ライフサイエンスコーパス: type IV

1 the most usual, followed by the lower-outer (type IV).

2 otor neuron loss and normal life expectancy (type IV).

3 xerts effects on erythroid physiology in CDA type IV.

4 haracteristic of human Bartter syndrome (BS) type IV.

5 e types consisted predominantly of type I or type IV.

6 en type VI and, to a lesser extent, collagen type IV.

7 are mutated for the alpha1 chain of collagen type IV.

8 R8L barttin variant associated with human BS type IV.

9 Type I: 6, Type 2: 6, Type III: 6, Type IV: 11, Type V: 4.

10 , with type III (29.6%), type II (14.5%) and type IV (5.7%).

11 Mutations in TRPML1 lead to mucolipidosis type IV, a severe lysosomal storage disorder.

12 f BL proteins-laminin, fibronectin, collagen type IV, agrin, and perlecan-on adhesion and TEER was as

13 from pruritic skin lesions, caused by type-I/type-IV allergic reactions accompanied by prominent eosi

14 mode has proven challenging and Type III or Type IV allosteric inhibitors may present a more promisi

15 Collagen type IV alpha 1 and alpha 2 chains form heterotrimers ([

16 on of fumarate hydratase (FH), and collagen, type IV, alpha 5 and collagen, type IV, alpha 6 (COL4A5-

17 and collagen, type IV, alpha 5 and collagen, type IV, alpha 6 (COL4A5-COL4A6) deletions.

18 Collagen type IV alpha1 (COL4A1) and alpha2 (COL4A2) form heterot

19 ntional T cells have also been implicated in type IV (also termed delayed-type or T cell-mediated) hy

20 PAD4 (peptidylarginine deiminase type IV), an enzyme essential for neutrophil extracellul

21 I, II, and VI were smaller than those of MPS type IV and of the control subjects.

22 ypes I and VI were smaller than those of MPS type IV and of the control subjects.

23 or other closely related systems such as the type IV and tight adherence pilus systems.

24 rate, mesangial cell expansion, and collagen type IV and transforming growth factor-beta expression w

25 2015 presented with a greater percentage of Type IV and Type V cases.

26 These included proteins such as type IV and type VI collagen, laminin, nidogen and perle

27 I), Porphyromonas and Peptoniphilus species (type IV), and Propionibacterium acnes (type V).

28 ive lysosomal storage disorder mucolipidosis type IV, and a gain-of-function mutation (Ala419Pro) in

29 Semaphorins, specifically type IV, are important regulators of axonal guidance and

30 esent in congenital dyserythropoietic anemia type IV as a result of dominant mutations in the second

31 e high diversity microbiota, Community State Type IV-B, was the most prevalent in both HIV-negative (

32 ically consisting of mixtures of type II and type IV beta-turns.

33 cs activity is regulated by AcuA (SaAcuA); a type-IV bGNAT.

34 Calcium/calmodulin-dependent protein kinase type IV (CaMKIV) is a key sensory/effector in excitatory

35 ildren vs adults (79.7% vs 64.9%; P = .003); type IV CCs predominated in the adult population (23.9%

36 umvallate papillae, TRPV4 colocalized with a type IV cell and epithelial cell marker but not type I,

37 Type IV cells (nonproliferative "basal cells") have a nu

38 basally situated, immature postmitotic type, Type IV cells.

39 es congenital dyserythropoietic anemia (CDA) type IV, characterized by severe anemia and non-erythroi

40 re in changing blue-green environments using type IV chromatic acclimation (CA4).

41 hesion, and reduced detachment from collagen type IV-coated plates but also, with decreased ability t

42 self-antigens fibronectin (FN) and collagen type-IV (Col-IV).

43 elf-epitope derived from the alpha3 chain of type IV collagen (alpha3135-145).

44 e I collagen (C1M), type III collagen (C3M), type IV collagen (C4M) and a pro-peptide of type III col

45 gh previous work has shown that VWF can bind type IV collagen (collagen 4), little characterization o

46 lymeric networks - one of laminin and one of type IV collagen (Figure 1, bottom).

47 of the basement membrane components, alpha1-type IV collagen and alpha2-type IV collagen, gamma1-lam

48 erium tuberculosis (Mtb) causes breakdown of type IV collagen and decreases tight junction protein (T

49 ron microscope analysis, the distribution of type IV collagen and effects of fibrosis on myocyte memb

50 inverted polarized cysts, with no laminin or type IV collagen assembly at cell/extracellular matrix c

51 ation (PRO-C3 and PRO-C5), basement membrane type IV collagen formation (PRO-C4) and degradation (C4M

52 Type IV collagen is critical for BM function, yet how it

53 ion and cell-matrix adhesion by showing that type IV collagen is essential for inter-adipocyte adhesi

54 l fibrillation and that CLICs and structural type IV collagen may interact on each other to promote t

55 supramolecular matrices built on laminin and type IV collagen networks that provide structural and si

56 nent in the gonadal BM, where it facilitates type IV collagen removal during BM expansion and tissue

57 ne-aminated poly(glycidyl methacrylate)) and type IV collagen targeted peptide (ColIV) for targeted n

58 ellular channel (CLIC) 1, 4, 5 and a rise in type IV collagen were revealed.

59 d cornea (type I collagen) and lens capsule (type IV collagen) were dissected from mouse eyes, and mu

60 Type IV collagen, a major constituent of BMs, is critica

61 xtracellular matrix proteins fibronectin and type IV collagen, and loss of podocyte markers WT1 and s

62 ed the possible interaction between CLIC and type IV collagen, confirmed by protein structure predict

63 mponents, alpha1-type IV collagen and alpha2-type IV collagen, gamma1-laminin and beta2-laminin, were

64 me (AS), a rare disease of basement membrane type IV collagen, impacts the kidneys, ears, and eyes.

65 ajor components of the GBM include laminins, type IV collagen, nidogens and heparan sulfate proteogly

66 AM-OOC contained 2 chambers connected by type IV collagen-coated microchannels, allowing independ

67 gene was COL4A2 (P = 1.41 x 10(-8)) encoding type IV collagen.

68 rface also showed positive stain results for type IV collagen.

69 icroscopy to show positive stain results for type IV collagen.

70 mposed of a mixture of laminin, entactin and type-IV collagen (LEC matrix) 'resets' these endothelial

71 transwell cell culture inserts coated with a type-IV collagen membrane on which an IOL (one-piece Tec

72 itch construct using the dynamic activity of type IV competence pili in V. cholerae as a model system

73 Type IV coupling protein (T4CP) is a hexameric ATPase th

74 Here we test the ability of 200 type III and type IV effector proteins from six Gram-negative bacteri

75 gg-Dube syndrome, Marfan syndrome, vascular (type IV) Ehlers-Danlos syndrome, alpha-1 antitrypsin def

76 The type IV family of P-ATPases, or P4-ATPases, establishes

77 pread homologous families of machines is the type IV filament (TFF) superfamily, comprised of type IV

78 The T2SS is part of an ancient type IV filament superfamily that may have been present

79 Type IV filaments (T4F), which are helical assemblies of

80 e class of filamentous nanomachines known as type IV filaments (Tff).

81 A type IV functional response (i.e. dome-shaped relationsh

82 We report a type IV functional response observed in heifers grazing

83 The type IV functional response resulted from changes in bit

84 Whole genome sequencing of 70 type IV GBS and subsequent phylogenetic analysis elucida

85 and capsid protein (CP) characteristic of a type IV genome organization.

86 Glycogen storage disease type IV (GSD IV) is a rare autosomal recessive disorder

87 eneous early-onset glycogen storage disorder type IV (GSDIV) or the late-onset adult polyglucosan bod

88 including the effector phase of the classic type IV hypersensitivity reaction.

89 We previously observed a cutaneous type IV immune response in nonhuman primates (NHP) with

90 ases and thus are potential target sites for type IV inhibitors.

91 Viral mRNA sequences with a type IV IRES are able to initiate translation without an

92 etic analysis elucidated the localization of type IV isolates in a SNP-based phylogenetic tree and su

93 The lead compounds identified are type-IV kinase inhibitors and represent an ideal framewo

94 follicular adenoma and carcinoma on Bethesda type IV lesions.

95 te (c-di-GMP) and requires production of the type IV mannose-sensitive hemagglutinin (MSHA) pilus.

96 n with their cognate PLXNB family receptors, type IV members have been increasingly shown to mediate

97 including NPC1, mild cases of mucolipidosis type IV (ML4) (TRPML1-F408), Niemann-Pick type A (NPA) a

98 Mucolipidosis type IV (MLIV) is a lysosomal storage disease characteri

99 sosomal storage disorder (LSD) mucolipidosis type IV (MLIV), contributes to upregulate autophagic gen

100 the lysosomal storage disorder mucolipidosis type IV (MLIV), we examined MLIV patient fibroblasts and

101 t TRPML1 (ML1), a protein that is mutated in type IV mucolipidosis (ML-IV), is a tubulovesicular chan

102 f-function mutations are the direct cause of type IV mucolipidosis, an autosomal recessive lysosomal

103 Phospholipid flippases in the type IV P-type ATPase (P4-ATPases) family establish memb

104 aryotic organisms typically express multiple type IV P-type ATPases (P4-ATPases), which establish pla

105 ase (NE(-/-)) or peptidyl arginine deiminase type IV (Pad4(-/-)) (enzymes that formation of neutrophi

106 dies of neonatal peptidyl arginine deiminase type IV (Padi4)-specific thymocytes reveal disparate fat

107 ts into the molecular basis of mucolipidosis type IV pathogenesis.

108 e II patients tend to have plateau iris; MPS type IV patients are vulnerable to open-angle glaucoma;

109 etion systems and are linked to extrusion of type IV pili (T4P) and to DNA uptake.

110 Type IV pili (T4P) are among the key virulence factors u

111 Type IV pili (T4P) are bacterial appendages composed of

112 Type IV pili (T4P) are filamentous appendages found on m

113 Bacterial type IV pili (T4P) are polymeric protein nanofibers that

114 Type IV pili (T4P) are ubiquitous bacterial cell surface

115 Type IV pili (T4P) are very thin protein filaments that

116 ansduction systems that regulate motility by type IV pili (T4P) can be markedly more complex than rel

117 Type IV pili (T4P) contain hundreds of major subunits, b

118 Neisseria meningitidis use Type IV pili (T4P) to adhere to endothelial cells and br

119 ith long, exquisitely thin appendages called type IV pili (T4P), dynamic filaments that are rapidly p

120 oscopic (cryo-EM) structures of two archaeal type IV pili (T4P), from Pyrobaculum arsenaticum and Sac

121 Bacteria can move across surfaces using type IV pili (T4P), which undergo cycles of extension, a

122 ic autotransporter adhesins, O antigens, and type IV pili (T4P).

123 eins, toxins, or filamentous phages; extrude type IV pili (T4P); or take up DNA.

124 Type IV pili (Tfp) are functionally versatile filaments,

125 Type IV pili (Tfp) are highly conserved macromolecular s

126 In the diverse world of bacterial pili, type IV pili (Tfp) are unique for two reasons: their mul

127 Type IV pili (TFP) function as mechanosensors to trigger

128 oups through the extension and retraction of type IV pili (TFP) on solid surfaces, which requires bot

129 we characterized the receptor recognition of type IV pili (Tfp), a key adhesive factor present in num

130 s have the ability to walk on surfaces using type IV pili (TFP), a motility mechanism known as twitch

131 inosa move across surfaces by using multiple Type IV Pili (TFP), motorized appendages capable of forc

132 Type IV pili (Tfp), which are key virulence factors in m

133 Type IV pili (Tfp), which have been studied extensively

134 recently emerged as a model for the study of type IV pili (Tfp)-exceptionally widespread and importan

135 lified by a positive feedback that increases type IV pili activity, thereby promoting long-term surfa

136 terium Synechocystis sp. PCC 6803 moves with Type IV pili and measures light intensity and color with

137 ling system that regulates the production of type IV pili and potentially other systems in certain ga

138 s powered by the extension and retraction of type IV pili and requires the presence of exopolysacchar

139 The broad conservation of the type IV pili and their importance in pathogens for host

140 The hair-like cell appendages denoted as type IV pili are crucial for biofilm formation in divers

141 Type IV pili are extracellular polymers of the major pil

142 Type IV pili are flexible filaments on the surface of ba

143 The genes encoding type IV pili are found universally in the Gram-negative,

144 Type IV pili are important virulence factors for many pa

145 Type IV pili are important virulence factors on the surf

146 at the envelope protein PilY1 and functional type IV pili are required mechanosensory elements.

147 odelled as groups of spherical particles and Type IV pili attached to bacteria are modelled as dynami

148 hese nanowires were previously thought to be type IV pili composed of PilA protein.

149 phosphatases; a set of 20 genes required for type IV pili function; and several conditionally essenti

150 unction of the structurally related archaeal type IV pili is unknown.

151 enzymogenes revealed that the production of type IV pili required the presence of the Le2152 gene, w

152 its key antifungal virulence factors is the type IV pili that are required for twitching motility.

153 Bacteria such as Pseudomonas aeruginosa use type IV pili to move across surfaces.

154 ry pathogen Streptococcus pneumoniae deploys type IV pili to take up DNA during transformation.

155 es a variety of virulence factors, including type IV pili, bacterial extracellular appendages often e

156 flagella, and twitching motility powered by Type IV pili, little is known about gliding motility.

157 face motility is independent of flagella and type IV pili, suggesting a novel mechanism of cell migra

158 IV filament (TFF) superfamily, comprised of type IV pili, type II secretion systems (T2SSs), archael

159 Movement depends on Type IV pili, which are extended, adhere to the substrat

160 liding motility, conditionally important for type IV pili-dependent motility and required to complete

161 s on surfaces as structured swarms utilizing type IV pili-dependent social (S) motility.

162 trast to their biofilm-promoting function in type IV pili-producing heterotrophic bacteria.

163 ation between cyanobacteria and well-studied type IV pili-producing heterotrophic bacteria.

164 or export of substrates and/or extrusion of type IV pili.

165 ish it from both the bacterial flagellum and type IV pili.

166 but the directional motive force comes from Type IV pili.

167 intracellular photoreceptors and mediated by Type IV pili.

168 SSs were relatively recently repurposed from type IV pili.

169 S. aureus colonies through the action of the type IV pili.

170 is dependent on extension and retraction of Type-IV pili (T4P) and production of extracellular polys

171 ovement in the pilMNOPQ operon, encoding the type IV pilin biogenesis apparatus.

172 gly, ComGC(SS) and ComGC(SP) exhibit a novel type IV pilin fold that is purely helical.

173 l structure of PilEDelta1-28 shows a typical type IV pilin fold, demonstrating how it may be incorpor

174 nii subsets produce morphologically distinct type IV pilin glycoproteins.

175 our results provide the first structure of a type IV pilin protein involved in the formation of compe

176 on in Caulobacter crescentus We identify the type IV pilin protein PilA as the primary signaling inpu

177 te the remarkable structural diversity among type IV pilin proteins.

178 encode two O-OTases, one devoted uniquely to type IV pilin, and the other one responsible for glycosy

179 omolog of the N-terminal domain of bacterial type IV pilin, showing once again how proteins can be re

180 a highly flexible and structurally divergent type IV pilin.

181 nas aeruginosa express one of five different type IV pilins (T4P) (5) , two of which are glycosylated

182 ments (T4F), which are helical assemblies of type IV pilins, constitute a superfamily of filamentous

183 rvation, defining a monophyletic group among type IV pilins.

184 m Thermus thermophilus produces two forms of type IV pilus ('wide' and 'narrow'), differing in struct

185 otein E (PE) and the majority subunit of the type IV pilus (PilA), two major antigens of nontypeable

186 an important model system for the studies of Type IV pilus (T4P) because it is motile by social (S) m

187 The bacterial type IV pilus (T4P) is a versatile molecular machine wit

188 ent homology with the evolutionarily related type IV pilus (T4P) system(4,5), we show that their over

189 With a specific focus on the NTHI type IV pilus (T4P), which we have previously shown bind

190 BPS is a type IV pilus (T4P)-inhibited acidic polymer built of ra

191 ce motility powered by the retraction of the type IV pilus (T4P).

192 The type IV pilus (Tfp) of nontypeable Haemophilus influenza

193 ce-associated cells to QS is affected by the type IV pilus (TFP) retraction motors and the minor pili

194 Type IV pilus assembly involves a conserved group of pro

195 The protein complex responsible for type IV pilus assembly is homologous with the type II pr

196 cts several processes, including phototaxis, type IV pilus biosynthesis, photosystem II levels, biofi

197 e PilF ATPase enzymatic activity that powers type IV pilus extension but remain inefficient on the AT

198 with bacterial type II secretion system and type IV pilus formation were shown to specifically bind

199 through the ComE pore through which the NTHI type IV pilus is expressed.

200 n genes are located directly downstream of a type IV pilus operon in strongly cellulolytic members of

201 -gene mannose-sensitive hemagglutinin (MSHA) type IV pilus operon), had reduced infectivity of A. cyt

202 he velocity-force relation of DNA uptake and type IV pilus retraction, we can exclude pilus retractio

203 emisolid surfaces are powered by a conserved type IV pilus system (T4P).

204 NA from the environment, is supported by the type IV pilus system in most species.

205 nd Vibrio cholerae are among the simplest of Type IV pilus systems and possess only a single minor pi

206 nderstanding filament growth in more complex Type IV pilus systems as well as the related Type II sec

207 The Type IV pilus systems of enterotoxigenic Escherichia col

208 ngatus, including genes encoding a conserved Type IV pilus, genes known to be associated with compete

209 bit flagellum-driven motility and upregulate type IV pilus-dependent twitching motility of P. aerugin

210 evidence implies that the JPC is a modified type IV pilus-like structure encoded for in part by gene

211 ty is mediated by the archaellum, a rotating type IV pilus-like structure that is a unique nanomachin

212 identification of compounds able to decrease type IV pilus-mediated interaction of bacteria with endo

213 stitutively undergoes diversification of the Type IV pilus.

214 For twitching, powered by type-IV pilus retraction, we find that individual cells

215 Type IV REases tend to target modified DNA sites, and E.

216 s observed in proteins (with a generic class Type IV representing the rest).

217 f the most abundant protein in pBM, collagen Type IV, requires prolidase, an exopeptidase cleaving th

218 Therefore, a type IV response of intake rate not directly related to

219 The first reported Type IV restriction endonuclease (REase) GmrSD consists

220 ated adenine Recognition and Restriction), a Type IV restriction endonuclease (REase), as instigator

221 Various type IV secreted effectors (T4SEs) have been experimenta

222 hila is a bacterial pathogen that utilises a Type IV secretion (T4S) system to inject effector protei

223 Type IV secretion (T4S) systems are versatile bacterial

224 A type IV secretion effector of E. chaffeensis blocks mito

225 not lose expression or translocation of six type IV secretion effectors (e.g., SidM) that are well k

226 effector protein (CagA) and components of a type IV secretion system (Cag T4SS).

227 acter pylori (Hp) strains that carry the cag type IV secretion system (cag-T4SS) to inject the cytoto

228 within the cell decrease the activity of the Type IV secretion system (T4SS) and subsequently the cap

229 tro work indicated that the cell biology and type IV secretion system (T4SS) dependence of B. neotoma

230 The Dot/Icm type IV secretion system (T4SS) is one of the key virule

231 kinases that are translocated by the Dot/Icm type IV secretion system (T4SS) of several Legionella pn

232 conjugation systems are members of the large type IV secretion system (T4SS) superfamily.

233 he cag pathogenicity island, which encodes a type IV secretion system (T4SS) that injects the CagA on

234 The cag PAI encodes a type IV secretion system (T4SS) that mediates delivery o

235 ic cag pathogenicity island, which encodes a type IV secretion system (T4SS) that translocates a pro-

236 and subsequent pathology require the Dot/Icm Type IV Secretion System (T4SS) to deliver effector prot

237 olar macrophages, C. burnetii uses a Dot/Icm type IV secretion system (T4SS) to generate a phagolysos

238 ique niche, C. burnetii requires the Dot/Icm type IV secretion system (T4SS) to translocate a cohort

239 permissive host cells by employing a Dot/Icm type IV secretion system (T4SS) to translocate effector

240 ing context-dependent human pathogens, use a type IV secretion system (T4SS) to translocate effectors

241 models often leads to downregulation of the type IV secretion system (T4SS), typically by recombinat

242 nce of L. pneumophila depends on its Dot/Icm type IV secretion system (T4SS), which delivers more tha

243 s via its cag pathogenicity island (cag PAI) type IV secretion system (T4SS).

244 A into the extracellular environment using a type IV secretion system (T4SS).

245 ctly into the host cytoplasm using a Dot/Icm type IV secretion system (T4SS).

246 presses the elaboration of the H. pylori cag type IV secretion system (T4SS).

247 n delivery of CagA into host cells through a type IV secretion system (T4SS).

248 at the cytoplasmic entrance of the F-encoded type IV secretion system (T4SS).

249 e proteins is paired with a highly conserved type IV secretion system (T4SS).

250 The bacterium Brucella abortus uses a type IV secretion system (VirB T4SS) to generate a repli

251 ove-described findings were dependent on the type IV secretion system (VirB) and the secreted BPE005

252 ctor protein that is secreted by the Dot/Icm type IV secretion system and interferes with the caspase

253 Aggregation Substance, PrgC) and the Prg/Pcf type IV secretion system and, in turn, conjugatively tra

254 results indicate that NLRC4 and a functional type IV secretion system are crucial for the production

255 are delivered into host cells by the Dot/Icm type IV secretion system during infection.

256 autophagosomes induced by the E. chaffeensis type IV secretion system effector Etf-1, which traffic t

257 us, which induces ER stress by injecting the type IV secretion system effector protein VceC into host

258 Helicobacter pylori type IV secretion system injects the oncoprotein CagA in

259 The H. pylori cag type IV secretion system is an oncogenic locus that tran

260 rgB (aggregation substance) and PrgC - and a type IV secretion system through which the plasmid is de

261 le trafficking/Intracellular multiplication) type IV secretion system to enable its replication in ta

262 vironment for replication and uses a Dot/Icm type IV secretion system to generate the large PV.

263 ncluding Legionella pneumophila, rely on the type IV secretion system to translocate a repertoire of

264 ive agent of Legionnaire's disease, uses its type IV secretion system to translocate over 300 effecto

265 on of the conserved conjugation machinery (a type IV secretion system), and the potential to transfer

266 es for tetracycline resistance [tet(O)], the Type IV secretion system, conjugative transfer and the T

267 other conjugative systems, which depend on a type IV secretion system, Streptomyces requires only Tra

268 a neotomaein vitro model system for study of type IV secretion system-dependent (T4SS) pathogenesis i

269 ellular destruction by restricting fusion of type IV secretion system-dependent Brucella-containing v

270 AP kinase phosphorylation and autophagy in a type IV secretion system-dependent fashion.

271 ted with the presence of a novel chromosomal type IV secretion system.

272 nslocated into the host cell via the Dot/Icm type IV secretion system.

273 into the host cell by the pathogen's Dot/Icm type IV secretion system.

274 into the invaded host cell by the bacterial type IV secretion system.

275 ilm formation, nutrient acquisition, and the type IV secretion system.

276 Bacterial type IV secretion systems (T4SSs) are molecular machines

277 Bacterial type IV secretion systems (T4SSs) can mediate conjugatio

278 Gram-negative bacteria use type IV secretion systems (T4SSs) for a variety of macro

279 Mobile genetic elements (MGEs) encode type IV secretion systems (T4SSs) known as conjugation m

280 A large subfamily of the type IV secretion systems (T4SSs), termed the conjugatio

281 rom one bacterium to another, is mediated by type IV secretion systems (T4SSs).

282 cretion apparatus in Brucella belongs to the type IV secretion systems present in many pathogenic bac

283 envelope, providing insights into the F-like type IV secretion systems.

284 VI secretion, and, unexpectedly, conjugative type IV secretion within competing bacteria, induce P. a

285 Given the central role of VirD4 in type IV secretion, our study provides mechanistic insigh

286 ore complex (TraC and TraG) with homology to type IV secretion-like systems.

287 ducts (HR 3.711, P=0.008), Bismuth-Corlette type IV stricture (HR 2.082, P=0.008), obstruction due t

288 tal bile duct obstruction, Bismuth- Corlette type IV stricture, biliary obstruction caused by gallbla

289 ATPase 2 (ALA2) and the related ALA1 in the type IV subfamily of P-type ATPases as key components of

290 Numerous plasmids and some prophages encode type IV systems, with similar predicted properties, that

291 _2071 encodes an ATPase homologue of type II/type IV systems.

292 e (necrotizing) form of hypophysitis through type IV (T-cell dependent) and type II (IgG dependent) i

293 his was due, in part, to the production of a Type IV Tad pilus (Iam).

294 er risk for follicular carcinoma in Bethesda type IV thyroid nodules but their absence does not allow

295 Legionella pneumophila utilizes the Dot/Icm type IV translocation system to proliferate within a vac

296 that increasing acylsucrose amount output by type IV trichomes and selecting for particular FA profil

297 A secondary analysis of Fitzpatrick skin types IV, VI, and VI demonstrated a sustained interrater

298 3, 1.08), or Fitzpatrick skin phototype (for type IV vs. type I, multivariable-adjusted RR = 0.99, 95

299 Microbiome type IV was not detected in healthy controls.

300 linical diagnosis of osteogenesis imperfecta type IV, we identified two homozygous variants in SPARC