ライフサイエンスコーパス: typhimurium

1 ith another nontyphoidal serovar, Salmonella Typhimurium.

2 wer inflammation and disease in vivo than S. Typhimurium.

3 challenged with Salmonella enterica serovar Typhimurium.

4 thogens, such as Salmonella enterica serovar Typhimurium.

5 lular bacterial pathogen Salmonella enterica Typhimurium.

6 S. aureus, E. coli, L. monocytogenes and S. Typhimurium.

7 formed a single clade distinct from other S. Typhimurium.

8 perfringens and Salmonella enterica serovar Typhimurium.

9 omparison with the model S. enterica serovar Typhimurium.

10 an HEK293 cells and the bacterium Salmonella Typhimurium.

11 cantly less invasive infections than serovar Typhimurium.

12 tles had renal granulomas associated with S. Typhimurium.

13 f porins and in countering bile stress in S. Typhimurium.

14 of mice with influenza virus and Salmonella typhimurium.

15 ty of Zur, ZntR, RcnR and FrmR in Salmonella Typhimurium.

16 ic separation (IMS) for detecting Salmonella typhimurium.

17 e Na(+)/H(+) antiporter NhaA from Salmonella Typhimurium.

18 nvasive pathogen Salmonella enterica serovar Typhimurium.

19 ared with enterocolitis-causing strains of S Typhimurium.

20 s from Salmonella Choleraesuis in Salmonella Typhimurium.

21 particularly E. coli and Salmonella enterica Typhimurium.

22 t conducive to the intracellular growth of S Typhimurium.

23 s with the intracellular pathogen Salmonella typhimurium.

24 om primary human monocytes in response to S. typhimurium.

25 hila, Pseudomonas aeruginosa, and Salmonella typhimurium.

26 tact S Enteritidis but did not bind intact S Typhimurium.

27 pses the DeltapH of intracellular Salmonella Typhimurium.

28 almonella serotypes Newport, Oranienburg and Typhimurium.

29 ections, such as Salmonella enterica serovar Typhimurium.

30 n with Ags prepared from S Enteritidis and S Typhimurium.

31 ammasome activation during infection with S. Typhimurium.

32 5 toxin encoded by the broad-host Salmonella Typhimurium (15) .

33 f all NTS isolates (n = 258), followed by S. Typhimurium (31.7%, n = 213).

34 These consisted of 85 Salmonella Typhimurium, 58 Salmonella Enteritidis, 32 other nontyph

35 athogens such as Salmonella enterica serovar Typhimurium (7.8%), Listeria monocytogenes (3.88%) and E

36 Salmonella enterica serovar Typhimurium, a Gram-negative bacterium, can cause infect

37 during the stepwise evolution of invasive S. Typhimurium across Africa.

38 To directly detect S. typhimurium after IMS, a sandwich immunoassay was implem

39 Typhimurium, allowing the bacteria to compete with the m

40 ve regulator of innate immune responses to S Typhimurium and a previously unrecognized substrate of C

41 The inactivation of S. Typhimurium and A. flavus along with quality degradation

42 e treatment of mice infected with Salmonella typhimurium and affording preliminary promising results

43 n of bacteria after oral administration of S Typhimurium and after administration of dextran sodium s

44 of low levels of Salmonella enterica serovar typhimurium and enteritidis in blood samples without cul

45 t NTS serovars, Salmonella enterica serovars Typhimurium and Enteritidis, were assessed using lipopol

46 ife in biofilms with competing strains of S. Typhimurium and Escherichia coli.

47 8 in response to NLRC4 activators Salmonella Typhimurium and flagellin, canonical or non-canonical NL

48 Salmonella enterica subsp. enterica serovars Typhimurium and its four closest relatives, Saintpaul, H

49 Salmonella Typhimurium and its monophasic variant S.

50 tidrug resistant (MDR) strains of Salmonella Typhimurium and Klebsiella pneumoniae.

51 Additionally, Salmonella serotypes Typhimurium and Newport also formed floccular biofilms.

52 In contrast, restriction of intracellular S Typhimurium and production of IL-18 are dependent on cas

53 ctor TMD-chaperone complexes from Salmonella Typhimurium and Pseudomonas aeruginosa, respectively, re

54 action was seen with Abs generated against S Typhimurium and S Enteritidis.

55 ia coli O157:H7, Salmonella enterica serovar Typhimurium and S.

56 High seroincidences of S. Typhimurium and S.

57 oD) as low as 86 CFU/mL and 94 CFU/mL for S. typhimurium and S. enteritidis, respectively, that could

58 terial pathogens-Salmonella enterica serovar Typhimurium and Shigella flexneri.

59 ir antimicrobial activity against Salmonella Typhimurium and Staphylococcus aureus.

60 re investigated with detection of Salmonella Typhimurium and Staphylococcus aureus.

61 ples resulted in a 7.389 log reduction in S. Typhimurium, and 6.182 log reduction in A. flavus.

62 intestinalis, Ruminococcus obeum, Salmonella typhimurium, and Clostridium difficile) to quantify, exp

63 rial pathogens (Escherichia coli, Salmonella typhimurium, and methicillin-resistant Staphylococcus au

64 ng latex beads, Escherichia coli, Salmonella typhimurium, and Mycobacterium tuberculosis in human and

65 from antigens of enterotoxigenic E. coli, S. typhimurium, and V. parahaemolyticus was produced and fo

66 LTBentero) containing epitopes from ETEC, S. typhimurium, and V. parahaemolyticus was produced in pla

67 Salmonella Enteritidis and Salmonella Typhimurium are major causes of bloodstream infection an

68 lthough S. enterica serovars Enteritidis and Typhimurium are responsible for most of the human infect

69 erichia coli and Salmonella enterica serovar Typhimurium as model microbes, a common redox active ary

70 ith Serratia marcescens in water, Salmonella Typhimurium ATCC 14028 was inoculated in water, 2% milk,

71 ganisms such as Escherichia coli, Salmonella typhimurium, Bacillus subtilis and Saccharomyces cerevis

72 pediatric patients with invasive Salmonella Typhimurium bacteremia (n = 7) and those with Staphyloco

73 S Typhimurium bacteria increase their OM cardiolipin conce

74 ceptible to systemic infection by Salmonella Typhimurium because of loss-of-function mutations in Nra

75 mediated clearance of catheter-associated S. Typhimurium biofilms.

76 Salmonella enterica serotype Typhimurium (S. Typhimurium) boasts a broad host range and can be transm

77 ion augmented lysosomal functions, reduced S Typhimurium burden, and diminished inflammation in vitro

78 Salmonella enterica serovar Typhimurium can inject effector proteins into host cells

79 Salmonella Typhimurium can invade and survive within macrophages wh

80 Most lineages of the S. enterica subspecies Typhimurium cause gastroenteritis in humans; however, th

81 Salmonella Typhimurium causes a self-limiting gastroenteritis that

82 Conversely, Salmonella enterica serovar Typhimurium causes gastroenteritis in humans and thrives

83 h as that mediated by l-asparaginase II of S Typhimurium causes suppression of activation-induced T c

84 ability to kill Salmonella enterica serovar Typhimurium compared to that of macrophages isolated fro

85 related with the logarithm of the Salmonella typhimurium concentration in the sample.

86 am infections by Salmonella enterica serovar Typhimurium constitute a major health burden in sub-Saha

87 n Salmonella enterica serovar Typhimurium (S Typhimurium) contains a complex disulfide bond (Dsb) cat

88 T313 pathovar of Salmonella enterica serovar Typhimurium contributes to a high burden of invasive dis

89 Deletion of the S Typhimurium copper exporters, CopA and GolT, was found t

90 nstrate that the Salmonella enterica serovar Typhimurium core promoter is more active than previously

91 atory response induced by Salmonella serovar Typhimurium creates a favorable niche for this gut patho

92 We also uncovered the S. Typhimurium D23580 and 4/74 genes that showed expression

93 ella Typhimurium LT2, and a clinical isolate Typhimurium D23580.

94 ellular pathogen Salmonella enterica serovar Typhimurium decreases H-NS amounts 16-fold when inside m

95 introduced into three constructed Salmonella Typhimurium Deltaasd mutants: SLT11 (DeltarfbP), SLT12 (

96 e potentiometric immunosensor for Salmonella typhimurium detection based on the blocking surface prin

97 Salmonella Typhimurium diarrhea serves as a paradigm, and complemen

98 cles (OMVs) from Salmonella enterica serovar Typhimurium displaying the variable N terminus of PspA (

99 In Salmonella enterica serovar Typhimurium, DSFs repress the activity of HilD, an AraC-

100 4,[5],12:i:- and S. Typhimurium DT104 clades, the two dominant pandemic clon

101 two MDR lineages, one of which resembled S. Typhimurium DT104, were predicted to have emerged circa

102 lutamyl-meso-diaminopimelic acid]) and the S Typhimurium effector protein SopE.

103 Typhimurium effector, SlrP, prevented anorexia caused by

104 la pneumoniae or Salmonella enterica serovar Typhimurium, enhanced translocation.

105 Salmonella Typhimurium, Escherichia coli and Pseudomonas aeruginosa

106 orescens, Salmonella Enteritidis, Salmonella Typhimurium, Escherichia coli).

107 . Typhimurium) in mildly acidic pH, which S. Typhimurium experiences inside macrophages.

108 Salmonella enterica serovar Typhimurium exploits the host's type I interferon (IFN-I

109 th flagellins of Salmonella enterica serovar Typhimurium, FliC and FljB, are methylated at surface-ex

110 nd II genes competes poorly with wild-type S Typhimurium for colonization of target tissues.

111 f qnrE1 (identified for the first time in S. Typhimurium from food chain), qnrB19, qnrS1, bla(CTX-M-8

112 Typhimurium from infecting the host.

113 to immune serum identified a repertoire of S Typhimurium genes that, when interrupted, result in incr

114 orescence induction to screen the Salmonella Typhimurium genome for loci that respond, at the single-

115 artate/malate can trigger initial Salmonella Typhimurium gut-lumen colonization in mice, providing in

116 ilis, Legionella pneumophila, and Salmonella Typhimurium has demonstrated the capability of this sens

117 to infection by Salmonella enterica serovar Typhimurium has just been published in Nature Microbiolo

118 ysis along the colonization trajectory of S. Typhimurium implicates the C4-dicarboxylate antiporter D

119 ublished paper-based detection method for S. typhimurium in bird feces and whole milk.

120 ally confirmed by capturing and detecting S. typhimurium in ground chicken and ground beef.

121 strict growth of Salmonella enterica serovar Typhimurium in host tissues by causing magnesium depriva

122 sed platform was applied for detection of S. typhimurium in inoculated Starling bird fecal samples an

123 However, the response to S. typhimurium in primary human monocytes has not been stud

124 ofilms formed by Salmonella enterica serovar Typhimurium in vitro and in vivo.

125 Salmonella enterica serovar Typhimurium (S. Typhimurium) in mildly acidic pH, which S. Typhimurium e

126 /PhoQ in both S. bongori and ssrB-lacking S. Typhimurium, indicating that the SsrB requirement for Ph

127 that transfected ultrapure flagellin from S Typhimurium induced cell death and cytokine secretion in

128 slow-replicating persister population of S. Typhimurium induced within the macrophage intracellular

129 inhibitor (stefin B) suppressed S. enterica Typhimurium-induced cell death.

130 Additionally, under these conditions S. typhimurium-induced IL-1 release occurred independently

131 Typhimurium-induced inflammation is the production of ox

132 However, S Typhimurium-induced mTORC2 signaling led to phosphorylat

133 The impaired ability to respond to S Typhimurium-induced oxidative stress results in reactive

134 iently attenuate Salmonella enterica serovar Typhimurium-induced pyroptosis and proinflammatory cytok

135 Salmonella Typhimurium induces inflammatory diarrhea and uptake int

136 When S Typhimurium-infected Lacc1(Deltamye) mice were injected

137 downstream of IFN-I and RIP3 signaling in S Typhimurium-infected macrophages.

138 model of sepsis, we observed that Salmonella typhimurium-infected mice exhibited simultaneous impaire

139 e methodology on Salmonella enterica serovar Typhimurium-infected murine bone-marrow-derived macropha

140 rest due to its high abundance at loci of S. Typhimurium infection and MLN disruption.

141 e susceptible to Salmonella enterica serovar Typhimurium infection in a NOD1-dependent manner, measur

142 human IECs and transformed human IECs upon S Typhimurium infection in vitro We show that caspase-1 is

143 Salmonella typhimurium infection is reported to activate NLRP3 and

144 ity of disease induced by enteric Salmonella Typhimurium infection is strongly modulated by microbiot

145 tion-driven thrombosis induced by Salmonella Typhimurium infection of mice.

146 y, we investigated the role of TRIM21 upon S Typhimurium infection of murine bone marrow-derived macr

147 Typhimurium infection via Ab.

148 Here, we show that Salmonella Typhimurium infection was accompanied by dysbiosis, decr

149 f2 function and antioxidative responses to S Typhimurium infection, eventually leading to cell death.

150 During Salmonella enterica serovar Typhimurium infection, host inflammation alters the meta

151 During Salmonella Typhimurium infection, intestinal CX3CR1(+) cells can ei

152 and function is severely disrupted during S. Typhimurium infection.

153 analyse MLN tissue from a murine model of S. Typhimurium infection.

154 subsequent phosphorylation in response to S Typhimurium infection.

155 ant for commensal E. coli diminishing the S. Typhimurium infection.

156 ndant role of NLRP3 and NAIP/NLRC4 during S. Typhimurium infection.

157 t instead utilizes caspase-4 to respond to S Typhimurium infection.

158 y Salmonella enterica serovar Typhimurium (S Typhimurium) inhibits T cell responses and mediates viru

159 port that an intestinal pathogen, Salmonella Typhimurium, inhibits anorexia by manipulating the gut-b

160 ellular pathogen Salmonella enterica serovar Typhimurium initiates an anti-inflammatory transcription

161 In this study, we showed that S Typhimurium interacted with CD209s, leading to the invas

162 standard DNA and 10(1) copies of Salmonella typhimurium InvA gene sequences (cloned in E. coli and a

163 Salmonella enterica serovar Typhimurium is a facultative intracellular pathogen that

164 Salmonella enterica serovar Typhimurium is an intracellular bacterial pathogen that

165 Salmonella Typhimurium is metabolically adaptable and can harvest e

166 Typhimurium is unclear, although as IgG2a is induced by

167 Murine infection with S. Typhimurium is used as a typhoid model, but its relevanc

168 Salmonella enterica serovar Typhimurium (S Typhimurium) is a Gram-negative bacterium that induces c

169 ined that, in a murine model of infection, S Typhimurium lacking both l-asparaginase I and II genes c

170 Salmonella enterica serovar Typhimurium (S. Typhimurium) leads to inflammasome activation.

171 ection of either MDR K. pneumoniae or MDR S. Typhimurium led to 40% and 60% survival, respectively, c

172 of DMG to mice previously inoculated with S. Typhimurium led to a 50% survival rate, while 100% of in

173 Gram-negative pathogens, such as Salmonella Typhimurium, leptin receptor (Lepr) expression increased

174 a systemic immune stimulation by Salmonella typhimurium lipopolysaccharide (LPS).

175 for inactivating Salmonella enterica serovar Typhimurium LT2 (ST2) in tender coconut water (TCW).

176 nella Typhi Ty2, the nontyphoidal Salmonella Typhimurium LT2, and a clinical isolate Typhimurium D235

177 compartment from Salmonella enterica serovar Typhimurium LT2, one of the most widely studied microcom

178 glucose has been shown to be required for S. Typhimurium macrophage survival, one possible hypothesis

179 Thus, we propose that similar to HIV, S Typhimurium may also utilize APCs via interactions with

180 lso promising, especially against Salmonella typhimurium (MBC = 0.44 mg/mL).

181 However, in contrast to the murine S. Typhimurium model, neither the PhoPQ two-component syste

182 A Salmonella Typhimurium mutant deficient in flagellin methylation is

183 he virulence defect of a S. enterica serovar Typhimurium mutant specifically defective in its ability

184 Typhimurium mutants as surrogates for expression of tran

185 We also observed that Salmonella Typhimurium needs lipid metabolism genes in proinflammat

186 e TSP confers specificity for the Salmonella Typhimurium O-antigen.

187 Thus, S. Typhimurium obtains fumarate by DcuABC-mediated import a

188 killing was evaluated by plating Salmonella typhimurium on agar plates and showed that the catholyte

189 is study was to investigate the effect of S. typhimurium on inflammasomes in primary human monocytes.

190 (Lacc1(Deltamye)) were given oral Salmonella Typhimurium or dextran sodium sulfate.

191 les, but S. enterica subsp. enterica serovar Typhimurium or lesions associated with Salmonella are ra

192 s consisting of closely related clones of S. Typhimurium or S.

193 coli (EHEC) and Salmonella enterica serovar Typhimurium, or the surrogate murine infection model for

194 with IBS, larger numbers of E coli HS and S typhimurium passed through the epithelium than in biopsi

195 Typhimurium penetrating the epithelium were significantl

196 Typhimurium porins including outer membrane protein OmpD

197 ce IgG1, whereas Th1 Ags, such as Salmonella Typhimurium, predominantly induce IgG2a.

198 Upon entering the host, S Typhimurium preferentially colonizes Peyer's patches, a

199 was able to quantitatively detect Salmonella typhimurium ranging from 1.4 x 10(2) to 1.4 x 10(6) CFU/

200 Like many bacteria, S. Typhimurium rapidly responds to changing environments by

201 Typhimurium, reducing virulence while increasing transmi

202 Salmonella enterica serovar Typhimurium (S Typhimurium) relies upon the inner membrane protein PbgA

203 gens such as Shigella flexneri or Salmonella Typhimurium remains incompletely understood [5, 6].

204 is important for restricting intracellular S Typhimurium replication and initiating IL-18 secretion i

205 ukin-18 (IL-18) release and restriction of S Typhimurium replication in the mouse cecum.

206 y impact additional pathways that restrict S Typhimurium replication.

207 While it is clear that Salmonella Typhimurium requires access to glucose during systemic i

208 Oranienburg/S. Typhimurium, respectively.

209 of phage P22 in Salmonella enterica serovar Typhimurium, revealing how a channel forms to allow geno

210 e human pathogen Salmonella enterica serovar Typhimurium (S Typhimurium) contains a complex disulfide

211 e II produced by Salmonella enterica serovar Typhimurium (S Typhimurium) inhibits T cell responses an

212 Salmonella enterica serovar Typhimurium (S Typhimurium) is a Gram-negative bacterium

213 Salmonella enterica serovar Typhimurium (S Typhimurium) relies upon the inner membra

214 Salmonella enterica serovar Typhimurium (S.

215 onocytogenes and Salmonella enterica serovar Typhimurium (S.

216 Salmonella enterica serotype Typhimurium (S. Typhimurium) boasts a broad host range a

217 tem PhoP/PhoQ of Salmonella enterica serovar Typhimurium (S. Typhimurium) in mildly acidic pH, which

218 macrophages with Salmonella enterica serovar Typhimurium (S. Typhimurium) leads to inflammasome activ

219 ered mice susceptible to invasive Salmonella typhimurium (S.t.) infection.

220 and PrgK in the Salmonella enterica serovar Typhimurium Salmonella pathogenicity island 1 (SPI-1) ty

221 In contrast, the S Typhimurium sapA mutants had similar sensitivity to osmo

222 Salmonella enterica subsp. enterica serovar Typhimurium secretome (STS)-induced outcomes in human in

223 Salmonella Typhimurium sequence type (ST) 313 causes invasive nonty

224 ontyphoidal Salmonella disease is Salmonella Typhimurium sequence type (ST)313.

225 However, S. Typhimurium sequence type 313 (ST313) lineage 2 strains,

226 In Salmonella enterica serovar Typhimurium, siroheme is produced by a trifunctional enz

227 Flagellin isolated from the Salmonella Typhimurium SJW1660 strain, which differs by a point mut

228 generated defined mutant derivatives using S Typhimurium SL1344 as the host.

229 we show that infection of host cells with S Typhimurium specifically induces the ubiquitination of t

230 ST11, Salmonella Heidelberg ST15, Salmonella Typhimurium ST 19, and Salmonella II 42:r:- ST1208 that

231 ections of Gram-negative Salmonella enterica Typhimurium (ST), a major source of human food poisoning

232 Schwarzengrund ST96, S. Typhimurium ST19, S.

233 in East Africa, but not of human Salmonella Typhimurium ST313 infection.

234 ntributes to the invasive pathogenesis of S. Typhimurium ST313 lineage 2.

235 Salmonella Typhimurium ST313 was isolated exclusively from human sa

236 a Enteritidis ST11 and 62 (36.0%) Salmonella Typhimurium ST313.

237 aecalis, Pseudomonas fluorescens, Salmonella typhimurium, Staphylococcus aureus); and fungal enzymes

238 ergence and evolutionary paths-of Salmonella Typhimurium (STM) from nine years of Australian disease

239 -term pathogen persistence during Salmonella Typhimurium (STm) infection.

240 ntracellular infections caused by Salmonella Typhimurium (STm) requires IFN-gamma and the Th1-associa

241 f infection with Salmonella enterica serovar Typhimurium (STM) to identify changes in intestinal immu

242 cterial pathogen Salmonella enterica serovar Typhimurium (STm).

243 with the trimeric porin OmpD from Salmonella Typhimurium (STmOmpD) protects against infection.

244 ring bile salt-induced stress and that an S. Typhimurium strain lacking cspE (DeltacspE) displays dos

245 tuberculosis, in Salmonella enterica serovar Typhimurium strain SL3261.

246 Typhimurium strain that specifically targeted the CX3CR1

247 Specifically, we screen Salmonella typhimurium strains expressing and delivering a library

248 ture of metabolically competitive Salmonella typhimurium strains in microfluidic devices.

249 dairy cattle farms as a model system, 87 S. Typhimurium strains isolated from 1999 to 2016 from eith

250 We report that Salmonella Typhimurium strains lacking lipid metabolism genes were

251 Typhimurium succinate utilization genes contribute to ef

252 Typhimurium survival mechanisms in macrophages, and can

253 r results reveal a novel strategy in which S Typhimurium T3SS effectors broaden their functions throu

254 w that human NAIP also senses the Salmonella Typhimurium T3SS inner rod protein PrgJ and that T3SS in

255 the highest antimutagenic activity toward S. typhimurium TA98 and TA100.

256 more susceptible to systemic infection by S Typhimurium than wild-type mice.

257 activity against Salmonella enterica serovar Typhimurium that is not shared by the related purine met

258 y labeled Escherichia coli HS and Salmonella typhimurium that passed through from the mucosal side to

259 vered as an essential gene in E. coli and S. typhimurium that plays a critical role in their virulenc

260 at during systemic infection, the Salmonella Typhimurium that relies upon host lipids to replicate is

261 together with l-asparaginase I to provide S Typhimurium the ability to catabolize asparagine and ass

262 Salmonella enterica subsp. enterica serovar Typhimurium through liver-resident immunoregulatory CD4(

263 Typhimurium tissue colonization and consequently disease

264 Typhimurium to H2O2L and H2O2H, and the results were val

265 This cycle allows S. Typhimurium to harvest energy by H(2)/fumarate respirati

266 flagellin facilitates adhesion of Salmonella Typhimurium to hydrophobic host cell surfaces, and contr

267 ing transposon insertion mutant library of S Typhimurium to immune serum identified a repertoire of S

268 d the ability of Salmonella enterica serovar Typhimurium to infect the central nervous system and cau

269 amily members of Salmonella enterica serovar Typhimurium to link the constitutively expressed CspC an

270 briae (Lpf), which facilitate adherence of S Typhimurium to M cells.

271 majority of AMR acquisition events in NYS S. Typhimurium to the twentieth century, largely within the

272 Typhimurium to utilize a variety of carbon sources, incl

273 Typhimurium traversing the epithelium did not differ bet

274 solution in situ structure of the Salmonella Typhimurium type III secretion machine obtained by high-

275 the needle filament protein of a Salmonella Typhimurium type III secretion system that are involved

276 llum and the needle protein PrgI from the S. Typhimurium type III secretion system.

277 Typhimurium undergoes an incomplete tricarboxylic acid (

278 In summary, S Typhimurium uses PbgA to influence LPS assembly during s

279 n of a pathogenic species such as Salmonella typhimurium, using a luminometer assay.

280 Attenuation of S Typhimurium virulence was due to overproduction of c-di-

281 lipopolysaccharide (LPS) core as a ligand, S Typhimurium was able to bind human dendritic cell-specif

282 ng this system, the limit of detection of S. typhimurium was found to be 10(2) CFU mL(-1) in culturin

283 Typhimurium was significantly reduced after the adoptive

284 and Q); however, Salmonella enterica serovar Typhimurium was the most predominant serovar, accounting

285 S. Typhimurium was the only Salmonella serovar detected in

286 ng Toxoplasma gondii and Salmonella enterica Typhimurium we demonstrate HRMAn's capacity to recognize

287 the intracellular human pathogen Salmonella Typhimurium, we analyzed their expression in varied stre

288 Typhimurium, we found that ArtB binds human glycans, ter

289 Here, by studying Salmonella Typhimurium, we show that the E3 ligase LUBAC generates

290 cytogenes V7 and Salmonella enterica serovar Typhimurium were used as model pathogens to evaluate the

291 e O determinant, which S Typhi shares with S Typhimurium, were present in the sera of immunized mice

292 he phagosome mediates host defense against S Typhimurium, which is counteracted by copper export from

293 ability to kill Salmonella enterica serovar Typhimurium, which was rescuable after experimentally in

294 ds, could efficiently concentrate Salmonella Typhimurium with a capturing efficiency of 95%.

295 eractions of the enteric pathogen Salmonella Typhimurium with host cells and its fitness in zebrafish

296 ctiveness against S. aureus, E. coli, and S. typhimurium, with minimum inhibitory concentration value

297 used to capture a food pathogen, Salmonella typhimurium, with starting concentrations as low as 10(0

298 of low levels of Salmonella enterica serovar Typhimurium without culture enrichment.

299 od was confirmed to be highly specific to S. typhimurium without interference from other pathogenic b

300 The S Typhimurium yfgA mutant lost the characteristic Salmonel