ライフサイエンスコーパス: unclear

1 to ensure efficient spindle assembly remains unclear.

2 n such intriguing additional capacity remain unclear.

3 rganizes to function throughout the colon is unclear.

4 s undergoing viral lytic reactivation remain unclear.

5 llows RcsF to reach the surface has remained unclear.

6 a differential effect on BP control remains unclear.

7 m specific risk factors for liver disease is unclear.

8 TerS binds DNA and the TerL protein remains unclear.

9 e of GR in the pathophysiology of AD remains unclear.

10 duced symptoms and inflammation over time is unclear.

11 How the two processes are coordinated is unclear.

12 d phenotype, although its mechanism remained unclear.

13 nctions of these glucose-sensing neurons are unclear.

14 se patients in contemporary practice remains unclear.

15 isms of these tissue-specific actions remain unclear.

16 ations reflect fiber or fiber source remains unclear.

17 ssociated with slower metabolic rate remains unclear.

18 1-mediated cancer cell proliferation remains unclear.

19 mline and the underlying pathophysiology are unclear.

20 and the molecular mechanism(s) involved are unclear.

21 disparate functions of Sema3A signaling are unclear.

22 d their heterogeneous susceptibility, remain unclear.

23 therapy can improve hepatic fibrosis remains unclear.

24 tion but the mechanism of repression remains unclear.

25 disease outcome and long-term health remains unclear.

26 nical management and patient outcome remains unclear.

27 omes that support tissue homeostasis remains unclear.

28 inhibition and its role in pathogenesis are unclear.

29 neural circuit assembly and function remain unclear.

30 the underlying psychological mechanisms are unclear.

31 ed glomerular filtration rate (eGFR) remains unclear.

32 biannual treatment with azithromycin remain unclear.

33 g mechanism for the network assembly remains unclear.

34 merely represents a bystander effect remains unclear.

35 body and yet the reasons for this are still unclear.

36 a high degree of genomic instability remain unclear.

37 o the bone marrow neutrophil lineage remains unclear.

38 avior, and children's brain structure remain unclear.

39 The underlying pathomechanism is unclear.

40 ion and recruitment to the injured heart are unclear.

41 ses in damaged or aggregated proteins remain unclear.

42 but how these centers are specified remains unclear.

43 , but its role in virus replication remained unclear.

44 G output nuclei and in downstream targets is unclear.

45 to infection, and COVID-19 dynamics remains unclear.

46 How these events are coordinated is unclear.

47 he relevant EP2-expressing cell types remain unclear.

48 ther factors support this dynamic process is unclear.

49 ally contaminated, although the mechanism is unclear.

50 h the UBQLN2 mutations cause disease remains unclear.

51 r, the mechanisms underlying its role remain unclear.

52 cco dependence but underlying mechanisms are unclear.

53 euronal circuit in its native environment is unclear.

54 ng the trajectory of ADHD over time is still unclear.

55 slow (<60 Hz) and fast gamma states remains unclear.

56 d the risk of subsequent reinfection remains unclear.

57 However, its efficacy and safety remain unclear.

58 tance of mRNA localization to centrosomes is unclear.

59 nducing ocular and neuronal pathogenesis are unclear.

60 ctly how mTORC1 promotes cell growth remains unclear.

61 in the context of amyloid pathology remains unclear.

62 neural elements mediating symptom relief are unclear.

63 he time required to attain maximal effect is unclear.

64 ays a role in GnRH-1 neuronal development is unclear.

65 of BFCNs support different functions remains unclear.

66 causal relationship of these events remains unclear.

67 ll adapt to evade neutralizing antibodies is unclear.

68 colon polyps at baseline colonoscopy remains unclear.

69 chronization and its global organization are unclear.

70 ons, but its impact on liver cancer had been unclear.

71 ms by which it regulates angiogenesis remain unclear.

72 e, but the function of PKD2 in cilia remains unclear.

73 ut its role in tauopathy progression remains unclear.

74 ive induction of immunity is, however, still unclear.

75 hich NS1 antagonizes RIG-I signaling remains unclear.

76 Underlying mechanisms remain unclear.

77 are selectively targeted for degradation is unclear.

78 olarity are regulated within neurons remains unclear.

79 ty but the molecular mechanisms involved are unclear.

80 easuring adherence to asthma therapy remains unclear.

81 tigen (PSMA) PET/CT, a newer examination, is unclear.

82 repertoire of traits encoded on MGEs remain unclear.

83 er, the role of CAFs in this context remains unclear.

84 ormation in surface and groundwaters remains unclear.

85 e contribution of the latter is particularly unclear.

86 hanisms causing such symptoms remain largely unclear.

87 , yet its impact on patient outcomes remains unclear.

88 kground interacts with Mendelian loci remain unclear.

89 the molecular basis of this polarity remains unclear.

90 the mechanism of CPT's beneficial effects is unclear.

91 smitter dopamine (DA) on inflammation remain unclear.

92 factors and the molecular mechanisms remain unclear.

93 er these are parallel or dependent events is unclear.

94 interact with MR1-antigen complexes remains unclear.

95 nd optimal immunosuppressive regimens remain unclear.

96 ceptor, TRPV1, outside of sensory nerves are unclear.

97 transmitted to the gene expression level is unclear.

98 those of the central nervous system, remains unclear.

99 and the impact on surgical outcomes remains unclear.

100 o mast cell degranulation and anaphylaxis is unclear.

101 trol of autophagy gene transcription remains unclear.

102 t and response to antipsychotic treatment is unclear.

103 n animals and humans, its structure has been unclear.

104 ese potentially antagonistic requirements is unclear.

105 , as well as those ameliorated by CR, remain unclear.

106 ns to the therapeutic effects of DBS remains unclear.

107 resulting health implications are currently unclear.

108 stander effects in radiation therapy remains unclear.

109 e or a consequence of hypertrophic growth is unclear.

110 anism by which they improve outcomes remains unclear.

111 percutaneous coronary intervention (PCI) is unclear.

112 LOAD genetic architecture and prediction are unclear.

113 d bond, but the underlying mechanisms remain unclear.

114 s, mechanisms underlying such phenotypes are unclear.

115 ecise role of the bza operon protein CobT is unclear.

116 the origin of extraembryonic mesoderm remain unclear.

117 between progenitor and stem cell states are unclear.

118 , antibodies to the new species in humans is unclear.

119 ed inflammation during DENV pathogenesis are unclear.

120 how this architecture influences activity is unclear.

121 daptations to drive metastatic cancer remain unclear.

122 However, its effects on OPOA remain unclear.

123 ctivation during glutamate release are still unclear.

124 he context of the chromatin organization, is unclear.

125 minants of CHK1 inhibitor sensitivity remain unclear.

126 improving grain productivity remain largely unclear.

127 tion of general cellular homeostasis remains unclear.

128 s that govern cocaine potency in vivo remain unclear.

129 that their roles in programme delivery were unclear.

130 tion events regulate BCR functions in GCs is unclear.

131 ctors to total phenotypic variance are still unclear.

132 utations can eradicate premalignant cells is unclear.

133 ion of tumor-specific CD8(+) T cells remains unclear.

134 ucture during early neurodevelopment remains unclear.

135 hritis, but the benefits of this therapy are unclear.

136 te specificity leading to drug resistance is unclear.

137 tween lipid metabolism and longevity remains unclear.

138 g mechanisms and disease consequences remain unclear.

139 ion origin but the association state remains unclear.

140 , the pathways driving this phenotype remain unclear.

141 dendritic microtubule organization is still unclear.

142 nisms of AXL overexpression in cancer remain unclear.

143 isms behind these host differences are still unclear.

144 specific role played by each ligand has been unclear.

145 t several aspects of this association remain unclear.

146 severity of peanut-allergic reactions remain unclear.

147 rcuit mechanisms underlying this ability are unclear.

148 ZH2 from a gene repressor to an activator is unclear.

149 rowth in the remodeled tumor-promoting BM is unclear.

150 pment, but the underlying mechanism remained unclear.

151 The significance of these features remains unclear.

152 l proliferate following Ag encounter remains unclear.

153 ns and reducing LPS-induced inflammation are unclear.

154 y erythropoiesis and hemoglobin switching is unclear.

155 but their relationship to brain circuits is unclear.

156 onnected across the brain and spinal cord is unclear.

157 grain yields of intercrop species are still unclear.

158 in the treatment of prostate cancer remains unclear.

159 er, its impact on endometrial cancer remains unclear.

160 es impacts the self-assembly process remains unclear.

161 ural circuits contribute to epilepsy remains unclear.

162 ary with bioenergy crop species also remains unclear.

163 factor against FA, but its mechanisms remain unclear.

164 of, and connection between, these events is unclear.

165 el consequences of such reactivations remain unclear.

166 but their effect in patients with TB remain unclear.

167 eir function in the ALT pathway has remained unclear.

168 wever, what causes these connections remains unclear.

169 es "activated" (hyperproliferative), remains unclear.

170 Although the molecular mechanisms remain unclear, altered distribution and function of gap juncti

171 cal relevance of this cleavage event remains unclear, although it is thought to inhibit activation of

172 animal populations is a concern that remains unclear and controversial.

173 chanisms of ubiquitin chain formation remain unclear and include a sequential addition model, in whic

174 echanism of antidepressant action of BoNT is unclear and maybe hypothetically attributed to the disru

175 ular mechanisms driving this response remain unclear and the effects on lipid metabolism are relative

176 o undergo concurrent resection of CLM/EHD is unclear and the impact of somatic mutations has not been

177 ion against a primary Chlamydia infection is unclear and was a focus of this work.

178 ion of predicted sensory information remains unclear, and studies thus far have revealed a mixed patt

179 ll chloride/bicarbonate exchanger pendrin is unclear, as are potassium's role in this response and th

180 effects of exercise on tumor progression are unclear, as are the cellular actors involved.

181 band power (SBP) and neural activity remains unclear, as does the capability of using the SBP to deco

182 or through an intermediate host is currently unclear, as is the potential for this virus to infect co

183 amental predictability of structures remains unclear, as networks' complex underlying formation dynam

184 controls T(FH) biology has largely remained unclear, at least in part due to the intrinsic challenge

185 storage in LDs with cellular metabolism are unclear but relevant to obesity-related diseases.

186 ical scenario maximizing uncertainty with an unclear clinical and molecular diagnosis.

187 ond to such treatment or if the diagnosis is unclear, endoscopy, esophageal manometry, and esophageal

188 rotein with no known structural homologs and unclear ER function.

189 ng with an increase in receptor activator of unclear factor kappa-B ligand, periostin, and peroxidasi

190 oding of foreseeable outcomes is potentially unclear from the analysis of units activity and their pa

191 r structures and mechanisms of action remain unclear, hampering drug development.

192 etic structure of root neighborhoods remains unclear, hindering the understanding of belowground comm

193 elucidation of the complexoform, it remains unclear how and whether cellular circuits control the di

194 At present it is unclear how common reinfection with SARS-CoV-2 is and ho

195 In nature, it remains unclear how defensive traits such as crypsis, activity l

196 timated in several ways, but it is currently unclear how different measures of WUE relate, and how we

197 However, it is unclear how distinct structural features of the two PA2G

198 ies is extremely challenging, and it is thus unclear how eco-evolutionary dynamics drive the evolutio

199 However, it is unclear how ecotypes arise and how their distinctive com

200 of GM-CSF in autoimmune disease, it remains unclear how GM-CSF is regulated at sites of tissue infla

201 he heterotrimeric G(s) protein(2), but it is unclear how GPR52 and G(s) couple for signal transductio

202 tes to advanced age-related disorders, it is unclear how Kruppel-like factor 5 (Klf5), an essential t

203 der Parkinson's disease (PD); however, it is unclear how mitochondrial impairment and alpha-synuclein

204 ional bursting have been studied, it remains unclear how morphogen gradients affect this dynamic prop

205 However, it remains unclear how neocortex maintains this asynchronous spikin

206 Nonetheless, it is unclear how such biological changes are linked to releva

207 However, it remains unclear how such ecologic competition via cross-immunity

208 However, it remains unclear how the marine P cycle would respond to the chan

209 Until now, it has been unclear how the origin DNA is guided by ORC-Cdc6 and ins

210 esponsible for object recognition, but it is unclear how the representation of visual objects is orga

211 However, it remains unclear how these findings relate to categorization in m

212 It remains unclear how these pathways interact to contribute to con

213 is important for protection, however, it is unclear how these protective antibodies function.

214 ted quality of life (VRQoL), but it is still unclear how this relationship varies with age across the

215 plex that includes beta-catenin, it remained unclear how this would contribute to blocking the metast

216 rol association studies, but it is currently unclear how to best combine case-control status and fami

217 peech integration occurs in the brain, it is unclear how visual and auditory cues are combined to imp

218 It remains largely unclear, however, what defines the range and specificity

219 However, it is unclear if alpha5-GABA(A)Rs are altered in vivo or relat

220 sed lineage heterogeneity within GMPs, it is unclear if committed progenitors already exist among the

221 However, it remains unclear if EAAT2 responds to environmental cues to speci

222 However, it is unclear if exceptional polymer length of vHMM-HA is impo

223 ften diagnosed after advanced disease, it is unclear if laminin-111 protein therapy at an advanced st

224 To date, it is unclear if other prenatal steroids also contribute to au

225 tion and Alzheimer's disease (AD), but it is unclear if sleep duration is a manifestation of the AD d

226 It is unclear if that silence requires minimum H3K27me3 levels

227 It is unclear if the flu vaccine exacerbates immune events in

228 t small-molecule additives (excipients) with unclear impacts on human physiology, drug safety, and re

229 g subsequent antiretroviral therapy still is unclear.IMPORTANCE The antiretroviral drug rilpivirine w

230 surgery (ie, surgical hip fractures) remains unclear in elderly patients who are suspected to have su

231 ed hereditary hemochromatosis, but risks are unclear in mostly undiagnosed p.C282Y homozygotes identi

232 ctions between antibiotics and phages remain unclear, in particular, whether and when phages can driv

233 of entry for shrimp-related pathogens remain unclear, infectious diseases are difficult to prevent an

234 Though its therapeutic mechanisms remain unclear, it has been suggested that antidromic activatio

235 nts at the SSIS and 19 away from it (2 cases unclear location; 7 patients with >1 recurrence).

236 l contribution to pathogenesis of ICL remain unclear.METHODSWe hybridized 34 and 51 ICL patients' ser

237 lthough the precise molecular details remain unclear owing to the lack of an analytical method for gl

238 n hair bulbs of affected human scalp remains unclear, oxidative stress sensing appears to be a key fa

239 variation in dispersal across species remain unclear, partly because standardised estimates of disper

240 of RESTV for pigs has remained elusive, with unclear pathogenicity in naturally infected animals and

241 emission of a coughing superemitter, who for unclear reasons emitted up to two orders of magnitude mo

242 However, it is unclear to what extent metabolic health changes over tim

243 It is unclear to what extent sensory processing areas are invo

244 It remains unclear under what climatic conditions these species cou

245 intraspecific trait differences, it remains unclear under which circumstances the effects of environ

246 eering cloud seeding experiments, it remains unclear what makes a material a good ice former.

247 re altered in the context of diabetes, it is unclear what regulates localization of insulin granules

248 However, it is unclear what role these factors play in compacting the b

249 However, it remains unclear whether a causal relationship exists between lip

250 ely studied in colorectal cancer, it remains unclear whether activity in the tumor-associated stroma

251 It is currently unclear whether and how these repulsive and attractive b

252 hown to be recruited by VWM paradigms, it is unclear whether any portion of cerebellum actively encod

253 tion are hallmarks of 2i exposure, but it is unclear whether epigenetic alterations are required to a

254 tivity in visual cortex of carnivores, it is unclear whether experience exerts a permissive or instru

255 te ion (Pi) uptake remain unknown, and it is unclear whether foliar P applications can meet plant nut

256 It is currently unclear whether hearing impairment may impair the abilit

257 At present, it's unclear whether HY5 uses additional mechanisms to inhibi

258 lly identical population of cells, but it is unclear whether it has a role in the development of mult

259 However, it remains unclear whether learning is facilitated by non-rapid eye

260 Moreover, it is unclear whether or not recognition and quality control o

261 It remains unclear whether POAF is associated with an increased ris

262 It is unclear whether PRDM16 plays a similar role in neurogene

263 However, it is unclear whether RFSs also utilize MiDAS and to what exte

264 However, it is unclear whether staffing levels are more likely to be pe

265 However, it is unclear whether super-spreading events occurred during t

266 n activity-dependent plasticity, it is still unclear whether synaptic plasticity rules inferred from

267 , given the predicted trade-offs, it remains unclear whether systemic lifespan-extending intervention

268 It is unclear whether the ACA is also associated with increase

269 ell migration behaviors, and it is presently unclear whether the mechanisms used for migration in 2D

270 It is unclear whether the number of steps per day and the inte

271 t of this and other therapies for SMA, it is unclear whether the paralysis associated with the diseas

272 However, it is unclear whether the transfer of auditory information to

273 sease (CHD) and depression is evident, it is unclear whether the two diseases have shared underlying

274 However, it is unclear whether these changes would be accompanied by hi

275 G: Tripsacum dactyloides L.), but it remains unclear whether these effects are the same between crops

276 It is unclear whether these features respond to anti-VEGF ther

277 However, it is unclear whether this applies to social contexts, and whe

278 However, it remains unclear whether this caspase signaling plays a key role

279 However it is unclear whether this finding can be replicated in a larg

280 or deterministic choice outcomes, it remains unclear whether this is also the case when integrating m

281 ltiple sclerosis clinical outcomes but it is unclear whether this is mediated by an excess of extracr

282 nd community-acquired pneumonia (CAP), it is unclear whether this remains the case in the setting of

283 However, it is unclear whether this specific function is related to the

284 However, it remains unclear whether this trend will continue, preventing for

285 ndrome coronavirus 2 illness, but it remains unclear whether underlying asthma is associated with wor

286 osis, and cell proliferation; but it remains unclear whether variants in this gene cause a genetic di

287 tebrates and other neglected taxa, and it is unclear whether well-studied taxa, such as vertebrates,

288 However, it remains unclear which aspects of attention are processed by fSTS

289 g extracorporeal membrane oxygenation, it is unclear which cannulation method carries a higher risk o

290 ecline in some species, it generally remains unclear which changes in climate actually cause extincti

291 localize to the nucleus; however, it remains unclear which contain transcriptional effectors.

292 Particularly, it is unclear which defects are responsible for the emergent m

293 In many cases, it is unclear which experimental features or model assumptions

294 50% of global photosynthesis, yet it remains unclear which genes and proteins are required to produce

295 However, it is unclear which motifs are prone to impact transcriptional

296 Thus, it remains unclear which patterns of hyporeactivity represent a tra

297 However, it is unclear which subpopulations of B cells express GZMB und

298 It is unclear why some individuals develop AN while most do no

299 nd death counts continue to rise, it remains unclear why some individuals recover from infection, whe

300 Contemporary incidence remains unclear with few recent nationwide studies and limited d