type B) live vaccine strains, thereby demonstrating the

218 of TRPM7 with NS8593 or waixenicin A in wild-type B lymphocytes results in a significant decrease in

219 (high reflectivity with optical shadowing), type B (medium reflectivity with partial visualization o

220 Whereas type B modification is not required for flagellar assemb

221 as enthalpically driven, which is typical of type B modules.

222 The recurrent type A, type D, and type B mutations require 1, 2, and 3 N-nucleotide extens

223 levels of intracellular replication of wild-type B. neotomae were significantly stimulated by coinfe

224 ts that are focused in space and time, while type B neurons integrate across these dimensions.

225 s: for species of type A, in environments of type B, nonadaptive radiations may emerge.

226 difies its flagellin with either a type A or type B O-linked glycosylation system, which has a contri

227 Aortic dissection, commonly type B, occurs in an appreciable proportion of Marfan pa

228 nstitute of Standards and Technology (NIST) "Type B On Bias" approach and yielded consensus values in

229 Sanfilippo syndrome Type B or Mucopolysaccharidosis IIIB (MPS IIIB) is a neu

230 Forty-eight hours after infection, wild-type B. parapertussis bacteria but not the O antigen-def

231 ass spectrometry analysis revealed that wild-type B. parapertussis lipid A consists of a heterogeneou

232 The type B pattern is most common and could be diagnosed wit

233 hil extracellular traps (NETs), whereas wild-type B. pertussis does not, suggesting that ACT suppress

234 rsors (IELps) include PD-1+ type A and Tbet+ type B populations, which differ in their antigen-recept

235 lin-lacking mutant MM36 compared to the wild-type B. pseudomallei strain 1026b.

236 tective immunity against challenge with wild-type B. pseudomallei, suggesting that the genes identifi

237 istically affecting the MAPK pathway in wild-type B-Raf cells.

238 , they induce conformational changes to wild-type B-Raf kinase domain leading to heterodimerization w

239 In tumors with wild-type B-Raf, vemurafenib paradoxically activates downstre

240 idepressants, alters gamma-aminobutyric acid type B receptor (GABABR) expression and signalling, to i

241 Interestingly, the ephrin type B receptor 2 (EphB2), a tyrosine kinase receptor as

242 gamma-Aminobutyric acid type B receptor autoimmunity deserves consideration in p

243 main specific to the gamma-aminobutyric acid type B receptor subunit 1a (GABA(B)R1a).

244 that inhibited the Edn type a receptor, Edn type b receptor, angiotensin type 1 receptor, mineraloco

245 tional consequences of circuit-specific GABA type-B receptor (GABABR) manipulation.

246 Here we show that ephrin type-B receptor 1 (EphB1) is upregulated in injured moto

247 The protein tyrosine kinase Ephrin type-B receptor 3 (EPHB3) counteracts tumor-cell dissemi

248 The GABA(B) (gamma-aminobutyric acid type B) receptor is one of the principal inhibitory neur

249 ABA Type A receptors), but not GABABRs (GABA Type B receptors).

250 mococcal vaccine serotypes and H. influenzae type b remain associated with bacterial meningitis among

251 ch consequently evolved to cagPAI type-A and type-B, respectively; importantly, all multi-cagA genoty

252 ne kinase activity and mutants involving the type B response regulators.

253 ssion of cytokinin primary response genes by type-B response regulator (RRB) transcription factors.

254 the activation of proteins belonging to the type-B Response Regulator family of cytokinin response a

255 f a key family of transcription factors, the type-B response regulators (RRs), in cytokinin signaling

256 ARR1 is one of the most abundantly expressed type-B Response Regulators.

257 f Arabidopsis response regulators (RRs): the type-B RR (RRB) transcriptional activators that promote

258 Novel as well as conserved roles for type-B RRs in the growth and development of a monocot co

259 s associated with decreased activity of rice type-B RRs included effects on leaf and root growth, inf

260 Farther down the signaling pathway, the type-B RRs were found across all plant clades, but many

261 reation of CHA-ud at cagA upstream in cagPAI type-B strains followed by its duplication to cagA downs

262 rtussis, hepatitis B, Haemophilus influenzae type b, Streptococcus pneumoniae, rotavirus, measles, me

263 present in both pellicle and planktonic wild-type B. subtilis cells and in strains with deletions in

264 tracts of chromosomally SNAP-tagged and wild-type B. subtilis strains with protein standards of known

265 bunits are lacking at the postsynapse, while type-B subunits cluster correctly.

266 type IIIB syndrome (also known as Sanfilippo type B syndrome) is a lysosomal storage disease resultin

267 Compared with Type A, Type B system has smaller droplet size and velocity in t

268 Meanwhile the electrospray process in Type B system is more stable than that in Type A with it

269 pectrometry to compare the secretome of wild-type B. thailandensis to that of a mutant harboring a no

270 , and it is proposed that in this monolayer (type B), the molecular axes are tilted by 40-45 degrees

271 AFF-positive cells (mostly B cells); and (2) type B, the main type in GOLD stage IV COPD, characteriz

272 lesion (FGL; 17/47 eyes); 2) progression to type B then type C FGL (17/47 eyes); and 3) persistence

273 ted BthetaOM T cells more strongly than wild-type B. thetaiotaomicron Despite similar levels of Bthet

274 c silencing by these sugars outcompeted wild-type B. thetaiotaomicron in mice fed a diet rich in gluc

275 onventional cooperative/dual photocatalysis (type B), this new class of unconventional PCs operates v

276 WNT1), trimeric intracellular cation channel type b (TRIC-B), and old astrocyte specifically induced

277 sarium graminearum, a fungus able to produce type B trichothecenes on cereals, including deoxynivalen

278 ough the cognate tropomyosin receptor kinase type B (trkB) receptor occurs in substantia nigra pars c

279 oform of tropomyosin-related receptor kinase type B (trkB) receptors expressed on astrocytes.

280 ng downstream of tropomyosin receptor kinase type B (trkB), namely, phosphorylation of Akt and riboso

281 probed the Schu S4 array with sera from 241 type B tularemia cases in Spain.

282 igens would also have utility for diagnosing type B tularemia caused by strains from other geographic

283 c cation transporter 3 and monoamine oxidase type B, two key proteins for DA uptake and metabolism.

284 e anti-A surface selectivity, solutions of O-type, B-type, A-type and AB-type red blood cells (RBCs)

285 tivated intestinal symbiont Epulopiscium sp. type B using fluorescent in situ hybridization.

286 hesized vitamin D in response to ultraviolet type B (UVB) light.We tested the hypothesis that, in vit

287 ter the introduction of routine H influenzae type b vaccination.

288 measles, rubella, and Haemophilus influenzae type b vaccine antigens were comparable between the 2 gr

289 occal serogroup A and Haemophilus influenzae type b vaccine each in 1 patient).

290 meningitis was observed after H. influenzae type b vaccine introduction.

291 nactivated poliovirus/Haemophilus influenzae type b vaccine; age 6/10/ 14 weeks) and 13-valent pneumo

292 uman and monkey cells as efficiently as wild-type B virus.

293 fluenza A subtypes, A(H1N1) and A(H3N2), and type B viruses co-circulate in humans and infection with

294 all influenza virus infections are caused by type B viruses, and these infections can be severe, espe

295 e detection of seasonal H1N1pdm09, H3N2, and type B viruses, as well as highly pathogenic H5 and H7 v

296 1 virus, a type A:H3N2 virus, and one or two type B viruses.

297 The LAIV4 effectiveness against type B was greater than against type A and greater than

298 susceptibility titers against type A but not type B were consistent with this observation.

299 t H. pylori types, termed pre-type-A and pre-type-B, which consequently evolved to cagPAI type-A and

300 epatitis B virus, and Haemophilus influenzae type b), yellow fever, measles, and tuberculosis.