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1 anisation specifically through the action of tyramine.
2  process is not facilitated by the substrate tyramine.
3 m the electrooxidation and polymerization of tyramine.
4 nylalanine to phenethylamine and tyrosine to tyramine.
5 rine DHPG/NE ratio and the pressor effect of tyramine.
6 e were achieved with these concentrations of tyramine.
7  a promising biosensor for quantification of tyramine.
8 ditions, the most concentrated BA was always tyramine.
9 makes it a promising tool for quantifying of tyramine.
10 es and ratiometric response was obtained for tyramine.
11 ers and terpenes, as well as amino acids and tyramine.
12 ER) activation, including the biogenic amine tyramine.
13 s the plant PPO's acceptance or rejection of tyramine.
14  be reduced and legal limits established for tyramine.
15 sed to 47% with increasing concentrations of tyramine (0.129 to 25.8 mmol/L; P<0.001).
16 along with two natural monomers N-E-caffeoyl tyramine (1) and N-E-feruloyl tyramine (2), using centri
17  increased after gastrointestinal digestion: tyramine (13-fold), tryptamine (9-fold), others (2.4-4.2
18  in which the intermediate 4-[(18)F]fluoro-m-tyramine ([(18)F]16) was prepared using a diaryliodonium
19 s N-E-caffeoyl tyramine (1) and N-E-feruloyl tyramine (2), using centrifugal partition chromatography
20 igher levels of asymmetric dimethylarginine, tyramine, 2-hydroxybutyric acid, phosphatidylcholine (32
21  percentage reductions in FVC in response to tyramine (-24 +/- 7 vs. -55 +/- 6 %), phenylephrine (-12
22  young men, the vasoconstrictor responses to tyramine (-25 +/- 1 versus -56 +/- 6%), phenylephrine (-
23  did not significantly blunt the response to tyramine (-30 +/- 2 versus -36 +/- 7%; P = 0.4) or pheny
24         Maximal vasoconstrictor responses to tyramine (-30 +/- 3 versus -41 +/- 3%), phenylephrine (-
25                                              Tyramine (4-hydroxyphenethylamine), which is a monoamine
26 r compared with young men in the presence of tyramine (-6 +/- 7 versus -31 +/- 6%), phenylephrine (-3
27 mpact courtship drive and were controlled by tyramine-a biogenic amine related to dopamine, whose rol
28                                 Furthermore, tyramine accumulation was higher in TAAR1-KO versus wild
29                                          How tyramine acts on neuronal, cellular and circuit levels i
30 logical precursor and functional antagonist, tyramine, adjust motor behavior to different nutritional
31  artery for intra-arterial administration of tyramine, an agent which displaces noradrenaline from th
32 ulation, we infused graded concentrations of tyramine, an indirect presynaptic norepinephrine release
33 ight response activates neurons that release tyramine, an invertebrate analogue of adrenaline and nor
34 -trans-feruloyl tyramine, and N-cis-feruloyl tyramine, an unreported N-feruloyl tyramine dimer was ch
35 rong inducers of bfeA transcription, whereas tyramine and 3,4-dihydroxymandelic acid demonstrated low
36 Use of leaves of some species with prunasin, tyramine and beta-p-arbutin, may be limited in food appl
37 rations of bark lignans, coumarins, proline, tyramine and defensive proteins, and was characterized b
38  used as a carbon and nitrogen source, while tyramine and dopamine can be used only as sources of nit
39 amine (PEA) and its hydroxylated derivatives tyramine and dopamine.
40                                              Tyramine and histamine are the biogenic amines (BA) most
41                               Extremely high tyramine and histamine contents, 1700 and 700 mg/kg, res
42              The synergistic cytotoxicity of tyramine and histamine should be taken into account when
43                            Co-treatment with tyramine and histamine was associated with a stronger cy
44  of dietary BA, the in vitro cytotoxicity of tyramine and histamine was investigated.
45 erivatization with benzoyl chloride, whereas tyramine and histamine were analysed by fluorescence det
46                                              Tyramine and histamine were toxic for HT29 intestinal ce
47 logically active biogenic amines, especially tyramine and histamine, in fermented beverages.
48                                              Tyramine and histamine, the most toxic biogenic amines (
49 selection bias requires bacterially produced tyramine and host octopamine signalling.
50              To further clarify the roles of tyramine and its metabolite octopamine, we have cloned t
51             Results showed that N-E-feruloyl tyramine and N-E-caffeoyl tyramine exhibited the highest
52 h 4-chlorobutyl oleate, oleamide, myricetin, tyramine and N-feruloyltyramine, found for the first tim
53                             We conclude that tyramine and octopamine act as neurotransmitters/modulat
54   Flies with a mutation in dTdc2 lack neural tyramine and octopamine and are female sterile due to eg
55                                              Tyramine and octopamine released from neurons expressing
56 ions (CFSs) of lactic acid bacteria (LAB) on tyramine and other biogenic amine production by differen
57 tyrosine and phenylalanine were converted to tyramine and phenethylamine by tyrosine and phenylalanin
58           The reproducibility of delta15N of tyramine and phenethylamine measured by GCC-IRMS average
59 opposite trend was observed, with histamine, tyramine and putrescine being the most abundant in Pigno
60 ere was prevalence of cadaverine followed by tyramine and putrescine; and total amine levels were low
61 re-activity analyses of APP derivatives with tyramine and the neurotoxin 1-methyl-4-phenylpyridinium
62                                              Tyramine and tryptamine were found in cheese from high S
63 l SCC categories contained spermine; whereas tyramine and tryptamine were only detected in cheese fro
64 thylamine, putrescine, spermine, spermidine, tyramine and tryptamine) in fish tissues.
65 cadaverine, spermidine, spermine, histamine, tyramine and tryptamine) were determined.
66 osine decarboxylase to convert tyrosine into tyramine and tyramine beta-hydroxylase to convert tyrami
67     Only two biogenic amines (tryptamine and tyramine) and two polyamines (spermidine and spermine) w
68 own serotonin, norepinephrine, dopamine, and tyramine, and because the lung is a major metabolic orga
69                            Phenylethylamine, tyramine, and dopamine are oxidized by TynA to the corre
70 ic acid, rutin, scopoletin, N-trans-feruloyl tyramine, and N-cis-feruloyl tyramine, an unreported N-f
71 ve and dietary monoamines such as serotonin, tyramine, and phenylethylamine.
72 mine, naTA, N-acetylserotonin, OA, dopamine, tyramine, and serotonin also have been consistently iden
73 ding L-3,4-dihydroxyphenylalanine, dopamine, tyramine, and serotonin were identified and their relati
74      We conclude that TyrR is a receptor for tyramine, and suggest that it serves to curb high levels
75 noreactive with antisera against octopamine, tyramine, and the enzymes required for their synthesis,
76 yramine, combined the patterns seen with the tyramine- and octopamine antisera.
77 nity to tyramine releasing sites, a specific tyramine antibody was applied.
78 fandel red wines revealed that histamine and tyramine are produced during yeast and malolactic fermen
79 zed on the surface of gold transducers using tyramine as a linker.
80                       Our findings establish tyramine as a neurotransmitter in C. elegans, and we sug
81 e legal limit, increases the cytotoxicity of tyramine at concentrations frequently reached in some fo
82 t from one isolate, S. thermophilus produced tyramine at low (47 isolates) and medium (10 isolates) l
83 rovides direct evidence that octopamine- and tyramine-based neuromodulation can be mediated by astroc
84 opic correlations that demonstrated feruloyl tyramines being incorporated into the lignin biopolymers
85  synthesis, tyrosine decarboxylase (TDC) and tyramine beta-hydroxylase (TBH).
86 probably converted to octopamine by the host tyramine beta-hydroxylase enzyme.
87 ns tyrosine decarboxylase gene, tdc-1, and a tyramine beta-hydroxylase gene, tbh-1.
88 xylase to convert tyrosine into tyramine and tyramine beta-hydroxylase to convert tyramine into octop
89                                              Tyramine beta-monooxygenase (TbetaM) belongs to a family
90                                   The enzyme tyramine beta-monooxygenase (TbetaM) belongs to a small
91                                              Tyramine beta-monooxygenase (TbetaM) catalyzes the synth
92                                              Tyramine beta-monooxygenase (TbetaM), the insect homolog
93 idue, Met471, coordinating the Cu(M) site of tyramine beta-monooxygenase (TbetaM).
94    BA with one amino function (isoamylamine, tyramine, beta-phenylethylamine), in general, showed a w
95            We further show that the level of tyramine-beta-hydroxylase (TBH), the enzyme that convert
96 e the gut, bypasses the requirement for host tyramine biosynthesis and manipulates a host sensory dec
97  We identify the genes that are required for tyramine biosynthesis in Providencia, and show that thes
98 pine-induced increase in egg laying requires tyramine biosynthesis.
99 e crises owing to peripheral accumulation of tyramine, but the role of tyramine in the central effect
100  followed by transformation of dopamine to m-tyramine by a molybdenum-dependent dehydroxylase from Eg
101 edominant before in vitro digestion, whereas tyramine, cadaverine and spermidine after digestion.
102                                   Therefore, tyramine can act as a classical inhibitory neurotransmit
103             This has led to a model in which tyramine can bind to either the Cu(I) or Cu(II) forms of
104                                              Tyramine can bind to two characterized receptors in hone
105            Owing to its chemical properties, tyramine can react with nitrite, a common food additive,
106 eir mode of action was also different, while tyramine caused cell necrosis, histamine induced apoptos
107 conformation) with either Na(125)I or (125)I-tyramine cellobiose, also known as the cell-trapping lig
108 g for TDC, the enzyme converting tyrosine to tyramine, combined the patterns seen with the tyramine-
109  increased synthesis of the hydroxycinnamoyl-tyramine conjugates, feruloyl-tyramine (FT) and coumaroy
110  samples ranged from 3.3 to 30 mg/L, and the tyramine content ranged from 1.0 to 3.0 mg/L.
111                     Increased cadaverine and tyramine contents were found in samples with good sensor
112                      Activation of LGC-55 by tyramine coordinates the output of two distinct motor pr
113 ugates, feruloyl-tyramine (FT) and coumaroyl-tyramine (CT).
114  previously reported to incorporate feruloyl tyramine derivatives.
115                                              Tyramine detection was linearly proportional in response
116 ontrast, two substrates of DAT, dopamine and tyramine, did not significantly impact cross-linking.
117 -feruloyl tyramine, an unreported N-feruloyl tyramine dimer was characterized as the most abundant po
118 that the vasoconstrictor responses evoked by tyramine during exercise or adenosine were repeatable in
119 ith their sum were putrescine, histamine and tyramine, even if reached levels were below toxicity thr
120                    The low and high doses of tyramine evoked significant reductions in FVC (vasoconst
121 to brachial artery infusions of two doses of tyramine (evokes endogenous noradrenaline release) in 10
122              FVC responses to intra-arterial tyramine (evokes endogenous noradrenaline release), phen
123 es to local intrafemoral artery infusions of tyramine (evokes noradrenaline (NA) release), phenylephr
124  that N-E-feruloyl tyramine and N-E-caffeoyl tyramine exhibited the highest DPPH scavenging activity
125  at the lower end of NET inhibition, whereas tyramine exhibits a linear relation, with NET inhibition
126 ntained four amines; there was prevalence of tyramine followed by histamine, phenylethylamine and put
127 droxycinnamoyl-tyramine conjugates, feruloyl-tyramine (FT) and coumaroyl-tyramine (CT).
128                               The inhibitory tyramine-gated chloride channel, LGC-55, induces head re
129 rward locomotion through the activation of a tyramine-gated chloride channel, LGC-55.
130 f LGC-55 in C. elegans, the first identified tyramine-gated chloride channel.
131 o inhibit egg laying, in part via the LGC-55 tyramine-gated Cl(-) channel on the HSNs.
132                                Surprisingly, tyramine had a stronger and more rapid cytotoxic effect
133 ontribute to a model in which octopamine and tyramine have distinct and separable neural activities.
134       The two biogenic amines octopamine and tyramine have recently been shown to modulate various pa
135 tants, with altered levels of octopamine and tyramine, have a locomotion deficit.
136           In most of the analyzed genotypes, tyramine, histamine, dopamine, serotonin, spermidine, an
137 hand, the bacterial-related biogenic amines, tyramine, histamine, tryptamine and beta-phenylethylamin
138 ses to excitatory cholinergic input, both by tyramine(honoka) receptor activation and by downstream d
139  this article: Tyramine (PubChem CID: 5610); Tyramine hydrochloride (PubChem CID: 66449); Poly(vinyl
140                              Octopamine- and tyramine immunostaining in the central complex differed
141                     In each brain hemisphere tyramine immunostaining was found in four neurons innerv
142 not restore the vasoconstrictor responses to tyramine in contracting muscle during heavy rhythmic han
143  HILIC-UPLC-MS approach was elevated urinary tyramine in galN-treated rats, reflecting disturbed amin
144  our technique was demonstrated by analyzing tyramine in spiked serum and milk.
145 al accumulation of tyramine, but the role of tyramine in the central effects of MAOIs remains elusive
146                                The amount of tyramine in the cheeses with the addition of the strain
147 en various potential roles of octopamine and tyramine in the larval brain, such as locomotion, learni
148 the biosensor exhibited a linear response to tyramine in the range 10-120 uM and the limit of detecti
149 ntrations of phenethylamine, tryptamine, and tyramine in the sauerkraut fermented with caraway (and c
150 nses to endogenous noradrenaline release via tyramine in the skeletal muscle circulation, similar to
151              These observations suggest that tyramine, in interaction with glutamate, is involved in
152 s, particularly of cadaverine, histamine and tyramine, in low-salt products.
153                       In both protocols, the tyramine-induced increases in deep venous noradrenaline
154                                     However, tyramine-induced mobilization of norepinephrine stores p
155                     Endotoxemia lessened the tyramine-induced reduction in leg vascular conductance (
156 emia + hypoxia (-17% +/- 21%) attenuated the tyramine-induced reduction in leg vascular conductance c
157 al BH(4) supplementation augments reflex and tyramine-induced VC in aged skin, suggesting that reduce
158 O: -17 +/- 2% DeltaCVC(base); P < 0.001) and tyramine infusion (Y: - 33 +/- 4% DeltaCVC(base), O: -15
159 er: 17 +/- 3% Delta CVC(base); P < 0.01) and tyramine infusion (young: 41 +/- 3, older: 21 +/- 4% Del
160 in temperature (T(sk)) = 30.5 degrees C) and tyramine infusion, respectively.
161 conductance) was evaluated by femoral artery tyramine infusion.
162                           Here, we show that tyramine inhibits head movements and forward locomotion
163 -hydroxylase (TBH), the enzyme that converts tyramine into octopamine in aminergic neurons, is increa
164 ine and tyramine beta-hydroxylase to convert tyramine into octopamine.
165                                              Tyramine is a biogenic compound derived from the decarbo
166 ansmitter in C. elegans, and we suggest that tyramine is a genuine neurotransmitter in other inverteb
167                                              Tyramine is an important neurotransmitter, neuromodulato
168 been reported; therefore, the measurement of tyramine is an urgent concern.
169 stomach lumen, the most favoured reaction in tyramine is C-nitrosation, which generates mutagenic pro
170 e shown that the product of C-nitrosation of tyramine is mutagenic, in the present article tyramine n
171                     The trace biogenic amine tyramine is present in the nervous systems of animals ra
172 PSS/AuNP/1-m-4-MP) for the quantification of tyramine is presented.
173                         Bacterially produced tyramine is probably converted to octopamine by the host
174                                 We show that tyramine is released into the CNS to reduce motoneuron i
175                         The trace amine para-tyramine is structurally and functionally related to the
176                                              Tyramine is synthesized from tyrosine by the enzyme tyro
177 u(II) form of TbetaM is also reduced at high tyramine, leading us to propose the existence of a bindi
178  using mutants having altered octopamine and tyramine levels and by genetic interference with both sy
179 tive feedback mechanism for TAAR1 in sensing tyramine levels.
180                                    Moreover, tyramine-like immunoreactivity was also found in brain r
181                                              Tyramine-like labeling was observed in AmTAR1-IR positiv
182 nd urocortins, together with the trace amine tyramine, may have a role in the human cardiovascular sy
183                      However, instead of the tyramine moiety that is present in methanofuran cofactor
184 n of Fru(M) by transformer expression in OCT/tyramine neurons changes the aggression versus courtship
185                       The regulation of this tyramine neurotransmitter was found to be linked to pati
186 yramine is mutagenic, in the present article tyramine nitrosation mechanisms have been characterized
187  structurally similar to the bVMAT substrate tyramine, none of them were measurably transported into
188 enzyme capable of synthesizing morphine from tyramine, norlaudanosoline, and codeine.
189                                 Furthermore, tyramine normalized the doubled leg vascular conductance
190 Surprisingly, however, mon1 functions in the tyramine/octopaminergic neurons (OPNs) and not in the ov
191                       This central effect of tyramine on motoneurons is required for the adaptive red
192 hod revealed robust accumulation of striatal tyramine on TCP administration.
193                               Application of tyramine or octopamine to live preparations silenced dop
194           There was no evidence for feruloyl tyramines, or homologues thereof, accumulating as a chem
195 roup were structural features that increased tyramine oxidase inhibition.
196                          Characterization of tyramine oxidase, carried out prior to the inhibition ex
197  induces transcription of genes encoding the tyramine oxidase, TynA, and the aromatic aldehyde dehydr
198 sayed using commercially available microbial tyramine oxidase.
199 und to be a weak noncompetitive inhibitor of tyramine oxidase.
200  ethyl ester in dry THF with phenethylamine, tyramine, p-methoxyphenethylamine, 2-(p-tolyl)ethylamine
201 d metabolites, such as tyrosine, tryptamine, tyramine, phenethylamine, dopamine, 3-methoxytyramine, s
202 was impaired using iontophoretic delivery of tyramine, phentolamine, and bretylium followed by a nore
203 cadaverine, spermine, spermidine), aromatic (tyramine, phenylethylamine) or heterocyclic (histamine,
204               The decrease in the content of tyramine, phenylethylamine, and dopamine in laboratory-s
205 spermine, spermidine, putrescine, histamine, tyramine, phenylethylamine, cadaverine and serotonin) we
206 onitoring of six biogenic amines (histamine, tyramine, phenylethylamine, tryptamine, putrescine, and
207                                 We show that tyramine plays a specific role in the inhibition of egg
208                       The binding of heparin-tyramine polymer (HT) onto the polycarprolactone (PCL) s
209                           Phenethylamine and tyramine prepared from a mixture of 18 amino acids were
210 n Caenorhabditis elegans, the neuromodulator tyramine produced by commensal Providencia bacteria, whi
211                              Locally infused tyramine produced dose-dependent pressor responses, pred
212 50% CFS of P. acidophilus was the highest on tyramine production (55%) by Listeria monocytogenes, fol
213                                              Tyramine production by Escherichia coli was also inhibit
214 50% CFS of Pediococcus acidophilus inhibited tyramine production up to 98% by Salmonella paratyphi A.
215                    Therefore, octopamine and tyramine provide global but distinctly different mechani
216  Chemical compounds studied in this article: Tyramine (PubChem CID: 5610); Tyramine hydrochloride (Pu
217                       Significant amounts of tyramine, putrescine, and cadaverine occurred especially
218                          Of the BA examined, tyramine, putrescine, histamine and cadaverine showed hi
219 late Ca(2+) increases through the octopamine/tyramine receptor (Oct-TyrR) and the transient receptor
220 o serotonin receptors (ser-1 and ser-4), one tyramine receptor (ser-2), and two dopamine receptors (d
221 traction-response, whereas in solitary ones, tyramine receptor (TAR) signaling mediated repulsion-res
222 ccurring non-coding polymorphisms in tyra-3 (tyramine receptor 3), which encodes a G-protein-coupled
223 stigated the spatial brain expression of the tyramine receptor AmTAR1 using a specific antibody.
224 ramine, which indicates that this ionotropic tyramine receptor functions in tyramine signaling in viv
225 terns of different splice forms of the ser-2 tyramine receptor locus.
226                             We show that the tyramine receptor SER-2 acts through a Galphao pathway t
227 tly, we analyze a mutant allele of the ser-2 tyramine receptor, a target of the ttx-3 Lhx gene in the
228 dopamine receptor, LGC-55 is a high-affinity tyramine receptor, and LGC-40 is a low-affinity serotoni
229 aviors, we have cloned a putative octopamine/tyramine receptor.
230 obe and in the gnathal ganglion suggest that tyramine receptors are involved in modulating gustatory
231 ensor technology, we found that both TCP and tyramine reduced glutamate release in the substantia nig
232 lasticity of neuromuscular synapses, whereas tyramine reduces locomotor speed, but the underlying cel
233 rs, such as heat or oxidative stress, reduce tyramine release and thereby allow the induction of cyto
234 pressing brain structures are in vicinity to tyramine releasing sites, a specific tyramine antibody w
235                      The dose of intravenous tyramine required to raise systolic blood pressure by 30
236 1 and TYRA-3, for NPY-like neuropeptides and tyramine respectively, do not appear to be involved in o
237 1) and female sex (P=0.02) predicted blunted tyramine responses.
238                      The patients followed a tyramine-restricted diet during the medication trial and
239 al-time cell analysis, whether histamine and tyramine show synergistic toxicity towards intestinal ce
240                    Finally, the N-E-caffeoyl tyramine showed the highest reducing power (EC50=26mug/m
241                   Putrescine, cadaverine and tyramine showed very good correspondence with the irradi
242                   Putrescine, cadaverine and tyramine showed very good correspondence with the level
243 is ionotropic tyramine receptor functions in tyramine signaling in vivo.
244  isolation, the activities of octopamine and tyramine signalings were respectively correlated with at
245 droxylation was observed for the reaction of tyramine solely with Met471Cys TbetaM.
246                             We knocked out a tyramine-specific receptor, TyrR, which was expressed in
247 obes, antennal lobes), indicating that other tyramine-specific receptors may be expressed there.
248 trescine, cadaverine, histamine, serotonine, tyramine, spermidine and spermine), as well as microbiol
249                                              Tyramine stimulates the insulin-IGF-1 signalling (IIS) p
250 st, these neurons synthesize octopamine from tyramine stress-dependently.
251 ng behavior and reproduction, dependent upon tyramine synthesis.
252     Here, we report that the biogenic amines tyramine (TA) and octopamine (OA) in Drosophila melanoga
253                   In Caenorhabditis elegans, tyramine (TA) and octopamine (OA) inhibit aversive respo
254                           The biogenic amine tyramine (TA) regulates many aspects of invertebrate phy
255 s ample evidence linking octopamine (OA) and tyramine (TA) to several neurophysiological functions in
256 opamine (OA), L-3, 4-dihydroxyphenylalanine, tyramine (TA), and serotonin as well as metabolites 5-hy
257 e interactions, we have defined the roles of tyramine (TA), octopamine, and neuropeptides in the inhi
258 nvertebrate-specific octopamine-OARalpha and tyramine-TAR signalings respectively mediate attractive
259                                              Tyramine temporally coordinates the different phases of
260 levels of serotonin, dopamine, histamine and tyramine than pulps.
261 t common tyrosinase substrates (tyrosine and tyramine), the enzyme is classified as a catechol oxidas
262 in just two pairs of neurons that synthesize tyramine, the RIM and RIC interneurons, induced intestin
263 red to FD (from 1mgkg(-1) for putrescine and tyramine to 4mgkg(-1) for histamine); MS/MS method showe
264 assage of eggs through the vulva and release tyramine to inhibit egg laying, in part via the LGC-55 t
265 mmunostaining for TBH, the enzyme converting tyramine to octopamine, in contrast, was strikingly simi
266 yme that catalyzes the beta-hydroxylation of tyramine to yield octopamine.
267 reagent for the derivatization of histamine, tyramine, tryptamine and 2-phenylethylamine, which elimi
268 cadaverine, spermidine, spermine, histamine, tyramine, tryptamine and phenylethylamine) were determin
269 wine samples analysed had histamine (HIM) or Tyramine (TYM) levels above the limits considered as a p
270 vascular responses to noradrenaline (NA) and tyramine (TYR) are related to SNA such that individuals
271                                              Tyramine (TYR) is a vasoactive biogenic amine found in f
272 was to develop an amperometric biosensor for tyramine (Tyr) measurement in food and beverages.
273      BAs concentrations increased with time, tyramine (TYR), putrescine (PUT) and cadaverine (CAD) we
274 ablished that the major product, MftA*, is a tyramine-valine-cross-linked peptide formed by MftC thro
275           AIY memory retrieval neurons sense tyramine via the SER-2 receptor, which is essential for
276 nhanced in frequency by the neurotransmitter tyramine via the TyrRII receptor.
277 on in FBF in response to the highest dose of tyramine was blunted in older men compared with young me
278 osation of ethylbenzene, phenethylamine, and tyramine was carried out, using UV-visible absorption sp
279                                              Tyramine was found mainly in red wines at <1-3.4 mg/L, w
280 vasoconstriction caused by administration of tyramine was inversely related to workload.
281                                              Tyramine was isolated selectively and pre-concentrated b
282 was not influenced by ageing conditions, and tyramine was only detected in some samples after 43days
283                                     Overall, tyramine was the prevalent amine, followed by putrescine
284 polamine, scopoline, tropine, tropinone, and tyramine, was accomplished by comparison of the in-sourc
285 asoconstrictor responses to the high dose of tyramine were determined in eight young adults during no
286 genous noradrenaline release (intra-arterial tyramine) were significantly blunted during moderate exe
287 sponses to local intra-arterial infusions of tyramine (which evokes endogenous NE release), phenyleph
288 C) responses to brachial artery infusions of tyramine (which evokes endogenous noradrenaline release)
289  (whole-body cooling) and pharmacologically (tyramine, which displaces noradrenaline from axon termin
290 enylacetic acid (DOPAC), methyldopamine, and tyramine, which gave negligible signals under conditions
291 ctive in a behavior that requires endogenous tyramine, which indicates that this ionotropic tyramine
292 m biogenic amines, especially histamine, and tyramine, which is an important consideration when selec
293           RIM synthesizes the neuromodulator tyramine, which is required in the L1 stage for learning
294 is associated with a large dietary intake of tyramine while taking MAO inhibitors has been reported;
295 increases in the tyrosine-derived metabolite tyramine, whose exogenous application elicits cell death
296         In addition to this, co-injection of tyramine with DOPA, the precursor of melanin, had a stro
297 O: -34 +/- 2% DeltaCVC(base); P < 0.001) and tyramine (Y: -38 +/- 4% DeltaCVC(base), O: -35 +/- 3% De
298 ion (cold, young: 38 +/- 4, older: 31 +/- 3; tyramine, young: 36 +/- 3, older: 36 +/- 5 Delta %CVC(ba
299 ine (cold, young: 37 +/- 4, older: 34 +/- 4; tyramine, young: 40 +/- 4, older: 45 +/- 4% Delta CVC(ba
300 (4) (cold, young: 37 +/- 3, older: 36 +/- 3; tyramine, young: 41 +/- 2, older: 36 +/- 3% Delta CVC(ba

 
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